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1 still controversial whether gliotransmitters are released from a cytosolic pool or by Ca(2+)-dependen
6 small amount of the studied metal oxides NPs was released from abrasion of the textiles coated by the
11 the body's principal hyperglycaemic hormone, is released from alpha-cells of the pancreatic islet.
13 ollectively these results suggest that ABCF1 is released from and binds to shed POSs in an autocrine
18 ted by ambient D-serine, whereas glycine may be released from astrocytes in response to afferent impu
19 inhibit itch and show that dynorphin, which is released from B5-I neurons, is a key neuromodulator o
22 , 9.4 GBq of (240)Pu and 29.7 GBq of (241)Am were released from both fire events corresponding to a s
24 tion assays show that in solution zebularine is released from CDA (k(off) > 0.14 s(-1)) much more rap
25 iments in vitro suggest that 2-aminoacrylate is released from CdsH following cysteine desulfhydration
27 s consisting of endogenous and exogenous Tau are released from cells and demonstrate their potential
29 data suggest that the different sgp130 forms are released from cells into their immediate surrounding
30 ) and some members of the Enterovirus genus, are released from cells nonlytically in membranous vesic
34 ibe three basic mechanisms by which GLUC can be released from cells: first, classical secretion by vi
38 calize with Kv2.1 clusters in live cells and are released from channels activated by voltage stimuli.
40 The mechanism whereby gaseous protein ions are released from charged solvent droplets during electr
41 they demonstrate in HeLa cells that once RNA is released from chromatin, the modifications are surpri
43 ts using a series of sodiated N-glycans that were released from commercially available glycoproteins
44 ations can be explained if ACh and glutamate are released from common vesicles onto spatially segrega
45 nt studies suggest that interleukin 6 (IL-6) is released from contracting skeletal muscles; however,
49 diates proteasomal degradation of slGFP that is released from cytosolic protein handling centers.
50 "danger signal" adenosine triphosphate (ATP) is released from damaged cells and promotes proliferatio
52 e proinflammatory danger signal IL-33, which is released from damaged or dying cells, achieves its ef
54 d even by non-CpG DNA and by self-DNA, which is released from dead cells and complexes with antimicro
55 geting tyrosine kinase inhibitor, masitinib, was released from degradable polymer microspheres delive
56 itical microRNAs that regulate inflammation, are released from dendritic cells within exosomes and ar
60 tamate is a second synchronizing signal that is released from DN1s and perceived in LNvs via the meta
70 e to environmental changes, TEs are known to be released from epigenetic repression and to become tra
73 tion showed that ~89-100% of As in porewater was released from exchangeable and specifically adsorbed
74 by the bioaffinity mechanism; 26% of the IgG were released from films embedded with pAG(MG) after fiv
75 the heterotrimeric G-protein complex that is being released from G-protein-coupled receptors on vagal
76 ulating form of the MIF receptor, CD74, that is released from hepatic stellate cells and that binds M
77 the very low density lipoprotein pathway and are released from hepatocytes as entities varying in the
78 enzyme Cajal body-associated protein, TCAB1, was released from hTR in mitotic cells coincident with T
79 endogenous oils have a higher propensity to be released from hypertrophied visceral fat in MUO indiv
82 throcytes, producing progeny merozoites that are released from infected cells via a poorly understood
83 HIV transactivator of transcription (Tat) is released from infected cells and has the ability to r
84 cated to 1.4-fold higher titers (P = 0.004), were released from infected cells 4.2-fold more efficien
85 s become necrotic immediately after rhizobia are released from infection threads into symbiotic cells
86 corticotropin-releasing hormone (CRH), which is released from inflamed tissues by cellular stress sig
87 nt in the extracellular space, but that PEPD is released from injured cells and tissues and that such
89 is signal is conveyed by a neuropeptide that is released from intestinal cells and acutely depolarize
91 hed E. coli and 8% of attached Ent. faecalis were released from IOCS columns during draining and rewe
92 ients with chronic spontaneous urticaria and was released from isolated basophils following either an
94 s separate from the rest of the root cap and are released from its edge as a layer of living cells.
96 We found that small amounts of UDP-sugars were released from keratinocytes and that UDP-glucose (U
100 g alternative ribosomes when M. tuberculosis is released from macrophages, to allow survival in the e
103 yme and its citrullinated substrate proteins are released from mast cells on activation with ATP.
