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1 ative contribution to the tissue destruction is unknown.
2 6 years, although the clinical significance is unknown.
3 umans but its significance for visceral pain is unknown.
4 of mitochondria from prion-infected animals is unknown.
5 um retention and salt-sensitive hypertension is unknown.
6 en allotypes, but the mechanism of selection is unknown.
7 of vitamin D reduces the risk among children is unknown.
8 beyond these boundaries would be beneficial is unknown.
9 The role of Hap10 in BD pathogenesis is unknown.
10 ive conformation whose ability to polymerize is unknown.
11 Whether MR1 binds other ligands is unknown.
12 rulence regulator, CovR, in UTI pathogenesis is unknown.
13 he mechanism of strain-specific cell tropism is unknown.
14 reduces VTE risk beyond aspirin monotherapy is unknown.
15 he mechanism by which DENV induces lipophagy is unknown.
16 ategy or active recruitment by the parasites is unknown.
17 g information to or between cortical regions is unknown.
18 cells, but its role in embryonic stem cells is unknown.
19 d-leading cause of death by cancer in women, is unknown.
20 entiation, although the underlying mechanism is unknown.
21 unctional differences between gamma-tubulins is unknown.
22 derlies the regulation of PICK1-Ago2 binding is unknown.
23 -term risk of developing metabolic disorders is unknown.
24 However, the specificity of these effects is unknown.
25 ation schedule in lung transplant recipients is unknown.
26 educes risk of coronary artery disease (CAD) is unknown.
27 on of T helper (TH) lineage-defining factors is unknown.
28 abilitation, though their combinatory effect is unknown.
29 basal and reflex cardiorespiratory function is unknown.
30 cell-specific role of Nox2 in BP regulation is unknown.
31 The mechanism by which this occurs is unknown.
32 ses, but a role in host response to microbes is unknown.
33 polypeptide (IAPP), its role in the disease is unknown.
34 nd MLR-associated proteins on neuronal aging is unknown.
35 -modifying enzymes in craniofacial disorders is unknown.
36 identity and/or network-dependent processes is unknown.
37 elated hospitalizations and associated costs is unknown.
38 manifestations, but the underlying pathology is unknown.
39 m distant metastases in primary colon cancer is unknown.
40 cognition by PCs), but the order of cleavage is unknown.
41 ting metabolic changes induce EC activation, is unknown.
42 structure and function of cortical networks is unknown.
43 however, the neurobiological basis for this is unknown.
44 ant PIK3CA during early stages of malignancy is unknown.
45 virulence and persistence in this population is unknown.
46 iation in non-alcoholic beverage preferences is unknown.
47 Whether this is true for other biomarkers is unknown.
48 arning, but how they encode this information is unknown.
49 tabolic stress and associated cardiomyopathy is unknown.
50 these observations to adult ovarian function is unknown.
51 but the full spectrum of the evoked changes is unknown.
52 mal traits and their intraspecific variation is unknown.
53 differentiate between high and low abilities is unknown.
54 t effective administration-to-birth interval is unknown.
55 the production of proinflammatory cytokines is unknown.
56 somal proteins regulate GABARAP localization is unknown.
57 mmunosuppressed adults with critical illness is unknown.
58 of breast cancers explained by these factors is unknown.
59 rotective in other models of synucleinopathy is unknown.
60 ncer-associated venous thromboembolism (VTE) is unknown.
61 estoratively have changed with this evidence is unknown.
62 ystem, but whether nonpathogenic ones spread is unknown.
63 obust mitotic rounding within packed tissues is unknown.
64 rucial components of epileptogenic networks, is unknown.
65 nationally representative, mixed-race sample is unknown.
66 of T cells, but how ROS influence NKT cells is unknown.
67 BV infection in the context of DAA treatment is unknown.
68 relative contribution to tissue destruction is unknown.
69 glioma, but the mechanism of its activation is unknown.
70 ntial association with intermittent exposure is unknown.
71 itative variation found in respiratory rates is unknown.
72 patients with low baseline vitamin D status is unknown.
73 action analyses are implemented in the brain is unknown.
