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1 rulation, but mutants of two, ylxY and ylyA, were outcompeted by a wild-type strain under sporulation

2 The nudA mutant was outcompeted by about fourfold by the parent in compe

3 nsfer models confirmed that Ahr(-/-) B cells are outcompeted by Ahr(+/+) cells.

4 competitive infections, the Deltairs4 mutant was outcompeted by CAI-12 in each mouse at >/=6 h (compe

5 sion model (i.e., the resident strain cannot be outcompeted by challengers) was discarded in favor of

6 he pathogen to the intestinal lumen where it was outcompeted by commensals.

7 IV-1 isolates from long-term survivors (LTS) were outcompeted by control strains and were significant

8 tative limiting cofactor of TEF-1 that could be outcompeted by exogenous TEF-1 only in a MSLN-overexp

9 coming environmental constraints and yet can be outcompeted by faster proliferating competitors, resu

10 r altruism, host bacteria promoting transfer are outcompeted by hosts with lower transfer rate, an as

11 competitive, such that the antagonist could be outcompeted by increasing doses of the ligand.

12 alveolar macrophages showed that the mutant was outcompeted by its parent 3-fold in 24 h and 24-fold

13 The lvgA mutant was outcompeted by its parent about sixfold in guinea pi

14 ophila pneumonia showed that the nudA mutant was outcompeted by its parent in both lung and spleen.

15 limited because smaller beneficial mutations are outcompeted by larger beneficial mutations that occu

16 tant outcompeted MGAS2221 in normal mice but was outcompeted by MGAS2221 in neutropenic mice and had

17 rference causes some beneficial mutations to be outcompeted by more-fit mutations that occur in the s

18 mutants overtake unicellular populations but are outcompeted by multicellular, soma-producing strains

19 in situ could not maintain hematopoiesis and were outcompeted by normal HSCs.

20 usceptible cell line, the subtype C isolates were outcompeted by other subtypes, as observed in exper

21 e-deficient Deltahma DeltachuA double mutant was outcompeted by siderophore receptor mutants specific

22 y, CAT-expressing pneumococci in mouse lungs were outcompeted by susceptible cells even during Cm tre

23 absence of Pchlide, and that its import can be outcompeted by the addition of the pSS.

24 g within epithelial cells, and a sitA mutant is outcompeted by the wild type in cultured epithelial c

25 e in the urinary tract; the znuC::Kan strain was outcompeted by the wild type during a cochallenge ex

26 itive to zinc limitation than the wild type, was outcompeted by the wild type in minimal medium, disp

27 mutant failed to colonize porcine lungs and was outcompeted by the wild-type strain (median competit

28 g independent challenge, each of the mutants was outcompeted by the wild-type strain to various degre

29 Mutants in feoB were outcompeted by the wild type during mixed colonizat

30 All of these mutants were outcompeted by the wild type in coinfection experim

31 monocytes deficient for TNF or TNF receptors are outcompeted by their wild-type counterpart.

32 H. pylori tlpC mutants are outcompeted by wild type during stomach colonisation

33 of protein synthesis and proliferation, and are outcompeted by wild-type cells in C57BLKS<-->ROSA26

34 nese hamster ovary (CHO) cell monolayers and is outcompeted by wild-type strains in mixed infections.

35 The DeltasisA DeltasisB mutant was outcompeted by wild-type CFT073 during cochallenge i

36 Most of the deletion strains were outcompeted by wild-type bacteria in either mouse s

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