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1 pts associated with arrested RNA polymerases are protected from 3'-5' degradation and thus, unstable
2                 In addition, Gsdmd(-/-) mice are protected from a lethal dose of lipopolysaccharide.
3 eceptor (TLR) adaptor Mal (encoded by TIRAP) are protected from a number of infectious diseases, incl
4  because we show that vaccinated animals can be protected from a mucosal challenge with a heterologou
5 with plasmids or given nonhuman-primate sera were protected from a lethal challenge with purified Tcd
6 irus A/California/7/2009 (H1N1) strain (Cal) were protected from a lethal challenge with the heterolo
7 ina using a prime-boost vaccination strategy were protected from a subsequent lung challenge with P.
8                       The 3' U-tract of scR1 is protected from aberrant processing by the La homologu
9 t manner while the conjugated-ubiquitin (Ub) is protected from active deubiquitination.
10                                   These mice were protected from acute podocyte foot process effaceme
11 wild-type littermates, hCD39 transgenic mice were protected from acute renal injury at 24 hours, but
12 male) mice deficient in Arg-II (Arg-II(-/-)) are protected from age-associated glucose intolerance an
13  necrosis factor (TNF)-deficient mice, which are protected from age-associated inflammation, age-rela
14 sulin levels following glucose challenge and were protected from age-related reductions in GSIS and g
15                 Mislocalized proteins (MLPs) are protected from aggregation by the Bag6 complex and d
16 ar fat mass and energy balance, M(IL10) mice were protected from aging-associated insulin resistance
17                    WT but not Ido1(-/-) mice were protected from AIA by IFN-alpha, and Kyn, the main
18 ted that integrin alphavbeta6-deficient mice are protected from airway hyperresponsiveness, due in pa
19  inflammasome components NLRP3 and caspase-1 were protected from aldosterone-induced vascular damage.
20                   In contrast, NT4(-/-) mice were protected from allergen-induced mucus overproductio
21                                  All animals were protected from an intrarectal SIVmac239 challenge,
22        We hypothesized that host cells would be protected from anthrax toxins if anthrax toxin recept
23 e conserved HIV-1 site of coreceptor binding is protected from antibody-directed neutralization by co
24                Within DCs A. terreus conidia were protected from antifungals, whereas A. fumigatus co
25  immunized with a single dose of this vector were protected from any signs of disease following letha
26  report that active spindle assembly factors are protected from APC/C-dependent degradation by microt
27  cells that received Tax-containing exosomes were protected from apoptosis through activation of AKT.
28  that 13-lined ground squirrel tubular cells are protected from apoptotic cell death during IBA.
29 k-out mice that lacked PEMT showed that they were protected from atherosclerosis, diet-induced obesit
30              Such critical functions need to be protected from attack by pests and pathogens or from
31                NOD mice treated with AZD1480 were protected from autoimmune diabetes, and diabetes wa
32                                    Transport is protected from back-scattering, possibly reflecting g
33  in response to different learning tasks and are protected from being eliminated when multiple tasks
34        ZDF rats with global deletion of CB1R are protected from beta-cell loss, hyperglycemia, and ne
35 n-G, amoxicillin, ampicillin, and cefazolin, are protected from beta-lactamase hydrolysis via the for
36 tic Ppp1r3b upon long-term fasting (12-36 h) were protected from blood ketone-body accumulation, unli
37 cient in the double-stranded DNA sensor AIM2 are protected from both subtotal body irradiation-induce
38                  Interestingly, mitochondria are protected from both the deficiency in NAD(+) biosynt
39 p to 1,200 microg/mL in serum, and up to 70% were protected from both i.v. and mosquito bite challeng
40  absence of target, aptamer coated particles are protected from capture on the test line and are inst
41                      PGC-1beta knockout mice are protected from carcinogenesis.
42 eletal muscle-specific EcSOD transgenic mice are protected from cardiac hypertrophy, fibrosis, and dy
43 v integrin is depleted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced sk
44 us for the ancestral C allele (EE genotype), are protected from cardiovascular disease (CVD), showing
45 nvestigated how glutamate in a neural tissue is protected from catabolism.
