ライフサイエンスコーパス: be explained by

1 eration in the PDZ domain-ligand interaction was explained by 3D structural models, which showed a ho

2 ic differences in PDE11A4 protein expression are explained by a coding single-nucleotide polymorphism

3 The experimental data are explained by a family of random-walk-based models an

4 Both cell types are explained by a model in which ILDs are computed with

5 These data are explained by a modified wormlike chain model incorpo

6 bally observed negative Vs anomaly (-4%) can be explained by a 0.7% melt fraction in peridotite at th

7 ge effect of PEG 20 on cold denaturation can be explained by a change in water activity, according to

8 glycinergic and GABAergic inhibition and can be explained by a circuit of local serial inhibition.

9 t that high HPV prevalence and diversity can be explained by a combination of a lack of homologous im

10 natural kind and that its experience cannot be explained by a constructionist account of emotion.

11 Among the PI3K-Akt targets, 75% could be explained by a deactivation of adenylate-uridylate-ri

12 gh thermoelectric figure of merit, which can be explained by a dislocation scattering model.

13 hat many of these empirical principles could be explained by a divisive normalization mechanism opera

14 stanoids in the PVAT-induced contraction can be explained by a greater release of thromboxane from PV

15 er survival in women who received NACT could be explained by a higher prevalence of limited performan

16 Those patterns could be explained by a lengthening growth season (during whic

17 eutral modifiers onto different ions and can be explained by a model which considers the transient bi

18 atively more heterogeneous rates that cannot be explained by a neutral model, with rapid pulses in th

19 ved experimentally in these brain states can be explained by a passage through a bifurcation combined

20 y responses in entorhinal cortex (EC) cannot be explained by a previously proposed relay from AC base

21 I near promoters and enhancers that can best be explained by a rapid decrease in termination.

22 The observed order of splicing cannot be explained by a simple kinetic model.

23 ode COMPAS to show that all three events can be explained by a single evolutionary channel-classical

24 e effects for both home and away teams could be explained by a single measure, home runs allowed.

25 s mitral and external tufted cells and could be explained by a single pool of slowly recycling vesicl

26 This may be explained by a temporal integration process in which

27 ugh a large portion of these differences can be explained by a variety of sociobehavioral and socioec

28 hat a nearby association with severe malaria is explained by a complex structural rearrangement invol

29 at transfer enhancement on textured surfaces is explained by a deterministic model that involves thre

30 eased backscattering from the base of the OS is explained by a model combining cytoplasmic swelling,

31 amagnetism in the oxoammonium salt solutions is explained by a redox equilibrium as shown between oxo

32 This is explained by a stabilizing effect on the transition s

33 This unexpected core structure is explained by a strong coupling of the phase and ampli

34 inct behavior in the thermoelectric response is explained by a strong deviation from the linear dispe

35 This was explained by a corresponding cyclorotation of the oc

36 as observed by the addition of saliva, which was explained by a salting out effect, due to the presen

37 owed over time and if such differences could be explained by acute resuscitation survival, postresusc

38 this account of human amygdala function can be explained by alternative views, which posit that the

39 in 2 (4E-BP2) protein in [Formula: see text] are explained by an excluded volume effect, similar to t

40 n of N-(3-pyridyl)aldimines with olefins can be explained by an asynchronous [4+2] cycloaddition; in

41 shows that beta-cell dysfunction in T2D can be explained by an impaired amplifying pathway with no n

42 evel of adaptation in diverse streams cannot be explained by an increased recolonization of sensitive

43 We show that the polarization's rotation can be explained by an optical splitting parameter appearing

44 Ws on the growth parameters investigated has been explained by an analytical relationship between the

45 Their age and formation processes have now been explained by an improved generation of galaxy-forma

46 This is explained by an increased susceptibility of field-rea

47 rmal, such avolition-like behavior could not be explained by anhedonia or behavioral despair.

