ライフサイエンスコーパス: be likely due to

1 es, after conversion to Stratus equivalents, are likely due to a change in retinal thickness rather t

2 a putative Protein Kinase C consensus site, are likely due to a post-translational modification requ

3 showed that establishment of HIV-1 infection is likely due to a biological mechanism that restricts t

4 71R mutation in 3.9% of our study population is likely due to a British founder effect.

5 s performance gap is not well understood but is likely due to a combination of compromises in acousti

6 on genetic diversity in the abundant species is likely due to a combination of demographic factors, i

7 This discrepancy is likely due to a combination of the differences betwee

8 The diminished virulence is likely due to a combination of the morphogenesis defe

9 This is likely due to a complex interaction between tradition

10 The survival benefit is likely due to a cumulative effect of N-cadherin's rol

11 The growth defect is likely due to a defect in capsid assembly, since DNA

12 ve recently demonstrated that this phenotype is likely due to a defect in induction of the metanephri

13 This is likely due to a defect in p97/VCP-mediated autophagos

14 wn and root morphology and amelogenesis that is likely due to a functional role of Irf6 in organizati

15 The molecular mechanism for this effect is likely due to a large conformational change elicited

16 breast cancer cells and breast cancer tumors is likely due to a marked loss of this Z-DNA-mediated tr

17 ells, indicating that the observed phenotype is likely due to a mechanotransduction defect.

18 sponse in ECs versus ART-treated individuals is likely due to a more intact humoral immune response i

19 unced hindered diffusion of IgG in brain ECS is likely due to a number of general factors associated

20 Inhibition of JMJD2A by K-bZIP is likely due to a physical interaction which blocks sub

21 This scarcity of robust methods is likely due to a realistic preference for simple and a

22 The decrease in inhibitory transmission is likely due to a reduced number of gamma-aminobutyric

23 The chirality transfer from guest to host is likely due to a restricted guest motion inside the ti

24 ving in such a diverse range of environments is likely due to a single and strong instance of local a

25 This is likely due to a stereoelectronic preference for cycli

26 ously determined fluorination trend at W6.48 is likely due to a sulfur-pi interaction with the side c

27 The disease is likely due to a toxic gain of function caused by the

28 This was likely due to a diminished inflammasome activation t

29 es, indicating that the increase in c-di-GMP was likely due to a loss in phosphodiesterase activity.

30 lere test for detection of influenza A virus was likely due to a study set that included many low-pos

31 We also found that the deficits are likely due to abnormal presynaptic Ca(2+) regulation

32 This effect is likely due to accelerated resolution of inflammation

33 The lower fetal blood concentrations are likely due to active efflux transport of tacrolimus

34 evere than that of those lacking alphavbeta3 is likely due to added failed activation of beta1 integr

35 nfections and acute bacteremia and that this is likely due to agr-independent and -dependent pathways

36 Mycobiome shifts during puberty are likely due to alterations in sebaceous gland activat

37 flammatory response in adipose tissue, which is likely due to alterations of both fat mass quantity a

38 nance at chronically disturbed shallow sites is likely due to an abundance of coral taxa which are to

39 This is likely due to an incomplete understanding of how lase

40 The increase in lacrimation is likely due to an increase in cool cell activity.

41 d by the decrease in particle size alone but is likely due to an increase in the defect density prese

42 This is likely due to an increase in the frequency of mTGFbet

43 of breastfeeding-associated HIV transmission is likely due to antiviral factors in breast milk.

44 The reduction in CD4(+)CD25(+) T cells was likely due to apoptosis driven by increased caspase-

45 he beneficial effect of Klotho gene delivery is likely due to attenuation of T-cell infiltration in p

46 This was likely due to augmented expression and activation of

47 These differences are likely due to biases or misunderstandings about the

48 Depletion in DMS following re-submersion was likely due to biologically-driven conversion of DMS

49 n total levels of PCBs after 365 days, which was likely due to biostimulation of the indigenous popul

50 The red-shift in low-energy absorption is likely due to both a decrease in ionization potential

51 139H interferes with HY TME infection, which is likely due to both strains targeting the same populat

52 dicating reduced mitochondrial Ca(2+) uptake was likely due to both lower psim and MCU activity.

