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1 etiopathogenesis of this devastating disease is not fully understood.
2 cient amplitude to trigger action potentials is not fully understood.
3 ne pathology during acute malaria infections is not fully understood.
4 , but their importance in vascular functions is not fully understood.
5 e, but how these pathogens are so successful is not fully understood.
6 ilience to outbreaks of generalist pathogens is not fully understood.
7 hanisms responsible for its tight regulation is not fully understood.
8 ical finding and its relation to clinical SM is not fully understood.
9 mechanism of PRC2 recruitment to CpG islands is not fully understood.
10 formation in Alzheimer's disease (AD) brain is not fully understood.
11 s component of inflammation and angiogenesis is not fully understood.
12 dically over low-lying areas of the WAIS and is not fully understood.
13 phospholamban and sarcolipin regulate SERCA is not fully understood.
14 re translocated to the forming autophagosome is not fully understood.
15 ients with breast cancer, yet its trajectory is not fully understood.
16 n/stability of another, within the sequence, is not fully understood.
17 efficient division through a mechanism that is not fully understood.
18 tep in the viral lifecycle, but its assembly is not fully understood.
19 driving extrinsic apoptosis and necroptosis is not fully understood.
20 ch HSA binds Abeta monomers and protofibrils is not fully understood.
21 TIC induction or maintenance in lung cancer is not fully understood.
22 ral and molecular basis of this encephalitis is not fully understood.
23 l pollution is important, even if its impact is not fully understood.
24 rate recognition and acetylation in cohesion is not fully understood.
25 However, its mechanism of action is not fully understood.
26 of exposure to electronic cigarettes (eCig) is not fully understood.
27 ns, but why this occurs at a given cell size is not fully understood.
28 er, the mechanism of EGCG-induced remodeling is not fully understood.
29 amino acid metabolism on cancer progression is not fully understood.
30 with asthma, but how these proteins interact is not fully understood.
31 energy metabolism in mitochondria, however, is not fully understood.
32 signals control mitotic spindle orientation is not fully understood.
33 anscriptional regulation of TIA biosynthesis is not fully understood.
34 phosphorus acquisition from the environment is not fully understood.
35 uses them to form a coherent unitary entity is not fully understood.
36 specificity for WFA recognition of LacdiNAc is not fully understood.
37 duces genomic instability, but the mechanism is not fully understood.
38 How cells recognize membrane curvature is not fully understood.
39 DX3 in placental trophoblast cells under ICP is not fully understood.
40 w they are positioned close to these lesions is not fully understood.
41 ng underlying these distinct cellular states is not fully understood.
42 of these connections vary inside stem cells is not fully understood.
43 al role of the TGR5 receptor in the pancreas is not fully understood.
44 ndometrial and breast safety during TSEC use is not fully understood.
45 chanism underlying this clinical observation is not fully understood.
46 ver, the precise mechanism of nephrotoxicity is not fully understood.
47 dritic cells promote this cell-fate decision is not fully understood.
48 APC/C specifically recognizes its substrates is not fully understood.
49 hich this process contributes to the disease is not fully understood.
50 Regulation of mitochondrial Drp1 recruitment is not fully understood.
51 However, the mechanism of this repression is not fully understood.
52 ver, the mechanism underlying this phenotype is not fully understood.
53 rmeabilizes the mitochondrial outer membrane is not fully understood.
54 to cellular vitamin B6 homeostasis in plants is not fully understood.
55 thway in disease development and progression is not fully understood.
56 ted inhibition of osteoclast differentiation is not fully understood.
57 cleus and VCSL on cortical visual processing is not fully understood.
58 eflex function both in normal and CHF states is not fully understood.
59 cal impact of increased translational errors is not fully understood.
60 te of a tissue is an important question that is not fully understood.
61 order structures confers different functions is not fully understood.
62 KLRG1-induced inhibition of NK cell function is not fully understood.
63 ort, the underlying nature of this mechanism is not fully understood.
64 dvantage of producing such similar isotoxins is not fully understood.
65 tilage is induced and utilized during repair is not fully understood.
66 egree of Tfh cell heterogeneity and function is not fully understood.
67 ed by HIV, although how this is accomplished is not fully understood.
68 d progression of the humoral immune response is not fully understood.
69 sis of LRRK2-linked Parkinson's disease (PD) is not fully understood.
70 nt system to produce an intermediate radical is not fully understood.
71 ram during cancer initiation and progression is not fully understood.
72 r basis of TRPV1 activation and deactivation is not fully understood.
73 tic and affective signs of opiate withdrawal is not fully understood.
74 c, but the basis of their antitumor activity is not fully understood.
75 (Treg cells) control lymphocyte homeostasis is not fully understood.
