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1 y dynamic structure of the mitochondrion can be remodeled to accommodate a change in oxidative phosph
2  in formation of these binding sites and can be remodeled to accommodate different non-cognate substr
3 by an extracellular matrix (ECM), which must be remodeled to accommodate growth and morphogenesis.
4 dues 297 to 362 of the C-terminal arm had to be remodeled to accommodate the higher curvature of the
5 central to providing resiliency, but how ECM is remodeled to accommodate growth is poorly understood.
6 across germ layer lineage boundaries and can be remodeled to adopt the characteristics of a new cell
7  by a peptidoglycan (PG) cell wall that must be remodeled to allow cell growth.
8 ric MuA transposase with recombined DNA must be remodeled to allow subsequent DNA replication.
9 plex DNA, and a conserved aromatic-rich loop is remodeled to bind ssDNA.
10 am of the injury degenerate, while the nerve is remodeled to direct axonal regrowth.
11 onical side chain enabled the active site to be remodeled to enable more efficient stabilization of t
12 between cytosolic factors and membranes that are remodeled to encapsulate autophagic cargo within an
13 dented mode in which the Ras effector domain is remodeled to expose an extended pocket that connects
14 n may stabilize early synapses that can then be remodeled to express a different cadherin and that ca
15 bition and the mechanisms by which chromatin is remodeled to facilitate the regulation of gene expres
16 epithelium of the tip and the adjacent trunk is remodeled to form new branches.
17 nfection, the host cell endosecretory system is remodeled to form the cytoplasmic virion assembly com
18 eta-intercalated (HCO(3)(-)-secreting) cells are remodeled to functionally resemble alpha-intercalate
19 attachment in the uterus, where the arteries are remodeled to supply the fetus with oxygen and nutrie
20 M with diverse physico-mechanical properties is remodeled to support blood vessel formation has remai

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