ライフサイエンスコーパス: be unchanged

1 he electrodes, whereas classically it should be unchanged.

2 serum cytokine levels measured were found to be unchanged.

3 control cells, but the number of organelles is unchanged.

4 while the overall cell density of the cortex is unchanged.

5 protection, even if the rest of the dynamics is unchanged.

6 with RNAs of different lengths, the protein is unchanged.

7 riage and that invasiveness of the NVT group is unchanged.

8 the auditory percept, although motor output is unchanged.

9 anner, whereas the maximally activated force is unchanged.

10 t the activities of concurrent NADPH sources are unchanged.

11 ulation, while those expressed only in cones are unchanged.

12 angiotensin II (ANGII), and renin activities are unchanged.

13 ated levels of Aiolos, whereas Ikaros levels are unchanged.

14 eins, such as claudin-1, ZO-1, and occludin, are unchanged.

15 ations from the original 2016 ASCO guideline are unchanged.

16 age, but bias in the energy risk coefficient was unchanged.

17 tion peptide, whereas proelastase activation was unchanged.

18 expression of other TPR-containing proteins was unchanged.

19 0.003); right ventricular ejection fraction was unchanged.

20 DS mortality, whereas the excess risk in MSM was unchanged.

21 Intrinsic myocardial function was unchanged.

22 ates were decreased, but the biofilm biomass was unchanged.

23 , whereas glucose-induced oxygen consumption was unchanged.

24 e mutants; however, the p53 expression level was unchanged.

25 ir capacity to stimulate collagen production was unchanged.

26 s sodium-independent taurocholic acid uptake was unchanged.

27 reference stream, total invertebrate biomass was unchanged.

28 the apparent Ca(2+) dependence of exocytosis was unchanged.

29 However, efficiency was unchanged.

30 ing columns; whisker movement representation was unchanged.

31 tion, whereas the root elemental composition was unchanged.

32 ery increased while renal oxygen consumption was unchanged.

33 e management, survival to hospital discharge was unchanged.

34 In these motors, stator-binding was unchanged.

35 ncreased to 4/10, while BCVA of the left eye was unchanged.

36 bone scintigraphy and radiotracer therapies was unchanged.

37 subfamily, member 1 (KCNQ1) mRNA expression was unchanged.

38 ncreased by 24 h, whereas C-reactive protein was unchanged.

39 rowth of IAV in all of the cell types tested was unchanged.

40 2, whereas the expression of gamma-tubulin-1 was unchanged.

41 er time, whereas the use of active vitamin D was unchanged.

42 tly improved control even when feedback rate was unchanged.

43 d room size, although the vocal-motor output was unchanged.

44 um ratio, a surrogate marker for citratemia, was unchanged.

45 stribution of absorbing and emitting species was unchanged.

46 efault") cleavage by the 20S core proteasome was unchanged.

47 lcium/calmodulin-dependent protein kinase II was unchanged.

48 hile the affinity for a fully matched target was unchanged.

49 Toll-like receptor 2 expression was unchanged.

50 tion and breathing; hypoxic hyperventilation was unchanged.

51 heating times while phytoene and phytofluene were unchanged.

52 but the numbers of megakaryocytic precursors were unchanged.

53 whereas rates of proliferation and apoptosis were unchanged.

54 rious Abeta-clearing and -degrading proteins were unchanged.

55 une cell infiltrates and liver transaminases were unchanged.

56 white matter lesions, although CBF responses were unchanged.

57 mber, size, and fatty acid respiration rates were unchanged.

58 mice, whereas soluble monomeric Abeta levels were unchanged.

59 moderately decreased whereas Abeta40 levels were unchanged.

60 resistance, and mean arterial blood pressure were unchanged.

61 ated during sound making, while P2 responses were unchanged.

62 After multivariable adjustment, results were unchanged.

63 sphingomyelins, and lysophosphatidylcholines were unchanged.

64 tb-stimulation, but Shh levels in astrocytes were unchanged.

65 Abeta1-x and Abetax-42 plaque deposits were unchanged.

66 not MCI subjects, whereas cognate receptors were unchanged.

67 at week 4 when renal function and histology were unchanged.