76 How protein structure encodes functionality is not fully understood.
77 frequency bursts, but the function of bursts is not fully understood.
78 se cations affect charge storage performance is not fully understood.
79 he physical basis for its target specificity is not fully understood.
80 e mesenchymal stem cell (MSC) mechanosensing is not fully understood.
81 tegration of PAK1 into the estrogen response is not fully understood.
82 f Rho-GAP cellular localization and function is not fully understood.
83 n growth and bone accretion during childhood is not fully understood.
84 the nature of the transport band gap in CNTs is not fully understood.
85 ppropriate content and dosage of transcripts is not fully understood.
86 and animal studies, but the direct mechanism is not fully understood.
87 cell identity is established and maintained is not fully understood.
88 cation on gingival connective tissue biology is not fully understood.
89 ling through phosphorylation and acetylation is not fully understood.
90 regulate airway smooth muscle contractility is not fully understood.
91 t and organization is a complex process that is not fully understood.
92 microbial infection and disease progression is not fully understood.
93 environment, but the basis for their effects is not fully understood.
94 the role of the pericytes in BRB regulation is not fully understood.
95 , the pathogenesis caused by the new variant is not fully understood.
96 vents are orchestrated in the aging progress is not fully understood.
97 interplay in orchestrating physical activity is not fully understood.
98 d opiate dependent individuals in particular is not fully understood.
99 L-4), hence class switching to IgE and IgG1, is not fully understood.
100 underlying mechanism of such co-localization is not fully understood.
101 and how sensory systems process backgrounds is not fully understood.
102 TBI are supportive, and the pathophysiology is not fully understood.
103 ic and interpopulation reproductive barriers is not fully understood.
104 atterns associated with spontaneous seizures are not fully understood.
105 ion after injury and link new and old tissue are not fully understood.
106 ween sensory recognition and motor execution are not fully understood.
107 ern PD1 expression and exhaustion in T cells are not fully understood.
108 but the mechanisms triggering its pathology are not fully understood.
109 in the absence of stabilizing ClC-1 current are not fully understood.
110 anisms regulating renal epithelial autophagy are not fully understood.
111 utions of and interplay among these features are not fully understood.
112 rol TRM cell differentiation and homeostasis are not fully understood.
113 e pathways controlling Mo-DC differentiation are not fully understood.
114 ces in cocaine self-administration behaviors are not fully understood.
115 during herpes simplex virus (HSV) infection are not fully understood.
116 l system, but the cell types underlying this are not fully understood.
117 the mechanisms conducting Opi1p localization are not fully understood.
118 ining apoptotic resistance of RA macrophages are not fully understood.
119 st IFITM inhibition, the detailed mechanisms are not fully understood.
120 s that drive the pro-tumor functions of TAMs are not fully understood.
121 ver, their identities and interrelationships are not fully understood.
122 d and fine-tuned energy conversion mechanism are not fully understood.
123 ypically sulfur, S), their synergistic roles are not fully understood.
124 on, the functions of VIP cell depolarization are not fully understood.
125 underlying their downstream output responses are not fully understood.
126 ribed, these processes and their fine-tuning are not fully understood.
127 embrane protein partitioning to raft domains are not fully understood.
128 sometimes affect activity, but these effects are not fully understood.
129 proliferation and activation of fibroblasts are not fully understood.
130 egative impact of each remodelling mechanism are not fully understood.
131 nctions and the mechanisms by which they act are not fully understood.
132 ehavioral effects of these early experiences are not fully understood.
133 these effector elements in parasite immunity are not fully understood.
134 ctions of extrinsic tissues in tendon repair are not fully understood.
135 such changes on harmful algal blooms (HABs) are not fully understood.
136 hat allow for a persistent E. coli infection are not fully understood.
137 Unlike Cxs, the functions of Panxs are not fully understood.
138 vironmental signals with endogenous pathways are not fully understood.
139 ever, neuroanatomic and molecular mechanisms are not fully understood.
140 omologs in noncanonical autophagic processes are not fully understood.
141 he mechanisms responsible for RA homeostasis are not fully understood.
142 tissue pathology, but underlying mechanisms are not fully understood.
143 s both upstream of and downstream from FOXP1 are not fully understood.
144 ie this disease, but the pathogenic pathways are not fully understood.
145 by which Atgl is induced during adipogenesis are not fully understood.
146 hat mediate the transition from use to abuse are not fully understood.
147 muscle cells, but the underlying mechanisms are not fully understood.
148 rojections from the MLR to brainstem neurons are not fully understood.
149 proanthocyanidin (PA) biosynthesis in plants are not fully understood.
150 the mechanisms that control their repression are not fully understood.
151 ulate this pathway during memory acquisition are not fully understood.
152 he precise mechanisms underlying its control are not fully understood.
