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1 O(2)) pulse (absolute increase: 0.4+/-1.4 mL/beat).
2  by antibodies based on the T-cell receptor (BEAT).
3 ponse; (6) supraventricular tachycardia (>15 beats).
4 a smaller SR Ca2+ release and AP in the next beat.
5 ocardiogram-derived QT interval for the same beat.
6 echanical energy efficiency of the flagellar beat.
7  introduced in an AP sequence by a premature beat.
8 ollections of sER curves generated from each beat.
9 , humans can easily identify and move to the beat.
10 stion of how we learn to generate and hold a beat.
11 ternal motors that shape the flagellum as it beats.
12 usly during passive listening to isochronous beats.
13  modulations during listening to isochronous beats.
14  position is biased and dependent on ciliary beating.
15  fluid flow in these organs via synchronized beating.
16 tial roles in generating coordinated ciliary beating.
17 ssembly and its role in regulating flagellar beating.
18 firmed the importance of TTC29 for flagellar beating.
19 or TTC29 axonemal localization and flagellar beating.
20 g them perpendicular to the plane of ciliary beating.
21 mechanisms by which the CA regulates ciliary beating.
22 1 was initiated after onset of cardiomyocyte beating.
23  motile cilia together to ensure coordinated beating.
24 esponded to tapping to every fourth auditory beat (0.625 Hz).
25 en (-2.3 +/- 1.4 vs. -3.3 +/- 1.4 bursts.100 beats(-1) mmHg(-1) , P = 0.007), while this sex differen
26  lift their feet in synchrony with a musical beat [2,3], but humans move to music using a wide variet
27 brillation; (4) ventricular tachycardia (>15 beats); (5) atrial fibrillation with rapid ventricular r
28              Despite the key role of beat-to-beat action potential (AP) variability in the onset of v
29 impaired barrier formation and reduced cilia beat activity.
30 pating accelerating, decelerating, or steady beats after direction-specific visual cues.
31 nd we show that the optimal quantum strategy beats all classical strategies by running multiple equiv
32        Cardiac alternans refers to a beat-to-beat alternation in contraction, action potential (AP) m
33 lular level alternans is observed as beat-to-beat alternations in contraction, action potential (AP)
34                   Analysis of the TARGET and BEAT AML databases identifies a correlation between CD82
35 nificantly different between patients on the Beat AML sub-studies and those receiving SOC (induction
36 ficantly longer for patients enrolled on the Beat AML sub-studies versus those who elected SOC.
37 ML sub-study; 224 (56.7%) were enrolled on a Beat AML sub-study.
38  within 7 d and were centrally assigned to a Beat AML sub-study; 224 (56.7%) were enrolled on a Beat
39 s were prospectively enrolled on the ongoing Beat AML trial (ClinicalTrials.gov NCT03013998 ), which
40                           In this laser, the beating among the coherent modes oscillating inside the
41 r relationships of 3-beat average with index-beat analysis (R=0.94, 0.94, and 0.94 for LA strain, LA
42                                      Beat-to-beat analysis of up to 4 full days of electrocardiograph
43             The CP coordinates the flagellar beat and defects in CP projections are associated with m
44  driving force of net IVC flow was the heart beat and not respiration.
45 inhibition significantly reduced the beat-to-beat and short-term variabilities of APD.
46 ambered muscle pumps demonstrated macroscale beating and continuous action potential propagation with
47 ent tissues exhibited defective synchronized beating and ineffective generation of directional flow l
48                 Reducing cardiac workload in beating and nonbeating Langendorff perfusions had no eff
49 plication for flagellar growth influenced by beating and provide possible explanations for the experi
50 protein required for flagellar structure and beating and that TTC29 mutations are a cause of male ste
51 cilium, could perceive forces during ciliary beating and transfer these signals to locally regulate t
52 ruption efficiency and yields comparing bead-beating and ultra-sonication of different biological mat
53  their seed sites on Drd1 mimicked the cilia-beating and ventricular deficits.
54  microRNA in 22q11DS mice normalizes ciliary beating and ventricular size.
55 automatic temporal processing of isochronous beats and explicit, temporally-oriented attention.
56 xperiments, comparing original BEAT, mutated BEAT, and "knobs-into-holes" interfaces, suggested a coo
57 perties of the motor system, we propose that beat anticipation relies on action-like processes consis
58 +) -activated Cl(-) channels reduced beat-to-beat AP alternations, but prolonged APD and failed to su
59                                      Quantum beats are confirmed to unambiguously reflect the exciton
60           In addition, premature ventricular beats arising from the Purkinje tissue as ventricular fi
61 ike representations as what they are without beating around the bush.
62 orations on Hawaii, highlighting undergrowth beating as highly efficient method.
