ライフサイエンスコーパス: be responsible for

1 Scabies was responsible for 0.21% of DALYs from all conditions s

2 ly, the croplands that are likely to be lost were responsible for 3-4% of worldwide crop production i

3 In Belgium and the Netherlands, DCDs were responsible for 30% of deceased donor liver transpl

4 ntation with a median delay of 50 months and were responsible for 5 deaths.

5 access to food resources, we found that ants were responsible for 52% of total bait removal whilst ve

6 I estimate that warming over the last 30 y is responsible for 59,300 suicides in India, accounting

7 s (37%), while for deeper regions arterioles are responsible for 61% of the total pressure drop.

8 We propose that mutations in TBC1D23 are responsible for a form of PCH with small, normally p

9 of the DREAM complex, had been suggested to be responsible for a continued ability to arrest by inhi

10 and together with the SPINK 1 mutation, may be responsible for a more severe course of the disease.

11 ough increased intrinsic noise is thought to be responsible for a range of perceptual deficits in amb

12 gonal to the heterojunction and find that it is responsible for a large interfacial nonlinearity prob

13 However, tamoxifen resistance is responsible for a large proportion of breast cancer d

14 , together with disrupted receptor function, is responsible for a lethal startle phenotype in a novel

15 Microbial enzymatic chain shortening was responsible for a shift in ethoxymer molecular weigh

16 Gram-positive bacteria were responsible for a high proportion of infections amo

17 anisms and extracellular carbonic anhydrase, is responsible for active transport-mediated disposal of

18 cycle, driven by the tachyzoite life stage, is responsible for acute toxoplasmosis.

19 matopoietic expression of TREM2 was found to be responsible for aggravated hepatitis, indicating a no

20 eta-glucosidase active against xyloglucan 1) is responsible for all or most of the soluble activity,

21 3' of the Nkx3.1 transcription start site to be responsible for alterations in Nkx3.1 expression patt

22 ibility that a reduction in neuronal density is responsible for alterations in the tissue-wide editin

23 n 10 (K10) resulting from Cdk5 knockdown may be responsible for an abnormal epidermal structure.

24 n induces a structural change in a loop that is responsible for antibody binding.

25 (BatF3)-dependent and BatF3-independent DCs are responsible for antigen exchange and cross-presentat

26 In arthropods, RNA interference (RNAi) is responsible for antiviral defense.

27 Asthma is responsible for approximately 25,000 deaths annually

28 Tropical deforestation is responsible for around one tenth of total anthropogen

29 n candidate driver (CD) genes are thought to be responsible for at least some cases of CH.

30 The maturation protein is responsible for attaching the virus to an F-pilus and

31 of the structure of the HCV envelope, which is responsible for attachment and fusion, could aid in t

32 he decrease in anthropogenic organic aerosol is responsible for averting 180,000 (117,000-389,000) pr

33 Histone chaperones are responsible for binding the highly basic histone pro

34 Mitochondria are responsible for bioenergetics, metabolism and apopto

35 or flavones such as chrysin and apigenin and is responsible for biosynthesis of baicalein and scutell

36 as exchange and low light levels under water is responsible for both a carbon and an energy crisis ul

37 show that zygotically expressed MOM-2 (Wnt) is responsible for both interactions.

38 tional subunit of the Rad3-Rad26 complex and is responsible for bringing the kinase to sites of DNA d

39 Furthermore, base excision repair (BER) is responsible for causing CAG repeat contractions downs

40 Islet amyloid polypeptide (IAPP) is responsible for cell depletion in the pancreatic isle

41 The MAPK pathway is responsible for cell proliferation and plays an impor

42 e involved in oxidative phosphorylation that are responsible for cellular energy production.

43 are small, double-stranded DNA viruses that are responsible for cervical, oropharyngeal, and various

44 cence spectra analysis, Mcp was confirmed to be responsible for chemotaxis towards As(III) and to bin

45 Group B coxsackieviruses are responsible for chronic cardiac infections.

46 The pduCDE enzymes are responsible for cleaving ethoxylate chain units into

47 , which sustain minimal residual disease and are responsible for CML relapse following discontinuatio

48 o) is the most abundant enzyme in plants and is responsible for CO2 fixation during photosynthesis.

49 antly slower, longer-range fluctuations that are responsible for complete randomization of the liquid

50 Our results suggest that Folfiri is responsible for concomitant muscle and bone degenerat

51 ut neurons of the central nervous system and are responsible for controlling muscle contraction.