153 echanisms underlying these responses in glia are not fully understood.
154 s in ECM proteins contribute to angiogenesis are not fully understood.
155 d nature of these deterministic interactions are not fully understood.
156 on and role in the activation of macrophages are not fully understood.
157 egative pleiotropic effects for reasons that are not fully understood.
158 ife, the evolutionary dynamics leading to it are not fully understood.
159 b11-positive vesicles at the plasma membrane are not fully understood.
160 he precise mechanisms underlying the disease are not fully understood.
161 s in the CNS; however, underlying mechanisms are not fully understood.
162 is activated and PAR is robustly synthesized are not fully understood.
163 fic contributions of KCs to plaque formation are not fully understood.
164 gical consequences of forming ER-PM contacts are not fully understood.
165 thrombus growth as a multicomponent process are not fully understood.
166 idual loci within a topological domain (TAD) are not fully understood.
167 mechanisms that control this important event are not fully understood.
168 coupling of receptor binding to F activation are not fully understood.
169 But the underlying molecular mechanisms are not fully understood.
170 ts only natural host (humans) are unique and are not fully understood.
171 the mechanisms underlying these alterations are not fully understood.
172 The underlying mechanisms are not fully understood.
173 r, the cellular mechanisms by which it works are not fully understood.
174 chanisms by which PKC induces synaptogenesis are not fully understood.
175 potential protective mechanisms of hepcidin are not fully understood.
176 the underlying cell and molecular mechanisms are not fully understood.
177 s) that contribute to neurofibroma formation are not fully understood.
178 gical mechanisms that underlie these changes are not fully understood.
179 r, the underlying neurobiological mechanisms are not fully understood.
180 al cells; however, the underlying mechanisms are not fully understood.
181 Mechanisms of these cardiotoxicities are not fully understood.
182 The underlying mechanisms, however, are not fully understood.
183 cells (HSCs) to maintain stem cell identity are not fully understood.
184 ndent mechanisms of myeloid cell recruitment are not fully understood.
185 mechanisms mediating its therapeutic effects are not fully understood.
186 contributing toward APC/C catalytic activity are not fully understood.
187 tes breast cancer cell motility and invasion are not fully understood.
188 associated junctions with spatial precision are not fully understood.
189 ogical mechanisms that maintain this barrier are not fully understood.
190 sms and the emotionally vulnerable phenotype are not fully understood.
191 f how this overexpression drives the disease are not fully understood.
192 transitions between these domains, however, are not fully understood.
193 potent growth inhibitor to a tumor promoter are not fully understood.
194 owever, the factors that disrupt homeostasis are not fully understood.
195 meditation and Ayurveda on human physiology are not fully understood.
196 roles in cancer, but the mechanisms involved are not fully understood.
197 enomenon; however, the underlying mechanisms are not fully understood.
198 and signalling dependence of ILC progenitors are not fully understood.
199 the many aspects of arterial stiffness which are not fully understood.
200 echanisms determining impacts on the N cycle are not fully understood.
201 sis for its contributions to metastatic risk are not fully understood.
202 isms that result in these noise correlations are not fully understood.
203 sms of acute pain transition to chronic pain are not fully understood.
204 hagus (BE) to esophageal adenocarcinoma (EA) are not fully understood.
205 l fates under such widely varying conditions are not fully understood.
206 m-atom correlations in the superfluid state, are not fully understood.
207 e, and its effects on CA1 synaptic function, are not fully understood.
208 details of how mural cells stabilize vessels are not fully understood.
209 g rise to their high temperature sensitivity are not fully understood.
210 e mechanisms that induce persister formation are not fully understood.
211 een microbiota and the intestinal epithelium are not fully understood.
212 role of agrp2 and its relationship to agrp1 are not fully understood.
213 iating effects of endogenous GLP-1 signaling are not fully understood.
214 otic cascade and events leading to extrusion are not fully understood.
215 As in glioblastoma cell growth and apoptosis are not fully understood.
216 s regulated in different biological contexts are not fully understood.
217 t the routes and mechanisms of sensitization are not fully understood.
218 g cell migration in prostate cancer patients are not fully understood.
219 n mechanism and composition of intermediates are not fully understood.
220 molecular players in these signaling events are not fully understood.
221 , its occurrence and mechanism of regulation are not fully understood.
222 na and the factors that regulate its actions are not fully understood.
223 ver, the precise mechanisms for this process are not fully understood.
224 ecular mechanisms underlying these phenomena are not fully understood.
225 chanism of the SLC25A46-related neuropathies are not fully understood.
226 a-cell development and functional maturation are not fully understood.
227 V-1) infection risk; however, the mechanisms are not fully understood.
228 hysiological implications of this adaptation are not fully understood.
