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1 romatin remodeling factor MTA1, continues to be poorly understood.
2 -55 The mechanism of action of GM-CSF in EAE is poorly understood.
3 is globally distributed, cryptic environment is poorly understood.
4 terial exposure on the human immune response is poorly understood.
5 of energy homeostasis, the precise mechanism is poorly understood.
6 eruptions around the world, yet their origin is poorly understood.
7 among black men who have sex with men (MSM) is poorly understood.
8 Fs) confer chemoresistance in ovarian cancer is poorly understood.
9 nate between different nucleosome substrates is poorly understood.
10 ty, but their functional phenotype during AD is poorly understood.
11 in other cellular functions in neutrophils, is poorly understood.
12 said innate and adaptive allergic responses is poorly understood.
13 riptional factors to the adipocyte phenotype is poorly understood.
14 tic basis for homotypic binding site synergy is poorly understood.
15 mation within the endoplasmic reticulum (ER) is poorly understood.
16 its uneven distribution within endemic areas is poorly understood.
17 lk between metabolic and DNA repair pathways is poorly understood.
18 However, the interaction between INT and MVP is poorly understood.
19 t initiates the synthesis of starch granules is poorly understood.
20 cing is maintained in subsequent generations is poorly understood.
21 kinase (PI3K); its role in vascular function is poorly understood.
22 er, the effect of K1 during animal infection is poorly understood.
23 ces on the foraging behaviour of pollinators is poorly understood.
24 ce that neurons exert on astrocytic function is poorly understood.
25 n several cancers, its epigenetic regulation is poorly understood.
26 urophysiological basis of this communication is poorly understood.
27 a fungal species with multiple cell conidia is poorly understood.
28 vironments on offspring molecular phenotypes is poorly understood.
29 role of arginine methylation in this process is poorly understood.
30 How this transition is operated in vivo is poorly understood.
31 which Cdx members impact their transcription is poorly understood.
32 macromolecular PIPs remains less studied and is poorly understood.
33 and how these two processes are coordinated is poorly understood.
34 thalamic pathway in circadian photoreception is poorly understood.
35 normal tissue damage following radiotherapy is poorly understood.
36 r, the regulation of MAVS-mediated apoptosis is poorly understood.
37 nd multiple organs of which the pathogenesis is poorly understood.
38 ransformed ECs respond to excess centrosomes is poorly understood.
39 als are translated into cell-level responses is poorly understood.
40 e regulatory cascade triggered by starvation is poorly understood.
41 ironment, the impact of which on RNA folding is poorly understood.
42 , and its mechanism of substrate recognition is poorly understood.
43 s are impacted by the amount of such linkers is poorly understood.
44 a group to specify a complex neural network is poorly understood.
45 (AD) pathology and expression of AD symptoms is poorly understood.
46 ne depletion and other forms of chemotherapy is poorly understood.
47 toward heterogeneous toxicological responses is poorly understood.
48 function but the underlying neural mechanism is poorly understood.
49 n the pathology of allergic chronic diseases is poorly understood.
50 ent in lupus nephritis (LN), but the process is poorly understood.
51 o called atopic dermatitis or atopic eczema) is poorly understood.
52 individual patients during AE disease flares is poorly understood.
53 these associations have changed across time is poorly understood.
54 cts of multisensory integration are achieved is poorly understood.
55 yield appropriate gene expression responses is poorly understood.
56 cling of this compound throughout ecosystems is poorly understood.
57 ectly at the plasma membrane; their function is poorly understood.
58 le of microbial communities in regime shifts is poorly understood.
59 s spatiotemporal availability in East Africa is poorly understood.
60 cations during this critical transition step is poorly understood.
61 g of memories, but its role during retrieval is poorly understood.
62 tratumoral distribution and efficacy of ADCs is poorly understood.
63 his process and the biochemistry they encode is poorly understood.
64 e, but the biological function of the ex-RNA is poorly understood.
65 ormation-the microscopic origin of fragility is poorly understood.
66 LT maintains telomere length in human cancer is poorly understood.
67 ) rhythms are linked to memory consolidation is poorly understood.
68 from activating to non-activating conditions is poorly understood.
69 oordinate Cu delivery to reproductive organs is poorly understood.
70 cy-dependent mobilization of these sub-pools is poorly understood.
71 ms, the involvement of microbial communities is poorly understood.
72 but the molecular regulation of this process is poorly understood.
73 of specific stages of thymocyte development is poorly understood.
74 ide-resistant alleles among weed populations is poorly understood.
