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1 of PAX5 fusion proteins in B-ALL development is largely unknown.
2 of altered brain states on this relationship is largely unknown.
3 but how they interact in lifespan regulation is largely unknown.
4 Glucocorticoid regulation of human ILC2s is largely unknown.
5 reside in the cell, and what role they serve is largely unknown.
6 ution of immune cells to disease progression is largely unknown.
7 regulation of alternative splicing in cancer is largely unknown.
8 enes still perform their ancestral functions is largely unknown.
9 and and water resources to realize this goal is largely unknown.
10 w immune cells influence the function of SCs is largely unknown.
11 sk factors on stroke incidence and mortality is largely unknown.
12 thelium, but its role in disease progression is largely unknown.
13 t its contribution in pro-survival signaling is largely unknown.
14 specific point mutations alter PTEN function is largely unknown.
15 motor neurons, but the underlying mechanism is largely unknown.
16 neural basis of these differences, however, is largely unknown.
17 tion of inflammatory pathways in fibroblasts is largely unknown.
18 utant p53 proteins on cancer cell metabolism is largely unknown.
19 of these "isletokines" in health and disease is largely unknown.
20 es ameliorates liver inflammation and injury is largely unknown.
21 -that is, treatment-naive metastatic disease-is largely unknown.
22 e pathophysiology of both movement disorders is largely unknown.
23 NAs (miRs) regulate IOP and glaucoma in vivo is largely unknown.
24 fection outcomes in HIV-associated pneumonia is largely unknown.
25 egulates the function of self-reactive Tregs is largely unknown.
26 but its association with long-term mortality is largely unknown.
27 lled the pyrenoid, whose protein composition is largely unknown.
28 he role of microRNAs (miRNAs) in the process is largely unknown.
29 the development of kidney disease, however, is largely unknown.
30 ly by NOD-like receptor (NLR) inflammasomes, is largely unknown.
31 However, the molecular basis for this is largely unknown.
32 failure (HF), but the outcome by type of AF is largely unknown.
33 diseases, but its role in skeletal disorders is largely unknown.
34 heir contribution to retinopathy-negative CM is largely unknown.
35 sponse to opportunistic bacterial infections is largely unknown.
36 t the molecular mechanism of this regulation is largely unknown.
37 dination is achieved and related to behavior is largely unknown.
38 r genetic stability under selective pressure is largely unknown.
39 ntially harmful action of apoptotic caspases is largely unknown.
40 ific advances support marketplace inventions is largely unknown.
41 is achieved by modulating receptor function is largely unknown.
42 he genome is remodeled in response to stress is largely unknown.
43 However, the detail at the molecular level is largely unknown.
44 ong-term refractive status of these children is largely unknown.
45 owever, how repair is initiated at telomeres is largely unknown.
46 mbrane mechanics of primary afferent neurons is largely unknown.
47 l killer (NK) cells and their alloreactivity is largely unknown.
48 ar, the impact of temperate forest on clouds is largely unknown.
49 neural mechanisms supporting this behaviour is largely unknown.
50 ects and mice, but their mechanism of action is largely unknown.
51 he effects of infection on neuronal function is largely unknown.
52 ng" characteristics are maintained afterward is largely unknown.
53 al lipid composition and membrane morphology is largely unknown.
54 multi-ethnic populations from Southeast Asia is largely unknown.
55 osphorylation, the AtRALF1 signaling pathway is largely unknown.
56 iptional and post-transcriptional regulation is largely unknown.
57 s quality of care, the accuracy of such data is largely unknown.
58 he impact of Abeta deposition on FPC regions is largely unknown.
59 oms have adapted to this extreme environment is largely unknown.
60 the effect of S deposition on the fate of As is largely unknown.
61 of QRS duration in non-European populations is largely unknown.
62 egulating intestinal epithelial cell renewal is largely unknown.
63 ent in the virus life cycle and pathogenesis is largely unknown.
64 for example automotive catalytic converters, is largely unknown.
65 these play a role in cholesterol metabolism is largely unknown.
66 ity and their function in natural ecosystems is largely unknown.
67 rough wild-type and mutant ferritin channels is largely unknown.
68 D95 in gammaherpesvirus-associated lymphomas is largely unknown.
69 influence of this admixture on human traits is largely unknown.
70 ar Ca(2+) signals in pancreatic acinar cells is largely unknown.
71 noma cells; however, its mechanism of action is largely unknown.
72 ception of pain emerges from neural activity is largely unknown.
73 s and cardiovascular risk factors to LA size is largely unknown.
74 e of CKD on the colonic microbial metabolism is largely unknown.
75 collective biofilm morphology during growth is largely unknown.
