ライフサイエンスコーパス: be restored to

1 P(f)(tiss) in AQP5-null mice was restored to 0.0015 cm/s after removal of the epithel

2 Adolescent anatomical plasticity can be restored to 1-year-old mice by conditional deletion o

3 The lytic activity of lysozyme was restored to 73.4+/-1.7%-92.5+/-1.2% during PEG chain

4 Intriguingly, ATPase values were restored to ~73% of wild-type and actin-sliding vel

5 A-I[delta(1-41)] or apoA-I[delta(1-59)], and was restored to 75-80% of the wild-type apoA-I control v

6 This was restored to 86% of the original value (200 U/mg) by

7 The expression of tdaC could be restored to 9 of the remaining 11 Tda(-) mutants-tdaA

8 roximately 50%, whereas the same interaction was restored to 90% of control when the DAX-1 transcript

9 at wild-type levels of coronatine production are restored to a DeltacmaL mutant when it is supplement

10 ACAM1 or form lumena when grown in Matrigel, are restored to a normal morphogenic program when transf

11 Human A3H protein expression could be restored to a normal level either by repairing this t

12 ons were hypersensitive to cocaine and could be restored to a normal locomotor response with GIRK2a e

13 , angiosperm chromosome numbers have usually been restored to a narrow range, as one element in a 'di

14 We found that long-term depression is restored to a normal level through inhibition of PI3K

15 absent in ankyrin-B-null cardiomyocytes and is restored to a normal striated pattern by rescue with

16 ned up or down so that the neuronal activity is restored to a physiological range.

17 iogenesis in isolated Deltapos5 mitochondria is restored to a significant extent by a small amount of

18 tion kinetics of CPY* in a hrd1 Delta strain is restored to a wild-type rate when CPY* is overexpress

19 -500 nm) observed between NAPc2 and bovine X was restored to a high affinity one in a recombinant chi

20 portant for flagellar function, and motility was restored to a LytC mutant by mutation of either lonA

21 V replication in FBP1-knockdown Huh7.5 cells was restored to a normal level by downregulation of eith

22 frequency trains was faster in old mice, but was restored to a normal time course by EGTA-AM treatmen

23 e of photoreceptor-BP connectivity, the ERG, was restored to a normal waveform.

24 Consistent with this, growth was restored to a ymr1Delta sjl2Delta sjl3Delta triple m

25 e a variant nucleotide (G(6016)) in NIa that was restored to A(6016) in eight of nine lineages by pas

26 Physiological levels of cystine (100-300 nm) were restored to acute tissue slices from the nucleus ac

27 Aggregation behavior can be restored to ahr-1-deficient animals by heat-shock ind

28 tingly, crossing over in mei-P22 mutants can be restored to almost 50% of wild-type by X irradiation.

29 Enzymatic activity could be restored to almost parental levels when SOD1 concentr

30 rogram change during ESC differentiation and are restored to an ESC-like state during somatic reprogr

31 at all features of a differentiated cell can be restored to an embryonic level of pluripotency withou

32 Processing efficiency is restored to an extent, but not completely, by the hig

33 s after infection with the P/V-CPI(-) mutant was restored to approximately 50% that of control HeLa c

34 In 86 limbs, straight inline flow was restored to at least one tibial vessel.

35 Monensin titers in the WD2 strain were restored to at least wild-type levels by plasmid-ba

36 Net GNG flux was restored to basal by 4 h, despite a substantial redu

37 When SNA was restored to baseline by blunting the cocaine-induced

38 Ejection fraction was restored to baseline in cell-treated pigs, whereas p

39 dex, and normalized very-low-frequency power were restored to baseline in the hypothermia group.

40 our (p< 0.001) during radiation therapy that were restored to baseline levels at 6 months and 1 year

41 EA, GQOL, and EM were restored to baseline levels during follow-up, where

42 ion, when absolute numbers of CD4(+) T cells were restored to baseline levels.

43 Transplanted liver fragments were restored to confluent tissue with normal hepatic ar

44 ionally, the elevated RyR2 levels in AD mice are restored to control levels with dantrolene treatment

45 ncreased caspases as well as NF-kappaB could be restored to control level.

46 (-/-) cells is dramatically impaired and can be restored to control levels by expression of wild-type

47 sion level in PGI-overexpressing cells could be restored to control levels by treatment with proteaso

48 d metabolic (3.9 +/- 0.3 mm) responses could be restored to control levels during fetal treatment wit

49 cGMP content in GAL4/UAS-PDE5 tubules is restored to control levels by treatment with sildenaf

50 that was observed after 52 h of wakefulness was restored to control levels during a 14-h recovery sl

51 synaptic terminal of neuromuscular junctions was restored to control levels in SMA-PLS3 mice.