68 Levels of most endogenous cellular proteins were unchanged.

69 rcadian activity, and despair-like behaviors were unchanged.

70 fin (EC) cell numbers (cells producing 5-HT) were unchanged.

71 hage and vascular smooth muscle cell content were unchanged.

72 and heart, but macrophage subset populations were unchanged.

73 dependence-like phenotype, whereas WKY rats were unchanged.

74 r TRF, even when caloric intake and activity were unchanged.

75 nditure rates and those after meal ingestion were unchanged.

76 ng antioxidants and cellular repair pathways were unchanged.

77 , whereas tissue levels of total cholesterol were unchanged.

78 revealed that, overall, pathologic findings were unchanged.

79 on-like peptide-1 receptor agonist exendin-4 were unchanged.

80 adiopharmaceutical use and bone scintigraphy were unchanged.

81 reas the cellular properties of interneurons were unchanged.

82 her-rated and self-rated antisocial behavior were unchanged.

83 study period, intravenous rehydration rates were unchanged.

84 but plasma zinc concentrations and EZP size were unchanged.

85 besity/T2D, while infections in control mice were unchanged.

86 erum levels in vivo, whereas other cytokines were unchanged.

87 LT uptake as well as TK1 and Ki67 expression were unchanged.

88 ntrast, GlyR IPSC and NMDAR-EPSC decay times were unchanged.

89 ivity, although other biophysical properties were unchanged.

90 e at decreased reward, while forward replays were unchanged.

91 82; 95% CI, 1.70-1.94), and private coverage was unchanged (21% and 19%; 0.96; .89-1.03).

92 004), and rates of terminal hospitalizations were unchanged (29% to 27%, P = .70).

93 mprovement at the last study visit, 23 (22%) were unchanged, 3 (3%) had a worsened clinical status, a

94 That local visual cortical excitability was unchanged across drug conditions suggests the involv

95 .99]) whereas the effect of medical staffing was unchanged across the range of patient acuity (1.00,

96 In-hospital mortality was unchanged (adjusted odds ratio, 0.96; 95% CI, 0.84-1

97 It was unchanged after ablation (P=0.57), remaining lower t

98 or sociodemographic factors; the excess risk was unchanged after adjustment for cognitive decline but

99 This result was unchanged after control for baseline body mass index

100 me of diagnostic tests and procedures billed was unchanged after conversion (median, +93; IQR, -20 to

101 chanical ventilation, meningitis, and death, was unchanged after introduction of the EOS calculator.

102 Insulin secretion was unchanged after LS/IMM but decreased after RYGB, exc

103 This relationship was unchanged after multivariable adjustment.

104 inity (KD = 6.2 nM) and specificity for METH was unchanged after nanoparticle conjugation.

105 This was unchanged after stratification on NIH trial particip

106 Blood pressure was unchanged after the HS diet, although it significant

107 90-120 min) myocardial DFA fractional uptake was unchanged after the LOWCAL diet (0.055 +/- 0.025 vs.

108 This relationship was unchanged after the policy change (adjusted relative

109 Whereas speech intelligibility was unchanged after WM training, subjects in the CL trai

110 and AMH (anti-Mullerian hormone) expressions were unchanged after 1month of cryopreservation.

111 d in regional strain between SSc and control were unchanged after adjusting for RV systolic pressure.

112 ages, whereas paclitaxel levels in the blood were unchanged after administering MSV-nAb-PTX or nAb-PT

113 rter (VAchT), and their nerve fiber density, were unchanged after buserelin treatment, but the relati

114 fter further adjusting for APRI, and results were unchanged after excluding subjects with suspected c

115 These results were unchanged after further adjustment for body mass in

116 These differences were unchanged after place avoidance training with the s

117 admissions (107 [4.9] vs 103 [4.4]; P = .42) were unchanged after the intervention.

118 ative morbidity, and postoperative mortality were unchanged (all P > 0.05).