76 The regulation of this process is largely unknown.
77 ly for distal reproductive tract development is largely unknown.
78 sorder in adults and children whose etiology is largely unknown.
79 st critical for food and livelihood security is largely unknown.
80 of the gene repertoire of fly Y-chromosomes is largely unknown.
81 erface to exert anti-inflammatory properties is largely unknown.
82 al resistance (AMR) in the wider environment is largely unknown.
83 ons of neurons and ultimately brain circuits is largely unknown.
84 omes yet the contribution of TEs to lincRNAs is largely unknown.
85 ate of this Hg during and following snowmelt is largely unknown.
86 molecular comparison within exosome subtypes is largely unknown.
87 flammatory responses of myeloid immune cells is largely unknown.
88 this is linked to their biological function is largely unknown.
89 s modify KCNQ1 channel gating so differently is largely unknown.
90 ow OGT regulates excitatory synapse function is largely unknown.
91 n patients with community-acquired pneumonia is largely unknown.
92 YAP has a role in innate antiviral immunity is largely unknown.
93 t the health of other neighborhood residents is largely unknown.
94 e molecular control of human spermatogenesis is largely unknown.
95 ntrol regions escape from this reprogramming is largely unknown.
96 d development of severe pathology of malaria is largely unknown.
97 the replication stress at these loci/regions is largely unknown.
98 ome is activated in this untreatable disease is largely unknown.
99 The etiology of perineuriomas is largely unknown.
100 nism by which intact NETs promote thrombosis is largely unknown.
101 and the specificity of processing enzyme(s) is largely unknown.
102 als with obsessive-compulsive disorder (OCD) is largely unknown.
103 al cell patterning (e.g. in neural circuits) is largely unknown.
104 eorganization in individual MOF nanocrystals is largely unknown.
105 isms that specify human microglia phenotypes are largely unknown.
106 grams and their impact on adult neurogenesis are largely unknown.
107 on the mechanical behaviour of crustal rocks are largely unknown.
108 d by RIG-I during infection with a DNA virus are largely unknown.
109 nts involved in transducing their perception are largely unknown.
110 by TAL1 and contribute to T-ALL pathogenesis are largely unknown.
111 zed, but the underlying molecular mechanisms are largely unknown.
112 s factors associated with long-term survival are largely unknown.
113 ke the onset and progression of the syndrome are largely unknown.
114 hanisms mediating adult enteric neurogenesis are largely unknown.
115 e detailed mechanisms for such NP assemblies are largely unknown.
116 olecular mechanisms underlying its formation are largely unknown.
117 nication mechanisms that mediate aggregation are largely unknown.
118 The underlying pathogenic mechanisms are largely unknown.
119 embly and function of the polycystin complex are largely unknown.
120 The pathogenic mechanisms of these viruses are largely unknown.
121 al calcium concentration in cardiac myocytes are largely unknown.
122 fects of protein binding on IAPP aggregation are largely unknown.
123 hat underlie symbiont-mediated host immunity are largely unknown.
124 n ecosystem functions and service provisions are largely unknown.
125 ous pathways, and the underlying mechanisms, are largely unknown.
126 However, its neural functions are largely unknown.
127 f their synaptic partners to disease process are largely unknown.
128 nism and factors controlling MARCH5 activity are largely unknown.
129 ion, the factors driving Treg specialization are largely unknown.
130 nderlying the pathological hyperexcitability are largely unknown.
131 cellular processes that mediate these events are largely unknown.
132 t their composition, dynamics, and functions are largely unknown.
133 al factors triggering the behavioral changes are largely unknown.
134 s, but the mechanisms by which ECD functions are largely unknown.
135 d spatial routes of each transport mechanism are largely unknown.
136 the various body patterns by the optic lobe are largely unknown.
137 w the LTR lncRNAs serve biological functions are largely unknown.
138 phate pathway, and the RNA and DNA backbone, are largely unknown.
139 ifferential integration into cortical layers are largely unknown.
140 te angiogenesis and the molecular mechanisms are largely unknown.
141 stress pathways involved in this recognition are largely unknown.
142 rus (CMV) DNA in infected asymptomatic hosts are largely unknown.
143 shape neuronal activity, their roles in mEC are largely unknown.
144 ng and the dynamics of the gating components are largely unknown.
145 s by which these loci influence disease risk are largely unknown.
146 tic triggers for gonadal sex differentiation are largely unknown.
147 ms determining its spatiotemporal regulation are largely unknown.
148 chymal stem cells' (MSC) suppressive potency are largely unknown.
149 spatiotemporal patterns at the global scale are largely unknown.
150 the processes determining these compositions are largely unknown.