52 l nonreducing polyketide synthases (NR-PKSs) are responsible for controlling the aldol cyclizations o

53 trated that MCPIP1 in nonhematopoietic cells is responsible for controlling skin pathology.

54 pses between IHCs and auditory nerve neurons are responsible for converting receptor potentials into

55 vival and milk production, as this community is responsible for converting plant-based feeds into acc

56 , is found widespread in marine bacteria and is responsible for converting TMA to TMAO.

57 sorting center of the cell, the Arf GTPases are responsible for coordinating vesicle formation.

58 waterways, lakes, and coastal marine beaches is responsible for costs that should be accounted for wh

59 dependent AhR-XRE-mediated Stc2 upregulation is responsible for cytoprotection against ectoplasmic re

60 red the understanding of mechanisms that may be responsible for defective skin repair.

61 on of free fatty acids (FFAs), which in turn is responsible for delayed suppression of hepatic endoge

62 est that polymorphism in the CagA C-terminus is responsible for differential alterations in some, but

63 he muscle-specific microRNA (myomiR) family, is responsible for direct and selective targeting of MCU

64 lex stress-related 1 (LHCSR1) protein, which is responsible for dissipation in green algae and moss.

65 elevation of the levels of TET enzyme, which is responsible for DNA demethylation in UVB-exposed skin

66 e results suggest that compartmental domains are responsible for domain structure in all eukaryotes,

67 ive trigger, a subinterval of magmatism must be responsible for driving deleterious environmental eff

68 scherichia coli, a single pair of replisomes is responsible for duplicating the entire 4.6 Mbp circul

69 ells that secrete IL-17A, but not IFN-gamma, was responsible for early IL-17A production.

70 capsid surface and in internal networks that are responsible for EC stability.

71 These minimal changes in the MCCB could not be responsible for effective active treatments failing t

72 ce muscle catabolism by activating TLR4, and are responsible for elevation of circulating cytokines.

73 IR is responsible for Endothelial Dysfunction (ED) through

74 Tuberculosis (TB) is responsible for enormous global morbidity and mortali

75 1-positive slow recycling pathway, which may be responsible for ensuring a persistent pool of GPRC6A

76 auses severe hemorrhagic fever in humans and is responsible for epidemics throughout sub-Saharan, cen

77 ossibly during the giant Moon-forming impact were responsible for establishing the observed abundance

78 Its peptide part is responsible for facilitated transport inside sensitiv

79 ys, with powerful enzymatic back-up systems, are responsible for fine-tuning physiological redox sign

80 Piezo1 is responsible for flow-sensitive non-inactivating non-s

81 and the associated ground-state aggregation is responsible for fluorescence modulation under certain

82 or sequential gene activation, and the other is responsible for freezing temporal rhythms into spatia

83 ng modules at the inter-domain interface may be responsible for global structural changes and a popul

84 are the primary support cells of the CNS and are responsible for glutamate clearance, metabolic suppo

85 DK1-positive tissue, we find that lncRNA H19 is responsible for glycolysis and BCSC maintenance.

86 %) of the interatrial septum was observed to be responsible for >/=80% of the final R-R and AH interv

87 se circuit adaptations are unknown and could be responsible for habitual behavior.

88 irst time, on regions within the 530 bp that are responsible for high-levels of ltxA expression.

89 The prefrontal cortex is responsible for higher order cognitive processing, an

90 tic extractions with Cellulase and Viscozyme were responsible for highest extraction yields (67-77%).

91 re is increasing evidence that these effects are responsible for HVEM-mediated pathogenesis in the ey

92 subtle interplay between these three factors is responsible for (i) the formation of non-IPR (isolate

93 vations showing that the osteoid matrix that is responsible for implant osseointegration originates f

94 ormed from a strong base and DMSO (solvent), is responsible for inducing the initiation by a photo-BH

95 transcriptional interference mechanism that is responsible for inhibiting expression of the coding N

96 nsporters are the best studied component and are responsible for initial high-affinity substrate bind

97 hree-way junction of stem II of the RRE that is responsible for initial Rev binding.

98 of long-range non-electrostatic interaction are responsible for intrafibrillar mineralization.

99 re consistent with a model in which MtMOT1.3 is responsible for introducing molybdate into nodule cel

100 infections to antifungal treatment and could be responsible for its high dissemination rates.