74 Their function is unknown.
75 of efferocytosis in metastatic tumor growth is unknown.
76 ing the Baby Boom years (1946-1964) or later is unknown.
77 ion; however, the basis for this selectivity is unknown.
78 ary for context-driven salt-seeking behavior is unknown.
79 bioavailability triggers activation of SczA is unknown.
80 whether DJ-1 can regulate human MC function is unknown.
81 d magnitude of groundwater use by vegetation is unknown.
82 ent; the molecular origin of this difference is unknown.
83 oxidative and substrate-level bioenergetics is unknown.
84 ntity of key 8p tumor-suppressor genes (TSG) is unknown.
85 n SMA but the underlying molecular mechanism is unknown.
86 candidemia of a urinary tract source (CUTS) is unknown.
87 their association with risk of CKD and ESRD is unknown.
88 eurohumoral activation during disease states is unknown.
89 lear, and its possible interaction with PIP2 is unknown.
90 is maintained and impacts tumor surveillance is unknown.
91 g route by which beta-DG reaches the nucleus is unknown.
92 and why this is defective in Fmr1 KO neurons is unknown.
93 stromal cells in adipose tissue inflammation is unknown.
94 therapy one can expect an improvement in QOL is unknown.
95 hough the underlying cause for this increase is unknown.
96 elative importance of these selective forces is unknown.
97 (NECs) of fish gills, whose embryonic origin is unknown.
98 different muscle subsets affect regeneration is unknown.
99 rrhythmia; however, the underlying mechanism is unknown.
100 weight loss on posttransplantation outcomes is unknown.
101 eatic cancer resistance to anticancer agents is unknown.
102 ion, but whether this translates to human DR is unknown.
103 e sorting sequence recognized by the microHD is unknown.
104 origenic cell undergoes to become malignant) is unknown.
105 but its effectiveness in real-world settings is unknown.
106 How TREM2 promotes such diverse responses is unknown.
107 er, the importance of these conditional APCs is unknown.
108 s, but the specific function of each subunit is unknown.
109 rophy, but how these mutations cause disease is unknown.
110 r circadian time influences body composition is unknown.
111 The molecular trigger of CNS myelination is unknown.
112 ation extends to related FOXO family members is unknown.
113 the mouse, yet the functional impact of most is unknown.
114 recisely how these mutations inactivate BAP1 is unknown.
115 priate activation of Cdks, yet the mechanism is unknown.
116 l edema; but what triggers this complication is unknown.
117 in elevated urinary arsenic concentrations, is unknown.
118 the United Kingdom (UK), but the prevalence is unknown.
119 esistance (IR) or impaired tissue metabolism is unknown.
120 for substrate recognition and translocation is unknown.
121 ium signalling apparatus in cardiac myocytes is unknown.
122 g allergy exist, but the quality of services is unknown.
123 es function in TLR4 responses of hepatocytes is unknown.
124 to rapid modulations in response to signals is unknown.
125 n's disease, but its role on skin microbiota is unknown.
126 revious suicide attempts in a soldier's unit is unknown.
127 brimonidine is used topically for hemostasis is unknown.
128 ry conditions, its potential role in allergy is unknown.
129 ically jagged-2, to the homeostasis of HSPCs is unknown.
130 s there and its function in these organelles is unknown.
131 low obstruction affect lung function decline is unknown.
132 ontributing to cognitive and memory decline, is unknown.
133 e (BAT) in regulating gestational metabolism is unknown.
134 ensure generation of high-affinity antibody is unknown.
135 nonrenal cell types, although the mechanism is unknown.
136 anism for the antiobesity effect of standing is unknown.
137 ediates Abeta-induced loss of surface AMPARs is unknown.
138 ukemic agent asparaginase, but the mechanism is unknown.
139 e possible activity of BAP1 in the cytoplasm is unknown.
140 ic oxide (NO) pathways with disease severity is unknown.
141 t tissues but its role in tissue maintenance is unknown.
142 ess and lower health-related quality of life is unknown.
143 by light-induced changes in SIG5 expression is unknown.
