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1 pts associated with arrested RNA polymerases are protected from 3'-5' degradation and thus, unstable
3 eceptor (TLR) adaptor Mal (encoded by TIRAP) are protected from a number of infectious diseases, incl
4 because we show that vaccinated animals can be protected from a mucosal challenge with a heterologou
5 with plasmids or given nonhuman-primate sera were protected from a lethal challenge with purified Tcd
6 irus A/California/7/2009 (H1N1) strain (Cal) were protected from a lethal challenge with the heterolo
7 ina using a prime-boost vaccination strategy were protected from a subsequent lung challenge with P.
11 wild-type littermates, hCD39 transgenic mice were protected from acute renal injury at 24 hours, but
12 male) mice deficient in Arg-II (Arg-II(-/-)) are protected from age-associated glucose intolerance an
13 necrosis factor (TNF)-deficient mice, which are protected from age-associated inflammation, age-rela
14 sulin levels following glucose challenge and were protected from age-related reductions in GSIS and g
16 ar fat mass and energy balance, M(IL10) mice were protected from aging-associated insulin resistance
18 ted that integrin alphavbeta6-deficient mice are protected from airway hyperresponsiveness, due in pa
19 inflammasome components NLRP3 and caspase-1 were protected from aldosterone-induced vascular damage.
23 e conserved HIV-1 site of coreceptor binding is protected from antibody-directed neutralization by co
25 immunized with a single dose of this vector were protected from any signs of disease following letha
26 report that active spindle assembly factors are protected from APC/C-dependent degradation by microt
27 cells that received Tax-containing exosomes were protected from apoptosis through activation of AKT.
29 k-out mice that lacked PEMT showed that they were protected from atherosclerosis, diet-induced obesit
33 in response to different learning tasks and are protected from being eliminated when multiple tasks
35 n-G, amoxicillin, ampicillin, and cefazolin, are protected from beta-lactamase hydrolysis via the for
36 tic Ppp1r3b upon long-term fasting (12-36 h) were protected from blood ketone-body accumulation, unli
37 cient in the double-stranded DNA sensor AIM2 are protected from both subtotal body irradiation-induce
39 p to 1,200 microg/mL in serum, and up to 70% were protected from both i.v. and mosquito bite challeng
40 absence of target, aptamer coated particles are protected from capture on the test line and are inst
42 eletal muscle-specific EcSOD transgenic mice are protected from cardiac hypertrophy, fibrosis, and dy
43 v integrin is depleted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced sk
44 us for the ancestral C allele (EE genotype), are protected from cardiovascular disease (CVD), showing
48 oth mucosal and serum IgA anti-toxin Abs and were protected from CDI upon rechallenge, with protectio
50 ntial sH1N1 influenza virus-infected ferrets were protected from challenge with a novel H1N1 influenz
51 operties, are strikingly enriched in AT, and are protected from chemotherapy by the GAT microenvironm
53 contrast, the stability of the XRCC1 monomer is protected from CHIP-mediated ubiquitylation by intera
54 e we demonstrate that mice deficient in Irp2 were protected from cigarette smoke (CS)-induced experim
58 intact complement negative regulatory system are protected from complement attack, whereas cells with
60 operties rivalling suspended graphene, while being protected from contamination and mechanical damage
61 ssemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD); (ii)
62 ionic doping, and the LLMO cathode materials are protected from corrosion induced by organic electrol
63 CPE-sensitive enterocyte-like cell line) can be protected from CPE-induced cytotoxicity by preincubat
65 ytochrome c oxidase, which have reduced COX, were protected from CS-induced pulmonary inflammation an
67 del, we report here that TRPV4-knockout mice were protected from D. farinae-induced airway remodeling
68 dling development, the shoot apical meristem is protected from damage as the seedling emerges from so
69 type in the heart during EAM, IL-4(-/-) mice were protected from DCMi like DeltadblGATA1 mice, and eo
70 effect of IL-5, as IL-5TgDeltadblGATA1 mice were protected from DCMi, whereas IL-5(-/-) mice exhibit
76 ergen extracts: The protein/DNA molecule can be protected from degradation, higher local concentratio
77 bstrate of the Lon-type protease and that it is protected from degradation by Nfs1, the sulfur donor
78 terious interaction with the Bam complex but was protected from degradation and eventually inserted i
79 subunit, the palmitoylated CaV2.2 I-II loop was protected from degradation, although oligoubiquitina
80 blueberry polyphenols complexed with protein were protected from degradation during 16weeks at 4 degr
81 of ultralow methylation at transposons that are protected from demethylation in the germline and ICM
85 did not occur in IL-17-deficient mice, which were protected from development of lupus autoantibodies
86 r, mice with liver-specific deletion of MPC2 were protected from development of NASH on this diet.