46 re deficient for both B cells and antibodies were protected from CAV.
47                               Germ-free mice are protected from CCM formation, and a single course of
48 oth mucosal and serum IgA anti-toxin Abs and were protected from CDI upon rechallenge, with protectio
49                 Differentiated keratinocytes are protected from cell death, whereas cells treated wit
50 ntial sH1N1 influenza virus-infected ferrets were protected from challenge with a novel H1N1 influenz
51 operties, are strikingly enriched in AT, and are protected from chemotherapy by the GAT microenvironm
52                               Leukemia cells are protected from chemotherapy-induced apoptosis by the
53 contrast, the stability of the XRCC1 monomer is protected from CHIP-mediated ubiquitylation by intera
54 e we demonstrate that mice deficient in Irp2 were protected from cigarette smoke (CS)-induced experim
55 ed cattle), despite vaccinated cattle having been protected from clinical disease.
56         Mice with PU.1 deficiency in T cells were protected from colitis, whereas treatment with anti
57                             TTPDeltaARE mice were protected from collagen antibody-induced arthritis,
58 intact complement negative regulatory system are protected from complement attack, whereas cells with
59 eficient mice, but not RIPK3-deficient mice, were protected from ConA-induced liver injury.
60 operties rivalling suspended graphene, while being protected from contamination and mechanical damage
61 ssemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD); (ii)
62 ionic doping, and the LLMO cathode materials are protected from corrosion induced by organic electrol
63 CPE-sensitive enterocyte-like cell line) can be protected from CPE-induced cytotoxicity by preincubat
64 al iron chelator or mice fed a low-iron diet were protected from CS-induced COPD.
65 ytochrome c oxidase, which have reduced COX, were protected from CS-induced pulmonary inflammation an
66         Endothelial cells and isolated lungs were protected from cytotoxin-induced death by stimulati
67 del, we report here that TRPV4-knockout mice were protected from D. farinae-induced airway remodeling
68 dling development, the shoot apical meristem is protected from damage as the seedling emerges from so
69 type in the heart during EAM, IL-4(-/-) mice were protected from DCMi like DeltadblGATA1 mice, and eo
70  effect of IL-5, as IL-5TgDeltadblGATA1 mice were protected from DCMi, whereas IL-5(-/-) mice exhibit
71                 The mechanisms by which CGIs are protected from de novo methylation remain elusive.
72             Until such foci were lost, cells were protected from death.
73 ly assumed that mRNAs undergoing translation are protected from decay.
74                                   Other FNWs were protected from decomposition with an amorphous carb
75 that viral genomes may in some circumstances be protected from degradation for centuries.
76 ergen extracts: The protein/DNA molecule can be protected from degradation, higher local concentratio
77 bstrate of the Lon-type protease and that it is protected from degradation by Nfs1, the sulfur donor
78 terious interaction with the Bam complex but was protected from degradation and eventually inserted i
79  subunit, the palmitoylated CaV2.2 I-II loop was protected from degradation, although oligoubiquitina
80 blueberry polyphenols complexed with protein were protected from degradation during 16weeks at 4 degr
81  of ultralow methylation at transposons that are protected from demethylation in the germline and ICM
82                         Growing microtubules are protected from depolymerization by the presence of a
83 ry diseases, ICER/CREM-deficient B6.lpr mice are protected from developing autoimmunity.
84           Ormdl3 knockout mice were found to be protected from developing allergic airways disease an
85 did not occur in IL-17-deficient mice, which were protected from development of lupus autoantibodies
86 r, mice with liver-specific deletion of MPC2 were protected from development of NASH on this diet.
87 red with WT, we demonstrated that Parp1(-/-) were protected from dextran-sulfate sodium-induced colit
88  upregulated when type 1 diabetes-prone rats are protected from diabetes development.
