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1 n of anthrahydroquinone (AnH(2)Q) appears to be not fully understood.
2 pulmonary infection and severity of disease is not fully understood.
3 The mechanism underlying this tight control is not fully understood.
4 ctors, but how their expression is regulated is not fully understood.
5 d navigation but its functional organization is not fully understood.
6 osis, the mechanisms mediating demyelination is not fully understood.
7 ver, early LR primordium (LRP) morphogenesis is not fully understood.
8 demonstrated, UDP-GlcNAc delivery by SLC35A3 is not fully understood.
9 mploy this potential immune evasion strategy is not fully understood.
10 ENV serotypes and ZIKV during DENV infection is not fully understood.
11 ole in carcinogenesis and cancer progression is not fully understood.
12 how each controls this core kinase cassette is not fully understood.
13 how LANA escapes host's immune surveillance is not fully understood.
14 the molecular basis for oligomeric assembly is not fully understood.
15 , but how these computations are coordinated is not fully understood.
16 -binding domain at serine 209 by Mst1 kinase is not fully understood.
17 topoietic hierarchy is specifically affected is not fully understood.
18 al cell polarity to achieve its oncogenicity is not fully understood.
19 rcellular coupling and cellular excitability is not fully understood.
20 This conundrum is not fully understood.
21 r informativeness for human complex diseases is not fully understood.
22 y which the cancer nucleus becomes misshapen is not fully understood.
23 concert in response to physical stimulation is not fully understood.
24 ting the stress response in follicular cells is not fully understood.
25 bosome assembly and other cellular processes is not fully understood.
26 ondrial respiration and stem cells' behavior is not fully understood.
27 hotoreceptors meet their high energy demands is not fully understood.
28 t the impact of nutrient limitation on xylem is not fully understood.
29 ype 2 diabetes mellitus among Chinese adults is not fully understood.
30 nd tau pathology in Alzheimer's disease (AD) is not fully understood.
31 apoptotic cells mediate such diverse effects is not fully understood.
32 esistance to EGFR inhibitors in this setting is not fully understood.
33 the transcriptional regulation of miR-17-92 is not fully understood.
34 systemic factors contribute to this process is not fully understood.
35 latory mechanisms controlling their function is not fully understood.
36 ioenergetic cost of matrix protein synthesis is not fully understood.
37 er, the detailed mechanism of UNC5B function is not fully understood.
38 ve group conversion between filler particles is not fully understood.
39 ach active zone protein to synapse functions is not fully understood.
40 metazoans to implement their body plans that is not fully understood.
41 ertension, but its role in disease pathology is not fully understood.
42 The dynamic behavior of phthalates indoors is not fully understood.
43 n and molecular function during this process is not fully understood.
44 chanism by which B-Myb alters the cell cycle is not fully understood.
45 DOP sources to microbial community P demand is not fully understood.
46 ct to control adult hippocampal neurogenesis is not fully understood.
47 y information to drive appropriate decisions is not fully understood.
48 e of actin in maintaining cell-cell adhesion is not fully understood.
49 usive, and thus, the mechanism of DDX action is not fully understood.
50 tion of angiogenesis, although the mechanism is not fully understood.
51 taneous nucleosome movements in this process is not fully understood.
52 physiologic and metabolic aspects of tumors is not fully understood.
53 selective response of Tregs to low-dose IL-2 is not fully understood.
54 selectively affected in Parkinson's disease is not fully understood.
55 rosstalk between GAMs and glioblastoma cells is not fully understood.
56 l disease state to advanced clinical disease is not fully understood.
57 the development of CNS-infiltrating B cells is not fully understood.
58 some binding proteins affect gene expression is not fully understood.
59 V gene expression and viral DNA replication, is not fully understood.
60 The regulation of SG dynamics is not fully understood.
61 sis; however, how these cells are maintained is not fully understood.
62 he metabolism of other amino acids in cancer is not fully understood.
63 on development; however, the function of Scx is not fully understood.
64 es affect the liberation of lutein from food is not fully understood.
65 ffect the functional proteome in lung cancer is not fully understood.
66 e appeals engage the brain to drive behavior is not fully understood.
67 the oncogenic effect of Arg882His in DNMT3A is not fully understood.
68 RD4 in normal hematopoiesis and inflammation is not fully understood.
69 ic bundle branch and atrioventricular blocks is not fully understood.
70 However, transcriptional regulation of ABI5 is not fully understood.
71 ymers are assembled in a coordinated fashion is not fully understood.
72 eir individual contribution to the tRNA pool is not fully understood.
73 of the aerosol alone on endothelial function is not fully understood.