53 The accelerated dechlorination was likely due to both Mg(OH)2 addition which limited pH

54 red to liposomes, in the circulation of mice is likely due to CD47-mediated protection of exosomes fr

55 glycosylation, and for tissue paper blotting is likely due to cell permeabilization.

56 We see heterogeneity between cohorts that is likely due to chance effects in smaller samples, or p

57 We show that this unintelligibility is likely due to changes in both cis- and trans-acting e

58 of RC formation upon DNAJC14 overexpression is likely due to chaperone dysregulation and that YFV pr

59 (2+) rise and activation of calcineurin, and is likely due to clathrin-dependent internalization of G

60 r and ssy5Delta-mediated life span extension is likely due to concomitantly increased mitochondrial a

61 ized the C(H)2 domain of the molecule, which is likely due to conjugation of DM1 on lysine residues i

62 Conduction slowing was likely due to connexin 43 (Cx43) downregulation, dec

63 per-mutation, notably CG to TG, the majority is likely due to context-dependent preferences for speci

64 breakdown of the rule of the geometric mean is likely due to coupled motion between the protein and

65 he phenotypes observed in these two patients are likely due to CTSC mutations.

66 ortant, and lower MBC at warmer temperatures is likely due to decreased carbon-use efficiency (CUE).

67 The reduced response by TSLPR-deficient mice is likely due to decreased frequency and reduced T cell

68 strate that generalized skeletal involvement is likely due to decreased osteogenesis.

69 This was likely due to decreased expression of cytokine and c

70 caused prolonged hypophagia; the hypophagia was likely due to decreased preference.

71 aneuploidy, and pachytene cell death, which are likely due to defects in DNA repair.

72 which indicates that the observed phenotypes are likely due to defects in glutamate receptor function

73 ed lower levels of cortical myosin II, which is likely due to defects in dephosphorylation of myosin

74 rts revealed a reduced respiration rate that is likely due to defects in electron transport chain com

75 This phenotype is likely due to deficiencies in auxin transport in the

76 These EEG dynamic differences are likely due to developmental factors including region

77 These discrepancies are likely due to differences in adherence.

78 individuals with GAG-7/7 compared to GAG-9/9 are likely due to differences in GCL regulation in RBCs.

79 r phenotypes caused by PXDN mutations, which are likely due to differences in genetic background and

80 ts suggest this change in relative intensity is likely due to differences in ionization efficiency ra

81 iation in PFC air concentrations by building is likely due to differences in the number, type, and ag

82 at the higher recurrence observed after LDLT is likely due to differences in tumor characteristics, p

83 gene expression variation among individuals are likely due to different mechanisms from those establ

84 ocal epilepsies caused by diverse etiologies are likely due to different pathogenic mechanisms.

85 subtype-specific effects of these mutations were likely due to differential amino acid residue netwo

86 This is likely due to difficulties associated with the nontar

87 ed that the mechanism of H2-O downregulation was likely due to direct protein degradation of H2-O as

88 and BMP combotherapy have been observed, and are likely due to distinct differences in their signalin

89 at the diversity of receptive fields in L2/3 is likely due to diversity in the underlying functional

90 s the source of the Co, whereas increased Ba was likely due to drilling mud used in the top kill atte

91 t the recent amplification in M. rufogriseus is likely due to duplications and concerted evolution ra

92 ncentrations found in earlier break-up years is likely due to earlier and/or increased foraging oppor

93 onal data indicate that the instability of 1 is likely due to ease of reduction of Ni(II) to Ni(I) an

94 uronal loss in the other hearing loss models is likely due to effects of the trauma on the sensory ne

95 the presence of ZnT8-RA rather than ZnT8-WA was likely due to effects of HLA-DQ2 and the SLC30A8 CC

96 +/-1sigma) of 44 +/- 22%; this underestimate is likely due to either HOOH destruction by Fe or a redu

97 CV replication in the humanized mouse models is likely due to either the lack of essential host facto

98 These functional effects were likely due to either steric blockade of downstream

99 reduce penile cancer and cervical cancer and is likely due to elimination of foreskin harboring human

100 flammatory responses between rHuPH20 and BTH is likely due to endotoxin contaminants in BTH.