151 and their potential roles in coral diseases are largely unknown.
152 diomics, clinical factors, and tumor biology are largely unknown.
153 regulate DNA replication, in breast cancers are largely unknown.
154 ng molecular and cellular mechanisms of PTSD are largely unknown.
155 es needed for meiotic hot spot specification are largely unknown.
156 in mice; however, the underlying mechanisms are largely unknown.
157 ildhood body size and future melanomagenesis are largely unknown.
158 ich AR-targeted therapy reduces tumor burden are largely unknown.
159 ns, the causative genes and variants for T1D are largely unknown.
160 , but the precise mechanisms of this process are largely unknown.
161 owever, the underlying regulatory mechanisms are largely unknown.
162 process driving adaptation-natural selection-are largely unknown.
163 ar mechanisms of their function in complexes are largely unknown.
164 t their effects on ecosystem-level processes are largely unknown.
165 ized, but the limiters of energy expenditure are largely unknown.
166 K20me3), in development and genome stability are largely unknown.
167 ut the resulting emissions and waste streams are largely unknown.
168 shape the temporal pattern of vertebrate MZT are largely unknown.
169 (RNAPII) elongation and alternative splicing are largely unknown.
170 erences mediating these infection phenotypes are largely unknown.
171 the interactive effects of these two factors are largely unknown.
172 alcohol seeking but the mechanisms involved are largely unknown.
173 ignals regulating neuronal versus glial fate are largely unknown.
174 hanisms leading to this regenerative failure are largely unknown.
175 However, the underlying mechanisms are largely unknown.
176 tion, the mechanisms guiding their formation are largely unknown.
177 l in the brain when taken as a drug of abuse are largely unknown.
178 the composition and functions of the conoid are largely unknown.
179 ion, but the underlying molecular mechanisms are largely unknown.
180 ion resistant, and the underlying mechanisms are largely unknown.
181 n plant cells, but their physiological roles are largely unknown.
182 isms by which breast cell fate are regulated are largely unknown.
183 regulation essential for brain regeneration are largely unknown.
184 ular complexities of the PH-PIP interactions are largely unknown.
185 The mechanisms of the latter are largely unknown.
186 ctors and mechanisms underlying food allergy are largely unknown.
187 tual mechanical properties of living tissues are largely unknown.
188 ike morphology to mature dendrite morphology are largely unknown.
189 diversification of eukaryotic NEET proteins are largely unknown.
190 s long-term cardioprotective actions in vivo are largely unknown.
191 r, the genetics of childhood body mass index are largely unknown.
192 s exert their key immunoregulatory functions are largely unknown.
193 two PAX6 isoforms, isoform-a and isoform-b, are largely unknown.
194 downstream targets of this signaling network are largely unknown.
195 ve and quantitative dynamics of this process are largely unknown.
196 ar mechanisms underlying acquired resistance are largely unknown.
197 rmance over several rounds of annual testing are largely unknown.
198 itory neuron to regulate cortical plasticity are largely unknown.
199 effects of TGF-beta during different stages are largely unknown.
200 t the pathogenic mechanisms of F. tularensis are largely unknown.
201 static cells with the brain microenvironment are largely unknown.
202 dulatory substrates of rejection experiences are largely unknown.
203 s of action and the responsible target genes are largely unknown.
204 s and the potential risk for human infection are largely unknown.
205 me kinase activity in plant light signalling are largely unknown.
206 which RTKs including PDGFRalpha mediate EMT are largely unknown.
207 x4 regulation, but the underlying mechanisms are largely unknown.
208 e molecular properties of protein aggregates are largely unknown.
209 supporting this form of learning in infants are largely unknown.
210 ntracellular factors controlling this growth are largely unknown.
211 erlying urothelial formation and maintenance are largely unknown.
212 the mechanisms that result in this phenotype are largely unknown.
213 ces of a permanent expression of these genes are largely unknown.
214 e exhausted states of immune cells, however, are largely unknown.
215 migratory behaviors for collective movement are largely unknown.
216 ential effects on the tumor microenvironment are largely unknown.
217 ying growth and dormancy in woody perennials are largely unknown.
218 erlie the direct effect of light on behavior are largely unknown.
219 motility, but the regulators of this process are largely unknown.
220 specific cues that impact c-di-GMP signaling are largely unknown.
221 rlying this fundamental mode of organisation are largely unknown.
222 they are assembled to create the exosporium are largely unknown.
223 g the microglial response to prion infection are largely unknown.
224 all 4 EHD proteins, their physiologic roles are largely unknown.
225 tion on the structure and dynamics of NMDARs are largely unknown.
226 ete sequences, genes of origin and functions are largely unknown.
