1 Direct MTBDRplus testing
was negative for 1,594/1,612 sputum samples that were cu
2 Posttreatment CT scans
were negative for 10 patients but showed persistent dise
3 Analyses were conducted among subjects who
were negative for 14 HPV types on day 1.
4 nal diagnosis of possible IMD, CTPA findings
were negative for 14 patients and were positive for 1 pa
5 WAT deposits
were negative for (
18)F-FMPEP-d2, consistent with the im
6 value; all trials negative at 1 year for DFS
were negative for 5-year OS.
7 These cells
are negative for a marker of ON bipolar cells and restri
8 sitive for RUB strains of both genotypes and
was negative for a panel of human viruses.
9 Conversely, they
were negative for a definitive endoderm marker (Sox17) a
10 ctinomycetemcomitans and 41 participants who
were negative for A. actinomycetemcomitans.
11 suboptimal in paucibacillary specimens that
are negative for acid-fast bacilli using smear microscop
12 ive of active tuberculosis but sputum smears
were negative for acid-fast bacilli on 3 consecutive day
13 ral mucosal fibroblasts and skin fibroblasts
were negative for active telomerase, as assessed accordi
14 All cases found to
be negative for acute aortic disorders were grouped acco
15 MR images
were negative for acute injury in 354 of the 366 patient
16 ell as conventional and real-time PCR tests,
were negative for adenovirus.
17 ide additional benefits, and may potentially
be negative, for adolescent bone.
18 ulatory domain 1-positive centrocytes, which
are negative for all the B cell transcription factors.
19 the biliary lineage and that rare cells that
are negative for all three markers are transitional cell
20 ve or negative, but long term responses will
be negative for all populations, with the timing of the
21 mere group, and +2.3 years in the group that
was negative for all antigens tested) (P = 0.027).
22 The infectious workup
was negative for all three patients, and antibiotics wer
23 2), 3 (3.1%) had MPL mutations, and 4 (4.2%)
were negative for all 3.
24 37 (15.0%) were TP positive, and 78 (31.7%)
were negative for all tests.
25 In contrast, patients whose tumors
were negative for all three markers and those tumors tha
26 yes were CMV positive; the remaining 27 eyes
were negative for all viruses on PCR analysis.
27 They
are negative for alpha-fetoprotein (AFP), intercellular
28 NCAM), cytokeratin (CK) 19, albumin +/-, and
are negative for alpha-fetoprotein (AFP).
29 Before coculturing, the BMSCs
were negative for alpha-actinin and exhibited a nucleus
30 All BRG1-deficient cases
were negative for alterations in known therapeutic targe
31 e (apical) and Na/K ATPase (basolateral) and
was negative for amino peptidase-N.
32 n on bone marrow biopsy and fat pad aspirate
was negative for amyloid light-chain deposition.
33 al control individuals (age 65-89 years) who
were negative for amyloidosis, hypometabolism, and hippo
34 colitis not associated with spiral bacteria
were negative for Anaerobiospirillum spp. in the same as
35 Instead, RO(+) GC B cells
were negative for Annexin V, comprised mostly (93%) of C
36 Further, men were more likely to
be negative for anti-SSA/Ro, anti-SSB/La, and antinuclea
37 At baseline, four of these five patients
were negative for anti-AAV2 serum antibodies and the fif
38 p visit were available from 4 patients; they
were negative for anti-HEV IgM, but levels of anti-HEV I
39 isk of PML was lowest among the patients who
were negative for anti-JC virus antibodies, with the inc
40 volving 8323 women 18 to 30 years of age who
were negative for antibodies to HSV-1 and HSV-2.
41 Analysis of his serum
was negative for antinuclear antibody (or ANA), cytoplas
42 neuronal or endothelial) and CaM antibodies,
were negative for any NOS isoform or CaM.
43 ondria as well as submitochondrial particles
were negative for any peptide from any NOS isoform.
44 tted significantly more urine specimens that
were negative for any type of drugs and tended to have l
45 pendicitis, nonvisualization of the appendix
was negative for appendicitis in 98% (95% CI: 71%, 100%)
46 Blood cultures
were negative for bacterial growth.
47 arance on routine microbiological media that
were negative for bacterial microorganisms.
