1 Mice aged 2, 8, and 18 months were fed 16 weeks of a low-fat diet or HFD.
2 d" mouse model where male mice from 7 inbred were fed a choline- and folate-deficient (CFD) diet for
3 ) and heterozygous knockout mice (sod2(+/-)) were fed a chow or high-fat (HF) diet, which accelerates
4 Fish reared at 21-22 degrees C (range) were fed a commercial diet at 3% body mass d(-1) (non-sa
5 F-344 rats or C57BL/6 mice were fed a diet containing T3 for 2-7 days.
6 Notably, Pstpip2(cmo) mice that were fed a diet rich in fat and cholesterol maintained a
7 -) mice, which recapitulate features of PXE, were fed a diet supplemented with warfarin and vitamin K
8 IQ scores of children who were fed a formula containing either LC-PUFAs or no LC-P
9 Adult male wild-type or RAG C57B6-6J mice were fed a high fat (60%) or normal chow diet for 8-10 w
10 When wild-type mice were fed a high fat diet for only 3 days, hepatic insuli
11 Pre-pubertal female pigs, age 35 d, were fed a high-energy diet (HED; n = 12), containing 15
12 Next, C57Bl/6J and Keap1-KD mice were fed a high-fat diet (HFD) to determine whether Keap
13 The mutant mice were fed a high-fat diet (HFD) to induce insulin resista
14 -/-) phenotype was exacerbated when the mice were fed a high-fat diet, which correlated with decrease
15 dysfunction (liver or muscle) when 79 humans were fed a low-choline diet.
16 TLR4(-/-) and wild-type mice were fed a low-fat, high-monounsaturated fat (HF(MUFA)),
17 Rats were fed a modified Lieber-DeCarli liquid diet with 38%
18 -/y) mice in an LDL receptor -/- background were fed a saturated-fat enriched diet, and infused with
19 Doxycycline-treated mice were fed a standard commercial diet for 18 weeks and the
20 Eight obese young adults were fed a standard hypocaloric diet to produce 10-15% w
21 Female C57BL/6J mice were fed a standard or obesogenic diet before and throug
22 seizure susceptibility, the three BS strains were fed a sulfonylurea drug (tolbutamide) known to both
23 NHPs were fed a WSD (36% fat) supplemented with 0.37% resvera
24 maternal nutrition restriction (MNR)] or who were fed ad libitum (control), were administered the pro
25 Female C57BL/6 or MIF-/- mice were fed an ethanol-containing liquid diet or pair-fed c
26 C57BL/6 mice were fed an obesogenic diet for 16 weeks, and concomitan
27 The strongest connections to V1 are fed back from neighbouring area V2 and from a networ
28 n vivo bioassays showed that mosquitoes that were fed blood with CSPBP antibody displayed a 25% and 9
29 ween iron loading and high TGs, Fischer rats were fed chow containing 1% carbonyl iron.
30 ation was even more pronounced when the mice were fed chow diet.
31 Pregnant baboons were fed control (ad libitum, n=11) or an MNR diet (70%
32 Thirty-one twin-bearing sheep were fed either a control (n=15) or low-protein diet (n=
33 G) and their nontransgenic counterparts (NT) were fed either a control diet (CD) or a high-fat diet (
34 ild-type (C57BL/6J) littermates when animals were fed either normal or sodium-deficient diets.
35 rexpressing OPN in hepatocytes (Opn(HEP) Tg) were fed either the control or the ethanol Lieber-DeCarl
36 mly divided into 2 groups (n = 65 each) that were fed folate-deficient (FD) or standard diets for 8 w
37 address that question, groups of BALB/c mice were fed high (10%) or low (2%) fiber diets and infected
38 uces atherosclerotic plaques in F1 mice that were fed high fat diet (HFD) after weaning.
39 e clinical signs of diarrhea developed, pigs were fed hLZ-milk or non-transgenic control goat milk th
40 Mice were fed liquid Lieber-DeCarli diet without or with alco
41 liothis virescens and Agrotis ipsilon larvae were fed on BvSTI leaves.
42 accumulated to higher levels in aphids that were fed on CYDV-infected plants compared to healthy pla
43 C57BL/6J male mice were fed regular chow or high fat diet for 8 weeks.
44 Thereafter, offspring were fed standard rodent chow.
45 tly more tumors than R group, and these mice were fed the same regular chow throughout the entire car
46 y (BSE)-contaminated meat and bone meal that was fed to cattle and precipitated the BSE epidemic in t
47 In the present study, low doses of BSE were fed to lambs of a range of ages (~24 h, 2 to 3 week
48 ive change from 18 mo to 6 y in children who were fed variable amounts of docosahexaenoic acid (0.32%
49 Mice were fed vitamin D-deficient or -sufficient chow for 6 w
50 Gravid female rats were fed with a resveratrol-enriched diet during gestati
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