48 While bimodality can hence be explained by anthropogenic edge effects and natural s

49 These stoichiometric responses could be explained by associated changes in growth, RNA:DNA, a

50 This could be explained by assuming that the concerted motions of t

51 d between alexithymia levels in coping style were explained by Avoidance strategies.

52 CC-induced inflammatory responses, which may be explained by BCD-mediated attenuation of complement a

53 st all of the PTOT spatial variability could be explained by BIOG, while BIOG and FARM explained 38%

54 ponents related to fast frequencies that can be explained by Born-Oppenheimer enzyme hypothesis (vibr

55 indicating that the reduction in CFU number is explained by cells entering into a Viable But Non-Cul

56 thirds of the decline in organic aerosol can be explained by changes in anthropogenic emissions, prim

57 These fear-inhibitory effects cannot be explained by changes in appetitive behavior.

58 gh much of plant morphological evolution can be explained by changes in gene expression, examining it

59 Incidence trends can be explained by changes in known risk factors, improved

60 Mechanistically, this could be explained by changes in the competitive interactions

61 is highly seasonal and this seasonality may be explained by changes in the weather, specifically, te

62 , a residual discard rate increase could not be explained by changes in these factors.

63 This result could be explained by charge transfer on the MoS2 channel and

64 66% of the variance in FIB removal rates can be explained by clean bed filtration theory (CBFT, 31%),

65 This shift cannot be explained by climatic changes.

66 Excess twin mortality cannot be explained by common risk factors for under-5 mortalit

67 (r = 0.43, p < 0.001), and this association was explained by common shared environmental influences

68 veal features of virus evolution that cannot be explained by comparing natural viral isolates.

69 Furthermore, bleeding phenotypes could not be explained by concurrent hemostatic defects.

70 imulations showed that this broadening could be explained by contrast-invariant single-unit tuning wi

71 success observed in animal populations could be explained by correlational selection, where the fitne

72 Vesta's surface roughness variations cannot be explained by cratering processes only.

73 al differences between fungal species cannot be explained by current knowledge obtained from genome s

74 an increase in fatty acid oxidation, cannot be explained by decarboxylated osteocalcin changes, sugg

75 report suggested much of the Kok effect can be explained by declining chloroplastic CO2 concentratio

76 This deficit can be explained by decreased expression of genes involved i

77 This behavior of MGs is explained by describing the competition between shear

78 The spectral evolution is explained by developing a theoretical model for the p

79 This is explained by DFT calculations which indicate that the

80 Although some of these disparities can be explained by differences in access to care, cancer sc

81 This microbiota signature could not be explained by differences in diet or use of medication

82 riation in rate between the sexes can mostly be explained by differences in hotspot magnitude, rather

83 e annual breeder) and test whether these can be explained by differences in life histories.

84 of femoral shaft fractures, which could not be explained by differences in patient case mix.

85 r patients with brain injury can only partly be explained by differences in patient characteristics.

86 e higher attenuation of this virus could not be explained by differences in the ability of the two NS

87 Finally, we provide evidence that this can be explained by differences in the kinetics of the rate-

88 s are less efficiently spliced, which cannot be explained by differences in U1 binding or the density

89 The effects could not be explained by differences in verbal IQ, intracranial v

90 nfunction between kidneys from uDCD and cDCD were explained by differences in the first warm ischemic

91 This circumstance could be explained by different stabilizing effects.

92 the past six to nine generations and cannot be explained by differential inbreeding, but are consist

93 These differences can be explained by differing levels of UV exposure.

94 d changes to community structure that cannot be explained by direct interspecific interactions among

95 igns with the CCFS and 41% of that with SARA were explained by disease duration, age at onset and the

96 s in patients with diabetes and may possibly be explained by dissimilarities in deglycation in turn l

97 of AMPP-T and AMPT-P over the last 33 years is explained by eCO2 (39% and 42%) almost equally than b

98 related genes and that most heritability can be explained by effects on genes outside core pathways.

99 The observed radio emission can be explained by either a collimated ultrarelativistic je

100 usly undocumented in this system-that cannot be explained by either mechanism in isolation.