48 Stool specimens
were negative for bacterial pathogens by culture and neg
49 ogy and PCR of the patient's blood and serum
were negative for Bartonella henselae, Bartonella quinta
50 patients (66%) tested positive and 59 (34%)
were negative for Bcl-2/IgH.
51 All strains
were negative for beta- and epsilon-toxin genes.
52 cultures showed that insulin-positive cells
were negative for beta-galactosidase.
53 with or without prior BMTx from C57BL/6 mice
were negative for BM-derived or extrarenal ECFCs.
54 d or wild-type hMSCs, whereas empty controls
were negative for bone formation.
55 at capacity at constant pressure, DeltaC(p),
was negative for both sites, suggesting the importance o
56 ere done in a restricted cohort of women who
were negative for both cervical HPV 16 and HPV 18 DNA an
57 fficacy against genital disease in women who
were negative for both HSV type 1 (HSV-1) and HSV-2 anti
58 Normal tendons
were negative for both Mphi and FPR2/ALX.
59 remaining 135 healthy first-degree relatives
were negative for both POCT and EMA.
60 +)/RPR(-), 6 were TPP(H)A(-)/RPR(+), and 254
were negative for both tests.
61 An exploratory cohort of FTH (n = 10)
was negative for BRAF(V600E) and NRAS codon 61 mutations
62 on between carriers of BRCA1/2 and women who
are negative for BRCA1/2 mutations.
63 These cells
were negative for Brn-3b and positive for both calretini
64 essed Brn3b, a vast majority of the M1 cells
were negative for Brn3b.
65 rcinoma in situ of the bladder, and 42 nodes
were negative for cancer.
66 alled for additional procedures and findings
were negative for cancer.
67 Blood samples
were negative for Capnocytophaga spp.
68 diac testing, laboratory workup, and imaging
were negative for cardiac or neurologic etiology.
69 as preferentially found in CD11b(+) DCs that
were negative for CD103.
70 8%, 93.8%, and 3.2%, respectively; all cells
were negative for CD11b, CD19, and CD45.
71 Germinal center B cells
are negative for CD300a expression.
72 ants were also able to infect U87 cells that
were negative for CD4, CD8, and common HIV coreceptors,
73 s demonstrated that most of the GFP(+) cells
were negative for CD45, a macrophage and hematopoietic c
74 Serology findings
were negative for celiac disease.
75 senting specific Ig and neutralizing factors
were negative for cellular response (and vice versa).
76 TRG@ PCR
was negative for clonal rearrangements in 29 of 31 cases
77 ncies of our cohort of infants with cCMV and
was negative for CMV in 47 (15.6%).
78 e use of the liquid-saliva PCR assay, 17,569
were negative for CMV, and the remaining 85 infants (0.5
79 with MCL morphology and immunophenotype that
were negative for cyclin D1 expression/t(11;14)(q13;q32)
80 indings, 65 cases were positive and 95 cases
were negative for dengue fever.
81 All patients tested
were negative for dengue virus infection as assessed by
82 sly reported to contain SV40 large T antigen
were negative for detection of the virally encoded oncop
83 methylation reveals that the male pronucleus
is negative for di- and trimethyl H3-K9 yet the female i
84 All recipients
were negative for donor HLA-specific antibodies before t
85 the highest ACR severity of each patient and
were negative for donor-specific antibodies (DSA), C4d,
86 th flat HGD (93.3%) and 2 of 45 samples that
were negative for dysplasia (4.4%).
87 Freshly isolated primary osteoblasts
are negative for E11 expression but begin to express thi
88 RNA shuttling can be detected in cells that
are negative for EBER shuttling, we demonstrate the shut
89 General areas of both ETCs
were negative for EBOV RNA.
90 iltrated by alpha-SMA-expressing CAFs, which
were negative for EGFP and the human Y-probe, but positi
91 AV-resistant human, murine, and simian cells
were negative for ELR1 expression but became susceptible
92 Blank dialysates
were negative for enzymatic activity that could cleave t
93 , the cytokeratin-positive cells in the SLNs
were negative for ER.
94 18%) prostate cancers were ERG-positive, and
being negative for ERG staining was associated with high
95 fold higher incidence of breast cancers that
are negative for estrogen receptor, progesterone recepto
96 ents to be diagnosed with breast cancer that
is negative for estrogen and progesterone receptors (ER/
97 1% EGFR positive versus 44% in controls) and
were negative for estrogen receptor (ERalpha; 32% ER neg
98 The tumors formed
were negative for estrogen receptor, progesterone recept
99 lasma and/or stool (EV(+)) and the remainder
were negative for EV and other viruses (EV(-)).