87 red with WT, we demonstrated that Parp1(-/-) were protected from dextran-sulfate sodium-induced colit
89 arp contrast, TLR3- and MyD88-deficient mice were protected from diabetes following the same treatmen
90 lar to those of WT STZ mice, TLR4KO STZ mice were protected from diabetes-induced bladder hypertrophy
92 mice and mice treated with a P2X7 inhibitor were protected from diabetes-induced TNF-alpha, IL-1beta
94 p66Shc that is not acetylatable on lysine 81 are protected from diabetic oxidative stress and vascula
95 deficient in small heterodimer partner (SHP) are protected from diet-induced hepatic steatosis result
96 more, adipose-specific Dnmt3a knock-out mice are protected from diet-induced insulin resistance and g
99 d and that Mfge8-deficient (Mfge8(-/-)) mice are protected from diet-induced obesity, steatohepatitis
102 , we found that mice lacking hepatic ZFP36L1 were protected from diet-induced obesity and steatosis.
104 heir severe fatty liver, the transgenic mice were protected from diet-induced obesity and type 2 diab
106 th wild type, whereas AdipoR2-deficient mice were protected from diet-induced weight gain and metabol
107 e a limited elastic range of 3-4% strain but are protected from direct stretch during physiological l
114 tatic and dynamic disorder, but how carriers are protected from efficient scattering with charged def
116 n of IL-37 (IL-37tg mice) with intact IL-1R8 were protected from endotoxemia, IL-1R8-deficient IL-37t
118 Eventually, ERK inhibitor treated cells are protected from ETO-induced nuclear envelope (NE) rup
120 e have decreased Th17 cell numbers, and they are protected from experimental autoimmune encephalitis,
122 ve defect in Ag-specific alphabetaTh17 cells were protected from experimental ARDS induced by a singl
124 pe-specific deletions of PTEN-targeting APCs were protected from experimental autoimmune encephalomye
126 ng facilities with large elderly populations are protected from extreme heat (for example through bac
127 genetic or pharmacological inhibition of BID are protected from Fas-mediated impairment of mitochondr
129 les cannot sing to attract females, but they are protected from fatal attack by an acoustically orien
134 Our results suggest that certain tissues are protected from functional deleterious effects of pro
135 tive Staphylococcus aureus cells appeared to be protected from GA by an increased formation of nm-sca
138 to wild-type mice, Cd74-deficient mice also were protected from glomerular injury and ensuing activa
142 te-specific Shp1 knockout mice (Ptpn6(H-KO)) are protected from hepatic insulin resistance evoked by
143 notype, adipose-specific 11beta-HSD1 KO mice were protected from hepatic steatosis and circulating fa
147 pe littermate control (M-JAK2(+/+)) mice and were protected from HFD-induced systemic insulin resista
148 le of binding leukocyte alphaMbeta2-integrin were protected from HFD-induced weight gain and elevated
149 homologous residue Thr1150 (InsrT1150A mice) were protected from high fat diet-induced hepatic insuli
152 Furthermore we found that male AhRR Tg mice were protected from high-dose TCDD-induced lethality ass
153 pontaneous NASH whereas AEG-1(DeltaHEP) mice were protected from high-fat diet (HFD)-induced NASH.