101 Such a scenario might be explained by encounters with cometary debris in Earth

102 is indicates that the role of heparin cannot be explained by enhancement of nucleation alone, as has

103 These findings can be explained by environmental statistics.

104 n increase in how well species distributions were explained by environmental conditions, from modest

105 Substrate acceptance and stereoselectivity were explained by extensive molecular modeling (MM) and

106 distribution as mean HAZ declined could not be explained by faltering limited to a growth-restricted

107 We examined whether this association could be explained by familial factors by comparing associatio

108 hierarchy stability on interview performance was explained by feelings of control and testosterone re

109 suscitation performance, which can partially be explained by fewer unsolicited cardiopulmonary resusc

110 ng in wild-type mice but weak in humans, may be explained by FEZF2-mediated cis-regulatory elements t

111 This discrepancy was explained by finding that BCR KO-induced changes in

112 the size of the larval population but cannot be explained by food depletion nor is it modulated by bi

113 show that the early difference in N e cannot be explained by food producers inhabiting more favorable

114 More than half of this variability was explained by forest descriptors, namely small variat

115 ity and that roughly half of this effect can be explained by functional annotations negatively correl

116 In contrast, regional gray matter increases were explained by GABAA receptor concentration in additi

117 t cancer subtype frequency differences could be explained by genetic variants.

118 stantial part of the variation in tooth loss is explained by genetic as well as environmental factors

119 ll phenotypic correlation the majority (78%) was explained by genetic factors.

120 c contributions to cancer and that which can be explained by genomic variants, both inherited and som

121 Variability among studies may be explained by geography and patient factors.

122 The statistical heterogeneity was explained by glioma subtype, echo time, and the prop

123 e total variance in right hippocampal volume is explained by HbA1c levels among Hp 1-1 carriers and t

124 levels among Hp 1-1 carriers and that 3.22% is explained by HbA1c levels among Hp 1-1 noncarriers.

125 prevalence of allergic sensitization by age was explained by high incidence and persistence.

126 This difference could be explained by higher aglycon content, while aglycon ap

127 The eed1 farnesol hypersensitivity can be explained by higher internal concentrations of farnes

128 Nonlinearities were explained by homogeneity of environmental condition

129 rtality in patients with diabetes, which may be explained by hypoglycemia-induced inflammation.

130 The findings can be explained by immunohistochemical data showing increas

131 This pattern cannot be explained by inbreeding avoidance or as a response to

132 e and waiting time) and late PVR (at repeat) are explained by incomplete transmurality and contiguity

133 ong cross-sensitivity to humidity, which can be explained by increased V-T relaxation.

134 This phenomenon can be explained by increased vascular permeability immediat

135 This difference has been explained by increased recipient mismatching for ma

136 ell contact between B cells and moDCs, which was explained by increased upregulation of costimulatory

137 The effect of DRB could not be explained by inhibition of DNA replication per se or

138 ater of the corresponding sediments can only be explained by initial deposition of this facies in a f

139 in ozone sensitivity among woody plants can be explained by interspecific variation in LMA and that

140 Thus ISE2's evolutionary conservation may be explained by its numerous roles in regulating chlorop

141 In CF-guided pulmonary vein isolation, PVR is explained by lack of both lesion depth and contiguity

142 etries, the phase transitions among them can be explained by Landau's symmetry breaking theory.

143 he variability in NZ sea lion pup production is explained by latent by-catch, and the population woul

144 Their formation can be explained by Lewis acid induced opening of the epoxy

145 en development of structure and function may be explained by life-long synaptic changes in human V1.

146 Instead, the richness gradient is explained by limited time for speciation in marine ha

147 A with diabetes risk in women that could not be explained by liver fat.

148 (EXAFS) spectra of these As-bearing pyrites are explained by local structure models obtained using d

149 cle activity on left and right sides, cannot be explained by M-cell circuit models.