100 entropies of activation for these reactions
are negative (for example, DeltaS() = -15 eu for 1Z and
101 All 16 SCHs examined by immunohistochemistry
were negative for expression of HIF-1alpha.
102 All
were negative for family history of PD and known pathoge
103 ol mice and early-stage PanINs from KPC mice
were negative for fascin, but approximately 6% of PanIN3
104 e located within the renal interstitium, but
are negative for Foxd1, an established marker of stromal
105 ained for CD4 and, less frequently CD25, but
were negative for FoxP3.
106 taining and culture of all BAL fluid samples
were negative for fungal infection.
107 All cultures
were negative for fungus.
108 Follow-up after six months
was negative for further recurrence.
109 ons in MYH, a base excision repair gene, and
are negative for germline mutations in the APC gene.
110 Tie2-GFP mouse DPSCs
were negative for GFP, indicating the absence of endothe
111 Tc17
are negative for granzyme B, perforin message, and cytol
112 GRN mutations (type A TDP-43 pathology) but
are negative for GRN mutations.
113 o positive for relative survival, whereas it
was negative for growth rate.
114 es of five or more consecutive cultures that
were negative for growth of M. tuberculosis.
115 lowing initiation of antimicrobial treatment
were negative for growth.
116 If the RP v1.7 results
were negative for HAdV, then the specimens were reflexed
117 3, 5, and 8 years, 85%, 88%, 87.0%, and 92%
were negative for HBsAg, respectively, and 95%, 99%, 100
118 mples (13.4%) were positive, and 207 (86.6%)
were negative for HCMV DNA.
119 urine specimens (4/6 specimens), and results
were negative for Hcp100 in all healthy control urine sp
120 associated HCC survive longer than those who
are negative for HCV.
121 HCV RNA, and 179 (26%) had test results that
were negative for HCV RNA.
122 OX9 and pancytokeratin (features of CCA) but
were negative for HepPar1 (a marker of hepatocellular ca
123 Genetic studies confirmed all seven cases
were negative for HFE mutations C282Y and H63D.
124 il 4 consecutive quarterly follow-up results
were negative for HGD and then biannually up to 5 years
125 nts on suppressive ART, 8 of 12 samples that
were negative for HIV-1 RNA by gSCA had detectable HIV-1
126 studied: (1) control mice, littermates that
are negative for hNox4 transgene but Cre positive; (2) c
127 ged 15-19 years, who had normal cytology and
were negative for HPV at recruitment from a single famil
128 All 14 FCDIIb specimens
were negative for HPV DNA with all 4 primer sets.
129 iated it from other types of phocomelia that
are negative for HR.
130 The selected 18 p16-positive cases tested
were negative for HR-HPV using mRNA ISH.
131 equate DNA isolated in 24 cases, 23 of which
were negative for HR-HPV.
132 All CSF samples
were negative for HSV.
133 Serum analysis
was negative for human immunodeficiency virus type 1 and
134 ed PEL-like lymphoma in an elderly woman who
was negative for human immunodeficiency viruses 1 and 2,
135 tor and progesterone receptor and those that
were negative for human epidermal growth factor receptor
136 estational age of fetus, 12 to 27 weeks) who
were negative for human immunodeficiency virus infection
137 nts who did not use tobacco, and tumors that
were negative for human papillomavirus (HPV) had more mu
138 Immunofluorescence
was negative for Ig deposits, although electron microsco
139 GP1 (n=240); and control group, patients who
were negative for IgA aB2GP1 (n=974).
140 Most patients
were negative for IgG aCL at baseline and remained so at
141 atients who received infliximab plus MTX and
were negative for IgG aCL at baseline were positive for
142 atients who received infliximab plus MTX and
were negative for IgM aCL at baseline were positive for
143 were detected only by individual testing and
were negative for IgM antibody, 29 percent were detected
144 nations, but only 15 of the 148 (10 percent)
were negative for IgM antibody.
145 lthy controls were positive for IgG, but all
were negative for IgM.
146 Lamina propria DCs containing parasites
were negative for IL-12p40.
147 Direct immunofluorescence study results
were negative for immune reactants.