229 However, the mechanisms by which this occurs are not fully understood.
230 e 1 (HDAC1) and its subcellular localization are not fully understood.
231 stic explanations of photoreceptor coactions are not fully understood.
232 nderlying mechanisms and cell types involved are not fully understood.
233 and mechanisms that guide this coordination are not fully understood.
234 ing mechanisms of PD-L1 expression in cancer are not fully understood.
235 e mechanisms underlying disease pathogenesis are not fully understood.
236 imary tumor to premetastatic niche formation are not fully understood.
237 ly persist throughout life, for reasons that are not fully understood.
238 chanisms leading to BCR over-activity in CLL are not fully understood.
239 f peripheral presentation of ocular antigens are not fully understood.
240 the mechanisms directing with this response are not fully understood.
241 pment of cardiac disease in type II diabetes are not fully understood.
242 e development and osteoblast differentiation are not fully understood.
243 eractions between different genetic pathways are not fully understood.
244 f antibiotic sensitive and resistant strains are not fully understood.
245 lecular mechanism underlying this modulation was not fully understood.
246 sm(s) for the vulnerability of DA neurons is(are) not fully understood.
247 help the processed PGs to achieve this goal were not fully understood.
248 echanisms underlying phagocytosis in the RPE are not fully understood, although dysfunction of this p
249 atopic dermatitis (AD) and food allergy (FA) is not fully understood, although a causal relationship
251 hages during tissue regeneration upon injury are not fully understood and their study lacks adequate
252 The mechanism of alpha-synuclein pathology is not fully understood and is a subject of active resea
253 ism of altering focus, termed accommodation, is not fully understood and seemingly conflicting theori
254 ysiology of autistic spectrum disorder (ASD) is not fully understood and there are no diagnostic or p
255 enicity in AAV vector-mediated gene transfer are not fully understood, and effective solutions for ov
256 (HF), the mechanisms behind this protection are not fully understood, and not all patients with HF r
257 vertheless, the formation of these compounds is not fully understood, and development of new compound
258 he killer cell Ig-like receptor (KIR) family are not fully understood, as the ligands for these recep
260 ecific B cells in patients with food allergy are not fully understood but are of major pathogenic and
261 in the neurogenic regions of the adult brain is not fully understood but it has been shown that vascu
262 e sequence of events leading to lung disease is not fully understood but recent data show that the cr
263 isms behind these nonspecific effects of BCG are not fully understood, but a shift from a TH2 to a TH
264 ecreate many anthocyanin blue hues in nature are not fully understood, but interactions with metal io
266 hich they involve change in genetic ancestry is not fully understood due to the lack of relevant anci
267 rganized interrelations in cytokine networks are not fully understood during acute HIV-1 infection (A
268 r, grain boundary effects on ionic transport are not fully understood, especially at the atomic scale
270 owever, the computational role of adaptation is not fully understood, especially when one considers n
271 port of H2O in the Earth's upper mantle, but is not fully understood for olivine ((Mg, Fe)2SiO4) the
272 t, and the parameters that impact cell entry are not fully understood, giving rise to variability and
274 into the pathogenesis of these diseases, it is not fully understood how distinct GJB2 mutations resu
276 f adaptive immunity are well established, it is not fully understood how innate cells integrate quali
281 ntributors to mutagenesis and cancer, but it is not fully understood how the chromosomal landscape in
283 t source for pancreatic cancer cells, but it is not fully understood how the tumor environment influe
284 mere length compared to normal cells, but it is not fully understood how WRN deficiency leads directl
285 anisms underlying CB sensitization during HF are not fully understood, however previous work indicate
286 mechanisms underlying human brain evolution are not fully understood; however, previous work suggest
287 Currently, the driver for this progression is not fully understood; in particular, it is not known
288 hobiology of vision limiting processes in RD is not fully understood, inflammation is known to play a
289 ng impaired uteroplacental vascular function are not fully understood, interventions aimed at enhanci
290 m for the biosynthesis of most modifications is not fully understood, owing, in part, to the difficul
292 biological mechanisms behind the association are not fully understood, particularly the role of DNA m
293 mechanism by which E7 induces carcinogenesis is not fully understood; specific anti-HPV agents are no
294 modeling in different physiological settings is not fully understood, there is a growing realization
295 patients will be drug sensitive or resistant are not fully understood; therefore, genomic assessment
296 s through which inflammation promotes cancer are not fully understood, two connected hypotheses have
298 In the case of V. vinifera ssp. vinifera, it is not fully understood what determines hermaphroditism
299 In the case of V. vinifera ssp. vinifera, it is not fully understood what determines hermaphroditism
300 anisms that maintain an oscillatory response are not fully understood, yet several models have been p
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