150 ng that grooming-handclasp style convergence is "explained by matrilineal relationship rather than co

151 in GIR, the largest fraction by far (70.2%) was explained by maximal oxygen uptake.

152 he observed population-level association can be explained by measured and unmeasured factors shared b

153 Sex-biased expression is explained by miRNA-specific regulation, including sex

154 The experimental observations cannot be explained by models that consider only mineral dissol

155 oves as the growth rate is increased and can be explained by more spatially regular nucleation as the

156 that the relationship with regional atrophy was explained by MTL tau.

157 46% of mortality risk was explained by multivariable modelling with these vari

158 ause PCH, and the vast majority of PCH cases are explained by mutations in TSEN54, which encodes a su

159 ies differences in PAH concentrations cannot be explained by normalizing them to the plant lipid cont

160 This behaviour is explained by noticing that the low-energy modes, resp

161 ng Bose gas, and we show here that it cannot be explained by only pairwise correlations.

162 d in patients with HOCM after surgery, which was explained by opposite changes in the septum (decreas

163 Homochirality is explained by origin of prebiotic life in one hemisphe

164 ere fatally struck, a pattern that could not be explained by other confounding factors (e.g. age or r

165 out, the previously observed association may be explained by other factors.

166 The behavior of such off-stoichiometric NCs was explained by our density functional theory calculati

167 tion of cAMP binding to the CNBD can largely be explained by partial competition between TRIP8b and c

168 genetic diversity within paleodrainages can be explained by past riverine properties (i.e., area and

169 While regional variation in 30-day outcomes were explained by patient and hospital factors different

170 erences in time to acceptance for transplant were explained by patients' demographic, cultural, psych

171 of the developing brain, those effects could be explained by patterns of neural activity that do not

172 red in the strong-excitation regime and have been explained by Pauli blocking of the optically filled

173 ersheds in potential spawning habitat change was explained by predicted increases in mean annual floo

174 95% CI, 0% to 8%; P = .03), neither of which were explained by preexisting trends.

175 This difference can be explained by preferential recruitment of the DNA mism

176 This effect could be explained by productive substrate binding that protec

177 Variations in attrition may be explained by program director attitudes, although lar

178 hypothesized that this seeming paradox could be explained by proteins that bind to and control the lo

179 e observed pattern of DOC quality change can be explained by redox-dependent adsorption/desorption of

180 This phenotype could be explained by reduced beta-cell apoptosis in db/db-PI3

181 PFC) tDCS induced an analgesic effect, which was explained by reduced perfusion to posterior insula a

182 hese changes in neutrophil recruitment could be explained by regulation of E-selectin on the cocultur

183 ed that CC and MCC behavioral patterns could be explained by reinforcement learning.

184 heterogeneity across study estimates, which was explained by risk group, world region, and HSV-2 exp

185 the association with chr5p15.33-Region 2 may be explained by rs36115365, a variant influencing TERT e

186 e observed variation in tree mortality could be explained by satellite-derived net primary productivi

187 ow that such negative kin discrimination can be explained by selection for unrelated targets to inves

188 eponderant prevalence of ASD might partially be explained by sex differences in clinical symptoms, et

189 eavily on D1 receptors, an effect that could be explained by sexual dimorphisms in receptor expressio

190 d socialization, the disposition to distrust is explained by shared socialization but not by heritabi

191 sitivity with morning fasting is unlikely to be explained by signalling proximal to Akt.

192 atios in surface waters of the North Sea can be explained by simple binary mixing models implying tha

193 and 58% of the racial difference in CMB risk was explained by sleep time and sleep efficiency, respec

194 (i.e., ambient plots), but this change could be explained by slow thaw in Control areas.

195 Plant growth promotion was explained by slow release of nutrients, although a m

196 virological failure among PWUD with TDR may be explained by some inappropriate ART prescribing, as w

197 ity of the variation in phylogenetic metrics was explained by spatially structured environmental vari

198 This difference might be explained by structural preservation after crush of I

199 low develops at the plastron, which can only be explained by surfactant gradients formed in the loadi

200 ured as activity correlations that could not be explained by task events.