75 However, the assembly of this oxidase is poorly understood.
76 non, known as the spacing effect for memory, is poorly understood.
77 nction is regulated by host cellular factors is poorly understood.
78 ism by which it contributes to this function is poorly understood.
79 tem (CNS) injury and disease, but their role is poorly understood.
80 y eliminated to maintain protein homeostasis is poorly understood.
81 The relationship between smoking and rosacea is poorly understood.
82 f cuticle biosynthesis during organ ontogeny is poorly understood.
83 disease, but the cellular function of MORC2 is poorly understood.
84 how a single cue is selected from among many is poorly understood.
85 Mechanistically, however, this process is poorly understood.
86 r, the mechanism involved in Fgf4 regulation is poorly understood.
87 to 5' end resection and/or 3' end extension is poorly understood.
88 m in processing limbic information, however, is poorly understood.
89 to enzymatic activity, but how they function is poorly understood.
90 he biological basis of these sex differences is poorly understood.
91 ammalian arcopallial and amygdala subregions is poorly understood.
92 s of such sequences are encoded in the brain is poorly understood.
93 PNPLA3 and its variant I148M in this process is poorly understood.
94 e pathobiology of early squamous lung cancer is poorly understood.
95 ing systems interact to regulate bone growth is poorly understood.
96 cortical oscillations and neuronal responses is poorly understood.
97 espiratory disease and lung cancer mortality is poorly understood.
98 dimerization impacts DNA-binding specificity is poorly understood.
99 c interactions with crop quality parameters, is poorly understood.
100 tagmin-1 (syt1) in regulation of endocytosis is poorly understood.
101 How ER shaping is linked to neuronal defects is poorly understood.
102 ogical diseases, its role at nerve terminals is poorly understood.
103 ely how it communicates with neural elements is poorly understood.
104 hologically distinct plasma membrane domains is poorly understood.
105 new connections, a process whose regulation is poorly understood.
106 regulation of differentiation-related genes is poorly understood.
107 n of specific residues in the nervous system is poorly understood.
108 sduce sensory stimuli into action potentials is poorly understood.
109 es to cellular behavior, but their interplay is poorly understood.
110 ut the mechanism underlying this association is poorly understood.
111 en the human pathology and NMDAR dysfunction is poorly understood.
112 CeA circuits underlying appetitive behaviors is poorly understood.
113 development, but regulation of this process is poorly understood.
114 on CD4 T cells and in HIV-infected children is poorly understood.
115 tal to plant cell growth, and its regulation is poorly understood.
116 cally limited nervous system of 302 neurons, is poorly understood.
117 culations resolve to form compact myocardium is poorly understood.
118 s alter tumour progression, their regulation is poorly understood.
119 The role of archaea in microbial mats is poorly understood.
120 e function following cardiac transplantation is poorly understood.
121 myelin-forming glia influence nodal assembly is poorly understood.
122 ted by energy deprivation, but the mechanism is poorly understood.
123 ever, protein dynamics within the 3D nucleus are poorly understood.
124 fy subgroups of AD patients at risk for ADHD are poorly understood.
125 sms by which targeting improves Ab responses are poorly understood.
126 that allow pathogens to target these niches are poorly understood.
127 ular signals that coordinate this regulation are poorly understood.
128 which their therapeutic action is initiated are poorly understood.
129 forces that lead to grana stacking, however, are poorly understood.
130 mammary gland morphogenesis and homoeostasis are poorly understood.
131 ut the mechanisms that govern their assembly are poorly understood.
132 ver, the long-term effects of HCS deficiency are poorly understood.
133 acclimation in such fluctuating environments are poorly understood.
134 patients with classic Hodgkin lymphoma (cHL) are poorly understood.
135 crease in risk of severe malaria in general, are poorly understood.
136 e structural implications of phosphorylation are poorly understood.
137 physical forces in the bone microenvironment are poorly understood.
138 ls arises from prostate adenocarcinoma cells are poorly understood.
139 tissues during human growth and development are poorly understood.
140 ng in a distant intercontinental disjunction are poorly understood.
141 show paradoxical immune side effects, which are poorly understood.
142 la," which lack isolated representatives and are poorly understood.
143 lecular mechanisms regulating this transport are poorly understood.
144 s for the dynamics of ecological communities are poorly understood.
145 MDARs in pancreatic beta-cells, by contrast, are poorly understood.
146 s and, specifically, the role of Akt kinases are poorly understood.
147 mechanisms contributing to these differences are poorly understood.
148 f these changes for soil carbon (C) dynamics are poorly understood.
