ライフサイエンスコーパス: be turned on

1 phosphorescence of the hexathiobenzene core is turned on.

2 ing the dark and dephosphorylated when light is turned on.

3 s activating chromatin marks only when xol-1 is turned on.

4 re than an order of magnitude when the cloak is turned on.

5 the number of seeds produced once the switch is turned on.

6 itive progeny even before the zygotic genome was turned on.

7 day period, during which time the NIr device was turned on.

8 adation were delayed when expression of US11 was turned on.

9 ersive enclosed arm, a light and white noise were turned on.

10 imes (24-hour clock): 0700 (before the light was turned on), 1000 (3 hours into the light cycle), 100

11 increased further and Stat6 gene expression was turned on after P. gingivalis infection in BALB/cByJ

12 st tau pathology once soluble tau expression is turned on again.

13 g cycle, only a fraction of the fluorophores were turned on, allowing their positions to be determine

14 , such as cannibalism and biofilm formation, are turned on and a later phase in which genes that play

15 f sRNAs with their target mRNAs so that they are turned on and off together to maximize duplex format

16 ecially those such as phototransduction that are turned on and off with great rapidity.

17 mers, revealing strong interactions that can be turned on and off as a function of the electrochemica

18 ich expression of the mutant transgene could be turned on and off as needed.

19 e incandescent and discharge lamps, LEDs can be turned on and off at high speeds.

20 strength in vivo, homosexual behavior could be turned on and off by altering glutamatergic transmiss

21 n scattering beacons (SERS beacons) that can be turned on and off by long-range plasmonic coupling, i

22 te a reversibly operating nanovalve that can be turned on and off by redox chemistry.

23 n generate self-sustaining activity that can be turned on and off by synaptic inputs.

24 ion in vivo and to enable such expression to be turned on and off effectively.

25 ansgenic animals, enabling reporter genes to be turned on and off rapidly.

26 Proliferation can be turned on and off repeatedly simply by shifting the e

27 of the yeast PI-SceI homing endonuclease can be turned on and off using a redox switch.

28 at cell-signaling pathways can theoretically be turned on and off, both locally and globally, in resp

29 el that allowed Zap70-dependent signaling to be turned on and then off again.

30 of gene silencing antisense agents that can be turned on and turned off in mammalian tissue culture.

31 f odontoblasts requires unique sets of genes being turned on and off in a growth- and differentiation

32 re simultaneously present, the photoactivity is turned on and evidence is given for an electron trans

33 antibody affinity in an ongoing response; it is turned on and off during the differentiation of B lym

34 f the cAMP/PKA pathway and that PKA activity is turned on and off on the timescale of tens of seconds

35 Each switch is turned on and off sequentially to distribute and rout

36 ies have shown how this locus-control region is turned on and off.

37 ing the ORR, particularly when the fuel cell is turned on and off.

38 For only one group, the device was turned on and delivered a slow heartbeat-like vibrat

39 The ion flux was turned on and off, apparently because the water wett

40 and Perelson suggested that if hypermutation were turned on and off in a phasic manner, affinity matu

41 orientation, and conformation of the ligand are turned "on" and "off." Second, sampling of the ligan

42 evelop a photoswitchable derivative that can be turned "on" and "off" with low-intensity light to spa

43 which a GTPase acts as a bimodal switch that is turned "on" and "off" by external regulatory factors,

44 a protein switch, whereby protein production is turned "on" and "off" by using short noise pulses.

45 mation about when, where, or how much a gene is turned on, and by dissecting the modules driving a gi

46 s the clusters rotate when the energy source is turned on, and fall apart when the energy input is of

47 s of the assembly of MCC when the checkpoint is turned on, and of its disassembly when the checkpoint

48 the component parts of the 'acetate switch' are turned-on as a consequence of direct competition, on

49 face of adversity (stress), death mechanisms are turned on at a cellular (apoptosis) and organismal l

50 Transcription of P(QA) is turned on at the end of the DNA replication period, c

51 controlled by the Cdc25C phosphatase, which is turned on at the G(2)/M transition to catalyze Cdk1 a

52 gelB expression is turned on at the migrating epithelial front in the co

53 Promoter Px is turned on at the same time as promoter P3 during earl

54 of DnaA in Bacillus subtilis whose synthesis is turned on at the start of sporulation.

55 Transgene expression was turned on at different time points: in utero, at bir

56 dian rhythm: IOP decreased after room lights were turned on at 00:00 CT and increased after they were

57 ned off at 48 h, while "cell division" genes were turned on at 48 h.

58 Some genes that were turned on at a low dose of Spo0A either had a high

59 enes, including a number of protein kinases, were turned on at this stage.

60 ther improvement when finally the stimulator is turned on, but in the case of saccadic latency the pa

61 en disengaged interactions within a receptor are turned on by its guest, these intrahost interactions

62 t the specific endogenous apoptotic programs are turned on by loss of Dicer.

63 rials demonstrating phosphorescence that can be turned on by incorporating halogen bonding into their

64 ide the intact endocytic organelles, but can be turned on by redox activation after endolysosomal dis

65 g polymerization of epsilon-caprolactone can be turned on by the in situ opening of the triple-layer

66 1 cases) the expression of 20 genes that had been turned on by sigma(G) and represses the expression

67 erpolarizing light-gated channel, HyLighter, is turned on by a brief light pulse at one wavelength an

68 e the XBP-1 axis normally, as follows: XBP-1 is turned on by cues that trigger differentiation and no

69 been described as a biochemical switch that is turned on by increases in intracellular calcium to me

70 chanism in which an inactive degradation tag is turned on by induced conformational changes.

