ライフサイエンスコーパス: be turned off

1 0% (during the first min after the treadmill was turned off).

2 ies from solution and the conjugate addition is turned OFF.

3 lodes' as a travelling wave when the tweezer is turned off.

4 rospray emitters when the ionization voltage is turned off.

5 the upstream DNA damage checkpoint signaling is turned off.

6 t permanently in G2 phase when Ku expression is turned off.

7 h apart the spindle poles when poleward flux is turned off.

8 nt mechanism that engages when poleward flux is turned off.

9 prevents expansion of slp stripes after Eve is turned off.

10 rotein is expressed and the wild-type Calpha is turned off.

11 his process is reversed, and KLF2 expression is turned off.

12 osure to light that persists after the light is turned off.

13 he magnetic relaxation signal when the field is turned off.

14 magnetization, which relaxes when the field is turned off.

15 the future active X and persists until Xist is turned off.

16 ion when DNA-templated primary transcription was turned off.

17 d in 13 of 14 APs after radiofrequency power was turned off.

18 the expression of the Nrg1-ntfbeta transgene was turned off.

19 nents that was reversed after the high light was turned off.

20 ed moving through the x-ray beam or the beam was turned off.

21 d in the dark cycle), 1900 (before the light was turned off), 2200 (3 hours into the dark cycle), and

22 n and mRNA levels is transient and begins to be turned off 3-6 h after growth stimulation of cultured

23 When the electrostatic protein-water forces are turned off, a dewetting transition occurs in the int

24 If as few as three inappropriate genes are turned off, a normal cell can be converted into a ca

25 In contrast, how transcription is turned off after activation is unclear.

26 ion occurs only in subconfluent cultures and is turned-off after a polarized epithelium has formed.

27 ylation; the silencing persisted after p15AS was turned off, although methylation and heterochromatin

28 When STN DBS was turned off, an initial fast but partial loss of bene

29 the amphiphilicity of the copolymer that can be turned off and on by varying the polarity of the solv

30 all kinetochores come under tension, the SAC is turned off and APC/C(Cdc20) degrades cyclin B and sec

31 idic supramolecular receptors for fullerenes is turned off and on by changing the solvent, whereas th

32 the alphaHL channel, the DC driving voltage is turned off and the conductance of the channel is moni

33 However, the mechanism of how the pEMT phase is turned off and the redifferentiation phase is initiat

34 Then, the metronome is turned off and the subject continues at the establish

35 mpairment was ameliorated when the transgene was turned off and D2R levels were normalized.

36 rovement was maintained when the stimulation was turned off and was accompanied by a higher number of

37 ields of individual neurons, then the lights were turned off and the object was silently removed.

38 sis, when chromatin condenses, transcription is turned off, and almost all double-stranded DNA-bindin

39 roms, can be stable for hours after the beam is turned off, and ions that screen charges on the filam

40 rsed spontaneously when calcineurin activity was turned off, and expression of fetal genes reverted t

41 matrix and cornea-transparency-related genes were turned off, and a repertoire of macrophage genes we

42 eas those associated with body column tissue are turned off as the tentacle zone expands.

43 When attractive van der Waals forces are turned off as well, the dewetting in the interdomain

44 in undifferentiated embryonic stem cells but was turned off as these cells differentiate in vitro and

45 Furthermore, when P3H2 expression was turned off, as seen naturally in cultured SAOS-2 ost

46 driven by the distal enhancer can no longer be turned off at late stages in lin-1 and lin-31 mutants

47 is specifically seen in progenitor cells and is turned off at the onset of neuronal differentiation.

48 etermining gene Sox9 in mutant Sertoli cells was turned off at embryonic day 14.5 after Wt1 ablation,

49 When transgenic human RTN3 expression was turned off at young age, formation of RIDNs was esse

50 rned on at 00:00 CT and increased after they were turned off at 12:00 CT.

51 it was found that reserpine oxidation could be "turned off" at flow rates as low as 2.5 microL/min a

52 RIDNs was dependent on whether the transgene was turned off before or after the formation of RTN3 agg

53 early stages of differentiation (when Vsx-2 is turned off), before decreasing to an intermediate lev

54 This sheath or coaxial fluid was turned off between analysis of cells and on during c

55 remained stable even when the electric field was turned off but readily redissolved when counterions

56 Galpha subunits are turned off by GTP hydrolysis.

57 ic system and allows the dynamic exchange to be turned off by addition of 18-crown-6, which binds the

58 pose that the Cyp6a8 gene in 91-C strain may be turned off by an active repressor which might be inhi

59 The surge can be turned off by fixing the rate of fluid flow through t

60 Moreover, gene expression could be turned off by removal of the inducing stimulus.

61 ransporters require PIP2 to function and can be turned off by signaling pathways that deplete PIP2.