52 RL KO mice than in heterozygous controls and was restored to control values by infusion of PRL, sugge

53 BP was restored to control values with pertussis toxin Gi-s

54 antly reduced 10 days following vagotomy and were restored to control levels by 30 days and 60 days,

55 compared with controls), but these variables were restored to control levels by anti-TNF.

56 These effects of maternal allopurinol were restored to control levels during fetal NO blockade

57 lated in the liver of alcohol-fed mice, they were restored to control levels upon ChREBP silencing.

58 during hypoxia (all P < 0.05), effects that were restored to control levels with fetal NO blockade.

59 tuted ribosomes in which purified L7/L12 had been restored to core particles.

60 he chemokine receptor CCR2, wild-type growth was restored to DeltayopM Y. pestis in both organs.

61 Significant activity is restored to E101A and E101A/H96A by adding the lipoph

62 dria in vitro, but full binding and activity was restored to each mutant by disrupting the disulfide

63 Cyclic beta glucan export can be restored to feuP mutant cells by constitutive express

64 substituted with alanine is inactive and can be restored to full activity by substitution of wild-typ

65 y synthesized DNA possessing discontinuities is restored to full size by a "copy choice" type of DNA

66 g Ile77 for Phe77), zolpidem sensitivity can be restored to GABAA receptors in chosen cell types.

67 When UL133/8 was restored to HCMV laboratory strain AD169, which othe

68 FimY production in this mutant could be restored to high levels when fimU was introduced on a

69 ontext was abolished by VTA inactivation but was restored to high control levels after saline microin

70 Acute rejection of cardiac allografts was restored to IgKO recipients by passive transfer of p

71 Protective immunity was restored to immunized C3(-/-) mice by transferring u

72 PS-direct and Zn(2+) were restored to inactive PS under mildly denaturing con

73 Duplication can be restored to inhibited extracts by addition of CaMKII

74 levels of testosterone, evoked GnRH release was restored to intact levels.

75 hus persists with modified kinetics that can be restored to its normally complex waveform by perchlor

76 er, it has not been determined how chromatin is restored to its undamaged state when DSB repair is co

77 on the presence of an L domain when NC-p1-p6 was restored to its C terminus.

78 eficient cells, bipolar spindle assembly can be restored to Kif2b-deficient cells by simultaneous def

79 tablish that early events in T lymphopoiesis are restored to Lck-deficient mice by provision of a tra

80 rienes, whereas arthritis susceptibility can be restored to leukotriene-deficient mice by intravenous

81 that the expression of proinflammatory genes is restored to levels comparable to the acute response t

82 ion transport was rebalanced, and PCL volume was restored to levels adequate for lung defense.

83 eplication of 73.Stop in p53-deficient cells was restored to levels comparable to those of 73.MR.

84 lation in the distal gene 50 promoter, which was restored to levels similar to those of littermate co

85 ansion of LCMV-specific LTalpha(-/-) T cells were restored to levels comparable to those of +/+ T cel

86 , constitutive NOS activity, and cGMP levels were restored to levels found in the control rats.

87 n), Cx43 GJs reformed and intracellular Cx43 were restored to levels observed before treatment.

88 Conventional B cell populations were restored to levels similar to those in lyn(-/-) mic

89 ntegrin-dependent difference in angiogenesis was restored to LLC cells by expression of PLGF, strongl

90 after a mean of 6 years; 5 of these patients were restored to MFS by secondary surgery and 1 spontane

91 protein labeled when the GlcNAc-transferase was restored to mutant extracts, Skp1 apparently mediate

92 CL can be restored to near wild-type levels in stationary phase

93 activity levels in these former cells could be restored to near-normal levels by electroporation wit

94 ling human heart, (2) thin-filament function is restored to near normal levels after LVAD support, an

95 Sam-S) or the histone methyltransferase Set1 is restored to near normal levels when SIN3 is also redu

96 Nucleosome positioning was restored to near wild-type levels with (CTG)(3), whi

97 is necessary for PARP-1-induced MPT, NAD(+) was restored to near-normal levels after PARP-1 activati

98 ome-wide reduction in DNA methylation, which was restored to near-normal levels in regenerated trees.

