ライフサイエンスコーパス: be relieved by

1 s apparent inhibition of channel conductance was relieved by 1 microM diazepam.

2 with dinitrogenase causes inhibition, which is relieved by 2OG.

3 bes and associated metabolites, but symptoms are relieved by a Bacteroides fragilis probiotic.

4 ion of class III/IV flagellar genes that can be relieved by a gain-of-function mutation in flbD calle

5 inhibition by its C-terminal tail, which can be relieved by a phosphoinositide to stimulate flippase

6 the gxySBA operon, the function of which can be relieved by a subset of the transported sugars, inclu

7 the inhibition placed upon the output domain is relieved by a conformational change that alters inter

8 mediated transcriptional repression in vitro is relieved by a match polyamide but not by a mismatch p

9 onal strain in an IHF-mediated DNA kink that is relieved by a nick (at least 0.76 kcal/mol) and pinpo

10 in the SOD-competent parental strain, and it was relieved by a cell-permeant mimic of SOD activity; h

11 hieved by showing that the growth inhibition was relieved by a mutation in a single residue in the ca

12 ifferentiated until Pax3-mediated repression is relieved by activated beta-catenin.

13 xpression of G alpha(o), and this inhibition is relieved by activation of alpha2A-adrenergic receptor

14 9-induced platelet aggregation by Ro 31-8220 was relieved by activation of the G(i) pathway by select

15 This inhibition is relieved by added H2, resulting in a lowering of the

16 This metabolic block could be relieved by addition of the alpha-ketoglutarate precu

17 )-1- or Cu-stimulated VEGF expression, which is relieved by addition of excess Cu.

18 from the prgQ promoter, and this repression was relieved by addition of exogenous cCF10.

19 iate receptor stimulation in the VTA and can be relieved by administration of the dopamine receptor a

20 7-OH substituent and the substrate C2 carbon is relieved by adopting the globally strained structure.

21 This depression was relieved by amacrine cell inhibitory feedback that a

22 l rickettsial infection, and the suppression was relieved by aminoguanidine.

23 This inhibition is relieved by AMP, an allosteric activator of this regu

24 tive effect displayed by the HA2 protein can be relieved by an additional mutation in HA2 RNase T tha

25 Profilin-mediated INF2 inhibition is relieved by an anti-N-terminal antibody for INF2 that

26 The experience of depression being relieved by another pregnancy may be a previously

27 and that many of the features of the illness are relieved by antibiotic therapy.

28 datively stressed and oxidative stress could be relieved by antioxidant treatment, leading to improve

29 agitation and stereotypical behaviors, which are relieved by antipsychotics.

30 for its activity, but this dependence could be relieved by any of six amino acid substitutions in th

31 ical obstruction to left ventricular outflow is relieved by aortic valve replacement.

32 hese two steps in the cross-linked actin can be relieved by appropriate factors.

33 gically important effect), this obstacle can be relieved by at least two strategies for exposing regu

34 inhibitory effect of secretin and ACh could be relieved by atropine, and by the protein kinase C (PK

35 ADP inhibition of bacterial Hsp90 can be relieved by bacterial Hsp70 and an activating client

36 Second, the resultant membrane strain is relieved by bending and by creation of exovesiculated

37 This novel mode of autoinhibition is relieved by binding of another ligand to WW3.

38 he Vps4p MIT domain and that this inhibition is relieved by binding of either Type 1 or Type 2 MIM el

39 basal catalytic activity of PLCdelta1, which is relieved by binding of Gbetagamma subunits to PLCbeta

40 were CD8(+)CTLA-4(+), and their suppression was relieved by blockade of CTLA-4, but not IL-10 or TGF

41 Thus, the barriers posed by 53BP1 and RAP80 are relieved by BRCA1 and POH1, respectively.

42 l factors to achieve growth regulation could be relieved by bringing the E2F1 activation domain to th

43 n EPS biosynthesis, and that this repression is relieved by c-di-GMP.

44 xin does not function as a fusion clamp that is relieved by Ca(2+)-synaptotagmin.

45 Furthermore, C2B inhibition is relieved by calcium binding to C2B, while the neighbo

46 MEF2 activity only when repression by HDACs is relieved by CaMK signaling to the DNA-binding domain.

47 ree-generation Canadian family in which pain is relieved by carbamazepine (CBZ).