89 arp contrast, TLR3- and MyD88-deficient mice were protected from diabetes following the same treatmen
90 lar to those of WT STZ mice, TLR4KO STZ mice were protected from diabetes-induced bladder hypertrophy
91                    Glomeruli from G4 KO mice were protected from diabetes-induced hypertrophy, mesang
92  mice and mice treated with a P2X7 inhibitor were protected from diabetes-induced TNF-alpha, IL-1beta
93 DBim(-/-)) mice developed less insulitis and were protected from diabetes.
94 p66Shc that is not acetylatable on lysine 81 are protected from diabetic oxidative stress and vascula
95 deficient in small heterodimer partner (SHP) are protected from diet-induced hepatic steatosis result
96 more, adipose-specific Dnmt3a knock-out mice are protected from diet-induced insulin resistance and g
97                           Mice lacking Tlr4s are protected from diet-induced insulin resistance and i
98                            Mice lacking LR11 are protected from diet-induced obesity associated with
99 d and that Mfge8-deficient (Mfge8(-/-)) mice are protected from diet-induced obesity, steatohepatitis
100 functional gene encoding MGAT2 (Mogat2(-/-)) are protected from diet-induced obesity.
101                              Stk25(-/-) mice were protected from diet-induced liver steatosis accompa
102 , we found that mice lacking hepatic ZFP36L1 were protected from diet-induced obesity and steatosis.
103                    PDZ-RhoGEF-deficient mice were protected from diet-induced obesity and T2D.
104 heir severe fatty liver, the transgenic mice were protected from diet-induced obesity and type 2 diab
105              Notably, Il5 (Tg) /CD300f (-/-) were protected from diet-induced weight gain and glucose
106 th wild type, whereas AdipoR2-deficient mice were protected from diet-induced weight gain and metabol
107 e a limited elastic range of 3-4% strain but are protected from direct stretch during physiological l
108               In contrast to humans, rodents are protected from disease on infection with ebolaviruse
109 vial fluids after injury and Cxcr3(-/-) mice being protected from disease development.
110 tis, because Nfil3/Rag1 double-knockout mice were protected from disease.
111                        These precursors must be protected from DNA damage-induced cell death, however
112                Furthermore, Sh2d1a(-/-) mice were protected from EAE and exhibited greatly decreased
113           Nevertheless, TPL-2-deficient mice were protected from EAE, which correlated with reduced i
114 tatic and dynamic disorder, but how carriers are protected from efficient scattering with charged def
115                 By 6 months, Mmp28(-/-) mice were protected from emphysema.
116 n of IL-37 (IL-37tg mice) with intact IL-1R8 were protected from endotoxemia, IL-1R8-deficient IL-37t
117 ting becomes viable when a quantum state can be protected from environment-induced error.
118      Eventually, ERK inhibitor treated cells are protected from ETO-induced nuclear envelope (NE) rup
119 ic core of cyclodextrins and therefore, they are protected from exogenous stress.
120 e have decreased Th17 cell numbers, and they are protected from experimental autoimmune encephalitis,
121                     Kv1.3-knockout (KO) mice are protected from experimental autoimmune encephalomyel
122 ve defect in Ag-specific alphabetaTh17 cells were protected from experimental ARDS induced by a singl
123                  Furthermore, CD43(-/-) mice were protected from experimental autoimmune encephalomye
124 pe-specific deletions of PTEN-targeting APCs were protected from experimental autoimmune encephalomye
125    Both talin1(L325R) and talin1(W359A) mice were protected from experimental thrombosis.
126 ng facilities with large elderly populations are protected from extreme heat (for example through bac
127 genetic or pharmacological inhibition of BID are protected from Fas-mediated impairment of mitochondr
128 ogen synthesis, and as a result, CD36Tg mice were protected from fasting hypoglycemia.