119 ed to NZW mice, while ferroportin expression was unchanged and ferritin expression increased, consist

120 At 12 months from baseline, sensory scores were unchanged and below-level neuropathic pain became p

121 In contrast, heat and mechanical pain are unchanged, and results show that, in striking contra

122 MAPK signaling was unchanged, and GSK3beta-Ser(9) phosphorylation was r

123 ity revealed that extraocular motor function was unchanged, and immunohistochemistry revealed no abno

124 More importantly, the PK behavior was unchanged, and neither anti-uricase nor anti-PCB ant

125 Blood pressure was unchanged, and no adverse clinical events occurred.

126 ent electroencephalography and brain imaging were unchanged, and a fluorodeoxyglucose F 18 positron e

127 sulin levels and the major milk compositions were unchanged, and the transgenic animals had no appare

128 contrast, the percentages of CD8(+) T cells were unchanged, and these cells showed an activated phen

129 r with plasma cholesterol and triglycerides, were unchanged as a function of genotype.

130 euronal network within the responsive barrel was unchanged, as characterized by c-Fos upregulation in

131 Nevertheless, liver utilization rates are unchanged at around 80%.

132 or the strongly consider radiotherapy cohort was unchanged at 26.9% until 2007.

133 threshold after terminal cleaning (71 RLU), was unchanged at the completion of injection clinic and

134 ver, dough extensibility and dough stability were unchanged at elevated [CO2].

135 In contrast, in TRN neurons, AP properties are unchanged between LTS bursts and tonic firing and, a

136 ive receptive field parameters were found to be unchanged between the two states, such as most aspect

137 HER2 amplification and/or mutation frequency was unchanged between local disease and metastases acros

138 urgitation (4.3% at 1 year; 4.4% at 2 years) was unchanged between the first and second year.

139 n the NEI VFQ-25 composite and its subscales were unchanged between baseline and 3-year follow-up, wh

140 neous firing of the presynaptic VIP+ neurons were unchanged between day and night, and their network

141 BKCa alpha-subunit expression was unchanged, BKCa beta1-subunit and sensitivity of BKC

142 e exceptional JRCSFvifH42/43D infection, vif was unchanged but replication proceeded after a long del

143 During beta1 -AR blockade, LV volumes were unchanged but blood pressure and heart rate were re

144 Liver DFA partitioning was unchanged, but liver fractional uptake of DFA tended

145 Myogenic tone was unchanged, but over a range of transmural pressures,

146 Circulating calcium and PTH levels were unchanged, but inorganic phosphate and 1,25(OH)2D3

147 In mutant CbN cells, IPSC kinetics were unchanged, but mutant males, unlike females, showed

148 B-cell FcgammaRIIb expression is unchanged by HD preculture, but B cells can support T

149 e dependence of stream microbial respiration is unchanged by nutrient enrichment, and that increased

150 re of this pi bond is that the bond strength is unchanged by substitution with electron-donating grou

151 tion was occluded by hypoxia (12% FIO2), but was unchanged by 6% FiCO2.

152 Importantly, CO and NO(*) binding was unchanged by AdoMet in a truncated form of CBS lacki

153 as dose-dependently increased, but ISF Abeta was unchanged by central insulin administration.

154 SleepEE was unchanged by overfeeding in the low-protein diet gro

155 lysis demonstrated that plasma arginase flux was unchanged by P. berghei infection.

156 he Orai crystal structure, and stoichiometry was unchanged by store depletion, coexpression with STIM

157 to the maximum observed in control mice but was unchanged by time of day or estradiol.

158 ia within the arteriovenous synthetic grafts was unchanged by treatment with fat/PGZ depots compared

159 Brain stimulation reward thresholds were unchanged by alcohol or naltrexone in 118AA mice.

160 tages (32.5-35.0; P = .47), and Marsh scores were unchanged by gluten challenge.

161 While many histone modifications were unchanged by Su(H) binding or Notch activation, we

162 on of low-frequency hippocampal oscillations were unchanged by this manipulation, indicating that sen

163 abolic and mitochondrial genes, whose levels are unchanged compared to NPCs, revealing distinct trans

164 ac unloading, capillary density and fibrosis are unchanged compared with loaded hearts.

165 mented by twofold in R192Q mice, whereas LTD was unchanged compared with wild-type mice.