155 show that GPR30 knockout (GPRKO) female mice were protected from high-fat diet (HFD)-induced obesity,
156 lacking both LDL receptor (LDLR) and Arhgef1 were protected from high-fat diet-induced atherosclerosi
157 note, adipocyte-specific gp130 knockout mice were protected from high-fat diet-induced hepatic steato
159 CreERT mice on different genetic backgrounds were protected from high-fat/ streptozotocin (STZ)-induc
161 nces of viral replication, and animals could be protected from HSE by acyclovir treatment provided 4
167 Here, we show that eosinophil-deficient mice were protected from induction of Th2-mediated peanut foo
168 These findings help explain why CF mice are protected from infection and nominate ATP12A as a po
170 factors that determine why some individuals are protected from infection while others go on to devel
171 tive cells engraft and traffic normally, and are protected from infection with CCR5- and CXCR4-using
174 igh-dose UV B radiation, IR/IGF-1R(MKO) mice were protected from inflammation, whereas controls devel
177 silon knockout mice on a high-fat diet (HFD) were protected from insulin resistance and showed altere
178 aRIIB globally or selectively in endothelium were protected from insulin resistance as a result of th
180 ing the fetal troponin I isoform, (ssTnI) to be protected from ischemia by increased glycolysis.
184 r expressed on myeloid cells 2 (Trem2 (-/-)) were protected from LCMV-induced hepatitis and showed im
185 ally, mice vaccinated with the DeltatolC LVS are protected from lethal challenge and clear challenge
187 ater column, seabirds, reptiles, and mammals are protected from lethal oiling at the surface, and mic
189 the CDV fusion and attachment glycoproteins were protected from lethal CDV challenge, whereas all an
191 myeloid cells and found that Blimp1 CKO mice were protected from lethal infection induced by Listeria
193 Fetal/neonatal progenitors may therefore be protected from leukemic transformation because they a
194 Plcbeta2-, Plcbeta3-, or Rac1-deficient mice were protected from lipopolysaccharide-induced lung inju
196 h VLPs and subsequently challenged with HMPV were protected from lung viral replication for at least
198 malaria pathogenesis because EphB2(-/-) mice were protected from malaria-induced liver fibrosis despi
202 ith a specific 11betaHSD1 inhibitor (UE2316) were protected from metabolic disturbances despite simil
203 ce vaccinated with the group 1 HA mini-stems are protected from morbidity and mortality against letha
204 (H5N2), A(H5N8), A(H6N1), or A(H7N9) viruses were protected from mortality and showed drastically red
207 treated with TAT2A during early adolescence were protected from MS-induced PVB loss and exhibited le
208 ut to reveal the mechanisms by which embryos are protected from mutant p53-induced transformation usi
209 n activating gene 1-deficient (Rag1-/-) mice were protected from NEC and transfer of intestinal lymph
210 iable region 1 yielded vector particles that were protected from neutralization by natural antibodies
211 he system was used to demonstrate that dsRNA is protected from nuclease digestion by virus-induced me
213 nt biological catalysts of H2 oxidation, can be protected from O2 damage upon integration into a film
214 cantly reduced intestinal fat absorption and are protected from obesity, hepatic steatosis and insuli
215 sue inflammation, cadherin-11-deficient mice were protected from obesity-induced glucose intolerance
216 U L3 XAS indicates that this U(V) species is protected from oxidation likely incorporated into oct
217 is Mast cell-deficient mice (Kit(W-sh/W-sh)) were protected from P. gingivalis-induced alveolar bone
218 Within these autophagosomes, the bacteria are protected from phagocytic killing, thus providing an
220 cient mice made diabetic with streptozotocin were protected from physiological and structural indices
222 ked as the most upregulated in vaccinees who were protected from Plasmodium falciparum infection.