99 Response to isoproterenol was restored to near-normal levels in the allopurinol gr

100 pression and RAI incorporation, all of which were restored to near basal levels upon discontinuation

101 ypic cultures was reduced and axonal lengths were restored to near normal by coculturing in the prese

102 Total myocardial NOS3 protein and activity were restored to near wild-type (WT) levels in NOS3(-/-)

103 cytes, as well as their excessive apoptosis, were restored to near-normal levels in the absence of ST

104 261 NMBA-dysregulated genes in rat esophagus were restored to near-normal levels of expression by BRB

105 lified at 50% phosphorylation of E1b, but it is restored to nearly half of the pre-phosphorylation le

106 Furthermore, the wall shear stress was restored to nearly homeostatic levels throughout mos

107 this mutant, the level of vir gene induction was restored to nearly wild-type level.

108 xidative stress conditions, and the activity was restored to nearly wild-type levels by adding Msr pl

109 Protein A production was restored to nearly wild-type levels by complementati

110 sphorylation, and E2F target gene expression were restored to nearly normal levels, rendering cells r

111 d with wild-type or vhs rescue viruses, they were restored to nearly wild-type levels of replication

112 The process by which the proper pattern is restored to newly formed tissues during metazoan rege

113 ated MYPT1 in telokin-null ileum homogenates was restored to nonphosphorylated MYPT1 levels by additi

114 Elevated levels of CGRP during migraine are restored to normal coincident with headache relief a

115 nd 8 pair, and that the levels of both pairs are restored to normal in var1 plants that overexpress A

116 Hematologic values are restored to normal levels and organ pathology is ame

117 in both wild-type and IGF-1 transgenic mice, are restored to normal levels at a faster rate in IGF-1

118 yocardial markers altered in Id mutant cells are restored to normal throughout the chimeric myocardiu

119 disease as an isolated abnormality that can be restored to normal form and function through medical

120 Of the five impaired alleles, four could be restored to normal functionality by elevating intrace

121 A manometrically defective LES can be restored to normal sphincter, whereas a normal LES re

122 ne myocytes remained evident, but this could be restored to normal upon direct application of poloxam

123 Sleep duration in qvr/sss-null mutants is restored to normal by a qvr/sss transgene that fully

124 dia mice, there is altered progression which is restored to normal by transferrin treatment which was

125 n is down-regulated in Tbx1(+/-) embryos and is restored to normal levels in Tbx1(+/-);Trp53(+/-) emb

126 creas development and islet differentiation, is restored to normal levels, and islet beta-cell prolif

127 In addition, when protein synthesis is restored to normal levels, Myc-overexpressing precanc

128 and defined by odorant receptor expression, is restored to normal or nearly so by 3 months after les

129 ormation was defective in C1qa(-/-) mice and was restored to normal after local application of C1q.

130 ardiomyocyte proliferation in mutant embryos was restored to normal at E14.5, concurrent with the est

131 ression in a mouse model of infection, which was restored to normal by an ectopic copy of rel(+) (Spn

132 OR, p-Raptor, and p-S6RP was observed, which was restored to normal by elevating ERK1/2 activity in t

133 e and cytotoxic T lymphocyte to Ld after DST was restored to normal by IL-2.

134 Inhibited cell motility as in i, iii, and iv was restored to normal level by addition of mAb 8E11, wh

135 culture, but the defect in Foxp3 expression was restored to normal levels after 60-72 hr.

136 had low baseline telomerase activity, which was restored to normal levels by exposure to androgens.

137 When myosin light-chain phosphorylation was restored to normal levels by phosphatase knockdown,

138 icit dramatically reduced endocytosis, which was restored to normal levels by PLS3 overexpression.

139 Cytokine production, however, was restored to normal levels by restimulation with phor

140 ion of the high affinity IgE receptor, which was restored to normal levels by the addition of soluble

141 The salt tolerance of sos2 was restored to normal levels by wild-type SOS2, but not

142 Maternal haematocrit was restored to normal levels only in animals given supp

143 y decreased in group 2 whereas in group 1 it was restored to normal levels.

144 ed ribosomes were eliminated and translation was restored to normal levels.

145 induced permeability transition pore opening was restored to normal levels.

146 ons of albumin, in which the ionized calcium was restored to normal plasma levels, had no significant

147 The sinI mutant was restored to normal swarming by 5 nM C(16:1)-HSL.

148 rregulatory response in STZ-diabetic animals was restored to normal with either local blockade of GAB

149 HRG-deficient mice, and accelerated clotting was restored to normal with HRG reconstitution.

150 Moreover, motor function was restored to normal within 1 month post-MPTP in BMT-t

151 that the recovery of the cone-driven a-wave was restored to normal, whereas recovery of the cone-dri

152 r ablation, although the activation sequence was restored to normal.