48 H-rev107 transcription in these cells could be relieved by chemically induced hypomethylation with 5

49 We demonstrate that strain in pBn can be relieved by cleaving the linker with a chemical reage

50 However, S6K1 sensitivity to osmotic stress was relieved by co-overexpression of RAPTOR1.

51 to cell proliferation; this inhibition could be relieved by coexpression of a cyclin B1 derivative th

52 This constraint is relieved by coexpression of SSP in trans, allowing tr

53 The block was relieved by coexpression of a U12 snRNA containing c

54 T expression in Xenopus oocytes was found to be relieved by coinjection of transcripts from other def

55 al rearrangement, whereby autoinhibition can be relieved by competitive sequestration of the autoinhi

56 MA-mediated inhibition is relieved by concentration-dependent Gag multimerizati

57 its own binding, and in that case repression is relieved by cooperative interactions with SNAP(c).

58 on fkh-6 expression and gonadogenesis, which is relieved by cyclin D activity.

59 ak at night when dopamine levels are lowest, are relieved by D3 agonists, and likely involve increase

60 These hypermorphic phenotypes could be relieved by deleting more of the N terminus, or in so

61 This suppression was relieved by deletion and mutation of the miR-206-bin

62 his system seems to be oxidative stress that is relieved by dipyridamole.

63 actions on the phosphate moiety of bound R1P are relieved by dissociation of R1P from PNP or by hydro

64 the stalling of PolB1 at the lesion site can be relieved by Dpo4.

65 nd the RASSF1A-induced cell cycle arrest can be relieved by ectopic expression of cyclin D1 or of oth

66 to participate in gene suppression that can be relieved by EGFR signal, but we found that cic also a

67 Both blocks in HIV-1 replication can be relieved by either expression of the appropriate huma

68 Autoinhibition of Ets-1 could be relieved by either protein partner(s) or posttranslat

69 icylic acid-dependent resistance pathway and is relieved by either a mutation in EDS1 or the presence

70 on both dividing and nondividing HeLa cells was relieved by either pharmacological or genetic disrup

71 These disruptive effects were relieved by either mutation of the catalytic residu

72 ss, and that the inhibitory activity of NIFL is relieved by elevated levels of 2-oxoglutarate when PI

73 nhibition of beta-actin folding by PDCL3 can be relieved by exchanging its acidic C-terminal extensio

74 ctivity of the promoter, and this repression is relieved by exogenous sphingosine.

75 ced by COX-2 inhibitors, and this inhibition was relieved by exogenous supplementation with PGE(2).

76 onsequences of the inhibition of PI 3-kinase are relieved by expression of a constitutively active fo

77 These effects could be relieved by expression in trans of the ltrA gene clon

78 ATG-mediated inhibition of transformation is relieved by expression of constitutively active Rac1,

79 The block is voltage dependent, is relieved by extracellular potassium and has rapid kin

80 e that the inhibitory activity of PTC124-AMP is relieved by free coenzyme A, a component present at h

81 This requirement of Sec14p is relieved by genetic inactivation of the cytidine diph

82 in the flipped-out nucleotide substrate that are relieved by glycosylic bond cleavage in the product

83 type; however, the acid-sensitive phenotype was relieved by growth in the presence of long-chain mon

84 how Ptc inhibits Smo and how such inhibition is relieved by Hh stimulation.

85 ation of core histones at promoter sites and is relieved by histone deacetylase inhibitors.

86 ted by the feoB mutant in Fe-deficient media was relieved by human holo-transferrin, holo-lactoferrin

87 In eukaryotic cells, this inhibition is relieved by hydrolysis of SAH to adenosine and homocy

88 Upon mitogen stimulation, this repression is relieved by hyperphosphorylation of Rb by the cyclin

89 This requirement can be relieved by inactivation of the cytosine 5'-diphospha

90 ration (median age 55.4 months), obstruction was relieved by incision of hypertrophied anomalous musc

91 of lateral root initiation in WT plants can be relieved by increased concentrations of external nitr

92 established but not understood, was found to be relieved by increased expression of a DUB that promot

93 orted by data showing that growth inhibition is relieved by increasing concentrations of tryptophan i

94 ts that strain, introduced by the disulfide, is relieved by increasing the twist of the long-pitch ac

95 diated mecA transcriptional repression could be relieved by induction through homologous but not hete

96 ng conferred by MeCP2 and methylated DNA can be relieved by inhibition of histone deacetylase, facili

97 ucture and a transcriptional repression that is relieved by inhibition of DNA methylation.