148 Although the TTC technique
was negative for infarct, histopathological analysis rev
149 4 BALF specimens (2 for each CARV case) that
were negative for infection and collected at a duration
150 The scan results
were negative for infection in 29 patients; 25 had fluid
151 patients; 25 had fluid culture results that
were negative for infection, and aspiration was unsucces
152 ease and who underwent standard testing that
was negative for infectious diseases, repeatedly donated
153 Although this trial
was negative for its primary and secondary endpoints, we
154 Although this exploratory study
was negative for its primary endpoint, VS/ALIC DBS demon
155 ile the third isolate expresses only LPS and
is negative for K1.
156 The urine examination
was negative for ketones.
157 sing cells exhibited high Ras expression and
were negative for Ki-67, whereas most late-passage H-Ras
158 e than those with allografts from donors who
were negative for KIR2DS1 (26.5% vs. 32.5%; hazard ratio
159 However, 15 to 50% of clinical isolates
are negative for known adhesins, making it difficult to
160 They
were negative for L-rhamnose and D-glucitol (sorbitol).
161 supported by positive results of rapid tests
were negative for leptospirosis on the basis of our diag
162 The target population was defined as
being negative for lineage markers and double-positive f
163 (CD)29, CD133, and stem cell antigen-1, but
were negative for lineage markers CD31, CD45, and F4/80.
164 These cells
were negative for lineage-specific markers (Lin(-)), exp
165 odendrocytes and astrocytes, but these cells
were negative for LSTc and did not bind virus.
166 bNOS-IR somas
were negative for LY, thus they were identified as ACs;
167 Tumor-free surrounding lung tissue
was negative for MAGE-A4.
168 rointestinal endoscopy-guided mucosal biopsy
was negative for malignancy.
169 The six dissected lymph nodes
were negative for malignancy.
170 Lancashire, UK) and river Tywi (South Wales)
were negative for MAP.
171 astric epithelial cells with DNA damage that
were negative for markers of apoptosis accounted for 42%
172 Nutrition Examination Survey, 1988-1994, who
were negative for markers of viral hepatitis B and C.
173 ptotic effect was detected in ALL cells that
were negative for MDM2 and wt-p53.
174 One sentinel node
is negative for metastasis.
175 s were clear, and three sentinel lymph nodes
were negative for metastasis.
176 Staging scans
were negative for metastatic disease.
177 Staging scans
were negative for metastatic disease.
178 ol specimens and the lymphoblastoid cultures
were negative for methylation of the three genes; (4) no
179 Finally, we show that PBL
is negative for MHC II.
180 cizumab acquired from compounding pharmacies
were negative for microbial contaminants and endotoxin.
181 In all, 35% (VTD) and 27% (VTDC) of patients
were negative for minimal residual disease (MRD) during
182 111 were significant even among patients who
were negative for minimal residual disease after remissi
183 t in whom peripheral blood had been found to
be negative for MMc on 4 occasions, and tissue from a su
184 Abcc6 null mice
were negative for Mrp6 expression in the liver, and comp
185 Cells that survive selinexor
are negative for multiple proliferation biomarkers, indi
186 ase III (POLR3)-related leukodystrophy cases
are negative for mutations in the previously identified
187 and all 89 patients tested by DNA sequencing
were negative for mutations in HER2 exon 20.
188 were compared with those of individuals who
were negative for mutations in THAP1.
189 performed in a cohort of LQTS patients that
were negative for mutations in the 11 known LQTS-suscept
190 Two CXN families, which
were negative for mutations in the NHS gene, were furthe
191 age was confirmed, and other candidate genes
were negative for mutations.
192 half of individuals positive for guaiac FOBT
are negative for neoplasia on colonoscopy.
193 NS3-positive cells
were negative for neuron and oligodendrocyte phenotypic
194 Molecular analyses
were negative for neurotropic viruses.
195 ough investigation of patients with LETM who
are negative for NMO-IgG may lead to an alternate cause
196 ine to l-[(14)C]citrulline conversion assays
were negative for NOS activity.
197 ant variants in most (16 of 21) samples that
were negative for NVP resistance by standard genotype, a
198 lls that delineate large axon fascicles, but
are negative for OEC markers.