153 ense, but not by sense, ODN infusions, which were restored to normal after BDNF coinfusions.

154 responses to acetylcholine in basilar artery were restored to normal after treatment with a scavenger

155 en blood glucose levels of the diabetic rats were restored to normal by insulin treatments, the AQP9

156 rther increased in 2- and 4-week MR dogs but were restored to normal in 4-week MR+beta1-RB dogs.

157 potential, and electron transport activities were restored to normal in the cybrid cells.

158 onduction velocity), disrupted by IR injury, were restored to normal level and the induction of ventr

159 ree proteolytic activities of the proteasome were restored to normal levels 7 days post-partum.

160 of B cells in Mdm2+/-Emu-myc transgenic mice were restored to normal levels in ARF+/-Mdm2+/-Emu-myc t

161 ng entropies and oscillations in both nuclei were restored to normal levels in the large-graft group.

162 cpcBA) promoter, PS I content and activities were restored to normal levels, and cells again produced

163 n mice heterozygous for IL-7 expression, but were restored to normal numbers in mice also lacking Bim

164 tive of the siRNA or an irrelevant siRNA and were restored to normal when a human codon-optimized der

165 cells, low levels of messenger RNA for CIC-1 were restored to normal.

166 ontrol values 24 h after the DO in the tanks was restored to normoxic levels.

167 NOR inducibility was restored to NorR null mutants by expressing NorR in

168 e-deficient mice in which dopamine signaling was restored to only the medial striatum by viral rescue

169 ntly higher energy output as soon as retinol was restored to physiological concentration, without the

170 eover, we find that end-joining activity can be restored to PNK-depleted extracts by addition of huma

171 serum urea nitrogen by -72.8%; these indices were restored to precastration levels by DHT.

172 All parameters were restored to precooling levels on rewarming.

173 co-accumbens synaptic and glial transmission were restored to predrug parameters for at least 2 wk af

174 cortisol levels initially increase and then are restored to prestress levels within several days of

175 nd discuss how these protective brakes might be restored to prevent absence seizures.

176 Phosphatase activity must subsequently be restored to promote mitotic exit.

177 reased plasma estradiol levels following T-H were restored to sham levels in the flutamide-treated T-

178 eral blood lymphocytes (PBL) to expand could be restored to some extent by coculture of these cells w

179 Dopamine production can be restored to specific brain regions by using adeno-ass

180 YF-); a mutant cell line, +Src, in which Src was restored to SYF- cells; and the mutant cell line (CS

181 In vitro reactivity can be restored to T cells from patients with suppressed HBV

182 ission phenotype of the vaccine strain could be restored to that of the St.

183 ing capacity of a nonpolar altA mutant could be restored to that of the wild-type strain by adding pu

184 egenerative performance of adult neurons can be restored to that of young neurons by gene transfer-me

185 ith the pcDNA vector, the GM3 synthase level is restored to that of control cells, and the ability of

186 Long term after surgery, outcome is restored to that of similar MI without PMR.

187 synthesis is reduced in most COX mutants but is restored to that of wild type by the same mss51 mutat

188 artial hepatectomy, hepatocyte proliferation is restored to that seen in WT animals.

189 The level of entry in both hosts was restored to that in strain JR32 by plasmid copies of

190 o the mtaR mutant, its growth rate in plasma was restored to that of the wild-type strain.

191 hich point food intake and glucose tolerance were restored to that of WT mice.

192 intermediate in RNA proofreading and repair, are restored to the forward position by the activity of

193 functional properties of the wounded tissue are restored to the levels before injury.

194 herapy is optimal when C1INH activity levels are restored to the normal range.

195 Tolerance could be restored to the CD1(-/-) hosts by infusing enriched T

196 d FDM biosynthesis, and FDM production could be restored to the fdmR1::aac(3)IV mutant by expressing

197 media or in Arabidopsis pop2-1 leaves could be restored to the gabT triple mutant by expression in t

198 D(-/-) mice to C. neoformans infection could be restored to the level of WT mice by increasing IL-5 a

199 rotein, as normal cell cycle progression can be restored to the mutant by expressing a mutant form of

200 t insights into how appropriate function can be restored to the nervous system after the critical per

201 and AT(1A)Rs with arginine substitutions can be restored to the plasma membrane by either using selec

202 e supports the notion that accommodation can be restored to the presbyopic eye, progress in this pote

203 Consequently, MHC class I molecules can be restored to the surface of DFTD cells in vitro by usi

204 artially stimulated by hMutSalpha but cannot be restored to the wild-type level.

