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1 he axon, and assembly of the node of Ranvier is significantly delayed.
2 due to the duration of production, treatment is significantly delayed.
3 tion of immune cells into parenchymal tissue is significantly delayed.
4 arly aggregation, but subsequent development is significantly delayed.
5 SWI/SNF, the initiation of DNA end resection is significantly delayed.
6 segregate chromosomes and (2) anaphase onset is significantly delayed.
7 plasts after MAPK activation, and cell death is significantly delayed.
8 ce in two T-DNA insertion lines of this gene is significantly delayed.
9 their response to a T-cell-dependent antigen was significantly delayed.
10 propria was markedly decreased and rejection was significantly delayed.
11 Relative skeletal maturation was significantly delayed.
12 veloped mammary tumors; however, tumor onset was significantly delayed.
13 In contrast, rejection of fetal skin grafts was significantly delayed.
14 the elimination of MPP(+) from the striatum was significantly delayed.
15 n a functional channel whose desensitization was significantly delayed.
16 filtration in the chemical peritonitis model was significantly delayed.
17 tual clearance of listeriae from both organs was significantly delayed.
18 n of MDSCs and fibrocytes, and wound healing was significantly delayed.
19 d signs of acute GVHD, but the time of onset was significantly delayed.
20 nce of injected 125I-labeled SAA from plasma was significantly delayed.
21 ased nodule formation, but nodule maturation was significantly delayed.
22 as dramatically increased and suture closure was significantly delayed.
23 but the rejection of subcutaneous allografts was significantly delayed.
24 chromosome alignment at the metaphase plate was significantly delayed.
25 plasia, although the appearance of dysplasia was significantly delayed.
26 cellular immunities, and their tumor growth was significantly delayed.
27 ased between 50 and 90%, and time to maximum was significantly delayed.
28 cells; however, the resolution of these foci was significantly delayed.
29 at 24 and 48 hr (3.3 +/- 0.31, 5.0 +/- 0.26) were significantly delayed.
30 observed than in young mice, whose responses were significantly delayed.
31 n of CB6F1 small-bowel allografts, rejection was significantly delayed (29.2 +/- 8.7 days vs. 16.5 +/
32 the catabolism of (125)I-apoB-VLDL and -LDL were significantly delayed, accounting for the 1.35-fold
33 ated by task-driven neural correlations that are significantly delayed across secondary somatosensory
34 on of lineage-specific transcription factors are significantly delayed after induction of Notch targe
35 virus from the lungs of infected BALB/c mice was significantly delayed after the transfer of virus-sp
36 length CFTR from the ribosome and translocon was significantly delayed after translation was complete
39 osO(2); and (ii) the peak expression of CD69 is significantly delayed and more sustained at physO(2)
43 Jkappa), as a RBP-Jkappa-deficient cell line was significantly delayed and inefficient in LANA transc
45 prid treatment, the time to larval emergence was significantly delayed and no pupae or adult workers
48 as phosphatidylserine externalization, which was significantly delayed and propagated more slowly in
49 however, the response in TLR2-deficient mice was significantly delayed and reduced, although not comp
51 ser extent RecQ, the recovery of replication is significantly delayed, and both the recovery and cell
52 In tPA-deficient mice, seizure progression is significantly delayed, and neuroserpin treatment does
53 red to that in chronically infected B6 mice, was significantly delayed, and this was associated with
54 t a distant site, the day of tumor emergence was significantly delayed, and tumor growth was reduced
55 ion, resistance to pyramided two-gene plants was significantly delayed as compared with resistance to
56 ous tumor nodules with reduced 6-O-sulfation is significantly delayed at the initial stages of tumor
57 Myelination of the nerve in the optic canal was significantly delayed at P15, and myelin was almost
58 in an allogeneic BMT model, GVHD development was significantly delayed behind recipients of WT CD4(+)
60 P30 op/op mice showed that nerve conduction was significantly delayed but not blocked with partial r
61 , and subsequent induction of neuronal death is significantly delayed by expressing a full-length Bak
66 anylylimidodiphosphate-mediated facilitation was significantly delayed by overexpression of different
69 We found that tumor formation driven by MT was significantly delayed by the loss of p110alpha expre
71 et of viremia for delta8-DR-infected animals was significantly delayed (by 2 to 5 days), and mean vir
72 esponse to TPZ treatment alone, tumor growth was significantly delayed, by up to approximately four d
73 quently, the temporal recovery of MZ B cells was significantly delayed compared to peripheral follicu
74 air of DSBs in cells lacking full-length NBN was significantly delayed compared with control cells, w
78 a corneal epithelial injury in Lum(-/-) mice was significantly delayed compared with Lum(+/-) mice.
79 tive transfer with NOD-Ncf1(m1J) splenocytes was significantly delayed compared with NOD splenocytes,
80 occludin in Madin-Darby canine kidney cells was significantly delayed compared with that of wild typ
81 reas in atopic children Treg cell maturation was significantly delayed compared with that seen in age
82 covery of the somatosensory-evoked potential was significantly delayed compared with the ChR2-mediate
83 of irradiated SCCNij202 and SCCNij167 tumors was significantly delayed, compared with controls (P < 0
84 etics of CXCL9 and CCL5 in CD4 KO recipients was significantly delayed, coupled with similarly delaye
85 FP-betagal mutant; however, its trafficking is significantly delayed during infection by the 480-1 m
87 however, both early and late gene expression are significantly delayed, even if the inhibitor is remo
88 In contrast, we found that polymerization was significantly delayed for both Ala variants and was
89 phase timing (simulated peak) of 24-hour IOP was significantly delayed for the older subjects in both
91 ed growth inhibition and gravitropic bending are significantly delayed in cngc14 compared to wild-typ
92 y without selection on chemotherapeutic drug are significantly delayed in the onset of apoptosis as d
93 the timing and mechanisms of this repression are significantly delayed in the wasp compared with the
94 strate for the first time that rejection can be significantly delayed in a large-animal composite tis
95 ids were tracked over pregnancy and found to be significantly delayed in Cox-1 null mice at term.