106 zed via a number of endocytotic pathways and are released from membrane-enclosed endocytotic organell
108 study, shaving experiments showed that Shu1 is released from membrane preparations when spheroplast
110 hniques, (1) showing that clean hydrogen can be released from Mg(BH4)2 under mild conditions and (2)
111 uced, conformationally destabilized proteins are released from mitochondria in a size-limited manner.
115 hysiological settings, the persulfide sulfur is released from NFS1 and transferred to a scaffold prot
118 Strikingly, we found that nucleoporins can be released from nuclear bodies and reintegrated into ex
120 inner plexiform layer (IPL), where glutamate is released from ON and OFF bipolar cell terminals in se
121 nge the direction of EE transport, kinesin-3 is released from organelles, and dynein binds subsequent
123 gging the question of how tightly bound cAMP is released from PKA to reset its signaling state to res
126 uring the process of blood coagulation, BDNF is released from platelets, which has led to its extensi
128 n the observation that fluorescent reporters being released from polymeric drug delivery systems poss
132 tive study a cohort of 4,357 individuals who were released from prison via an amnesty on July 16, 200
133 tion of ENM are entering an in-use stock and are released from products over time (i.e., have a lag p
134 cells during the host immune response, iron is released from proteins and can act as a catalyst for
139 rate that PKA-dependent phosphorylated HDAC4 is released from Runx2 bound to the MMP-13 promoter in t
141 es indicated that the addition of CS - which was released from scaffolds quickly - significantly upre
145 th intercellular adhesion molecule 1 (ICAM1) is released from senescent cells by microvesicles indepe
146 ary to exponential phase (outgrowth), 6S RNA is released from sigma(70)-RNAP, resulting in a fast inc
153 measurements revealed that Sucnr1(-/-) mice were released from succinate-induced inhibition of lipol
155 8 colocalize across the AML genome, and each is released from super-enhancer regions upon chemical in
157 In conclusion, our data indicate that CD154 is released from T-cells by ADAM10 and ADAM17 upon CD40
158 studies demonstrated that some gut peptides are released from taste buds by prolonged application of
162 uses, which preferentially enter through and are released from the apical surface of polarized human
165 eptide (FCAP) and cerebral peptide-2 (CP-2)] are released from the cholinergic command-like neuron ce
166 technique is typically applied, the peptides are released from the chromatography column by the gradu
169 y mediators, such as nitric oxide (NO), that are released from the endothelium in response to fluid s
170 atory factors such as nitric oxide (NO) that are released from the endothelium under the influence of
174 ged due to the presence of CN(-) ions, which are released from the ferri/ferrocyanide redox probe.
176 hotoproduct-containing oligonucleotides that are released from the genome during excision repair.
177 temporary adhesive secretory vesicles (TASV) are released from the gland cells into the antennule via
181 Cytochrome c and other apoptotic factors are released from the intermembrane space through these
182 stroke, various damage-associated molecules are released from the ischemic core and diffuse to the i
184 y, we show that significant amounts of lipid are released from the nanodiscs upon insertion of larger
186 vanov et al. report that chromatin fragments are released from the nuclei of senescent cells and are
187 amers representative of the native topology, are released from the precursor upon SID, significantly
188 of an inflammatory response, ions/particles are released from the surface of the implant into the bi
189 of broken, PM "bubbles" with exposed PS that are released from the surface of the otherwise intact ce
190 re extra U residues, the nascent transcripts are released from the transcription initiation complex,
191 e C. elegans demonstrated that ectosomes can be released from the cilium and can mediate the intercel
192 DISC, phosphorylated caspase-8 is unable to be released from the complex; this inhibits further cycl
194 favoring the production of a PAH, which can be released from the framework under acidic conditions i
195 h aspect ratios (>10(4)); such nanowires can be released from the glass matrix and show relatively hi
196 -PCL nanoparticles simultaneously, and could be released from the micelles in an extended period in v
197 In vitro release assays showed heparin to be released from the microparticles over 8-12 h and for
200 stR that promote the KstR-DNA interaction to be released from the operator, retaining its dimeric sta
201 s fibers in which dopamine and glutamate can be released from the same axons, but are not normally re
202 ith visible light, the ruthenium complex can be released from the surface of the nanoparticles by sel
204 n persist long after the nascent protein has been released from the ribosome, and that a sufficient l
205 and responding to endosomal acidification by being released from the complex and inserting into and d
212 at the targeting site, the CPP modified drug is released from the blockage by a second triggering age
213 ntrol subjects, suggesting that, in MS, ET-1 is released from the brain to the cerebral circulation.