205 of a derivative set of strains in which tetL is restored to the chromosome support earlier indication

206 Cyanide production is restored to the FRD1aceA mutant by addition of glyoxy

207 herapeutically imposed, wherein blood supply is restored to the previously ischaemic tissue.

208 Light sensitivity is restored to the resulting apo-opsin when it recombine

209 injury observed in RAG-1 knockout (KO) mice is restored to the wild-type (WT) level by adoptive tran

210 reduced in the ctbp-1 deletion mutant, which is restored to the wild-type level by RNAi inhibition of

211 Activity is restored to the wild-type level for Ycf1-S251E.

212 Expression of p21(Cip1) mRNA was restored to the control level in all the fusion cell

213 a-coated microtiter assay, biofilm formation was restored to the DeltasigB mutant through the additio

214 Cyanide production was restored to the FRD1dadA mutant by the addition of g

215 elastase removed 68% of MUC16, 78% of which was restored to the HCLE cell surface 24 hours after rel

216 protein level in PAX3-FKHR-expressing cells was restored to the level of control cells by treatment

217 h cognitive flexibility in abstinent smokers was restored to the level of nonsmokers following stimul

218 l markedly declined with age (P = 0.003) but was restored to the level seen in young rats when ALCAR+

219 Remarkably, virtually normal growth was restored to the mreB null mutant in the presence of

220 The algXDelta::Gm mutant was restored to the mucoid phenotype with wild-type P. a

221 ne in either orientation and origin activity was restored to the mutant c-myc replicator.

222 activating these proteases, biofilm capacity was restored to the mutant, demonstrating they are respo

223 ence of the Gq-selective inhibitor YM-254890 was restored to the normal extent achieved by PAR agonis

224 urprisingly, wild-type-like TTSS functioning was restored to the pnp mutant strain by expressing just

225 ectively reestablished when GPR88 expression was restored to the striatum.

226 The enzyme activity was restored to the wild-type levels when the mutant p66

227 The LOS structure of the kdtA::aphA-3 mutant was restored to the wild-type structure by complementati

228 , fruiting body formation and EPS production were restored to the levels observed in mutant strains l

229 rete CNF1 and invade HBMEC of the fdx mutant were restored to the levels of the parent strain by comp

230 In all cases, the double mutant phenotypes were restored to the luxS+ phenotype by the addition of

231 xception of dystrophin, other DGC components were restored to the sarcolemma including alpha-sarcogly

232 ent from IL-7(-/-) mice, TCRgammadelta cells were restored to the thymus and periphery by expression

233 but not ceramides and complex sphingolipids, were restored to the wild-type levels in the Cers2-rescu

234 s these ratchet-like epistatic substitutions are restored to their ancestral states, reversing the ke

235 d, the mechanisms by which its targets genes are restored to their preactivation state are less clear

236 6-fold; P < 0.01) that resolved when choline was restored to their diets.

237 Moreover, fertility was restored to these promoter IR-containing plants by e

238 ent was immediately reversed when blood flow was restored to these regions, indicating that parts of

239 However, functionality could be restored to this +1 leucine mutant by either insertin

240 embly and extracellular release of virus can be restored to this mutant with the addition of an N-ter

241 evels of GM3 synthase and corresponding mRNA are restored to those of control cells.

242 significantly enhanced ECCE rates that could be restored to those measured in wild-type cells after e

243 fibrosis observed in Kit(W-sh/W-sh) mice can be restored to those observed in WT mice after the adopt

244 heximide (CHX) or emetine, expression levels were restored to those observed in primary and immortal

245 ity to concentrate and organify iodide could be restored to TSHR-KO thyroids when cultured in the pre

246 isingly efficient class switch recombination is restored to ung(-/-) B cells through retroviral deliv

247 blasts, and also expression of MRPP1 protein was restored to values comparable to controls.