101 tended substrate-binding patch that we posit is responsible for its 3' --> 5' activity.

102 complex as the metabolite of disulfiram that is responsible for its anti-cancer effects, and provide

103 ge to generate reactive RO(*) species, which are responsible for key C-H hydroxylation within the sol

104 regulated in HNSCC-associated CAFs, where it is responsible for key pathogenic contributions in this

105 PLA2g1B is elevated in resistant mice and is responsible for killing tissue-embedded larvae.

106 the anterior-posterior axis of mouse embryos is responsible for left-right symmetry breaking.

107 enase-2-dependent lipid inflammatory pathway is responsible for lethality in F. novicida infection du

108 ding Niemann-Pick C1 (NPC1) protein, and GP2 is responsible for low pH-induced membrane fusion.

109 ta, we hypothesized that reduced Zhx2 levels are responsible for lower Mup expression in BALB/cJ mice

110 state even without orbital orderings and may be responsible for macroscopic spin-glass behavior.

111 role for linker O-glycans, namely that they are responsible for maintaining separation between order

112 Carotenoid pigments are responsible for many of the red, orange, and yellow

113 ine particulate matter (PM2.5) is thought to be responsible for many of these health impacts.

114 found that the ubiquitin/proteasome pathway was responsible for MeCP2 T158M degradation and that pro

115 iochemical analysis showed that DC2 and KCP2 are responsible for mediating the interaction between th

116 Instead, beta2 adrenoceptor activation was responsible for mediating gene silencing of IL-27p28

117 BP 3 and Staphylococcus aureus PBP 2A, which is responsible for methicillin resistance.

118 Polycomb repressive complex 2 (PRC2) is responsible for methylation of histone H3 lysine 27 (

119 Chlamydia is responsible for millions of new infections annually,

120 s, and its use, in the form of ALARA dosing, is responsible for misguided concerns promoting radiopho

121 tained within the 530 bp deletion that could be responsible for modulation of leukotoxin production h

122 isition of infection and may result in males being responsible for more between-group transmission.

123 ant metastases occur in half of patients and are responsible for most cancer-related deaths.

124 mice that inhibition of motility appears to be responsible for most, if not all, of the protection e

125 domain and four PDZ domains, with the latter being responsible for most of Scribble's complex formati

126 Cancer cell metastasis is responsible for most cancer deaths.

127 Metastasis is responsible for most cancer-related deaths, but the c

128 3 (n = 38 [36.2%]) and 19A (n = 30 [28.6%]) were responsible for most PCV13 failures.

129 In the lung, ENaC is responsible for movement of sodium.

130 Site diffusion is responsible for multilayer growth in the linear and i

131 revealed that the 2-methylimidazolate ligand is responsible for multiple redox transformations.

132 vones and total levels of phenolic compounds were responsible for nearly 51% of total data variabilit

133 nsmembrane O-methyltransferase (TOMT/LRTOMT) is responsible for non-syndromic deafness DFNB63.

134 pecific planar-polarised organisation, which is responsible for oriented cell intercalation.

135 This pathogen is responsible for over 95% of seafood-related deaths in

136 filament, known as the presynaptic complex, is responsible for pairing the ssDNA with homologous dou

137 lly, we show that the N-desmethyl metabolite is responsible for PK11195-mediated hCAR activation by f

138 LPS may be responsible for platelet activation and potentially c

139 )3](-) serves as the persistent radical that is responsible for predominant cross-coupling.

140 eses about immunological mechanisms that may be responsible for preventing HIV-1 acquisition.

141 mmunosuppressive activities, and its pathway is responsible for producing a nematode "food signal" in

142 ventricular (RV) impairment is postulated to be responsible for prominent systemic congestion in Chag

143 model and indicate that the same interaction is responsible for promoting integration and for inhibit

144 wever, dysregulation of the TGF-beta pathway is responsible for promoting the progression of several

145 machine composed of RNA and proteins and it is responsible for protein synthesis.

146 However, DENV4 has been responsible for recent large and sustained epidemic

147 identified the region of the SVV capsid that is responsible for receptor recognition using cryoelectr

148 , and short-wavelength sensitive (S)-opsins, are responsible for red, green and blue color recognitio

149 al ligament mesenchymal stem cells (PDLMSCs) are responsible for regeneration of the periodontium tha

150 o the Apicomplexan AP2 (ApiAP2) family, that is responsible for regulating the expression of genes in

151 DDIT4 activity could be responsible for resistance to mTOR inhibitors and is

152 er the course of a week, osteoclast activity was responsible for resorbing the necrotic bone, which i

153 f guinea pig and human sensory nerves, which are responsible for respiratory symptoms.