74 (TB) infection and progression to active TB is not fully understood.
75 to modulate synaptic plasticity and learning is not fully understood.
76 their excitability, although how this occurs is not fully understood.
77 e development of inflammatory bowel diseases is not fully understood.
78 may explain these discrepancies however this is not fully understood.
79 the mechanism behind this complication still is not fully understood.
80 iability impacts detection of sensory inputs is not fully understood.
81 , the underlying mechanism of their activity is not fully understood.
82 osine diphosphate (ADP) on flagellar beating is not fully understood.
83 However, the molecular basis is not fully understood.
84 Yet how Rsr1 regulates cell polarization is not fully understood.
85 al significance of polymicrobial bacteriuria is not fully understood.
86 unctional operation of neuronal interactions is not fully understood.
87 ERK activation, but its underlying mechanism is not fully understood.
88 o generate cell type-specific transcriptomes is not fully understood.
89 of CNS neurons to regulating those features is not fully understood.
90 g and glycolysis in liver cancer progression is not fully understood.
91 ne-bound Nogo-A is presented intercellularly is not fully understood.
92 The role of NIPP1 in tissue homeostasis is not fully understood.
93 er, the metabolic control of these processes is not fully understood.
94 r while moving through a complex environment is not fully understood.
95 olds human telomeric G-quadruplex structures is not fully understood.
96 eral density, a universal feature of ageing, is not fully understood.
97 ying mechanism of EDR impairment in diabetes is not fully understood.
98 oring growth by and through the nerve fibers is not fully understood.
99 siological homeostasis in the brain and body is not fully understood.
100 function, particularly during inflammation, is not fully understood.
101 f genome stability, yet their mode of action is not fully understood.
102 t the molecular mechanism of this protection is not fully understood.
103 teins interact to nick and unwind the duplex is not fully understood.
104 s recruit hundreds of further ParB to spread is not fully understood.
105 The mechanism is not fully understood.
106 uman cancers, but how PTEN is down-regulated is not fully understood.
107 However, the mechanism is not fully understood.
108 (CBS) domain, though the role of this domain is not fully understood.
109 However, the mechanism of drug resistance is not fully understood.
110 tion of beta2-adrenergic agonists at the NMJ is not fully understood.
111 stalk with other cell types mediating repair is not fully understood.
112 lesterol management on cellular biomechanics is not fully understood.
113 h signals are encoded within the spinal cord is not fully understood.
114 are regulated and activate defense signaling is not fully understood.
115 isease severity and the host immune response is not fully understood.
116 equired for nodulation and nitrogen fixation is not fully understood.
117 atory role of ECs in adipose tissue function is not fully understood.
118 how neuromodulation influences this activity is not fully understood.
119 enes, the genetic architecture of this trait is not fully understood.
120 ent components contribute to their formation are not fully understood.
121 archetypical sodium-ion conductor Na(3)PS(4) are not fully understood.
122 in aggressive behavior of TNBC, while means are not fully understood.
123 tation and attenuation of rotavirus vaccines are not fully understood.
124 of treatment of cutaneous leishmaniasis (CL) are not fully understood.
125 but the mechanisms of virulence attenuation are not fully understood.
126 overning these competing adhesive properties are not fully understood.
127 itching and maintenance of epigenetic states are not fully understood.
128 ns, but the mechanisms underlying its effect are not fully understood.
129 ing to these different individual strategies are not fully understood.
130 The mechanisms are not fully understood.
131 he underlying molecular mechanisms, however, are not fully understood.
132 ever, most of their functions and activities are not fully understood.
133 tivity in epithelial sodium channels (ENaCs) are not fully understood.
134 f the specifics of virus-vector interactions are not fully understood.
135 hanisms and their physicochemical properties are not fully understood.
136 nt for HTLV-1, and the osteolytic mechanisms are not fully understood.
137 e neurodegeneration, although the mechanisms are not fully understood.
138 nic entry and the factors that regulate them are not fully understood.
139 ever, the pathogenic mechanisms of psoriasis are not fully understood.
140 its physiological and pathological functions are not fully understood.
141 s, while the underlying molecular mechanisms are not fully understood.
142 wever, the relationships among these factors are not fully understood.
143 ion of the system and its control mechanisms are not fully understood.
144 tails regarding deamination target selection are not fully understood.
145 st for NPC and the mechanisms of NPC disease are not fully understood.
146 bolic adaptations required for thermogenesis are not fully understood.
147 f such interactions for particular functions are not fully understood.
148 ation of sensory evidence into motor output, are not fully understood.