98 sed by ethambutol or isoniazid treatment may be relieved by iniBAC to increase the chance of bacteria

99 nce spectroscopy that the autoinhibition can be relieved by integrin or integrin regulator migfilin.

100 eavage of domain A2, and that inhibition can be relieved by interaction of domain A1 with platelet GP

101 This inhibition can be relieved by interaction with protein partners or coop

102 esses its own binding to DNA, and repression is relieved by interactions with the Oct-1 POU domain th

103 rly block to HIV-1 infection in monkey cells is relieved by interference with TRIM5alpha expression.

104 of one insertion variant reveals that strain is relieved by intermolecular folding with an identicall

105 The transdominant phenotype can be relieved by introducing a second mutation in the DNA-

106 volves cooperation between DNMT1 and p53 and is relieved by introduction of DNMT1- or p53-specific sm

107 transcription from the irr promoter in vitro was relieved by Irr.

108 Here, we show that the polar barrier is relieved by ISW2, an ATP-dependent chromatin remodele

109 mediate gene repression, and this repression is relieved by JNK-mediated c-Jun N-terminal phosphoryla

110 The repression was relieved by laminaribiose, also in a dose-dependent

111 diated autoinhibition of the HECT domain can be relieved by linker post-translational modifications,

112 the repression of the fadBA5 operon by PsrA is relieved by long-chain fatty acids (LCFAs).

113 /C interaction on activation function 2 that was relieved by MAGE-11.

114 Repression by GAL4-MBD3L2 is relieved by MBD2 and vice versa, and repression by MB

115 epression by MBD2 from a methylated promoter is relieved by MBD3L2.

116 ver, the suppression of rdgA-tacZ expression is relieved by MC in the RecA+ E. coli strain MC4100, bu

117 ions 1 and 2, noted above, could conceivably be relieved by migration of the acyl group to the neighb

118 It is proposed that this steric strain can be relieved by migratory insertion, to give a five-coord

119 ATA6 represses MIR100HG, but this repression is relieved by miR-125b targeting of GATA6.

120 This actin inhibition is relieved by Mn(2+), providing a direct and experiment

121 ation; upon TNF stimulation, the suppression is relieved by Mule/ARF-BP1-mediated Miz1 ubiquitination

122 ethoprim (TMP) prevents growth, but this can be relieved by murine DHFR (mDHFR).

123 tially in G0/G1 thymocytes; this restriction is relieved by mutation of Thr-490 to alanine (T490A).

124 When repression of the H1t promoter was relieved by mutation of GC box 2, additional mutatio

125 Cell death induction was relieved by mutation of the BAR domain, a putative e

126 ere, we show that this catabolite repression is relieved by mutations that weaken the DNA site for Fi

127 on of the hTERT promoter and this repression was relieved by Myc overexpression.

128 when competition for H(2) inside the biofilm was relieved by nearly complete removal of NO(3)(-).

129 asm, and that the importin-alpha aggregation is relieved by NLS peptide binding, with the importin-al

130 The inhibitory activity of p85 is relieved by occupancy of the NH(2)-terminal SH2 domai

131 We show that this TFII-I inhibition is relieved by one of the previously defined viral activ

132 gy of approximately 8 kcal mol-1 which could be relieved by opening the s-cis lactone ester bond to a

133 teins avoids adventitious expression and can be relieved by other DNA-binding counter-silencing prote

134 nt on the p300-binding domain of E1A and can be relieved by overexpression of p300.

135 rature-sensitive growth, but this defect can be relieved by overexpression of YPT6.

136 to growth rate reduction, and this reduction was relieved by overexpression of tRNA(1)(Pro).

137 pneumophila in broth, and growth inhibition was relieved by overproduction of LppA or by metal ion t

138 p53-mediated repression of RelA is relieved by p300 overexpression and the increased Rel

139 main on binding of the Fyn SH3 domain to CD2 was relieved by peptides containing a phosphorylated YEE

140 Ser-473 phosphorylation and entry into G2/M was relieved by PHLPP phosphatase inhibition.

141 s that tethering of cross-bridges by cMyBP-C is relieved by phosphorylation of PKA sites in cMyBP-C,

142 TRIM28-mediated repression of STAT3 is relieved by phosphorylation of S473, and in vitro kin

143 This repression is relieved by phosphorylation of the N-terminal region

144 ates can inhibit kinase activation, and this is relieved by phosphorylation of these substrates by th

145 bition of transport substrate affinity, that is relieved by phosphorylation).