129 les cannot sing to attract females, but they are protected from fatal attack by an acoustically orien
130                     Mice deficient in IRAK-M were protected from fibrosis and displayed a diminished
131               Moreover, TRPV4-deficient mice were protected from fibrosis.
132 less nitrogen after 64 years than plots that were protected from fire.
133                Cyclic, unbranched structures are protected from fragmentation during the first oxidat
134     Our results suggest that certain tissues are protected from functional deleterious effects of pro
135 tive Staphylococcus aureus cells appeared to be protected from GA by an increased formation of nm-sca
136 mice, the tumor suppressor isoform of CUGBP1 is protected from Gank-mediated degradation.
137  proliferating cells in plant meristems must be protected from genome damage.
138  to wild-type mice, Cd74-deficient mice also were protected from glomerular injury and ensuing activa
139 A9 express reduced neutrophil chemokines and are protected from HCC.
140  female neuron-specific SOCS3 knock-out mice were protected from HCD-induced obesity.
141  stimulation, whereas Bex1 gene-deleted mice are protected from heart failure-promoting insults.
142 te-specific Shp1 knockout mice (Ptpn6(H-KO)) are protected from hepatic insulin resistance evoked by
143 notype, adipose-specific 11beta-HSD1 KO mice were protected from hepatic steatosis and circulating fa
144              Finally, HFD-fed AdSod2 KO mice were protected from hepatic steatosis, adipose tissue in
145 lation and preserved brown adipose mass; and were protected from hepatic steatosis.
146                 MPO knockout (MPO(-/-)) mice were protected from HFD-enhanced body weight gain and in
147 pe littermate control (M-JAK2(+/+)) mice and were protected from HFD-induced systemic insulin resista
148 le of binding leukocyte alphaMbeta2-integrin were protected from HFD-induced weight gain and elevated
149 homologous residue Thr1150 (InsrT1150A mice) were protected from high fat diet-induced hepatic insuli
150 y during the suckling period and, as adults, were protected from high fat diet-induced obesity.
151 e United States in 2004-2006, when consumers were protected from high WLLs.
152  Furthermore we found that male AhRR Tg mice were protected from high-dose TCDD-induced lethality ass
153 pontaneous NASH whereas AEG-1(DeltaHEP) mice were protected from high-fat diet (HFD)-induced NASH.
154                                 AKO/cTg mice were protected from high-fat diet (HFD)-induced obesity
155 show that GPR30 knockout (GPRKO) female mice were protected from high-fat diet (HFD)-induced obesity,
156 lacking both LDL receptor (LDLR) and Arhgef1 were protected from high-fat diet-induced atherosclerosi
157 note, adipocyte-specific gp130 knockout mice were protected from high-fat diet-induced hepatic steato
158                   Plxnd1-deficient zebrafish were protected from high-fat-diet-induced insulin resist
159 CreERT mice on different genetic backgrounds were protected from high-fat/ streptozotocin (STZ)-induc
160                In accordance, LRAT-null mice are protected from holo-RBP-induced suppression of insul
161 nces of viral replication, and animals could be protected from HSE by acyclovir treatment provided 4
162                 The data show that telomeres are protected from hyper-resection through the repressio
163             Panx1 knockout mice (Panx1(-/-)) were protected from hypersensitivity in two sciatic nerv
164 ike-overexpressing transgenic (Sike-TG) mice are protected from hypertrophic stimuli.
165 mmune privilege, in which immunogenic tissue is protected from immune attack.
166                             Ptpn22(-/-) mice were protected from immune complex-mediated arthritis, i
167 Here, we show that eosinophil-deficient mice were protected from induction of Th2-mediated peanut foo
168      These findings help explain why CF mice are protected from infection and nominate ATP12A as a po
169 carrying neutralizing antibodies against MuV are protected from infection by batMuV.
170  factors that determine why some individuals are protected from infection while others go on to devel
171 tive cells engraft and traffic normally, and are protected from infection with CCR5- and CXCR4-using
172 ve neutralizing antibodies against MuV might be protected from infection by BMV.