62 n healthy cells, the SR-BI apoptotic pathway is turned off by eNOS and HDL which prevents inappropria

63 hanism by which NF-kappaB-mediated signaling is turned off by its removal from the DNA in the presenc

64 Rh* is turned off by phosphorylation by rhodopsin kinase [G-

65 synthesis in both asexual and sexual spores is turned off by the NF-kB like fungal regulators VosA a

66 o distinct points in the cycle, one of which is turned off by the presence of P(i).

67 When hetN expression was turned off by transferring cells to media lacking co

68 h in PDL pancreas were impaired when ERalpha was turned off chemically or genetically (ERalpha(-/-)),

69 presence of doxycycline, and the gene could be turned off completely by removal of doxycycline.

70 Transgene expression in these mice can be turned off completely with low doses of doxycycline (

71 When the current is turned off during phases of the domain wall motion wh

72 a naive host, the activity of the M promoter is turned off early, preventing excessive synthesis of m

73 After the electric field is turned off, electrical current is measured and observ

74 Accordingly, miR-150 repression was turned off following treatment with the DNMT inhibit

75 ve from this "memory" after the illumination is turned off for an extended period of time.

76 clinical status when deep brain stimulation was turned off for 2 days, suggesting that deep brain st

77 When the smoke evacuator was turned off for 30 seconds, there was a more than 26-

78 When the transgene was turned off from conception to 3 months of age, no ph

79 tablished in the host, these virulence genes are turned off, fully attenuating the vaccine strain.

80 When spikes were turned off GOM recoveries returned to 60 +/- 19% of

81 scribed, but the pathway whereby the levator is turned off have not.

82 When the stimulus was turned off, his body partially tilted back at an int

83 Our findings indicate that the RASSF1A gene is turned off in a significant number of melanomas and t

84 In adults, Brg1 (also known as Smarca4) is turned off in cardiomyocytes.

85 ormation: miR-211 is a molecular switch that is turned off in melanoma cells, raising the hope that i

86 e demonstrate that (independent of EBV) TET2 is turned off in normal and malignant germinal center (G

87 downstream target of the GTPase Rheb, which is turned off in response to AMPK via the tuberous scler

88 Optx2 expression is turned off in the future pigment epithelium but remai

89 e expression of episome maintenance proteins is turned off in the predominant long-term latency reser

90 at of 738-lob2A1::Km, suggesting that lob-2A was turned off in this phase.

91 phloem fibers undergoing tension stress, but was turned off in tissues undergoing compression on the

92 of MCC that takes place when the checkpoint is turned off involves the conversion of C-Mad2 back to

93 eversed, if expression of the mutant protein is turned off, is unknown.

94 the LTi developmental pathway, but needs to be turned off later to avoid diversion to the T cell fat

95 from a regulated galactose-induced promoter was turned off, leading to recapitulation of the iron re

96 eory in which all interspecific interactions are turned off leads to analytical results that are in a

97 When the checkpoint signal is turned off, MCC is disassembled and the checkpoint is

98 When the checkpoint signal is turned off, MCC is disassembled, a process required f

99 When the light was turned off motoneuron firing and locomotor frequency

100 The XIST allele must be turned off on one X chromosome to maintain its activi

101 arge/polarity of an atom or functional group was turned off/on, and the resulting effects on binding

102 How the SAC signal is turned off once all the chromosomes have successfully

103 within cellular grafts after NT-3 expression was turned off possibly via reduction in L1 and laminin

104 isible after-images of the stimuli when they were turned off ('prime-sight').

105 y phase, when an active p53 program needs to be turned off quickly.

106 sient behavior that occurs after the forcing is turned off suggests that the double knocks of Campeph

107 s to turn on anion transport, which can then be turned off through addition of the blocker dodecyl su

108 mechanism for receptor diversification that is turned off when antigen receptors are cross-linked by

109 However, how this pathway is turned off when hepatocyte proliferation is no longer

110 tified by the integrated fluorophores, which are "turned off" when the particles become destructed an

111 significantly declined when NT-3 expression was turned off, whereas continued NT-3 expression sustai

112 oved subsequent peeling performance when AFF was turned off, with reductions in average force, SD of