166 P was undetectable; IL-10, IL-17A, and IL-33 were unchanged compared to diluent challenge.

167 h the expression of other ZnT family members was unchanged, cytoplasmic (n = 4 mice per genotype; p <

168 ue and seed numbers, but seed protein levels were unchanged, demonstrating the importance of AAP8 fun

169 s following hyperoxia, mean diffusivity (MD) was unchanged despite baseline values lower than control

170 toconversion-we find that the Dam1 submodule is unchanged during anaphase, whereas MIND and Ndc80 sub

171 s (Stat3(DeltaCD4)), the course of infection was unchanged during the innate lymphoid cell-dependent

172 mpectomy in the 3729 patient analytic sample was unchanged during the study.

173 ealed that RNA levels of cotransfected genes were unchanged during superinduction, yet Western blotti

174 and IV (COL1A1, COL3A1, COL4A1) transcripts was unchanged early but increased 5.7 +/- 2.5-, 3.2 +/-

175 Quality of life was unchanged (EQ5D: E 0.54(0.29) vs D 0.56(0.24), p = 0

176 nucleus, the position of the nucleus in FADD is unchanged, even when permutation reduces the complexi

177 hile alpha1-adrenoceptor-mediated excitation was unchanged, excitatory responses to AMPA were enhance

178 c stability with the CO2 adsorption isotherm being unchanged following 7 days of steam-treatment (>85

179 tionship and 50 ms paired-pulse facilitation were unchanged following COX10 removal from dentate gran

180 Mortality was unchanged for all hospitals.

181 ut was suppressed for C-25ms-Bi (by 31%); it was unchanged for Bi only and C only.

182 n (adjusted HR 1.99, 0.75-5.25; p=0.163) but was unchanged for MSM (2.24, 0.82-6.11; p=0.114).

183 Over time, the GSS was unchanged for the NP group and improved for the HP g

184 In addition, while total tau-soluble levels were unchanged for both groups, PD146176-treated mice ha

185 tatistically normal tend to have an IOP that is unchanged from baseline or even higher following cata

186 9 strain used in the live attenuated vaccine was unchanged from 2015-2016, the Advisory Committee on

187 obtained at the time of physical examination was unchanged from baseline.

188 thin normal limits, and an electrocardiogram was unchanged from baseline.

189 dient (median, 4.5 years after implantation) was unchanged from early post-TPV replacement, and all b

190 The safety profile was unchanged from the initial report.

191 At week 48 on cART, HIV DNA levels were unchanged from pre-cART levels, although a signific

192 ssion of the vesicular GABA transporter VGAT was unchanged; however, there was a significant increase

193 eliminated while the autologous NAb response was unchanged.IMPORTANCE Trimeric proteins are being dev

194 ington's disease models, NPM1 protein levels are unchanged in cultured cerebellar granule and cortica

195 riptionally, since the levels of nAChR mRNAs are unchanged in qvr/sss mutants.

196 r CD24 (nucCD24) and finding that its levels are unchanged in surCD24(-) cells.

197 gnitude of the protrusive force was found to be unchanged in response to cytoskeletal inhibitors of l

198 R and GPP equivalently, with the net outcome being unchanged in NEE.

199 We find that PTP is unchanged in PKC triple knock-out (TKO) mice in which

200 ness decreased by more than 10% in 47 (44%), was unchanged in 47 (44%), and increased by more than 10

201 F decreased in 13.0%, improved in 40.0%, and was unchanged in 47.0% of the patients.

202 After 4 years, visual acuity was unchanged in 6 of 10 patients.

203 ors (64%), decreased in 11 tumors (22%), and was unchanged in 6 tumors (12%).

204 unctional class had improved in 21 patients, was unchanged in 63 patients, and had deteriorated in 7

205 Similarly, the volume of the nLOT was unchanged in aged rats, except in layer 3 where it w

206 ibodies based on MFI of 2000, 4000, and 8000 was unchanged in all patients.

207 istant areas exposed to high SS; plaque size was unchanged in atheroprone areas, in which endothelial

208 A/NMDA ratio was reduced in ApoE KO mice, it was unchanged in bEKO mice compared with controls.

209 renic patients; overall use in no-cap states was unchanged in both cohorts.