173 e infected, while the 6 PrEP-treated animals were protected from infection.
174 igh-dose UV B radiation, IR/IGF-1R(MKO) mice were protected from inflammation, whereas controls devel
175 nism by which hosts with allergic asthma may be protected from influenza morbidity.
176 e one or more means by which transplants can be protected from injury.
177 silon knockout mice on a high-fat diet (HFD) were protected from insulin resistance and showed altere
178 aRIIB globally or selectively in endothelium were protected from insulin resistance as a result of th
179           TNF-receptor I (TNFRI) mutant mice were protected from intestinal barrier dysfunction and A
180 ing the fetal troponin I isoform, (ssTnI) to be protected from ischemia by increased glycolysis.
181       A primary means by which the intestine is protected from its microbiota is via multi-layered mu
182                               S1pr3(-/-)mice are protected from kidney IRI, because DCs do not mature
183     However, S1pr3(-/-) BMDC-pretreated mice were protected from kidney IRI.
184 r expressed on myeloid cells 2 (Trem2 (-/-)) were protected from LCMV-induced hepatitis and showed im
185 ally, mice vaccinated with the DeltatolC LVS are protected from lethal challenge and clear challenge
186                              Vaccinated mice are protected from lethal challenge with diverse influen
187 ater column, seabirds, reptiles, and mammals are protected from lethal oiling at the surface, and mic
188                         Gas6 null (-/-) mice are protected from lethal venous and arterial thromboemb
189  the CDV fusion and attachment glycoproteins were protected from lethal CDV challenge, whereas all an
190 usion protein-expressing recombinant viruses were protected from lethal CDV challenge.
191 myeloid cells and found that Blimp1 CKO mice were protected from lethal infection induced by Listeria
192                             Nlrp12(-/-) mice are protected from lethality during IAV infection and sh
193     Fetal/neonatal progenitors may therefore be protected from leukemic transformation because they a
194 Plcbeta2-, Plcbeta3-, or Rac1-deficient mice were protected from lipopolysaccharide-induced lung inju
195                 MRTF-A (Mkl1)-deficient mice are protected from lung fibrosis.
196 h VLPs and subsequently challenged with HMPV were protected from lung viral replication for at least
197        We show that aging mice lacking FGFR4 are protected from LVH.
198 malaria pathogenesis because EphB2(-/-) mice were protected from malaria-induced liver fibrosis despi
199                                 Target genes are protected from MazF activity by recoding the gene se
200 ommunities accumulate on teeth in areas that are protected from mechanical abrasion forces.
201 rgely confined to synaptic vesicles where it is protected from metabolic breakdown.
202 ith a specific 11betaHSD1 inhibitor (UE2316) were protected from metabolic disturbances despite simil
203 ce vaccinated with the group 1 HA mini-stems are protected from morbidity and mortality against letha
204 (H5N2), A(H5N8), A(H6N1), or A(H7N9) viruses were protected from mortality and showed drastically red
205  of contracting and transmitting the disease are protected from mosquito bites.
206                                      The CNS is protected from most virus infections by effective imm
207  treated with TAT2A during early adolescence were protected from MS-induced PVB loss and exhibited le
208 ut to reveal the mechanisms by which embryos are protected from mutant p53-induced transformation usi
209 n activating gene 1-deficient (Rag1-/-) mice were protected from NEC and transfer of intestinal lymph
210 iable region 1 yielded vector particles that were protected from neutralization by natural antibodies
211 he system was used to demonstrate that dsRNA is protected from nuclease digestion by virus-induced me
212                                     This DNA is protected from nucleases by a phage protein until Mu
213 nt biological catalysts of H2 oxidation, can be protected from O2 damage upon integration into a film
214 cantly reduced intestinal fat absorption and are protected from obesity, hepatic steatosis and insuli
215 sue inflammation, cadherin-11-deficient mice were protected from obesity-induced glucose intolerance
216    U L3 XAS indicates that this U(V) species is protected from oxidation likely incorporated into oct
217 is Mast cell-deficient mice (Kit(W-sh/W-sh)) were protected from P. gingivalis-induced alveolar bone
218    Within these autophagosomes, the bacteria are protected from phagocytic killing, thus providing an
219 sembly and how vulnerable assembly complexes are protected from photodamage are unknown.