63 ographies with micron-sized pillars that can beat at a programmable frequency and force level in an e
64 g for the primary prevention of egg allergy; BEAT (at 12 months, n = 42) and STEP (at 5 months n = 82
65 ated analysis of repeated 30 s recordings of beating atria in 381 live, intact zebrafish embryos at 2
66 ith anomalous bundles, ventricular premature beats, atrial flutter, atrioventricular nodal reentry, a
67 re strong positive linear relationships of 3-beat average with index-beat analysis (R=0.94, 0.94, and
68                                Consecutive 3-beat averaged values of strain and SR were used.
69                                          The BEAT (balloon versus self-expandable valve for the treat
70 namics in sensorimotor areas are involved in beat-based timing.
71 iving, especially when measured on a beat-to-beat basis.
72 cal stretch) eliciting premature ventricular beats because of a primary morphofunctional abnormality
73 ensitivity (BRS) with ambulatory and beat-to-beat blood pressure (BP).
74 rography, 12 paired recordings), and beat-to-beat blood pressure (BP; photoplethysmography) during th
75 soconstrictor firing while measuring beat-by-beat blood pressure and forearm vascular conductance.
76 l sequence over 5 years by recording beat-to-beat blood pressure and R-R intervals over 10 min.
77 nstrumented for electrocardiography, beat-to-beat blood pressure, respiratory rate, CO-Modelflow algo
78 e activity (MSNA; microneurography), beat-to-beat BP (photoplethysmography) and heart rate (electroca
79                           Ambulatory beat-to-beat BP was recorded using a portable device for multipl
80 ency domain HRV indices, BRS, office beat-to-beat BP, and heart rate (HR) were measured for 10 minute
81 g HRV, HR, BRS and ambulatory/office beat-to-beat BP.
82 eave philosophy, by describing Roald's being beaten by simulation.
83 sophy, beginning by describing Roald's being beaten by simulation.
84 hetic vasoconstrictor firing while measuring beat-by-beat blood pressure and forearm vascular conduct
85                     Custom software modelled beat-by-beat pressure (photoplethysmography) and R-R int
86 ars, subjects underwent a 6 min recording of beat-by-beat pulmonary artery diastolic pressure (PAD),
87 a sarcomere length-dependent manner and on a beat-by-beat time-scale.
88 scent of a cardiac crescent before forming a beating cardiac tissue near a putative primitive gut-lik
89 plied the AR-DIC analysis to a spontaneously beating cardiomyocyte (CM) tissue, and could provide cor
90 e differentiation, as indicated by no or few beating cardiomyocytes and reduced expression of cardiom
91 ect of antiarrhythmic drugs on spontaneously beating cardiomyocytes derived from hESC and hiPSC was g
92                                              Beating cardiomyocytes undergo fast morphodynamics durin
93 l recessive inheritance) that impair ciliary beating cause a variety of motile ciliopathies, a hetero
94 l interrogation of contractile parameters in beating cells.
95 EMOTION analyses showed proper cardiomyocyte beating characteristics on spider silk coatings, and car
96 flows at a similar frequency to the actively beating cilia can entrain cilia oscillations.
97 e PCF is pushed along by the ciliated cell's beating cilia, the PCF and its virus content are also pu
98 ding to progressively higher rate of beat-to-beat CL changes.
99 ns of tissue displacement and strain for the beating CMs utilizing physiologically-relevant, sarcomer
100 ) preserves the donated heart in a perfused, beating condition preventing cold storage-related ischem
101              Fluctuations of the human heart beat constitute a complex system that has been studied m
102 he surface of a multiciliated epithelia cell beat coordinately to protect the epithelium from bacteri
103  it active for a larger fraction of the tail beat cycle.
104                                    The cilia-beating deficit observed in brain slices and in vivo is
105  to binary strings using threshold crossing, beat detection and a novel feature detection parsing alg
106  noticed that zebrafish defective in ciliary beating develop abnormal body curvatures.
107                                    The heart-beating donor- and DCD-NEVLP-groups showed significantly
108                            Grafts from heart-beating donors (HBD), and from donors with 30 minutes (d
109  Pig LT was performed with livers from heart-beating donors or donation after circulatory death (DCD)
110 d in adults, the critical signals regulating BeAT during infant development need to be better defined
111 a reduction in number of ventricular ectopic beats during the ischaemic phase compared with acute tre
112 roaches for systematically assessing ciliary beat dynamics and for drug testing would improve the cha
113 h significantly attenuated the percentage of beating EBs in culture and expression of early and late
114 ions for the experimental observation that a beating flagellum is usually longer than its immotile mu
115 e most efficient drugs for restoring ciliary beating for each individual donor.
116 mechanical combination in real time recorded beating force of the CMs cluster and the triggering elec
117 ontractile speeds required to swim with tail-beat frequencies of 80-100 Hz.