149 , and on central nervous network dysfunction are poorly understood.
150 ll- and process-specific effects of TGF-beta are poorly understood.
151 on from this niche to cause invasive disease are poorly understood.
152 roles of RBPs in somatic cell reprogramming are poorly understood.
153 etween respiratory and photosynthetic growth are poorly understood.
154 cific diseases are definable, but mechanisms are poorly understood.
155 serpentinization and the controlling factors are poorly understood.
156 and Southeast Asia, details of Holocene RSL are poorly understood.
157 sed in the brain, but their individual roles are poorly understood.
158 ng pathways that fine-tune cardiac autophagy are poorly understood.
159 l signals to airway remodeling during asthma are poorly understood.
160 ment, the nature and timing of these changes are poorly understood.
161 involvement of RA signaling in this process are poorly understood.
162 ells that mediate this differential response are poorly understood.
163 vores and the conditions that can lead to it are poorly understood.
164 ng of sensory evidence (choice-bias control) are poorly understood.
165 ory consolidation, but underlying mechanisms are poorly understood.
166 ansmission along the Thailand-Myanmar border are poorly understood.
167 lity of survival motor neuron (SMN) isoforms are poorly understood.
168 although early steps in plastid integration are poorly understood.
169 e mechanisms accounting for hypersensitivity are poorly understood.
170 echanisms driving its biosynthesis in cancer are poorly understood.
171 iation of osteocytes from mature osteoblasts are poorly understood.
172 ng cell death, but the underlying mechanisms are poorly understood.
173 enetic determinants of body fat distribution are poorly understood.
174 ating the development of inhibitory synapses are poorly understood.
175 olesterol through the TICE pathway, however, are poorly understood.
176 efects associated with the majority of these are poorly understood.
177 ta senses and responds to environmental cues are poorly understood.
178 mechanisms responsible for this ATP release are poorly understood.
179 ory gene networks in the innate immune cells are poorly understood.
180 and incurable, yet the metastasis mechanisms are poorly understood.
181 n-host variation to global genetic diversity are poorly understood.
182 ng pancreatic beta cell survival in diabetes are poorly understood.
183 ated modulations of sensory cortex, however, are poorly understood.
184 tive contributions and underlying mechanisms are poorly understood.
185 for heterodimerization specificity of bZIP53 are poorly understood.
186 of melt and the active volcanoes they supply are poorly understood.
187 underlying history-dependent decision-making are poorly understood.
188 but the mechanisms that maintain homeostasis are poorly understood.
189 ver billions of years, their formation rates are poorly understood.
190 the mechanisms regulating their development are poorly understood.
191 s of contemporary tree-climate relationships are poorly understood.
192 l outcomes of IgG binding to astrocytic AQP4 are poorly understood.
193 heir spatiotemporal dynamics during behavior are poorly understood.
194 kable heterogeneity in disease manifestation are poorly understood.
195 The forces that drive lipid raft formation are poorly understood.
196 ular mechanisms that regulate their function are poorly understood.
197 ndence, yet mechanisms of nicotine avoidance are poorly understood.
198 w MITF-low cells in tumors escape senescence are poorly understood.
199 erations in glioblastomas with wild-type IDH are poorly understood.
200 ure of age-associated B cells (ABCs) in mice are poorly understood.
201 ution and diffusion properties in live cells are poorly understood.
202 erning the specificity of these interactions are poorly understood.
203 primary sensitization or new sensitizations are poorly understood.
204 e aging process to improve health in old age are poorly understood.
205 thers to remain in the same area year-round, are poorly understood.
206 y differ in latent and lytic KSHV infections are poorly understood.
207 oteins, neural function, and brain structure are poorly understood.
208 cular mechanisms that control this interplay are poorly understood.
209 ys of Cr(VI) reduction by such clay minerals are poorly understood.
210 s well as their broader physiological roles, are poorly understood.
211 stem and the signals received by the sensors are poorly understood.
212 by which CAFs regulate cancer cell migration are poorly understood.
213 hat regulate the body's response to exercise are poorly understood.
214 ferentiation subsequent to immune challenges are poorly understood.
215 rlying rearrangement of cell wall components are poorly understood.
216 isms that regulate endosome sorting of BACE1 are poorly understood.
217 stantially between patients for reasons that are poorly understood.
218 s PLPs at a structural and biochemical level are poorly understood.
219 ng overexpression of this receptor in cancer are poorly understood.
220 terparts; however, the underlying mechanisms are poorly understood.
221 Basic mechanisms for this association are poorly understood.
222 protein aggregates from more innocuous ones are poorly understood.