220 cient mice made diabetic with streptozotocin were protected from physiological and structural indices
221         Conversely, areas exposed to high SS are protected from plaque development, but the mechanism
222 ked as the most upregulated in vaccinees who were protected from Plasmodium falciparum infection.
223                      KEX1-immunized macaques were protected from Pneumocystis pneumonia, compared wit
224  and IL-1beta before intestinal manipulation were protected from POI.
225 nitrosylation, cyclic GMP, NO formation, and were protected from postinfarct and pressure overload HF
226 ct with cortactin, and Nox2-deficient hearts were protected from pressure overload-induced adverse my
227 V infection or SVR after antiviral treatment were protected from progressive liver disease and showed
228                      Finally, coexposed mice were protected from prolonged bacterial infection by adm
229 eptum to the adjacent cell pole where SpoIIE is protected from proteolysis and activated.
230                         Most circulating SAA is protected from proteolysis and misfolding by binding
231 etically deficient in PTP-alpha (Ptpra(-/-)) were protected from pulmonary fibrosis induced by intrat
232  mice had increased macrophage apoptosis and were protected from pulmonary fibrosis.
233  importance, mice that were deficient in MCU were protected from pulmonary fibrosis.
234 n contrast, SMPDL3b overexpressing podocytes were protected from radiation-induced cytoskeletal remod
235 All BCV-treated mice that survived infection were protected from rechallenge without additional treat
236                            Ddah1(PT-/-) mice were protected from reduced kidney tissue mass, collagen
237 ogen receptor alpha (ERalpha)-deficient mice are protected from renal disease and have prolonged surv
238  express only talin1(L325R) in myeloid cells were protected from renal ischemia-reperfusion injury.
239 Ksp-CreERT2(+) mice crossed to p62(-/-) mice were protected from renal tumor development.
240  with decoy IL-21 receptor Fc fusion protein are protected from reperfusion injury.
241                                Bacterial DNA is protected from restriction endonuclease cleavage by m
242  develop high intraocular pressure (IOP) but are protected from retinal ganglion cell (RGC) dysfuncti
243 rains, we have determined whether cattle can be protected from rinderpest by inoculation with vaccine
244 wever, only the animals given wild-type PPRV were protected from RPV challenge.
245                                  NSAID users were protected from SAB (OR = 0.78, 95% CI 0.56-1.10), w
246 ingle knockout IL-4(-/-) or IL-13(-/-) mice, were protected from Schistosoma-induced PH, with decreas
247             Interestingly, miR-155(-/-) mice were protected from SEB-mediated inflammation and lung i
248 g for more than 40 weeks, and these macaques were protected from several infectious challenges with S
249 he baboons in the P. knowlesi-only group and were protected from severe anemia.
250 contrast, animals vaccinated with MVA-H7-Sh2 were protected from severe disease.
251 an inoculum containing Nod1 x 2(-/-) T cells were protected from severe graft versus host disease.
252   C3H mice expressing WT Gusb as a transgene were protected from severe Lyme arthritis.
253 ozygotes and alpha(+)thalassemia homozygotes were protected from severe malaria (odds ratio [OR], 0.1
254                   Baboons vaccinated with aP were protected from severe pertussis-associated symptoms
255  of three MHC-congenic strains, of which two were protected from severe PIA.
256 le stress fibers, whereas contractile fibers are protected from severing.