199 The patient
was negative for onconeural (Hu, Yo, Ri, CV2, Tr, amphip
200 ntermolecular potential energy surface (PES)
is negative for one and two H(2) molecules in C(70) but
201 ts with antiphospholipid syndrome (APS) that
are negative for other isotypes.
202 le, harbors the colonization factor CS23 but
is negative for other known adhesins.
203 osinophilia, and multiple stool examinations
were negative for ova and parasites.
204 Liver, uterus, and spleen
were negative for P. gingivalis DNA among all other chal
205 fluid samples from infected and control dams
were negative for P. gingivalis DNA.
206 This tumour
was negative for p16 and positive for Rb protein.
207 e, associations with neurodevelopment scores
were negative for pairs with either high maternal, high
208 alysis, using a panel of degenerate primers,
was negative for papilloma family viruses.
209 All respiratory samples
were negative for PARV4.
210 lysis was performed in one large family that
is negative for pathogenic PRRT2 mutations.
211 r study of archived PDNS tissue samples that
were negative for PCV2 by IHC analysis identified 45 of
212 PD-L1 because of a genetic event will always
be negative for PD-L1 on cancer cells.
213 FL tumor cells
were negative for PD-1 ligands, but PD-L1(+) histiocytes
214 Overall, CT angiograms
were negative for PE in 1806 (90.16%) of 2003 patients.
215 violation in 34% of patients (68 of 200) and
were negative for peritoneal violation in 66% of patient
216 fact that most conventional mutagenic assays
were negative for PFOS, we propose that PFOS-induced mut
217 created a pure ON bipolar cDNA library that
was negative for photoreceptor unique genes.
218 nd to be positive and 13 (28%) were found to
be negative for pneumococci by both methods; each method
219 ed in a wild-type swine isolate and found to
be negative for poly-N-acetylglucosamine (PNAG)-like mat
220 The remaining 18 sera
were negative for potassium channel subunits and associa
221 All animal and environmental specimens
were negative for poxvirus and both patients had complet
222 om the subset of the mammary epithelium that
is negative for PR and probably ER as well.
223 Of 1,106 brain autopsies, 352 (32%)
were negative for prion disease, 304 of which had adequa
224 ional beta-catenin-positive hepatocytes that
were negative for progenitor markers were also observed
225 The infiltrates
were negative for proliferating cell nuclear antigen and
226 His medical history
was negative for psoriasis.
227 pe, SFFV gp55-sf-Stk-transformed fibroblasts
are negative for PU.1.
228 e detected in 2/4 stool extract samples that
were negative for PV in cell culture.
229 scence, and exhaustion marker expression and
were negative for regulatory CD8(+) T cell markers.
230 subjects received all three vaccinations and
were negative for relevant HPV types at enrollment, and
231 cytometry at the end of treatment, 41 (72%)
were negative for residual disease.
232 Family history
was negative for retinitis pigmentosa and haemoglobinopa
233 excluded because next-generation sequencing
was negative for ROS1 fusion.
234 lla cases collected on the day of rash onset
are negative for rubella virus-specific IgM.
235 or CD34, vimentin, and focally for CD68, but
were negative for S100 and SMA.
236 adult iliac bone, newly embedded osteocytes
were negative for sclerostin staining but became positiv
237 lance of the speciation and extinction rate,
is negative for selfing species.
238 to-Van Laere kindred, whose affected members
were negative for SLC52A3 mutations.
239 as but not in carcinomas, because the tumors
are negative for smooth muscle actin.
240 ng high levels of RXRalpha immunoreactivity,
are negative for stellate or smooth muscle cell markers.
241 d nucleic acid amplification test (NAAT) but
were negative for stx1 and stx2 following nucleic acid s
242 While glucagon-immunoreactive amacrine cells
were negative for substance P in central regions of the
243 However, some cancer cells selected to
be negative for surface CD24 (surCD24(-)) still retain a
244 Although they
were negative for surface IgM and CD5 expression, iPS-de
245 In both cases, skin prick tests
were negative for suspected seafoods.
246 Knockdown cells that
were negative for syndecan-1 expression became apoptotic
247 History
was negative for systemic disorders.
248 rphan nuclear receptor gammat and FoxP3, but
are negative for T-bet and GATA-3 transcription factors.
249 All 71 mesotheliomas
were negative for T-antigen transcripts by RT-PCR, and l
250 Of the clinical isolates, 27%
were negative for tdh and trh, while 45% contained both
251 Four subjects positive for both POCT and EMA
were negative for TG2-IgA.