201 g-bands within their shell walls that cannot be explained by temperature alone.

202 lthy male locusts, and propose that this may be explained by terminal reproductive effort and a lower

203 These results are explained by TFAM playing a role in promoter melting

204 These different patterns are explained by the different apparent solubility of ca

205 The results are explained by the elimination of the three-fold Pt si

206 They are explained by the existence of randomly occurring "de

207 The spectroscopic similarities are explained by the fact that, in both materials, photo

208 These interesting outcomes are explained by the influences of societal learning or

209 e change of the graphene temperature sensors are explained by the temperature dependent electron mobi

210 riability in genetically-identical cells can be explained by the accumulation of misincorporated comp

211 that mixed epistasis between a gene pair can be explained by the action of a third gene that modulate

212 icals colliding with rare-gas atoms that can be explained by the analytical Fraunhofer model.

213 the reactions was complicated and could not be explained by the Arrhenius equation.

214 itation in the general population appears to be explained by the association between atopy and having

215 majority of the kidney discard rate rise can be explained by the broadening donor pool.

216 genic Ras cooperates with Egfr, which cannot be explained by the canonical signalling paradigm.

217 ship between DELC and Abeta-positivity might be explained by the causative role of CSVD in Abeta accu

218 on of surface charge is 4 times too large to be explained by the change in dielectric constant upon a

219 lergy in the African continent cannot simply be explained by the change in public hygiene.

220 red increased nuclease resistance, which can be explained by the close proximity between 4'-OMe subst

221 his analysis reveals that Eph clustering can be explained by the combined contribution of polymerizat

222 These observations might be explained by the combined influence of several factor

223 gnetic susceptibility anisotropy that cannot be explained by the commonly used Bleaney's theory.

224 he two- or four-electron reduction of O2 can be explained by the constraint provided by the stannoxan

225 ot been observed in other animals and cannot be explained by the conventional and widely-used "piston

226 This can be explained by the coupling of ice and sediment dynamic

227 This terrestrial locus of innovation cannot be explained by the Cretaceous to recent expansion of di

228 This sensitivity can be explained by the dependency of pol delta mutants on C

229 ommunity variation of bacterioplankton could be explained by the distinct conditions of each lake and

230 ikingly, this asymmetric localization cannot be explained by the distribution of GapR binding sites o

231 me oxidative vascular diseases may very well be explained by the dysfunction of the Trx1-GC1 associat

232 rate but that this effect could only partly be explained by the effect of the two azoles on cytochro

233 ns, and shorter length of stays, which might be explained by the elective nature of surgery and earli

234 istic size-frequency scaling of icebergs can be explained by the emergence of a dominant set of drivi

235 istribution in the dC18:1+dC22:1 mixture can be explained by the energetic cost associated with compr

236 This discrepancy can be explained by the evolution of the crystal and electro

237 This can be explained by the fact that coarse and PE bran hold mo

238 esized that differences in performance would be explained by the following: (i) entorhinal cortex (ER

239 al modeling suggests the Fe(II) dynamics can be explained by the formation of a rapidly reducible Fe(

240 The favorable effects of fiber may be explained by the generation and distribution of one o

241 The formation of the cubosomes can be explained by the hierarchical interactions of the con

242 levated PCB-101 concentrations in winter can be explained by the high frequency of this transport reg

243 he MA and the smaller halide anions, but can be explained by the increase in the polarizability with

244 This abnormal condition can be explained by the inter-diffusion of Pb and Bi element

245 ltidecadal time scales, which is unlikely to be explained by the Interdecadal Pacific Oscillation.

246 ng Island identified a region that could not be explained by the known stratigraphy.