101 at the underlying mechanism of CDI reduction is likely due to enhanced channel opening within the Ca(

102 The intracellular action of Klotho is likely due to enhanced forward trafficking of channel

103 month of winter (September) is discussed as being likely due to environmental issues, rather than bi

104 ary for adequate Dpp signaling and that this is likely due to excessive ubiquitylation on a specific

105 nal SDP and offspring substance use/problems is likely due to familial background factors, not a caus

106 k solvent Tg, has low activation energy, and is likely due to fast vibrations that may be enabled by

107 The activation of autophagy including TFEB is likely due to fisetin-mediated mammalian target of ra

108 ncovered struts at flow divider sites, which is likely due to flow disturbances.

109 ions and the increase of SI in the DN, which is likely due to gadolinium retention.

110 integrity through loss of function, while 3 were likely due to gain of function and/or combinatorial

111 The enhanced ALA-related toxicity is likely due to generation of additional porphyrin inte

112 ific aspects of the WWTP community functions are likely due to high biomass concentration, turnover r

113 These antiproliferative effects are likely due to high molecular weight compounds contai

114 nges in geochemistry and community structure are likely due to high rainfall influx during the monsoo

115 itness of the low-predation risk populations is likely due to higher consumption of algae when invert

116 This result was likely due to higher and earlier onset of Aipl1 expr

117 vation by PC vesicles as opposed to micelles is likely due to hindered product release.

118 es in the distribution of molecular features are likely due to horizontal gene transfer and changes i

119 creased toxicity measured postbioremediation is likely due to hydroxylated and carboxylated transform

120 ar PPi levels and that the incisor phenotype is likely due to hyperostosis of mandibles, which distin

121 This is likely due to hypoperfusion of the spinal cord, which

122 nesis among CD105(low) adipose-derived cells is likely due to identification of a subpopulation with

123 The outbreak is likely due to immunity gaps among adults who were ina

124 lls, relative to other CcO assembly mutants, is likely due to impaired hemylation and subsequent misf

125 This reduction was likely due to impaired maturation of NK cells, as re

126 s determined using a Nur77-GFP reporter, but were likely due to impaired induction and maintenance of

127 hich indicates that excess He is present and is likely due to incomplete degassing of pre-existing He

128 These observed differences are likely due to increased stability and loss of flexib

129 The loss of GMPs is likely due to increased apoptosis and increased phago

130 ysical studies reveal that altered stability is likely due to increased dynamic motion in the N-termi

131 beta-LGNDs, and the reduction in body weight is likely due to increased energy expenditure.

132 t hospital-associated clones, which we found is likely due to increased expression of the accessory g

133 ation and increased apoptosis of HSCs, which is likely due to increased expression of the cyclin-depe

134 age-related neurodegeneration in these mice is likely due to increased iron availability rather than

135 ain mechanism for enhanced TRAIL sensitivity is likely due to increased recruitment of FADD to the re

136 and elevated cancer risk in these survivors is likely due to increased smoking prevalence.

137 The latter effect was likely due to increased oligomerization of PLM phosp

138 This increase in DA sensitivity is likely due to increasing the electrode surface area w

139 ow that ADEP's effectiveness in mycobacteria is likely due to inhibition of ClpP-dependent protease a

140 This is likely due to insulin resistance of protein metabolis

141 Prolonged postconcussive symptoms are likely due to interactions between the biological in

142 specificity; EBNA3A and EBNA3B specificities are likely due to interactions with other cell transcrip

143 Targeting is likely due to interactions between the Ty1 integratio

144 This is likely due to interference by the amide function of t

145 The need for hypoxic response is likely due to interrupted wound vasculature, as well

146 Follistatin's mode of action is likely due to its ability to block myostatin and enha

147 This effect of AgC10 is likely due to its ability to induce L-selectin sheddi

148 The beneficial effect of FA is likely due to its ability to prevent global loss of m

149 This phenotype is likely due to its inability to deposit BslO murein hy

150 e role of PTEN in neuronal insulin signaling is likely due to its protein phosphatase actions, which

151 DA; therefore, opr3 resistance to some pests is likely due to JA accumulation, and signaling activiti

152 We show that this toxicity is likely due to LacZ misfolding and that, in the absenc

153 ablished ranges beyond which SUV differences are likely due to legitimate biologic effects.