252 t may be driven primarily by individuals who
are negative for the established AD genetic risk factor,
253 ormal, are positive for ALDH1 expression but
are negative for the expression of ER.
254 lated from the hair-follicle bulge area that
are negative for the keratinocyte marker keratin 15 can
255 haviors such as urine drug test results that
are negative for the prescribed opioid should be fully i
256 The inclusions
are negative for the TAR DNA binding protein 43 and fuse
257 ajority of thalamotectal cells were found to
be negative for the calcium-binding proteins calbindin,
258 ation and 1573 scans from 754 women known to
be negative for the mutations.
259 ere suspected of being infected but shown to
be negative for the virus by PCR.
260 m 15 healthy donors previously determined to
be negative for the viruses.
261 The probe test
was negative for the 280 samples that were negative by t
262 onfigurations of decaalanine, and gamma = -5
was negative for the decaalanines.
263 normal human breast and prostate epithelium
was negative for the major isoform [reading frame-1 (RF1
264 This study
was negative for the primary end point, but findings for
265 nt model (p=0.24), indicating that the trial
was negative for the primary endpoint.
266 1.7% versus 6.2%, P=0.008) than patients who
were negative for the antibody.
267 The strains
were negative for the ctrA gene but were positive for th
268 Sequencing also showed that all strains
were negative for the FetA receptor gene.
269 Patients
were negative for the FIP1L1-PDGFRA fusion gene and requ
270 nuclear leukocyte (PMN) cell morphology that
were negative for the Gr-1 neutrophil maturation marker.
271 we assigned 481 patients (nearly all of whom
were negative for the human immunodeficiency virus) with
272 ardless of CSF1 expression status, most GAMs
were negative for the M2 polarization markers ARG1 and C
273 Fifty (1.6%) macrolide-resistant isolates
were negative for the mef and the erm resistance genes.
274 Neurones expressing functional B1 receptors
were negative for the neuropeptides CGRP and substance P
275 Most fecal specimens
were negative for the pathogens tested in both studies,
276 The remaining 19 patients
were negative for the posterior cingulate sign.
277 Gs from both depleted and mock-depleted mice
were negative for the presence of the LAT transcript.
278 ET, and all nine non-clear-cell renal masses
were negative for the tracer.
279 All patients
were negative for the VGKC-complex-associated proteins L
280 ethyl-D-aspartate receptor antibodies and 38
were negative for these antibodies.
281 r of follow-up among IDUs whose test results
were negative for these viruses at baseline (n = 2061 an
282 ere carried out, and an animal determined to
be negative for this reactivity was immunized by a skin
283 the mobile genetic elements Tn1545 and mega,
were negative for Tn1207.1, had tetracycline resistance
284 By contrast, most nuclei within true knots
are negative for transcriptional markers but positive fo
285 An exploratory abdominal laparotomy
was negative for traumatic injury.
286 s retinoic acid- and vitamin D-like activity
were negative for Tritan and PC migrates.
287 myocardial infarction, even in patients who
are negative for troponin T (<0.1 ng per milliliter) at
288 of myocardial necrosis (defined as those who
were negative for troponin T), the base-line myeloperoxi
289 lity (8.0% vs. 2.7%, P<0.001) than those who
were negative for troponin.
290 (18%) were positive for tumor and 186 (82%)
were negative for tumor.
291 ve by fliC PCR for serovar Typhi; 4 of these
were negative for tviA and tviB.
292 or positive results, while the blood samples
were negative for up to 1 x 10(8) CFU/ml of the Deltacaf
293 n the UK, The Netherlands and Australia that
were negative for variants in known predisposition genes
294 All blood samples
were negative for vector DNA.
295 analyzed among 14,527 adult participants who
were negative for viral hepatitis B and C and iron overl
296 All 16 donors
were negative for West Nile virus-specific IgM antibody
297 Rotateq and Rotarix), and yellow fever virus
were negative for XMRV and highly related MLV sequences.
298 All healthy blood donors
were negative for XMRV proviral sequences.
299 istance mutations, and an additional 6 women
were negative for Y181C after SD-NVP.
300 on but had no cerebral abnormalities, and 11
were negative for Zika virus but had cerebral abnormalit