225 nitrosylation, cyclic GMP, NO formation, and were protected from postinfarct and pressure overload HF
226 ct with cortactin, and Nox2-deficient hearts were protected from pressure overload-induced adverse my
227 V infection or SVR after antiviral treatment were protected from progressive liver disease and showed
231 etically deficient in PTP-alpha (Ptpra(-/-)) were protected from pulmonary fibrosis induced by intrat
234 n contrast, SMPDL3b overexpressing podocytes were protected from radiation-induced cytoskeletal remod
235 All BCV-treated mice that survived infection were protected from rechallenge without additional treat
237 ogen receptor alpha (ERalpha)-deficient mice are protected from renal disease and have prolonged surv
238 express only talin1(L325R) in myeloid cells were protected from renal ischemia-reperfusion injury.
242 develop high intraocular pressure (IOP) but are protected from retinal ganglion cell (RGC) dysfuncti
243 rains, we have determined whether cattle can be protected from rinderpest by inoculation with vaccine
246 ingle knockout IL-4(-/-) or IL-13(-/-) mice, were protected from Schistosoma-induced PH, with decreas
248 g for more than 40 weeks, and these macaques were protected from several infectious challenges with S
251 an inoculum containing Nod1 x 2(-/-) T cells were protected from severe graft versus host disease.
253 ozygotes and alpha(+)thalassemia homozygotes were protected from severe malaria (odds ratio [OR], 0.1
258 le explores the question of how the active X is protected from silencing by its own Xist locus, and t
259 d that mice lacking ephrin-B2 in fibroblasts are protected from skin and lung fibrosis and that a dis
260 n of Cosmc in 50% of crypts (IEC-Cosmc(+/-)) were protected from spontaneous inflammation and partial
261 hich also shows reduced muscle strength, but is protected from stretch-induced eccentric damage with
262 erm clearance of tumor after TLR7/RT therapy are protected from subsequent tumor rechallenge by the g
263 Vmac239 challenge, whereas only four animals were protected from subsequent intravenous SIVmac239 cha
264 ptible to WNV NS4B-P38G mutant infection but were protected from subsequent lethal wild-type WNV chal
265 ns were significantly higher in children who were protected from symptomatic malaria compared with th
266 ll-dependent antiviral immune responses, and are protected from T cell-mediated autoimmunity and allo
267 t neuronal cultures treated with Tat protein are protected from Tat-mediated cytotoxicity when treate
268 ned as "metabolically abnormal obese" (MAO), are protected from the adverse metabolic effects of weig
269 graphite die and the graphite punches, which are protected from the alumina fiber film by a graphite
270 In these dry films, the particle aggregates are protected from the environment during storage and ar
271 beta-HSD1 KO mice with circulating GC excess are protected from the glucose intolerance, hyperinsulin
272 caspases promote proliferation and how cells are protected from the potentially harmful action of apo
273 he lumen of beta-barrel OM proteins where it is protected from the hydrophobic membrane interior.
274 cetylated STAT3 emerged upon HDAC inhibition was protected from the proteasome-mediated degradation o
276 e, OVA323-339, mice that received MPO409-428 were protected from the development of humoral and cell-
277 that lack HMGB1 in the intestinal epithelium were protected from the development of lung injury, conf
283 nd mice lacking the endogenous receptor CD36 were protected from the neuroinflammatory and BBB permea
284 imilar organism burdens, but MyD88(-/-) mice were protected from the PcP-related respiratory impairme
286 e and C5aR were pharmacologically inhibited, were protected from these adverse effects and consequent
290 dramatically altered: encapsulated peptides are protected from trypsin hydrolysis, whereas physicoch
291 nfection, genitally infected IL-10(-/-) mice were protected from tubal pathologies and infertility, w
295 Surprisingly, DNase-accessible euchromatin is protected from UV, while lamina-associated heterochro
298 ate the mechanism(s) by which Pemt(-/-) mice are protected from weight gain and insulin resistance.
299 carrying the gene for the human PV receptor are protected from wild-type PV when immunized with the
300 deficiency in complement C3 or C3a receptor were protected from WNV-induced synaptic terminal loss.
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