144 in animal development and tissue homeostasis is unknown.
145 ficance in pain originating from the viscera is unknown.
146 the evolutionary reason for this association is unknown.
147 o absorbs salt and fluid and expresses ENaC, is unknown.
148 r autologous free flap breast reconstruction is unknown.
149 ty (HPR) in SVG PCI versus native lesion PCI is unknown.
150 e in homeostatic and pathological conditions is unknown.
151 ptomatology, but its role in predicting PTSD is unknown.
152 e contributions to sister chromatid cohesion are unknown.
153 g electron transfer to proton translocation, are unknown.
154 ouple nutritional state to adipokine release are unknown.
155 ms mediating Treg migration to neonatal skin are unknown.
156 far the effects of IL-31 on human basophils are unknown.
157 wist1 and Foxa1 in breast cancer progression are unknown.
158 and the regional drivers of deadwood stocks are unknown.
159 The neuromolecular bases for these delays are unknown.
160 s induce these bactericidal stress responses are unknown.
161 lying trehalose's effect on taste perception are unknown.
162 ancy induces CHD, but the underlying reasons are unknown.
163 ns of the motor system to sensory processing are unknown.
164 The reasons for this association are unknown.
165 nal significance of this localized signaling are unknown.
166 idities in contemporary youth-onset diabetes are unknown.
167 s on respiratory function in later childhood are unknown.
168 (heart rate, < 80 beats/min) in septic shock are unknown.
169 ular mechanisms underlying CRY2 interactions are unknown.
170 tion factors that maintain AT2 cell identity are unknown.
171 CTO percutaneous coronary intervention (PCI) are unknown.
172 al outcomes in patients with type 2 diabetes are unknown.
173 ymph, but the underlying signalling pathways are unknown.
174 ar mechanisms that mediate these differences are unknown.
175 l origin and mode of homeostatic maintenance are unknown.
176 of these two different types of hybrid ZIFs are unknown.
177 for the pathogenic effects of ADAR1 mutation are unknown.
178 enotype in mice, but the cell types involved are unknown.
179 although the mechanisms of these activities are unknown.
180 hat enable HD-PTP to regulate ESCRT function are unknown.
181 es LRH-1 conformation to drive transcription are unknown.
182 ut the mechanisms underlying this phenomenon are unknown.
183 hanisms that minimize proactive interference are unknown.
184 s and rate laws governing individual enzymes are unknown.
185 eptide-containing nerves in the mouse vagina are unknown.
186 own adipose tissue before environmental cold are unknown.
187 m childhood on midlife cognitive performance are unknown.
188 on to distinct mitochondrial subcompartments are unknown.
189 genetics associated with amelanotic melanoma are unknown.
190 However, the underlying molecular mechanisms are unknown.
191 ctors that determine their abundance changes are unknown.
192 usual care in patients with type 2 diabetes are unknown.
193 mosome Y (mLOY), and its clinical relevance, are unknown.
194 hich GLIS3 dysfunction causes hypothyroidism are unknown.
195 otonin reuptake inhibitors in these patients are unknown.
196 ge differentiation of c-kit(+) cells in vivo are unknown.
197 caspase involved in its secretion currently are unknown.
198 n process [2], but the underlying mechanisms are unknown.
199 viral avian leukosis virus (ALV) integration are unknown.
200 ion of avascular privilege in photoreceptors are unknown.
201 g the last century, but the underlying genes are unknown.
202 (miRNAs) involved in host resistance to APEC are unknown.
203 ape cooling-induced translational repression are unknown.
204 s governing beta-cell mitochondrial turnover are unknown.
205 functional connections between these regions are unknown.
206 but the mechanisms underlying this sex bias are unknown.
207 rgets that promote cancer cell proliferation are unknown.
208 e-specific effects of SRC-2 in these changes are unknown.
209 e neural pathways underlying these abilities are unknown.
210 anisms underlying the tumor type specificity are unknown.
211 ling the expression of Longus-encoding genes are unknown.
212 ences of loss of Lpd in the CNS on behaviour are unknown.
213 e to the accumulation of methylated arsenic, are unknown.