71 Fluorescence of the reporter dye is turned on by rapid removal of the quinone quencher, a

72 Posterior neural markers were turned on by bFGF in AC expressing DN-BR or chordin

73 Here, we show that striatal circuits can be "turned on" by acute Gq-protein signaling or "turned

74 ) agents has been developed where the signal is "turned on" by altering the longitudinal relaxation t

75 When NT-3 gene expression was turned on, cholera toxin beta-subunit-labeled sensor

76 reactive alkynes and whether reactivity can be turned "on demand." When stereoelectronic amplificati

77 e thus provided evidence that specific genes are turned on during catagen, which is therefore not sim

78 on of the mucoid exopolysaccharide alginate, are turned on during conversion to mucoidy.

79 sed genes found during this effort appear to be turned on during organogenesis, however, some genes w

80 No cues were turned on during testing.

81 hern blot analysis showed that AC expression was turned on early in development, by E7.0, and continu

82 erentiate into myofibroblasts when RhoA/ROCK is turned on, endothelial cells when turned off.

83 When expression of barnase is turned on, failure to form a complex between the muta

84 a completely quenched (19)F NMR signal that is "turned on" following reduction to Cu(I).

85 ch balance; thus, the angiogenic switch must be turned "on" for cancer progression.

86 We propose that when the checkpoint is turned on, free MCC is the precursor of APC/C-bound M

87 y complex is key for understanding how genes are turned on in response to a Notch signal.

88 ding sine oculis (so) and eyes absent (eya), are turned on in their respective domains by Dpp.

89 uminescence of the AuNCs@GSH-Fe(3+) can also be turned on in the presence of phosphate-containing met

90 ng in mice indicates that gastrin expression is turned on in a subset of differentiated beta-cells af

91 tivated and the maintenance promoter, SxlPm, is turned on in both sexes; however, only in females is

92 ay be part of a differentiation program that is turned on in senescent cells.

93 number of existing probes whose fluorescence is turned on in the presence of the class of biological

94 first salivary gland genes, fork head (fkh), is turned on in the primordium of this organ, the saliva

95 pression of the bHLH gene, Thylacine1, which is turned on in the PSM as cells mature and segment, in

96 r1 regulon responds to copper limitation and is turned on in zinc deficiency, and Crr1 is required fo

97 ormation of PSC when expression of DeltaFosB was turned on in adult animals by removing Dox.

98 When CRT was turned on initially, immediate changes occurred in L

99 Which switching mode is turned on is controlled by the molecular environment.

100 When the checkpoint is turned on, it promotes the formation of the mitotic c

101 Underscoring how classic doctrine can be turned on its head, the peptidoglycan layer of polar-

102 This appealingly simplistic notion has been turned on its head in recent years with the discove

103 The usual approach is turned on its side by using the mathematical descript

104 se growth in an El Tor strain, transcription is turned on later in growth to levels in excess of thos

105 nential-phase growth, while the ctxAB operon was turned on later, after the rise in toxT expression.

106 Adhesion/invasion genes were turned on later in the maturation process.

107 ystem, organic light-emitting diodes (OLEDs) are turned on locally where the surface is touched, and

108 vicious cycle such that the Hh pathway might be turned on nonclassically by stimuli from the tumor mi

109 n single cell clones, hMLH1 expression could be turned on, off, and on again by 5-azacytidine exposur

110 nce of UV light via diffusiophoresis and can be turned "on/off" in a controlled manner.

111 Galpha(q) protein, the activity of which can be turned on or off at will in cardiac myocytes, develop

112 rmation pathways activated by Galpha(12) can be turned on or off by the addition or removal of serum.

113 transgenic system in which Akt signaling can be turned on or off in the adult heart, we previously sh

114 r size allow gradient-controlled pathways to be turned on or off simply by altering the distance betw

115 that the ATPase activation switch appears to be turned on or off when L175C/N820 are clamped at dista

116 pecific signals, allowing kinase activity to be turned on or off.

117 san nanofibres, the flow of protonic current is turned on or off by an electrostatic potential applie

118 ptional regulation, the expression of a gene is turned on or off by changes in the concentration of a

119 Ion flow through PAL-modified channels is turned on or off by photoisomerizing PAL with differe

120 The neurostimulator was turned ON or OFF for the first one month and then to

121 ight, the switch of age-dependent cell death was turned on prematurely, as indicated by the accumulat

122 esorbable packaging and electronics that can be turned on remotely, after implantation, to provide th

123 opment in transgenic mice even when the gene is turned on shortly before the time of tumor developmen

124 in which all terms in the interaction energy are turned on simultaneously using 1/kT as the coupling

125 The flow was turned on suddenly so that the entire evolution of m

126 reased when the p53 transactivation activity was turned on, suggesting that PRC1 transcription could

127 The H-function can be turned on/switched off by the concentration of comple

128 When mismatching of atoms or bonds is turned on, the compute times increase as expected, an

129 When the checkpoint is turned on, the mitotic checkpoint complex (MCC) inhib

130 dian time and begins at 00:00 CT when lights are turned on.) The maximum difference in IOP between li

131 when upper room germicidal air disinfection was turned on throughout the ward.

132 If the angiogenic switch needs to be turned on to facilitate the tumor growth, the questio

133 the late part of the assigned period, lights were turned on twice for a 1-hour interval.

134 design, an autoinhibited coiled-coil switch is turned on upon proteolytic cleavage, which results in

135 These results illuminate how GIV-GEF is turned on upon receptor activation, adds GIV to the r

136 pletely CEST-silent in the oxidized form but was "turned on" upon reduction with beta-NADH.

137 The device is turned on when the temporary occupation of the core s

138 artificial radiation was designed; the lamp was turned on when solar UV-A irradiance was below 20 W

139 The bistable photoswitch can be turned on with millisecond-long pulses at one wavelen

140 The Scl gene is turned on within FLK1(+)CD4(-) cells to give rise to