227 on the abuse potential of opioids in humans are largely unknown.
228 r protrusions, but the underlying mechanisms are largely unknown.
229 its functional role and signaling mechanisms are largely unknown.
230 ellular mechanisms underlying these deficits are largely unknown.
231 e molecular bases of these sensory processes are largely unknown.
232 ch probiotics exert their beneficial effects are largely unknown.
233 shape and regulate their expression profiles are largely unknown.
234 he pathways that negatively regulate NETosis are largely unknown.
235 t and function, but their specific functions are largely unknown.
236 The structure and function of these proteins are largely unknown.
237 nd implications for intellectual development are largely unknown.
238 idence of primary biliary cholangitis (PBC), are largely unknown.
239 signaling pathways governing LKB1 activation are largely unknown.
240 functions of other ArabidopsisMAP65 isoforms are largely unknown.
241 ze this unique, cell-dense migration pathway are largely unknown.
242 tors, but their roles in peritoneal fibrosis are largely unknown.
243 erlie the sequestration of these sub-domains are largely unknown.
244 r functions of AMOTL2 besides YAP inhibition are largely unknown.
245 nature of onset and progression of symptoms are largely unknown.
246 ssues and their possible role in contraction are largely unknown.
247 )-associated intracerebral haemorrhage (ICH) are largely unknown.
248 However, the underlying genetic determinants are largely unknown.
249 ear factor kappa B, although its role in PCa was largely unknown.
250 responsible for tumour cell phagocytosis is(are) largely unknown.
251 proteins and RNAs in these compartments has been largely unknown.
252 of UNC-45A in neuronal differentiation have been largely unknown.
253 mal hepatitis B virus (HBV) replication have been largely unknown.
254 s of this, and of its precursor neutropenia, are largely unknown, although genetic factors have an im
255 ces of this exposure on the developing fetus are largely unknown, although in animal models we have f
256 ese nasal commensals in host innate immunity is largely unknown, although bacterial interference in t
257 ging characteristics of MOG antibody disease are largely unknown and it is unclear whether they diffe
258 olecular underpinnings of this heterogeneity are largely unknown, and there is a paucity of markers t
259 se after cessation of contingency management are largely unknown, and, until recently, an animal mode
260 xtent of population structure within Ireland is largely unknown, as is the impact of historical migra
261 ulation and disassembly of the 100S ribosome are largely unknown because the temporal abundance of th
262 specific physiological tau functions, which are largely unknown but could contribute to neuronal dys
263 munity structure in high latitude ecosystems are largely unknown but critical to understand in light
264 ms of damage search and location by Pol beta are largely unknown, but are critical for understanding
266 for the majority of cases, the causes of ASD are largely unknown, but it is becoming increasingly acc
267 how such a cue triggers a change in behavior are largely unknown, but mechanics is likely to be invol
269 m underlying the chaperone activity of AIPL1 is largely unknown, but involves the binding of isopreny
271 bacteria in freshwater ecosystems, however, is largely unknown, confounding assessments of their rol
272 ic underpinnings of these cognitive deficits are largely unknown, deficits in cortical dopamine funct
273 er, a role for zinc during hepatic ER stress is largely unknown despite important roles in metabolic
274 er correlated phenotypic world) between them are largely unknown, despite extensive data showing that
275 enile mice or primary neuronal cell cultures is largely unknown even though both are widely used mode
277 re activated and other neurochemical content is largely unknown hence whether stimulus specific popul
278 vitro has been extensively investigated, it is largely unknown how APE1 repairs AP sites in cells.
279 udied uptake mechanisms in cyanobacteria, it is largely unknown how Mn is distributed to the differen
282 erate linear mRNAs and circular RNAs, but it is largely unknown how the ratio of linear to circular R
283 and gastrointestinal motility during the MMC is largely unknown, however, as is its ability to stimul
286 limited disease models, the etiology of NBL is largely unknown, including both the cell of origin an
288 on of Dlg4/PSD95, or other plasticity genes, are largely unknown, limiting the development of targete
290 ecular mechanisms that lead to these changes are largely unknown, particularly for the spaceflight (S
291 advanced and relapsed peritoneal metastasis are largely unknown, precluding development of more effe
292 wever, the pathophysiology of these deficits is largely unknown; published studies have mainly examin
294 ature of human CD4(+) T cells that target RV is largely unknown, T cell epitopes of RV capsid protein
295 Although the exact function of many circRNAs is largely unknown, the cell type-and tissue-specific ci
296 inant of their phenotypic properties, yet it is largely unknown to what extent ecological association
299 re multiple services from ecosystems, but it is largely unknown whether trade-offs between ecosystem
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