248 Molting after the normal first instar period was restored to various degrees by feeding the mutants t

249 e elevated salicylic acid (SA) levels, which are restored to wild-type levels by expressing nahG, bac

250 ity, and replication, and these deficiencies are restored to wild-type levels with the introduction o

251 in by MgCl2 was inhibited strongly but could be restored to wild type actin levels by phalloidin and

252 ns in the N-terminal half of the toxin could be restored to wild type secretion levels if cultured in

253 The mutant phenotype can be restored to wild-type by the intact AtNAP, as well as

254 s diminished intramacrophage growth that can be restored to wild-type F. tularensis LVS levels by eit

255 We show that virions with nef deleted can be restored to wild-type infectivity by stimulating intr

256 usly shown that virions with nef deleted can be restored to wild-type infectivity by treatment to ind

257 bidopsis ipmdh1 T-DNA knock-out mutant could be restored to wild-type levels by constructs expressing

258 oliferation of the IMP1-null fibroblasts can be restored to wild-type levels by IGF2 in vitro or by r

259 reductase activity in the PrrA strain could be restored to wild-type levels by using nirK expressed

260 inished Env expression in cells, which could be restored to wild-type levels either by mutating the Y

261 a mir163 null mutant, but the repression can be restored to wild-type levels in complementation lines

262 Furthermore, transduction efficiency can be restored to wild-type levels in scid cells that are c

263 and attachment of a gcbA mutant strain could be restored to wild-type levels via overexpression of th

264 riable in a CpsA-dependent manner, but could be restored to wild-type levels when grown with lysozyme

265 d that sebaceous gland size and function can be restored to wild-type levels.

266 esults in a buildup of internal GroPCho that is restored to wild type levels by reintegration of GDE1

267 al development and iron content in Pcm brain is restored to wild-type levels by 7 weeks of age.

268 Growth of the triple mutant in culture is restored to wild-type levels by supplementation with

269 e in medium that contains trace zinc; growth is restored to wild-type levels by supplementing medium

270 CLN1, reduced nearly threefold in paf1Delta, is restored to wild-type levels in the rtf1Delta paf1Del

271 with 5-azacytidine, root growth of epi-lines is restored to wild-type levels, implicating hypermethyl

272 dified to contain an amber stop codon, which was restored to wild type by G to T transversion induced

273 ocyte recruitment and C1q-hemolytic activity was restored to wild type levels when CD93 was expressed

274 n grown on 1/2 MS medium lacking sugars, but was restored to wild-type (WT) levels by supplementation

275 Nucleosome density was restored to wild-type level by re-expressing wild-ty

276 4, dynein activity in pf18 and pf15 axonemes was restored to wild-type level.

277 es and primary macrophages; virus production was restored to wild-type levels after reintroduction of

278 dulin knockdown neurons, presynaptic release was restored to wild-type levels by expression of consti

279 lfate or glycosaminoglycan synthesis, uptake was restored to wild-type levels by incubating target ce

280 oduced a phenotype of increased sleep, which was restored to wild-type levels by pharmacological trea

281 did not show any defect in vivo Colonization was restored to wild-type levels in a luxO opaR double m

282 The severity of joint swelling was restored to wild-type levels in mice infected with a

283 creased in the SM-B null mesenteric vessels, was restored to wild-type levels in the double knockout

284 lies fed food without doxycycline; virulence was restored to wild-type levels when the strain was inj

285 Paraquat resistance was restored to wild-type levels, but zinc was not.

286 By P21, MBP expression was restored to wild-type levels, demonstrating that the

287 ith a wild-type copy of the asd gene, growth was restored to wild-type levels.

288 ed using in vivo magnetic resonance imaging, was restored to wild-type levels.

289 noclonal antibodies, however, granuloma size was restored to wild-type levels; this finding revealed

290 ony morphology, and serum sensitivity, which were restored to wild type by trans-complementation with

291 cid or to air exposure, and these phenotypes were restored to wild-type (WT) attributes upon compleme

292 ally, tRNA maturation and 9S rRNA processing were restored to wild-type levels in each of the three s

293 ermates, but memory and synaptophysin levels were restored to wild-type levels in Tg19959-MnSOD litte

294 exhibited by the mutants were reversed; both were restored to wild-type levels.

295 stunted growth phenotype, and leaf ABA level were restored to wild-type values, pointing to the redun

296 In contrast, lumbar BMD was restored to within 5% of pretransplant levels in the

297 vated [ATP] by 35%; both [ATP] and [5-InsP7] were restored to WT levels by overexpression of PPIP5K1,

298 TP were reduced in CD36-deficient hearts and were restored to WT levels by rescue of myocyte CD36.

299 cardiac infiltration of leukocytes after MI were restored to WT levels via lentiviral-mediated re-ex

300 is demonstrated that YopP and YspH secretion was restored to yopP mutants by complementation in trans