154 mulation often observed in older individuals are likely due to lifestyle factors rather than inherent

155 gregate formation in aneuploid yeast strains is likely due to limiting protein quality-control system

156 d melt production during continental rifting is likely due to localized thinning and melt focusing wh

157 mulation studies suggest that these findings are likely due to locus heterogeneity rather than random

158 eatening, slowly progressive disorder, which is likely due to loss of PIEZO2 protein in afferent neur

159 This low level of diversity is likely due to low mitochondrial mutation rates that a

160 ol-3-kinase pathways in Ecad-lo cells, which was likely due to lower EGFR expression in this subset a

161 T cell response to histoincompatible tissue is likely due to many small T cell populations respondin

162 of Sir2 protein to chromosomes under tension was likely due to methodological weakness in early chrom

163 y more frequent and larger synaptic currents are likely due to more numerous, larger, and more powerf

164 Although this excess risk during therapy is likely due to multiple factors, a growing body of evi

165 lusion of minorities from NIH-funded studies is likely due to multiple reasons, including insufficien

166 l sulfonate substrate under these conditions is likely due to neighboring group participation or dias

167 ICC1 mRNA, while in vitro, we show that this is likely due to neuronal activity-induced downregulatio

168 We found that protection was likely due to neutralizing mucosal IgA Ab.

169 These mutations, which are likely due to nonrandom selection of a distinct gene

170 This increased resistance is likely due to not only the consumption of disinfectan

171 ty in radiologists who graduated before 1940 is likely due to occupational radiation exposure.

172 /- 2.6 mm/y in the northwest Atlantic, which are likely due to ocean bottom pressure variations.

173 ene exchange, and provide evidence that this is likely due to ongoing recombination between clade mem

174 The remaining discrepancy is likely due to other sex-specific elimination routes t

175 o activate Chk1 in response to MVM infection was likely due to our observation that Rad9 failed to as

176 els of genomic heterozygosity, much of which is likely due to outcrossing.

177 This biomarker discrepancy is likely due to overexpression of amyloid precursor pro

178 This is likely due to overlapping particle tracks near the an

179 ent differences among reported study results are likely due to paradigm differences that tap into dif

180 This suboptimal outcome is likely due to physiological barriers that hinder deli

181 on suggests that covariation of these traits is likely due to pleiotropy, although physically linked

182 athematical model suggests these differences were likely due to pQBR57's lower intraspecific conjugat

183 %), although half of the corneal edema cases were likely due to pre-existing causes other than the aq

184 This specific phenotype of mutant animals is likely due to preferential TrkB expression in DRD2 MS

185 of diabetes mellitus in chronic pancreatitis is likely due to progressive disease rather than individ

186 The lung injury and acute cor pulmonale is likely due to pulmonary microvascular injury, the mec

187 herefore, the decline in self-administration was likely due to rapid CocH-mediated cocaine metabolism

188 w but detectable ADCC recognition of A(H7N9) was likely due to rare individuals in the population con

189 These changes are likely due to recent climate change and particularly

190 The underlying cause is likely due to reduced levels of Tub4, as overexpressi

191 CREB target gene expression in PS cDKO mice is likely due to reduced neuronal activity in these muta

192 The effect of MPS1 inhibition is likely due to reduction of closed MAD2 (C-MAD2), as e

193 of endogenous Abeta on plasticity and memory was likely due to regulation of transmitter release, act

194 This is likely due to relatively efficient carbon transfer th

195 cantly decreased over time (P < 0.03), which was likely due to repeated antibiotic exposure.

196 n biofilm-coated sand compared to clean sand is likely due to repulsive electrosteric forces between

197 indicated that reduced sensory regeneration is likely due to SC-derived CLU.

198 rence with glycated hemoglobin A1c reduction is likely due to selection bias.