210 Early microglia activation was unchanged in CCL2- and CCL3-deficient knockouts, but

211 Following hyperoxia DTI was unchanged in controls.

212 Plasma ADMA was unchanged in DDAH1(En-/-) mice, and cultured aortas

213 Cav1.3 and Cavbeta2 protein expression was unchanged in du/du IHCs, forming clusters at presyna

214 proximately 65% in Eln(+/-) (P = 0.002), but was unchanged in Eln(+/+) mice (P = 0.17).

215 The amount of Slc26a6 protein was unchanged in enamel organs of Ae2a,b- and Cftr-null

216 However, tau phosphorylation was unchanged in eNOS(-/-) mice (P>0.05).

217 In contrast, SERCA2 was unchanged in hearts of female mice, whereas phosphor

218 alization of wild-type and DeltaSIV channels was unchanged in HEK293 cells.

219 Femur bone density was unchanged in mice heterozygous for Drp1 deletion, an

220 However, warm water swim-induced analgesia was unchanged in Null vs. control mice.

221 Tetraselmis sp. when exposed to copper, but was unchanged in P. tricornutum (72-h exposure to 19, 40

222 Xylem putrescine concentration was unchanged in putrescine-treated plants, so the exoge

223 ation; however, proliferation of type I NSCs was unchanged in response to fluoxetine.

224 Whereas mitochondrial mass was unchanged in single SN neurons from IPD patients, we

225 function in one subject, whereas hotspot SUV was unchanged in subjects with stable graft function.

226 well as long-term synaptic plasticity (LTP) was unchanged in the absence of APLP1.

227 f luminal narrowing in the diseased segments was unchanged in the intervention group (Wilcoxon W = 76

228 pothesis, excitability of F-type motoneurons was unchanged in the mutant mice.

229 g frequency of bloody stools; bowel function was unchanged in the other groups.

230 il group (baseline vs. end of treatment) but was unchanged in the placebo group.

231 Instrument response was unchanged in the presence of matrix, indicating no n

232 After 48 months, radius bone mineral density was unchanged in the teriparatide to denosumab group (0.

233 TnI levels increased in 23.0%, were unchanged in 51.3%, and decreased in 25.7% of patie

234 frequency of intracellular Ca(2+) increases were unchanged in alpha-cells of alphaZnT8KO KO mice.

235 Findings were unchanged in analyses controlling for the presence

236 llowing blast TBI, phospho-Tau (pTau) levels were unchanged in ApoE3 mice, whereas in ApoE4 mice, lev

237 c neuritis, whereas optic radiation measures were unchanged in controls.

238 mendations were new, 1,339 were dropped, 881 were unchanged in COR and LOE, and 129 were revised.

239 vealed two peaks, M1 and M2, whose positions were unchanged in different buffers to indicate conforma

240 RF-induced EPSCs only in AC and PL-responses were unchanged in IL, a critical area for suppression of

241 SERCA2a and phospholamban protein were unchanged in MR versus control left ventricles.

242 ower in patients with apathy (P = 0.004) and were unchanged in patients without apathy.

243 X levels than healthy donors, whereas levels were unchanged in primary progressive MS and neuromyelit

244 d intake, muscle and bone mass and adiposity were unchanged in Sgta(-/-).

245 Inferences were unchanged in surveys regardless of world region, na

246 ival, climbing ability and neuronal function were unchanged in tau KO flies.

247 2 and -0.006 +/- 0.003 kg, respectively) but were unchanged in the DSE group (0.00 +/- 0.02 and 0.004

248 ion and activity as well as monoamine levels were unchanged in the projection areas of 5-HT neurons.

249 Rates of PMRT were unchanged in the radiotherapy recommended (29.9%) a

250 0%, P=0.005; PSCS, -3.5+/-4.3%, P=0.001) but were unchanged in those in SR during both assessments (b

251 sure, bronchoalveolar lavage cytokine levels were unchanged in TREK-1-deficient mice compared with co

252 ut basal average peak coronary flow velocity was unchanged, indicating LV stunning post balloon occlu

253 cin, although the sensitivity to papuamide A was unchanged, indicating preferential exposure of PE.