149 clear localization of individual chromosomes are not fully understood.
150 y, and functional capabilities of DC subsets are not fully understood.
151 hat correlate with recovery from RSV disease are not fully understood.
152 g abilities in dogs, their homing strategies are not fully understood.
153 cent cells contribute to disease progression are not fully understood.
154 rk conditions, but the associated mechanisms are not fully understood.
155 ral tolerance and prevention of autoimmunity are not fully understood.
156 l center (GC) B cell responses in the spleen are not fully understood.
157 ate oncogenesis, but the detailed mechanisms are not fully understood.
158 nd function of TET2-RNA interactions in vivo are not fully understood.
159 mechanisms underlying these adverse effects are not fully understood.
160 e strain-specific differences in GAG binding are not fully understood.
161 eceptor-mediated antidepressant-like effects are not fully understood.
162 increased fragility, whereas the mechanisms are not fully understood.
163 The reservoirs and transmission of both are not fully understood.
164 controlling its differentiation and function are not fully understood.
165 ogression, and the mechanisms of the disease are not fully understood.
166 es other peculiar responses whose mechanisms are not fully understood.
167 emical features associated with reticulation are not fully understood.
168 e face of noise or significant perturbations are not fully understood.
169 herms than ectotherms, the reasons for which are not fully understood.
170 eptor PD-1 (anti-PD-1) reinvigorates T cells are not fully understood.
171 -stage heart failure (HF) by mechanisms that are not fully understood.
172 to target because the underlying mechanisms are not fully understood.
173 al remodeling and its role in mechanosensing are not fully understood.
174 CNS targets after intranasal administration are not fully understood.
175 e mechanisms that regulate capsule thickness are not fully understood.
176 ere disease caused by newly emerging strains are not fully understood.
177 , yet its effects on the fibrinolytic system are not fully understood.
178 chanisms underlying this protective immunity are not fully understood.
179 nt, though their precise molecular functions are not fully understood.
180 and growth over timescales less than a month are not fully understood.
181 ity, but the underlying molecular mechanisms are not fully understood.
182 (THC) impact on threat processing in humans are not fully understood.
183 tion and its interplay with dopamine release are not fully understood.
184 plasmodesmata, but the molecular mechanisms are not fully understood.
185 f pycnofibres, their affinities and function are not fully understood.
186 actions that influence cell-to-cell adhesion are not fully understood.
187 , their roles in remodeling cilia properties are not fully understood.
188 well documented and the mechanisms involved are not fully understood.
189 of macrophages after APAP overdose, however, are not fully understood.
190 l activation of these two signaling pathways are not fully understood.
191 ction pathway whose components and mechanism are not fully understood.
192 re and biological mechanisms underlying mLOY are not fully understood.
193 ns, yet the requirements for their induction are not fully understood.
194 n and contribute to inflammation and disease are not fully understood.
195 , of altered haemoglobin affinity for oxygen are not fully understood.
196 SCLC), mechanisms of resistance to this drug are not fully understood.
197 lular components required for plant immunity are not fully understood.
198 which tumours become inhospitable to T cells are not fully understood.
199 ivity, and its effectiveness in treating HCC are not fully understood.
200 fference, but the mechanisms behind sex-bias are not fully understood.
201 cer risk; however, the underlying mechanisms are not fully understood.
202 I) resistance, but the underlying mechanisms are not fully understood.
203 h the origin and underlying cellular defects are not fully understood.
204 among patients with new musculoskeletal pain are not fully understood.
205 h different species tolerate these compounds are not fully understood.
206 ms of APAP-induced necrosis and liver injury are not fully understood.
207 et growth hormone-secreting pituitary tumors are not fully understood.
208 derlying this urotoxicity and its prevention are not fully understood.
209 ater childhood, but effects on lung function are not fully understood.
210 ogenic mechanisms underlying this phenomenon are not fully understood.
211 bosome flux along open reading frames (ORFs) are not fully understood.
212 direct interactions with photoactivated CRYs are not fully understood.
213 PIP2 mobility and thus its spatial patterns are not fully understood.
214 g setron binding and inhibition of 5-HT(3A)R are not fully understood.
215 , the intrinsic clinical implications of FTR are not fully understood.
216 echanisms and the origins of the selectivity are not fully understood.
217 breached in patients with autoimmune disease are not fully understood.
218 e the T cell inflamed tumor microenvironment are not fully understood.
219 onal cell death in Huntington's disease (HD) are not fully understood.
220 r, the supporting mechanisms of this process are not fully understood.
221 mechanisms by which this integration occurs are not fully understood.