154 orter (NSS) family of membrane proteins that are responsible for reuptake of neurotransmitters from t

155 Moreover, phosphatase PPP6C is responsible for RIG-I dephosphorylation.

156 Snf2 family protein HARP, mutations of which are responsible for Schimke immuno-osseous dysplasia.

157 e switch between two enhancers: One enhancer is responsible for sequential gene activation, and the o

158 ility control and that the SPOP/CUL3 complex is responsible for SETD2 polyubiquitination both in vivo

159 Variations in the ABCB4 gene are responsible for several biliary diseases, including

160 Genetic mutations in epigenetic regulators are responsible for several neurologic disorders.

161 ) are expressed in human atrial myocytes and are responsible for shaping atrial action potentials.

162 We hypothesized that purinergic signaling is responsible for shear-dependent increases in muscle p

163 ied by enhanced formation of arterioles, may be responsible for shift of Hmox1(-/-) myofiber size dis

164 in of the HpnN family of transporters, which is responsible for shuttling hopanoids to the outer memb

165 karyopherin transportin SR2 (TRN-SR2, TNPO3) is responsible for shuttling specific cargoes such as se

166 Mineral dust aerosols are responsible for some of the largest sources of uncer

167 IL-10 is responsible for STAT3 activation during the first rou

168 ic, biologically labile, "N-peak", which may be responsible for stimulating the observed phytoplankto

169 Plant diseases are responsible for substantial crop losses each year an

170 the g2.07 species, and not the g2.7 species, is responsible for substrate oxidation in the (L)Fe(II)/

171 ng the interesting question of which factors are responsible for such enhanced nested structure.

172 NK cells were responsible for such attenuated responses because d

173 tor-complex in the bacterial flagellar motor is responsible for surface-sensing.

174 le oligomeric Abeta has been hypothesized to be responsible for synapse damage, neurodegeneration, le

175 tion of the inner and outer nuclear membrane are responsible for the additional fluctuation process,

176 othesized that reactive oxygen species (ROS) are responsible for the association between chronic infl

177 nocrystal surfaces and nanocrystal core size are responsible for the crystallization process.

178 Axons traveling within the brachial plexus are responsible for the dexterous control of human arm a

179 marrow (BM) Gr-1(lo) immature myeloid cells are responsible for the elevated, pathological levels of

180 g between FeSe electrons and the STO phonons are responsible for the enhancement of Tc over other FeS

181 FAT family of transcription factors not only are responsible for the expression of many activation-in

182 into the combined graphene oxide sheets and are responsible for the highly conductive nature of thes

183 Capsaicinoids are responsible for the hot taste of chili peppers.

184 s of endogenous SRC-1 and apoA-IV expression are responsible for the impaired E2's anorectic action i

185 ress of circulating monocytes into the joint are responsible for the initial reduction of macrophages

186 Voltage-gated sodium (NaV) channels are responsible for the initiation and conduction of act

187 These features are responsible for the liquid crystalline smectic-like

188 to carbon capture and storage technologies, are responsible for the majority of freight emissions mi

189 Eggs produced by the mature female parasite are responsible for the pathogenesis and transmission of

190 ia Landau damping (during the plasmon decay) are responsible for the photocatalytic property of this

191 ow that the different PBDs of lipins 1 and 3 are responsible for the presence of phosphoregulation on

192 l metabolism components; however, which ones are responsible for the quantitative variation found in

193 The anomalous MS channels are responsible for the reverse-polarity current that ap

194 nt observations showed that while EMIC waves are responsible for the significant loss of ultra-relati

195 the N-terminal domains of these polymerases are responsible for the varying degrees of searching com

196 sm, rather than allosteric modulation, could be responsible for the adverse effects.

197 Decreased peak ICa-L was found to be responsible for the age-associated increase in SR Ca

198 e major antigenic site of HA was reported to be responsible for the antigenic drift.