146 Na(+)(o) inhibition of HERG current was relieved by physiological [K(+)](o), while Na(+)(o)

147 s [Na]i affinity, and this inhibitory effect is relieved by PKA activation.

148 ts as an inhibitory constraint on I-LTP that is relieved by PKA.

149 its apparent Na(+) affinity, an effect that is relieved by PLM phosphorylation.

150 e but show that the requirement for clpX can be relieved by point mutations, frame shifts, or deletio

151 ddition, suppression of CTGF mRNA expression is relieved by pretreatment with an antagonist of PPAR g

152 NaC by affecting channel gating, which could be relieved by proteases cleavage.

153 cis-acting autoinhibitory domains, which can be relieved by protein kinase-mediated phosphorylation o

154 Channel block was relieved by pyrophosphate (5 mM) and ATP (5 mM), two

155 d54 contains an autoinhibitory activity that is relieved by Rad51.

156 Auto-inhibition is relieved by recognition of two template features, the

157 nts, Drosophila and mammals; this repression is relieved by reducing copy number, leading to chromati

158 complex and a block in transcription, which is relieved by removal of Leo1 or Rtf1.

159 where the inhibited state of the holoenzyme is relieved by removal of sigmaN Region I sequences.

160 Pairing-sensitive silencing is relieved by removing the transvection protein Zeste,

161 two adjacent equatorial positions, and these are relieved by rotation toward the eclipsed conformatio

162 elated sigma factor and that this inhibition is relieved by RsrA binding, reaffirming that region 2 i

163 Itch is relieved by scratching, but the neural mechanisms tha

164 y through a substrate-directed effect, which is relieved by Ser-301 phosphorylation.

165 This repression is relieved by shortening day length resulting in up-reg

166 The checkpoint function of NCoR is relieved by signal-dependent phosphorylation of c-Jun

167 Accordingly, repression is relieved by small in-frame insertions before this sec

168 ication-stressed Pif1-deficient cells, which was relieved by spontaneous mutations that eliminated th

169 essed protein level in normoxic cells, which was relieved by stimulation with hypoxia, cobaltous ions

170 promoter with tandemly linked GAS motifs can be relieved by strengthening the avidity of protein-DNA

171 ence and a slowing of channel activation and is relieved by strong depolarization resulting in facili

172 La3+ block was relieved by strong depolarization in a time- and vol

173 This inhibition was relieved by strong depolarizing prepulses.

174 rc/suprasubduction-zone ophiolite obduction, is relieved by subduction polarity reversal (flip).

175 ed signal, this dynamic range constraint can be relieved by subtracting away components of the signal

176 a-butyrolactone, and its repressing function is relieved by succinate semialdehyde (SSA).

177 eurally mediated vasoconstriction, which can be relieved by systemic NOS inhibition with L-NMMA.

178 fore the G1/S restriction point, by p16INK4a is relieved by Tax.

179 Inhibition of delta-toxin deformylation was relieved by TCA cycle inactivation or by addition of

180 This inhibition is relieved by TGF- beta signalling, to which the future

181 eta repression of the SMA enhancer could not be relieved by TGFbeta1, whereas repression mediated by

182 The effect of the boronic acids could be relieved by the addition of boron-complexing sugars a

183 on protein A from M. thermoautotrophicum can be relieved by the addition of M. thermoautotrophicum ho

184 The defect could be relieved by the addition of magnesium to the swarm pl

185 n linked to the loss of ELO3 activities, can be relieved by the addition of metabolic sugars in the g

186 iated inhibition of the HCV IRES is shown to be relieved by the addition of recombinant human PTB in

187 The inhibition of Smo by Ptc can be relieved by the addition of Shh.

188 The thiamin requirement can be relieved by the addition of the thiamin precursor 5-h

189 ition of human and yeast cytosolic Hsp90 can be relieved by the cochaperone aha1.