222 ecular mechanisms deriving CRC tumorigenesis are not fully understood.
223 logical organization of proteins within IACs are not fully understood.
224 ying the anticancer action of iron chelation are not fully understood.
225 assembly and the dynamics of PIC components are not fully understood.
226 circadian clocks to cardiac arrhythmogenesis are not fully understood.
227 red immunity and effector-triggered immunity are not fully understood.
228 et the mechanisms coupling the two processes are not fully understood.
229 hanisms underpinning these changes in humans are not fully understood.
230 ecular mechanisms of macrophage polarization are not fully understood.
231 A-subunits that stabilize the FXIII-A2 dimer are not fully understood.
232 he occurrence and importance of this process are not fully understood.
233 PD-L1 inhibition elicits anti-tumor immunity are not fully understood.
234 V-D68 circulation and molecular epidemiology are not fully understood.
235 vern stiffness-dependent NSC fate commitment are not fully understood.
236 The causes of thalamic atrophy are not fully understood.
237 network dynamics underlying these phenomena are not fully understood.
238 sumably hinder their adaptation in ways that are not fully understood.
239 leading to pathogenic levels of the cytokine are not fully understood.
240 s in their expression patterns and functions are not fully understood.
241 , the functions of these intermediate states are not fully understood.
242 for mTORC1 signaling through mechanisms that are not fully understood.
243 yet the implications for transplant outcomes are not fully understood.
244 rlying chemical and physiological mechanisms are not fully understood.
245 ptibility translates into autoimmune disease are not fully understood.
246 ently of weight loss through mechanisms that are not fully understood.
247 ncoding genome, but their biological impacts are not fully understood.
248 mpact of specific 11q23/KMT2A rearrangements are not fully understood.
249 actors that govern various NK cell functions are not fully understood.
250 uman B cell and Ab responses during COVID-19 are not fully understood.
251 ion, particularly during cardiac remodeling, are not fully understood.
252 d the mechanisms governing these differences are not fully understood.
253 een suggested, but the underlying mechanisms are not fully understood.
254 lar and molecular mechanisms of this process are not fully understood.
255 nical studies, their combined effects on TME are not fully understood.
256 se exerts its effects, especially in humans, are not fully understood.
257 rates, but the basis for this discrimination was not fully understood.
259 The biological mechanisms leading to BPD are not fully understood, although an arrest in lung dev
260 onnection between chronic stress and disease are not fully understood and are likely multifactorial.
261 nmental factors such as soil moisture stress are not fully understood and are therefore poorly repres
262 quality of life, the causes of endometriosis are not fully understood and validated diagnostic marker
264 etastatic process of colorectal cancer (CRC) is not fully understood and effective therapies are lack
265 ional regulation mechanism of the Cyp3a gene is not fully understood and requires a thorough investig
266 arly 1990s, its pathophysiological mechanism is not fully understood and there is no LS-specific cure
267 ar events in response to severe hyperthermia are not fully understood, and research has focused mainl
268 rlie irAEs from immune checkpoint inhibition are not fully understood, and treatment strategies are c
269 The etiology and pathogenesis of the disease are not fully understood, and treatment with corticoster
271 r the regulation of their catalytic activity is not fully understood, and RPTPs are generally not the
272 f motor actions in reward seeking and taking is not fully understood, and there is little information
273 ever, the biological relevance of these PTMs is not fully understood because their regulation is larg
274 ng mechanisms regulated by such therapeutics are not fully understood but could underlie differential
275 The mechanisms driving timing differences are not fully understood but two opposing (but not mutua
276 OD1/2 are stimulated by such diverse stimuli is not fully understood but may partly rely on cellular
277 ls and research needs to improve EH practice are not fully understood, but 26% of EH professionals wo
280 The pathophysiology of diabetes-related AF is not fully understood, but is related to structural, e
282 ys through a common set of effectors, yet it is not fully understood how different receptors elicit d
285 y significantly contribute to resistance, it is not fully understood how tumor sub-clones interact wi
286 ical smoke associated with energy device use is not fully understood, however transmission is biologi
288 orresponding mechanical properties, however, are not fully understood, nor is it understood how micro
289 abuse interact with individual vulnerability is not fully understood, nor is it clear how individuals
290 nisms such as the reaction network structure are not fully understood, significant discrepancy exists
291 However, the genetics of their biosynthesis is not fully understood, since no gene has been identifi
293 story of research and a plethora of data, it is not fully understood what determines the biodiversity
296 matic diseases have complex aetiologies that are not fully understood, which makes the study of patho
297 une mediators against C. difficile infection are not fully understood, with data suggesting that infl