96 was monitored up to 5 days and was found to be significantly delayed in UCP2(-/-) mice after partial
98 fischeri waaL mutant has a motility defect, is significantly delayed in colonization, and is unable
99 ndent apoptotic response to genotoxic stress is significantly delayed in Dmp53 (Drosophila p53) mutan
100 teral fibers in the retinocollicular pathway is significantly delayed in gene knockout mice in which
102 A V. fischeri mutant unable to produce PepN is significantly delayed in its ability to colonize squi
103 own that the onset of neurologic progression is significantly delayed in patients with brain metastas
105 terized by great potential to make IFN-gamma is significantly delayed in the absence of B7:CD28 costi
107 e to the wild-type control mice, the process is significantly delayed in the EC-Nur77-DN mice, in whi
109 e, the time to complete thrombotic occlusion is significantly delayed in the ob/ob mice, and the thro
110 covalent inhibitory complex ACT-chymotrypsin is significantly delayed in the presence of SANT2 with n
112 we demonstrate that compact myelin formation is significantly delayed in TIMP-1 KO mice, a situation
113 The onset of post-transplant proteinuria was significantly delayed in a B cell-independent manner
115 version and neutralizing antibody production was significantly delayed in animals showing complete ti
116 Time to corneal re-epithelialization in vivo was significantly delayed in AQP3-null mice compared to
117 ention model, the growth rate of CEA+ tumors was significantly delayed in CEA-Tg mice orally immunize
118 resence Cr6+, whereas the onset of apoptosis was significantly delayed in cells overexpressing MTF1.
119 limited to parenchymal cells, but resolution was significantly delayed in chimeras deficient in IFN-g
120 aging revealed that transit of fecal pellets was significantly delayed in colons from P2ry1(-/-) mice
121 The healing of stabilized tibia fractures was significantly delayed in COX-2(-/-) mice compared wi
122 ith HCM and control patients, but untwisting was significantly delayed in HCM patients and longest in
128 ee joints after intra-articular Ag injection was significantly delayed in L-selectin-deficient and do
129 eir development into insulin-producing cells was significantly delayed in male compared with female r
133 f Staphylococcus aureus-infected skin wounds was significantly delayed in miR-142(-/-) mice compared
134 lids (28% +/- 27%) and liquids (22% +/- 18%) was significantly delayed in nNOS-deficient mice compare
143 ol-treated subjects, time to first PDR event was significantly delayed in subjects treated with FAc (
145 emigration of CD4+ cells into the allograft was significantly delayed in the CDR-OKT4A/hIgG1-treated
146 ks of age, atherosclerotic lesion initiation was significantly delayed in the double-knockout mice.
147 Moreover, her-2-induced mammary tumor onset was significantly delayed in the GnT-V null tumors, evid
151 aling that the clearance of the formulations was significantly delayed in the presence of CS-NAC and
152 rom baseline to a peak and to a steady state was significantly delayed in the two lower, but not the
156 reover, LH-induced oocyte meiotic resumption was significantly delayed in vivo, and this delayed resu
157 tein degradation and endosomal acidification were significantly delayed in gammadelta T-APCs compared
158 and several of the single-crossover strains were significantly delayed in the ability to initiate no
159 me time, no corresponding stimulus locations were significantly delayed in the diabetic females compa
160 n blood glucose level and islet inflammation were significantly delayed in these HR-deficient relativ
165 of onset for T-ALL in the SCL/LMO1/scid mice was significantly delayed (P < 0.001) compared with SCL/
166 ecystokinin response after the n-3 PUFA meal was significantly delayed (P < 0.001), and the glucagon-
169 se of the blood clearance of 99mTc-HYNIC-IgG was significantly delayed (p < 0.01) compared with 111In
171 cit times for each group, the diabetic males were significantly delayed (P < 0.025) at 23 correspondi
172 in Kc tissue culture cells, their expression is significantly delayed relative to that seen in the an
173 ruses, clearance of scSIV(mac)155T3 TM(stop) was significantly delayed relative to the other strains,
174 ion of the palatal processes of the palatine was significantly delayed, resulting in submucous cleft
175 verely reduced and outer membrane disruption is significantly delayed, suggesting that the spanin pat
178 DNA damage G(2) checkpoint, as this process was significantly delayed upon centrosomal tethering of
179 of the optic nerve lesion site by astrocytes was significantly delayed upon microglia depletion, spon
180 I:C <1%) treated with 25 pmol/L TF, clotting was significantly delayed versus normal, whereas replace
181 vo that Alzheimer's disease (AD) progression is significantly delayed when insulin-like growth factor
183 Plm inactivation by alpha(2)-antiplasmin was significantly delayed when Plm was preincubated with
184 l activation of MyoD transcriptional targets was significantly delayed, whereas targets of the Rb1/E2
187 (-/-) mice showed RV breakthrough times that were significantly delayed with respect to QRS complex o
188 Sporulation in strains harboring pASspo was significantly delayed, with sporulating cells exhibi
189 ut not TEM, CD8+ cells to inflammatory sites was significantly delayed without MCP-1, whereas both su
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