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1  are highly aggressive, and their activities are held in check by host developmental and epigenetic m
2  equilibrium (expansion of transformed cells is held in check by immunity); and escape (tumour cell v
3 gnizes and is poised to eliminate cancer but is held in check by inhibitory receptors and ligands.
4  differentiation are expressed by HSPCs, but are held in check by miRNAs until differentiation occurs
5 order in the cell wall reaction kinetics and is held in check by negative feedback inhibition of exce
6 es are primed for expression in ES cells but are held in check by opposing chromatin modifications.
7 ibute to the genesis of early neoplasms that are held in check by the alternate ability of c-Myc to i
8 sis is induced by cyclin B-bound Cdk1, which is held in check by the protein kinase, Wee1.
9  local immune responses in patients with HCC are held in check by tumor-infiltrating CD4(+)CD25(+) T-

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