190 atory suppression of fAbeta phagocytosis can be relieved by the coincubation with anti-inflammatory c

191 ion in a transient assay, and repression can be relieved by the deacetylase inhibitor trichostatin A

192 This repressive activity can be relieved by the HDAC inhibitor trichostatin A.

193 criptional repressor QS, and suppression can be relieved by the nontoxic small molecule quinic acid.

194 ost, but not all, such elongation blocks can be relieved by the re-addition of GTP.

195 ble epilepsy that is associated with HME can be relieved by the surgical treatment hemispherectomy, a

196 the tethered head of wild-type kinesin have been relieved by the long insertions.

197 The inherent repression of the 20S CP is relieved by the ATP-independent activators 11S and Bl

198 Autoinhibition is relieved by the binding of Ca(2+)-calmodulin to the c

199 This inhibition is relieved by the binding of the C-terminal region of c

200 abilize a distorted state of polymerase that is relieved by the binding of the two nucleotides requir

201 Transcriptional repression in vivo is relieved by the deacetylase inhibitor trichostatin A,

202 anscriptional repression by NK-3 and Groucho is relieved by the histone deacetylase inhibitor trichos

203 h is required for activation when repression is relieved by the inductive signal.

204 s with hexamer formation, an inhibition that is relieved by the insertion of the 56-kDa species.

205 ne 3T3 fibroblasts, the block to HIV-1 entry is relieved by the introduction of human CD4 and CCR5 or

206 undergo premature replicative arrest, which is relieved by the loss of p53.

207 The resulting acyl enzyme intermediate is relieved by the nucleophilic attack of the side-chain

208 lding wherein PDI acts as a placeholder that is relieved by the pairing of cysteines caused by substr

209 ugh increasing oxidative stress, this effect is relieved by the presence of Mn2+.

210 impose a replication fork pause, and pausing is relieved by the Rrm3p helicase.

211 s signal-peptide binding, but autoinhibition is relieved by the SecB chaperone.

212 This "stalled" UPR is relieved by the subsequent activation of compensatory

213 and inhibits translation, an inhibition that is relieved by the substrate acetyl-CoA.

214 ritical evidence of ER stress, which in turn is relieved by the UPR.

215 d with a decrease in oxygen availability and was relieved by the addition of nitrate.

216 this gene was restored when self-inhibition was relieved by the addition of plant surface waxes.

217 ition of DNA repair by the C terminus of p21 was relieved by the addition of purified PCNA protein.

218 tion in vitro by an excess of Fos-Jun dimers was relieved by the addition of TFIIE, indicating that i

219 endothelial cell growth, and this inhibition was relieved by the coaddition of either MMP inhibitors,

220 re of RUNX1c to allow nuclear colocalization was relieved by the deletion of amino-terminal residues

221 the occurrence of anaphylactic shock, which was relieved by the intramuscular injection of adrenalin

222 n, the growth defect of the DeltayopM mutant was relieved by the parent Y. pestis strain in a coinfec

223 matin template, however, and this repression was relieved by the phospho (Ser133) KID-dependent recru

224 This inhibition was relieved by the presence of a TR domain, which conta

225 ant mechanism by which mitral valve stenosis is relieved by this technique, and thus commissural morp

226 after infection by NRs, with this repression being relieved by titration of these repressors through

227 ntaneous insulin insensitivity, which cannot be relieved by TOPOBOP treatment.

228 ceptor can undergo self-squelching, and this was relieved by transfected c-Jun.

229 The repression of the promoter activity was relieved by treatment with DNA methyltransferase inh

230 This inhibition was relieved by treatment with estradiol but not by trea

231 The former effect of insulin was relieved by treatment with inhibitors of phosphatidy

232 This repression was relieved by treatment with the histone deacetylase i

233 Transciptional repression of targeted genes was relieved by treatment with the methyltransferase inh

234 ssors to suppress TrkA expression, which can be relieved by trichostatin A, an HDAC inhibitor.

235 Repression could be relieved by trichostatin A.

236 related Vav isozymes, auto-inhibition of Tim is relieved by truncation, mutation, or phosphorylation

237 This silence is relieved by TSA but not by RA.

238 Ume6p repression is relieved by ubiquitin-mediated destruction that is st

239 ginal activity by T3Rbeta and the repression is relieved by usp/T3Rbeta heterodimers.