215 e, elucidating the mechanisms by which CD154 is released from the cell surface following its interact
217 r cells convert glutamine to glutamate which is released from the cell through the system Xc- antipor
218 Finally, we report that the excised 30-mer is released from the chromatin in complex with the repai
219 n is phosphorylated at the N-terminus, c-Jun is released from the complex and cannot be ubiquitinated
220 r of normal nuclear import cargoes, HIV-1 IN is released from the complex with TRN-SR2 by RanGTP.
221 drophobic core in the intermediate state but is released from the core in the fully activated state,
222 rtex of cells in both embryos and larvae and is released from the cortex in a Wnt-responsive manner.
223 n, and completely native and soluble insulin is released from the depot in a well behaved, first orde
224 peptide 1 (GLP-1) is a peptide hormone that is released from the gut in response to nutrient ingesti
226 in which the cellular ribosomal protein L13a is released from the large ribosomal subunit soon after
227 tic exocytosis whereby the cryptococcal cell is released from the macrophage into the extracellular e
228 y, wherein the soluble catalytic sector, V1, is released from the membrane and its MgATPase activity
229 in contains a transcriptional regulator that is released from the membrane when engagement of the cog
232 poptosis, XIAP is antagonized by SMAC, which is released from the mitochondria upon caspase-mediated
235 uclear receptor retinoic acid receptor gamma is released from the nucleus to disrupt TNF initiated ce
239 The mechanism through which soluble endoglin is released from the placenta is currently unknown; howe
241 t, throughout seedling development, a factor is released from the plastid to the cytoplasm that indic
242 a soluble form of NRX-1's ectodomain, which is released from the post-synaptic membrane by the SUP-1
243 central nervous system, (S)-glutamate (Glu) is released from the presynaptic neuron where it activat
244 omain immediately before the nascent protein is released from the ribosome and decrease its chances o
246 to the initial ring radius if either myosin is released from the ring during contraction and actin f
247 , except when the duplicated terminus region is released from the septum and recoils to the center of
248 or, both in solution after the DNA-initiator is released from the solid support as well as directly o
252 llowing laser treatment, hydrophilic AMD3100 was released from the aqueous liposome chamber and then
254 potential (Deltapsimito) depolarized; Ca(2+) was released from the endoplasmic reticulum (ER), increa
256 ine-inducing, fully reduced isoform of HMGB1 was released from the ischemic brain in the hyperacute p
257 only 70% of the more hydrophobic doxorubicin was released from the material, whereas the more hydroph
260 , EFA6R lacking the CC domain (EFA6RDeltaCC) was released from the PM into the cytosol upon PIP2 depl
261 DEX, a powerful ameliorator of inflammation, was released from the polymer by external application of
262 OH acids occupied), and the absorbed peptide was released from the polymer for >2 weeks in a controll
268 extraction of recombinant virions, peptides were released from the coat protein by chemical cleavage
270 ective capture, the purified MC-LR molecules were released from the extractor nanoparticles within 5m
273 water, we find that vibrational bonds of C2 were released from the molecule containing carbon-carbon
274 , genes and a putative enhancer in LADs that were released from the periphery during T-cell activatio
278 ydrogen, electrostatic and covalently bonds, were released from the sandwiches after dissolving in di
279 During intermittent flow, fewer bacteria were released from the saturated column compared to the
286 capture support, the purified DNA sequences were released from this support upon treatment with tetr
287 Recently, lipids have been identified that are released from tissues and act locally or systemicall
289 rms of two substrate proteins, but while BiP is released from unfolded substrates in the presence of
290 receptors also sense endogenous ligands that are released from uninfected dying cells, thereby activa
292 junctions, the synapses, where transmitters are released from vesicles in a Ca(2+)-dependent fashion
294 ast Vo The structure indicated that, when V1 is released from Vo, the N-terminal cytoplasmic domain o
297 s a highly toxic priority pollutant that can be released from wetlands as a result of biogeochemical
298 not all polyphenolic compounds were able to be released from WPM, stronger interactions, possibly by
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