257                       IL-20R1-deficient mice were protected from short-term and long-term liver injur
258 le explores the question of how the active X is protected from silencing by its own Xist locus, and t
259 d that mice lacking ephrin-B2 in fibroblasts are protected from skin and lung fibrosis and that a dis
260 n of Cosmc in 50% of crypts (IEC-Cosmc(+/-)) were protected from spontaneous inflammation and partial
261 hich also shows reduced muscle strength, but is protected from stretch-induced eccentric damage with
262 erm clearance of tumor after TLR7/RT therapy are protected from subsequent tumor rechallenge by the g
263 Vmac239 challenge, whereas only four animals were protected from subsequent intravenous SIVmac239 cha
264 ptible to WNV NS4B-P38G mutant infection but were protected from subsequent lethal wild-type WNV chal
265 ns were significantly higher in children who were protected from symptomatic malaria compared with th
266 ll-dependent antiviral immune responses, and are protected from T cell-mediated autoimmunity and allo
267 t neuronal cultures treated with Tat protein are protected from Tat-mediated cytotoxicity when treate
268 ned as "metabolically abnormal obese" (MAO), are protected from the adverse metabolic effects of weig
269 graphite die and the graphite punches, which are protected from the alumina fiber film by a graphite
270  In these dry films, the particle aggregates are protected from the environment during storage and ar
271 beta-HSD1 KO mice with circulating GC excess are protected from the glucose intolerance, hyperinsulin
272 caspases promote proliferation and how cells are protected from the potentially harmful action of apo
273 he lumen of beta-barrel OM proteins where it is protected from the hydrophobic membrane interior.
274 cetylated STAT3 emerged upon HDAC inhibition was protected from the proteasome-mediated degradation o
275                Intriguingly, Spns2(-/-) mice were protected from the development of experimental auto
276 e, OVA323-339, mice that received MPO409-428 were protected from the development of humoral and cell-
277 that lack HMGB1 in the intestinal epithelium were protected from the development of lung injury, conf
278  receptor Fcgamma receptor IIB (FcgammaRIIB) were protected from the disorder.
279                             IFNAR1(-/-) mice were protected from the effects of hypoxia on the right
280                        As expected, HCR rats were protected from the HFD.
281             Mice lacking RANKL in osteocytes were protected from the increase in osteoclast number an
282              Mice transfected with nucleolin were protected from the lethal effects of agonistic anti
283 nd mice lacking the endogenous receptor CD36 were protected from the neuroinflammatory and BBB permea
284 imilar organism burdens, but MyD88(-/-) mice were protected from the PcP-related respiratory impairme
285              Here we discover how the embryo is protected from these genotoxins.
286 e and C5aR were pharmacologically inhibited, were protected from these adverse effects and consequent
287                                 Gpx1 Tg mice were protected from this aging-related enhanced suscepti
288 ptosis in vitro, and Ripk1(D138N/D138N) mice are protected from TNF-induced shock in vivo.
289          Instead, the HPRT locus appeared to be protected from transposon integration.
290  dramatically altered: encapsulated peptides are protected from trypsin hydrolysis, whereas physicoch
291 nfection, genitally infected IL-10(-/-) mice were protected from tubal pathologies and infertility, w
292 ice lacking nCDase treated with azoxymethane were protected from tumor formation.
293          This data suggests that hESC-EC may be protected from unwanted TLR4-mediated vascular inflam
294                                Because limbs are protected from use during a painful condition, this
295   Surprisingly, DNase-accessible euchromatin is protected from UV, while lamina-associated heterochro
296                                 Cancer cells are protected from VC-mediated cell death when co-cultur
297                    Medullary hyperosmolarity is protected from washout by countercurrent exchange and
298 ate the mechanism(s) by which Pemt(-/-) mice are protected from weight gain and insulin resistance.
299  carrying the gene for the human PV receptor are protected from wild-type PV when immunized with the
300  deficiency in complement C3 or C3a receptor were protected from WNV-induced synaptic terminal loss.

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