199 repair (MMR)-proficient tumors and appear to be responsible for the bulk of genomic instability in th

200 a distinct unknown enzyme was postulated to be responsible for the conversion of the glutamic acid t

201 These results suggest that these genes might be responsible for the critical bifurcated hydroxylation

202 ely used animal model of MS, in mice and may be responsible for the decreased incidence of MS in equa

203 of tetrahedral metal ions were suggested to be responsible for the different performance of these tw

204 er, it remains unclear what mechanisms could be responsible for the elicitation of particle and ion r

205 des from two groups of clock cells appear to be responsible for the fly's circadian neurons becoming

206 racene isomer implies that HACA alone cannot be responsible for the formation of PAHs in extreme envi

207 ation within the silica network was found to be responsible for the increase in the elastic modulus o

208 sibly owing to bisretinoid accumulation, may be responsible for the increased complement activation s

209 and DNA binding activities of IE2 appear to be responsible for the increased neural stem cell exit f

210 at local (within field) inoculum sources may be responsible for the initiation of CLS epidemics in mi

211 of compounds present in Stevia, which could be responsible for the nutraceutical effects ascribed to

212 a an intermediate state which is believed to be responsible for the onset of the aggregation process

213 hore deformations that are suggested to also be responsible for the optical modulation of naturally o

214 resence of extensive desmoplasia, thought to be responsible for the poor response of patients to syst

215 oxides particles have recently been shown to be responsible for the protection and burial of a large

216 ation, and a deterioration of this mechanism is responsible for the age-associated memory impairment.

217 His96 is responsible for the binding of two haem groups within

218 Sel1L-Hrd1 ER-associated degradation (ERAD) is responsible for the clearance of misfolded pro-argini

219 CSN5 is responsible for the cleavage of NEDD8 from CRLs, and

220 Diarrhea is responsible for the death of approximately 900,000 ch

221 e show that transforming growth factor-beta1 is responsible for the decreased levels of FGFBP1.

222 water-soluble peripheral part of the complex is responsible for the delivery of electrons to the prot

223 the up-regulation of the ALOX5 gene pathway is responsible for the development of the biochemical an

224 Vibrio cholerae is responsible for the diarrheal disease cholera that in

225 AT-6 family protein EsxL, encoded by Rv1198, is responsible for the down-regulation of CIITA/MHC-II b

226 CrACX2 is a genuine acyl-CoA oxidase, which is responsible for the first step of the peroxisomal fat

227 sequently accelerates contact activation and is responsible for the full procoagulant activity of FXI

228 vacancies by substitutional replacement and is responsible for the generation of the additional SrTi

229 ducts to cultured neurons suggested that C3b is responsible for the growth inhibitory and neurotoxic

230 rein we investigated whether this activation is responsible for the Hcy effect on neurodegeneration o

231 onstrate that the up-regulation of the ALOX5 is responsible for the Hcy-dependent worsening of the AD

232 cooperative binding between guest molecules is responsible for the high uptakes of acetylene and car

233 inhibitor (R)-BEL suggested that iPLA2gamma is responsible for the hydrolysis of oxidized CL and sub

234 We propose that this effect is responsible for the hypercontractile phenotype induce

235 The oceanic biological pump is responsible for the important transfer of CO2-C as PO

236 somal interactions, and centromere tethering is responsible for the inter-chromosomal interactions.

237 rich in basic residues at the C-terminal end is responsible for the interaction with RNA polymerase.

238 Epithelial cadherin (Ecadherin) is responsible for the intercellular cohesion of epithel

239 ting from a single locus, okra (l-D1), which is responsible for the major leaf shapes in cotton.

240 ed to Zip3/RNF212), together with MutSgamma, is responsible for the majority of COs.

241 racellular glutamate in the NAc, while GLT-1 is responsible for the majority of glutamate uptake.

242 in individuals infected with clade C, which is responsible for the majority of HIV infections worldw

243 r of protein and amino acid metabolism as it is responsible for the majority of proteins secreted in

244 he spread of circulating tumor cells (CTCs), is responsible for the majority of the cancer deaths.

245 gangliosidosis, indicating that this enzyme is responsible for the metabolic bypass of beta-hexosami

246 evidence that the silicon based 2D overlayer is responsible for the observed linear dispersed feature

247 on of a more spherical shape after power off is responsible for the observed threshold switching.