223 underlying determinants of this variability are poorly understood.
224 nce intervals of intraplate faults therefore are poorly understood.
225 hich such expansions cause neurodegeneration are poorly understood.
226 ons influencing risk and disease progression are poorly understood.
227 control expression of RP genes in metazoans are poorly understood.
228 underlying opposition to Polycomb silencing are poorly understood.
229 ense, and the spectrum of microbes affected, are poorly understood.
230 nisms that control its proapoptotic function are poorly understood.
231 sm such as the incorporation of molecular O2 are poorly understood.
232 ning antipsychotic efficacy and side effects are poorly understood.
233 hey alter the course of Leishmania infection are poorly understood.
234 d those resulting in return to theater (RTT) are poorly understood.
235 early disease in the context of prediabetes are poorly understood.
236 g domain, the mechanisms for Tie2 activation are poorly understood.
237 elation to antisocial behavior and adversity are poorly understood.
238 sorder (MDD) patients with suicidal ideation are poorly understood.
239 spective value in predicting clinical events are poorly understood.
240 southern peripheries of ancient Mesoamerica are poorly understood.
241 e mechanisms involved in disease progression are poorly understood.
242 e, but the mechanisms ensuring high fidelity are poorly understood.
243 lecular mechanisms underlying these findings are poorly understood.
244 signals controlling differential translation are poorly understood.
245 s directing specific proteins to this domain are poorly understood.
246 aque and abdominal aortic aneurysm stability are poorly understood.
247 vascular events after kidney transplantation are poorly understood.
248 disease (AD), but the underlying mechanisms are poorly understood.
249 iosynthesis, their release from the cell has been poorly understood.
250 sm, although their mechanisms of action have been poorly understood.
255 es during antibody-mediated rejection (ABMR) are poorly understood and could contribute to the delete
257 of zinc, but the transport mechanism of ZntB is poorly understood and based only on experimental char
258 ) regulate phloem unloading in monocot stems is poorly understood and particularly so for species sto
260 athophysiology of neurodegenerative diseases is poorly understood and there are few therapeutic optio
261 et, the health consequences of such exposure are poorly understood, and the scientific literature rem
263 pid aortic valve (BAV)-associated aortopathy is poorly understood, and no prognostic biomarker is cur
264 se determinants are delivered and positioned is poorly understood, and the upstream signal to initiat
265 esity, how ILC2s respond to high fat feeding is poorly understood, and their direct influence on the
266 The pathogenesis of osteoarthritis (OA) is poorly understood, and therapeutic approaches are lim
267 the coordination of TMs to the {P8W48} ring is poorly understood, and therefore largely unpredictabl
268 amics of bacterial growth in such conditions is poorly understood, and, unlike that for liquid cultur
269 results in such large slip near the surface is poorly understood as shallow parts of thrust faults a
270 n and persistence of such activity, however, are poorly understood, as is their role in the function
273 act of diet on the metabolism-epigenome axis is poorly understood but could alter gene expression and
274 e molecular and cellular mechanisms involved are poorly understood, but novel methods now make it pos
275 mechanisms triggered by these various agents is poorly understood, but it might explain the different
276 otein 1 (ARGLU1) is a protein whose function is poorly understood, but may act in both transcription
277 t of genetic variation on these associations is poorly understood, especially in children.We estimate
278 s of these communities following disturbance are poorly understood, given that they are often evaluat
279 n of transition zone squamous cell carcinoma are poorly understood, hence representing limited option
280 Environmental adaptation by RF Borrelia is poorly understood, however our previous studies indic
282 e pathophysiology of carcinoid heart disease is poorly understood; however, chronic exposure to exces
284 derlying altered beta-cell function in aging are poorly understood in mouse and human islets, and the
286 ough how the signal transmission takes place is poorly understood, it has been shown that flagella pl
287 is activates numerous PRRs, for reasons that are poorly understood LAP does not substantially contrib
288 eir importance in normal and disease states, are poorly understood, largely because of the inability
289 CM quantities in end-of-life vehicles (ELVs) are poorly understood, mainly due to a limited understat
290 hough how viruses influence asthma inception is poorly understood, much research has focused on the h
291 cellular control of morphogenetic apoptosis is poorly understood, notably the modulation of cell sen
293 atality ratios in children with tuberculosis are poorly understood-particularly those among children
294 urrents in molecular and non-molecular rings is poorly understood, partly because they are studied in
297 cause the economics of water quality testing are poorly understood, the extent to which cost may be a
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