247 ies with EcoSSB and that the differences may be explained by the lack of an acidic, disordered C-term

248 erved here and in the wider literature could be explained by the lbeta phase being out of equilibrium

249 vity to NLP cytolysins of monocot plants may be explained by the length of the GIPC head group and th

250 This can be explained by the limited reproducibility of US examin

251 to what extent auditory motion analysis can be explained by the linear combination of static spatial

252 ite fraction and magnesium concentration can be explained by the location of the magnesium in calcifi

253 The decrease in RM dose could be explained by the lower volume of distribution.

254 but reduced particle clearance, which could be explained by the marked rounded-up morphology of thes

255 ges; importantly, the hollowing event cannot be explained by the nanoscale Kirkendall effect, nor by

256 Some of these discrepancies can be explained by the nonselective application of relative

257 d pairs of transcription factors may instead be explained by the older duplication-degeneration-compl

258 The early sterilizing effects of PQ may not be explained by the preferential clearance of male gamet

259 electrical conductivities reaching 1 S/m can be explained by the presence of an interconnected networ

260 The resolved density variations can be explained by the presence of post-perovskite, chemica

261 dability phenotype of E8-SBD123 plants could be explained by the putative cell-wall loosening effect

262 as observed in the presence of NO and it can be explained by the radical consumption by NO as SOx and

263 at at least 80% of the discard rate rise can be explained by the recovery of kidneys from an expandin

264 crease in the motor neuron firing that could be explained by the reduction in the expression of the p

265 upon termination of enrichment ( 35%) cannot be explained by the reduction of leaf area ( 15%) and as

266 magnitude and timing of Heinrich events can be explained by the same processes that drive the retrea

267 lling specificity between these pathways may be explained by the spatial separation of FLS2 and BRI1

268 A remarkable improvement with fever may be explained by the temperature-dependent leftward shift

269 This paradox has been explained by the centromere drive hypothesis, which

270 This is explained by the different rates of increase of plasm

271 asynchrony of wind and hydropower resources is explained by the differential impact of the two modes

272 This cross-reactivity is explained by the sequence similarity of the two virus

273 f the interindividual variability in the MFO was explained by the [Formula: see text]O2 max, sex, and

274 Until recently, its reliability was explained by the assumption that it is hardwired; bu

275 ution of WSOCs with increasing carbonization was explained by the hydrophobic carbonaceous matrix act

276 This disease phenotype was explained by the presence of larger numbers of cytok

277 This behavior for acid whey was explained by the proximity of the pI of whey protein

278 matches between the TB&S and kmer ID results were explained by the close phylogenetic relationship be

279 at the synergistic potential of azoles could be explained by their effect on the biotransformation ra

280 l associations between different species can be explained by their genome composition.

281 h RA are at increased risk of HF that cannot be explained by their increased risk of IHD.

282 Reactivity abilities of these N-Lewis acids were explained by theoretical calculations.

283 varied greatly, with much of this variation being explained by thermal preference.

284 associated refolding processes often cannot be explained by thermodynamic effects alone.

285 menopausal and postmenopausal breast cancers are explained by these factors.

286 field loss in the secondly affected eye can be explained by these outcomes in the first eye.

287 The stability of such colloids cannot be explained by traditional electrostatic and steric mec

288 The mean trophic level pattern is explained by trophic replacement of herbivorous fish

289 We suggest that this apparent paradox can be explained by two compensatory water effects.

290 jority of variation in the timing of feeding was explained by two principal components.

291 This selectivity can be explained by unique structural features of the KIFC1

292 ate concentration on and off the equator can be explained by upwelling rate, with slower upwelling al

293 compound heterozygous variants, which could be explained by variable disease expression and warrants

294 ly 50% of genome-wide expression variability is explained by variation across individuals and identif

295 Variation in lags is explained by variation among species in physiological

296 ts; (2) vary by diagnostic category; and (3) are explained by variations in insurance, income, teachi

297 Several inconsistencies in the literature are explained by viewing power holders as more flexible

298 ite prevalence and host shell length, cannot be explained by Waltherian facies change, host availabil

299 radipine levels to protect SN DA neurons can be explained by weaker state-dependent inhibition of SN

300 imate simulations, we argue that this puzzle is explained by weaker atmospheric circulation in respon