248 that the point mutation C64T in the TYR gene is responsible for the OCA1 albino phenotype in the capu

249 conductor and suppresses bulk recombination, is responsible for the outstanding photocatalytic perfor

250 The proposal that reduced reticulon-1 is responsible for the poor inherent ability of macropha

251 We also show that commendamide is responsible for the previously described hemolytic ac

252 Cu(2+) is responsible for the reduction in antioxidants and per

253 cells, the ubiquitin-proteasome system (UPS) is responsible for the regulated degradation of intracel

254 The viral protein E2 is responsible for the replication of the virus.

255 rsity and the magnitude of myocardial injury is responsible for the resolving and nonresolving pathwa

256 y suggest that PKC activation by stolonidiol is responsible for the resulting potentiation of ChAT ac

257 By screening for the hydrolase that is responsible for the reversal of Ser-ADPr, we identifi

258 nsulin across the adipose tissue capillaries is responsible for the slow suppression of free fatty ac

259 The lowest energy pipi* state is responsible for the sub-picosecond lifetime observed

260 epidermal growth factor receptor (EGFRvIII) is responsible for the survival of non-EGFRvIII-expressi

261 as manifested in the classical size effect, is responsible for the thermal conductivity reduction.

262 t as loosely bound aggregates or "flocs" and is responsible for the transport and fate of sediment, c

263 ents suggest that small polaron localization is responsible for the ultrafast trapping of photoexcite

264 It is the existence of such modes which is responsible for the wonderful properties of a topolog

265 The activation of this pathway was responsible for the accumulation of tau oligomers af

266 induction of the cell cycle inhibitor Cdkn1a was responsible for the decreased proliferation in the k

267 sufficient NADPH due to GLS2 underexpression was responsible for the delayed metabolism of 5-ALA and

268 Whereas the activation of GABAA receptors was responsible for the direct inhibition of light-evoke

269 a mixture of cell cycle arrest and apoptosis was responsible for the observed decrease in cell viabil

270 encoding the type I collagen, alpha1 chain, was responsible for the phenotype.

271 re, we demonstrated that the AhR-IDO pathway was responsible for the preferential activation of non-c

272 ight, as we cannot exclude that this lineage was responsible for the production of Acheulean or Middl

273 ate nucleophilic aryl substitution mechanism was responsible for the rapid reaction of the faster sub

274 The Asian lineage of ZIKV was responsible for the recent epidemics in the Americas

275 kinase signaling in Wolffian duct epithelium was responsible for the retention of male structures in

276 Trimeric LHCII was responsible for the slowly activated quenching compo

277 A120G) in the N-terminal region of eIF2Bbeta was responsible for the TuMV resistance.

278 d tissue damage and subsequent IL-33 release were responsible for the activation of hepatic ILC2s tha

279 h respiration and root allocation parameters were responsible for the highest fraction of predictive

280 egulated by myosin light chain kinase (MLCK) were responsible for the intriguing turning behavior of

281 UTR sequences of the lymphotropic HCV strain were responsible for the lymphotropism.

282 These neutrophils were responsible for the pro-metastatic effect of MSCs s

283 ning between particles and colloids: OC + BC were responsible for the sorption of 65% of PBDEs, follo

284 In order to evaluate which compounds are responsible for these activities of five Spanish EVO

285 It is not known which Shank binding partners are responsible for these defects.

286 Sperm-specific SLO3 K(+) channels are responsible for these membrane potential changes cri

287 companying frontal hyperactivation appear to be responsible for these alterations.

288 We identify a key R146A residue that is responsible for these effects, and mutant CrPV(R146A)

289 amination and polyglutamination on TBA-7 may be responsible for this destabilization.

290 We identify the CsrA RNA binding protein as being responsible for this, and show that CsrA different

291 ly suggests that a particular type of enzyme is responsible for this modification.

292 encephalitis virus is the most significant, being responsible for thousands of deaths each year in A

293 Historically it was responsible for three pandemics: the Plague of Justi

294 The mucoadhesive layer is responsible for tight adhesion of the whole system to

295 eport that downregulation of STAT3 signaling is responsible for TLR7-mediated inhibition of Th17 cell

296 The human ubiquitous protein cystinosin is responsible for transporting the disulphide amino aci

297 that each active site of a transposase dimer is responsible for two hydrolysis and one transesterific

298 zed two-component system, named UzcRS, which is responsible for U-dependent activation of PurcA .

299 They are responsible for up to 50% of all N2 released from ma

300 ily of immunoglobulin-related receptors that are responsible for various aspects of T-cell immune reg