254 Extracellular volume fraction was unchanged, indicating the absence of fibrosis.

255 in a two-head-bound state, exchange kinetics were unchanged, indicating that rearward strain in the t

256 d the fusion kinetics and calcium dependence were unchanged, indicating that the effect of helix 12 e

257 Levels of oxidative damage were unchanged (liver) or reduced (brain and serum) in R

258 esponse element regions in the HAS2 promoter was unchanged or decreased.

259 ce of amyloid increased apoE3, whereas apoE4 was unchanged or decreased.

260 reased, whereas those that lacked Trp codons were unchanged or even decreased.

261 ivo reporters, however, show dCREB2 activity is unchanged, or decreased when sleep/activity patterns

262 nt scientific fields per year, has tended to be unchanged over time, at least until now.

263 creased disability although tremor frequency is unchanged over time.

264 % per quarter, whereas the rate of O-MCS use was unchanged over the study period.

265 Pre-sharing, status 1A listing was unchanged over time (adjusted odds ratio, 0.98; 95%

266 ent-elevation acute coronary syndrome trials was unchanged over time.

267 recommendations from the 2011 ASCO guideline are unchanged pending a full update.

268 atory markers in subcutaneous adipose tissue was unchanged postoperatively, although plasma CRP was d

269 line, although almost all other metabolites were unchanged relative to control.

270 However, GRP78 mRNA levels were unchanged, suggesting a posttranscriptional event.

271 time but total Ni concentrations in sediment were unchanged, suggesting changes in Ni partitioning th

272 ong-term and homeostatic synaptic plasticity were unchanged, suggesting that loss of DBN is not suffi

273 ts of both the standard and immunoproteasome were unchanged, suggesting the abnormalities we observed

274 spontaneous metastasis in vivo When fluidity was unchanged, the antimetastasis compounds could no lon

275 as largely unchanged and the mRNA half-lives were unchanged, the steady-state levels of the mRNA vari

276 hough total phospholamban protein expression was unchanged, there was a decreased expression of prote

277 Conversely, topo-II levels were unchanged through early development, and partial to

278 ese processes depends on local structure and is unchanged throughout most of the film, while the othe

279 ollowing an unreinforced food port entry and were unchanged throughout the NAcc when initiating inact

280 In contrast, S18-1 mRNA levels are unchanged to slightly elevated under Zn(2+)-limited

281 and subcutaneous white adipose tissue depots was unchanged upon cold acclimation.

282 In contrast, the nucleolar pattern of B23 was unchanged upon infection with an SBV recombinant mut

283 ficant findings and the many parameters that were unchanged, we can conclude that cataract surgery do

284 Results were unchanged when additionally adjusted for smoking, h

285 Copper levels were unchanged when AP-1 function was impaired, but cell

286 The results were unchanged when further adjusted for ejection fracti

287 Total crossovers measured by chiasmata were unchanged when heterozygosity was varied, consisten

288 Apoptotic index is unchanged, whereas proliferation index is significant

289 The level of PC in leaves was unchanged, whereas that of PE was reduced in all AAP

290 -positive, and Tau C3-positive NFT densities were unchanged, whereas only AT8-positive neuropil threa

291 model predictions, however, collision losses were unchanged, while hip work during swing initiation w

292 and 19%) and Medicaid (50% and 50%) coverage were unchanged, while private coverage decreased (13% an

293 and 52%) and Medicaid (18% and 18%) coverage were unchanged, while private coverage increased (4% and

294 ind that Ca(2+) stores and resting [Ca(2+)]i are unchanged with age.

295 Serum sST2 was unchanged with canagliflozin and placebo over 104 we

296 D-dimer declined with ATV/r and DRV/r and was unchanged with RAL.

297 and median percent time hypoglycemic on CGM were unchanged with CSII, SD glucose and CONGA4 reduced

298 mortality (HR, 1.12; 95% CI, 1.08-1.16) that were unchanged with further adjustment for NO2.

299 polar dynamics in the 100 fs to 100 ps range were unchanged with pH, although nanosecond yield, rates

300 and peak oxygen consumption and VE/CO2 slope were unchanged with sildenafil.