199 Defective trafficking of prenylated proteins is likely due to sequestration of prenyl binding protein

200 This was likely due to several factors including reduced resi

201 nal prepregnancy BMI and offspring bone mass is likely due to shared familial, genetic, and environme

202 This counterintuitive result was likely due to significantly delayed snowmelt over th

203 rences in epigenetic status found in cybrids are likely due to single nucleotide polymorphisms (SNPs)

204 es in access to living donor transplantation are likely due to socioeconomic factors rather than cult

205 The broad efficacy of antibody inhibition is likely due to steric competition because of its large

206 mental paradigm using mice that the increase is likely due to stress.

207 impairments present in psychiatric disorders are likely due to subtle neurochemical differences, and

208 ubstituents, the improvement in J(sc) and FF are likely due to suppressed charge recombination.

209 ited remarkable tumor control in vivo, which is likely due to sustained stepwise release of active tr

210 Further analyses showed that the changes are likely due to the activation of programmed cell deat

211 r levels of 18:3n-3 in DGAT1-silencing lines are likely due to the compensatory activity of a TAG-syn

212 markedly different fluorescence efficiencies are likely due to the differences in the shapes of the f

213 emission episodes at the investigated sites are likely due to the episodic ventilation of deep reser

214 repair defects of CtIP dimerization mutants are likely due to the failure in localization to chromos

215 led that most of the traits in affected boys are likely due to the genomic region spanning of MECP2 a

216 trends and difference in rate-limiting steps are likely due to the influence of the relative stabilit

217 Differences in limb proportions and scaling are likely due to the possession of display structures s

218 avored by large entropic contributions which are likely due to the reduced hydrophobic pyrimidine sur

219 lethality and the in vitro migration defects are likely due to the reduced levels of ILK-VT/GG and di

220 We show that these defects are likely due to the requirement for cell adhesion to f

221 This is likely due to the ability of nucleocapsids to follow

222 This is likely due to the absence of strong mixing, which is

223 This increased metabolic cost is likely due to the added force demand on the plantar f

224 It is likely due to the anisotropic bending energy of the L

225 ed responses, and the negative effect of PTI is likely due to the antagonism between BR and PTI signa

226 ystemic administration of BMSCs to MCAO rats is likely due to the cellular changes in blood and/or br

227 We propose that such performance is likely due to the complete transformation of MoS2 to

228 We show that this is likely due to the conservative species definition rat

229 This absence is likely due to the difficulty associated with measurin

230 The lack of a measure of proteome quality is likely due to the difficulty in producing one, a prob

231 This is likely due to the difficulty of pseudobase attack at

232 evealed that the early maturation of area MT is likely due to the disynaptic retinopulvinar input and

233 er timing of cell division, a mechanism that is likely due to the double negative feedback loop betwe

234 ous genes adjacent to yeast telomeres, which is likely due to the ectopic spreading of the Sir comple

235 The pleiotropic nature of this regulation is likely due to the elaborate control of TGF-beta produ

236 Their constitutive induction is likely due to the elevated DNA damage responses in sa

237 gation of donor molecules on the PTCDI fiber is likely due to the enhanced local electrical field bui

238 This difference in 2-MC toxicity is likely due to the fact that as a si-citrate synthase,

239 This is likely due to the fact that LDD allows for the presen

240 tiffening behavior of the material alone and is likely due to the fibrous nature of the proteins.

241 ce genetic profile has been determined; this is likely due to the fluid nature of the UPEC genome as

242 This is likely due to the formation of a Np(V) hydroxide prec

243 ustained oil accumulation occurring under SL is likely due to the formation of plastidial LDs.