214 logical mechanisms of this genetic influence are unknown.
215 ncer; however, the details of such functions are unknown.
216 Mps1's error correction-relevant substrates are unknown.
217 aptic function, neuronal growth and survival are unknown.
218 ever the mechanisms underlying these changes are unknown.
219 ircadian oscillator in a GI-dependent manner was unknown.
220 in the generalist's adoption of P. angulata was unknown.
221 hway(s) involved in neuronal loss thereafter was unknown.
222 ctural protein components of the basal layer was unknown.
223 nserved residues whose functional importance was unknown.
224 ocortical memory consolidation have thus far been unknown.
225 nd the basis for its selectivity have so far been unknown.
226 il now, the consequences of this interaction were unknown.
227 tions of plasmepsins IX and X (PMIX and PMX) were unknown.
229 e anorexigenic effects of oxytocin in humans are unknown and critical to study to consider oxytocin a
230 However, the exact functions of the N-Srcs are unknown and it is likely that N-Src signalling event
231 the RhopH complex in PSAC/NPP establishment are unknown and it is unclear whether any of the RhopH p
233 dulate the cell cycle, the precise mechanism is unknown and its impact on inflammation in vivo has no
234 daries, yet the full extent of these impacts is unknown and thus neglected in environmental licensing
236 ol the establishment of astrocyte morphology are unknown, and it is unclear whether impairing astrocy
237 he prevalence of THSD7A-Ab-positive patients is unknown, and it is unclear whether the clinical prese
238 utes exist in a regenerated microcirculation is unknown, and methodologies for answering this have be
239 chromosome 5q31-q33, the genetic cause of FE is unknown, and prior studies have failed to demonstrate
240 rength of microtubule-centrosome attachments is unknown, and the possibility that mechanical force mi
242 ever, awareness and knowledge about glaucoma is unknown at community level, making provision of inter
243 learning or an outcome of learning accuracy is unknown at this stage, but can now be addressed in fu
248 ospores and polymerized in specific patterns are unknown, but the primexine, a transient cell wall ma
249 e of idiopathic dilated cardiomyopathy (DCM) is unknown by definition, but its familial subtype is co
250 sponse of forest trees to such perturbations are unknown, especially given the lack of efficient dyna
252 accumbens core medium spiny neurons, but it is unknown how achieving drug use affects this plasticit
253 and ecosystem services benefit from fire, it is unknown how ecosystems will respond to increased burn
255 ite recent discoveries made in this area, it is unknown how the oral microbiome responds to short-ter
258 s defined the architecture of the family, it was unknown how a channel has adapted to cope with its d
261 inclusion body myositis (sIBM) pathogenesis is unknown; however, rimmed vacuoles (RVs) are a constan
266 which sulfurs are oxidized by these enzymes are unknown, in part because intermediates have yet to b
267 the mechanisms underlying these associations are unknown, it is important that all HEDS patients rece
269 t functions and its interactions with lipids are unknown or limited due to inability of obtaining sta
270 ndividuals, the genetic cause of the disease is unknown, suggesting that other loci are most likely i
274 t the effect of glycemic control on survival is unknown.We sought to determine the relationship betwe
276 accines to prevent Ebola virus disease (EVD) were unknown when the incidence of EVD was peaking in Li
280 conserved throughout the animal kingdom, it is unknown whether common signaling pathways regulate th
284 hibiting the mitochondrial complex I, but it is unknown whether manassantin inhibits mitochondrial re
286 preventing further fertilization events, it is unknown whether plant gametes prevent polyspermy by a
290 lear skin without a precursor lesion, but it is unknown whether these melanomas can arise from melano
291 genetic changes in colorectal tumors, but it is unknown whether these mutations phenocopy gain-of-fun
293 gnitive shifts could recruit the DMN, yet it is unknown whether this holds for nonhuman species.
295 AWR leads to improvement in function, but it is unknown whether this is specific to the abdominal wal
297 with reduced dorsal striatal volume, but it is unknown whether this relationship extends to adolesce
299 Despite the importance of this structure, it is unknown which factors are needed to activate cumulus
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