244 and that dominance within the TCR repertoire is likely due to the frequency or expansion and survival

245 This is likely due to the higher calcium content of Powder Ri

246 The cNOS-mediated reduction of IkappaB is likely due to the imbalance of nitric oxide/peroxynit

247 This is likely due to the important role that primary care pl

248 of heparan sulfate on the SPR surfaces; this is likely due to the inability of CCL7 to oligomerize be

249 s TRPM6(V(1393)I) and TRPM6(K(1584)E), which is likely due to the inability of the insulin signaling

250 This current is likely due to the increased expression of Kir2.1 chan

251 ted decline in otherwise healthy individuals is likely due to the interaction of multiple factors inc

252 relief of LIN-1-mediated repression in P6.p is likely due to the known MAPK-dependent phosphorylatio

253 f NT3 to activate Rac1-GTP-cofilin signaling is likely due to the labile nature of NT3/TrkA complexes

254 This is likely due to the large accumulation of carbon aboveg

255 higher ionization efficiency of the rf lamp is likely due to the larger area of high intensity light

256 diversity and recombination intensity, which is likely due to the limited number of loci sampled in o

257 The lack of gastrointestinal toxicity is likely due to the low absorbed doses delivered to the

258 This is likely due to the low frequency at which long-lived T

259 This signal gain is likely due to the more significant conformational cha

260 The high photoconductivity thus obtained is likely due to the optimal molecular design that enabl

261 e formation of the superoxide-like component is likely due to the porosity of the activated carbon us

262 This is likely due to the rather different structural and fun

263 This is likely due to the reduced migration of STAT6(-/-) eos

264 uring the early stages of virus entry, which is likely due to the relatively high expression of the h

265 cts on the associated visceral organs, which is likely due to the release of endogenous opioids.

266 This is likely due to the route of infection being introgress

267 This regional difference is likely due to the significant differences of tissue h

268 iorated hyperglycemia in treated mice, which is likely due to the suppression of gluconeogenesis by s

269 ted based on expression in other tissues and is likely due to the unique role the liver has in insuli

270 oxylation phenotype of MYB115 overexpressors is likely due to the up-regulation of both flavonoid 3',

271 This was likely due to the accumulation of atmospheric degrad

272 28-53% for the other treatment types), which was likely due to the combined influence of the rich sou

273 ated predominantly with R. leguminosarum and was likely due to the coordination of Zn with cysteine-r

274 The decline of mice in control drainages was likely due to the decline in shrub cover that they u

275 The AMPKalpha1-specific effect on myogenesis was likely due to the dominant expression of alpha1 in m

276 The reduced release kinetics was likely due to the formation of large surface-bound M

277 accine-induced T-cell responses, this effect was likely due to the improved antigen delivery afforded

278 ase in peak force in Tg-R145W muscles, which was likely due to the increased Ca(2+) transient duratio

279 The enhanced brain endothelial cell invasion was likely due to the increased surface exposure of the

280 rease in MT2 and HJV upon neogenin knockdown was likely due to the inhibition of cell surface MT2 and

281 dynamic toxicity observed in Raisin sediment was likely due to the instability of NiS in surface sedi

282 This was likely due to the silicate weathering-negative feedb

283 e prolonged treatment with Abeta1-6A2VTAT(D) was likely due to the TAT intrinsic attitude to increase

284 c aerosol concentration along the Gulf Coast were likely due to the DWH spill.

285 Molecular typing confirmed these changes were likely due to the ingress of the globally distribut

286 mple-positive/pooled sample-negative results were likely due to the M. tuberculosis concentration bei

287 on changes over the ocean unexplained by SST were likely due to the water vapor transport dominated b

288 responses for viewing hands and tools, which is likely due to their common role in object-directed ac

289 se DeltasagA mutants in the liver and spleen were likely due to their reduction in the systemic disse

290 ore, the immunogenicity of the split vaccine was likely due to TLR7 recognition of virion RNA, as its

291 locus, we showed that the protective effect was likely due to transcript stability as revealed by re

292 The pro-apoptosis/cell death function of ATR is likely due to transcription stress because the lethal

293 Coexpression of genes is likely due to transcriptional control.

294 This is likely due to triplet-singlet surfaces crossing occur

295 The resistance of these melanomas is likely due to tumor heterogeneity, which may be expla

296 ifferent lines of evidence suggest that this is likely due to tumor micro-environment instead of tumo

297 ALT activity was low, which was likely due to underlying vitamin B-6 deficiency, whi

298 ations, but we conclude that this difference is likely due to unique aspects of non-African populatio

299 This inactivity is likely due to virus-mediated interference since the t

300 AR 5, indicating that the AR 5 fluctuations are likely due to weakened inter-repeat stabilization fr