118                         No change in ciliary beat frequency (CBF) or airway surface liquid (ASL) meni
119 , and specialised responses, such as ciliary beat frequency (CBF).
120 a an increase in LRRC26 expression), ciliary beat frequency and airway surface liquid volume improved
121 c power balance, we infer the mean flagellar beat frequency and conjecture that its diurnal variation
122 /DeltaF508 and DeltaF508/-) based on ciliary beat frequency and coordination.
123 n planaria resulted in ciliary loss, reduced beat frequency and dyskinetic motion of the remaining ve
124 icated by calcium elevation, increased cilia beat frequency, and increased fluid and cytokine secreti
125 normal ciliary number, radial structure, and beat frequency.
126                     The high harmonic of the beating from the photothermal expansion mixes with the A
127 igitised kinematic descriptions of flagellar beating from videomicroscopy.
128                             Low rates (~1 ms/beat) generate a btb-ER dominated by steady-state rate d
129                                          The beat generator is endowed with plasticity allowing it to
130 , but not exact information, of how well the beat generator spike times match those of a stimulus seq
131 per, we introduce a neuronal framework for a beat generator that is capable of learning isochronous r
132 ollowing rank order: mutated BEAT > original BEAT > knobs-into-holes.
133 ssified in the following rank order: mutated BEAT > original BEAT > knobs-into-holes.
134           The study of beige adipose tissue (BeAT) has recently gained popularity because of its pote
135 tate-of-art approaches, where the entire ECG beats have been considered.
136                                      Beat-to-beat heart period, systolic blood pressure, and electrom
137 the instantaneous variability of the beat-to-beat heart rate): spontaneous swallowing 12.02 +/- 1.07
138 treated with vehicle or stimulatory CpG ODN (beating heart control and DCD stimulated with CpG ODN, B
139                                              Beating heart controls and DCD rats were pretreated with
140 ional fluorescence time-lapse imaging of the beating heart is extremely challenging, due to the heart
141  developmental and cellular processes in the beating heart, revealing the dynamics of the immune resp
142 chanical stress and metabolic demands of the beating heart.
143 scle cells (Tg[acta2:GFP]), to visualize the beating heart.
144 ution or electromechanical uncoupling of the beating heart.
145 me, conditioning later cell diversity in the beating heart.
146          Donor pigs were assigned to undergo beating-heart donation (n = 9) or donation after circula
147 splants; 14 successfully weaned from bypass (beating-heart donation versus donation after circulatory
148 n through which their bones, intestines, and beating hearts can be seen.
149                                          The BeAT-HF (Baroreflex Activation Therapy for Heart Failure
150 reflex Activation Therapy for Heart Failure [BeAT-HF]; NCT02627196).
151    Here, we provide recommendations from the BEAT-HIV Martin Delaney Collaboratory on which viral mea
152 ters of CM contraction to account for the CM beating homogeneity, synchronicity, and propagation as h
153       We study here the entrainment of cilia beat in multiciliated cells from brain ventricles.
154 n be used to characterize collective ciliary beating in a non-biased automated manner.
155 the origin and mechanistic function of these beats in photosynthetic light-harvesting has been extens
156               Since the discovery of quantum beats in the two-dimensional electronic spectra of photo
157 ichalcogenides have been measured as quantum beats in two-dimensional electronic spectroscopy, but th
158  Kcne5 deletion caused ventricular premature beats, increased susceptibility to induction of polymorp
159  between baseline and R-wave triggered paced beats increases consistently with an increase in the pac
160         Furthermore, we show how the mutated BEAT interface can be exploited for the routine preparat
161 ose that the superior cooperativity found in BEAT interfaces is the key driver of their greater perfo
162 ans; (c) SAN features can be calculated from beat-intervals obtained in-vivo, without intervention; (
163 and adenosine diphosphate (ADP) on flagellar beating is not fully understood.
164 r to as Ion-Bunch Energy Acoustic Tracing (I-BEAT), is a refinement of the ionoacoustic approach.
165 ifferent from the issue of how we perceive a beat, is the question of how we learn to generate and ho
166 oscopy with multiple equally spaced lines by beating it with a reference Fabry-Perot comb confirms th
167 e of APD (type 1), intermediate rates (~5 ms/beat) lead to a btb-ER similar to a single sER (type 2),
168                    Motile cilia are cellular beating machines that play a critical role in mucociliar
169                    An external sound/musical beat may have been initially uninvolved.
170 cise predictions of steady state from single-beat measurements.
171 aytime, sleeping, 24-hour and office beat-to-beat measurements.
172                     LA deformations by the 3-beat method are feasible and reproducible during AF.
173 art rates during dives were typically 4 to 8 beats min(-1) (bpm) and as low as 2 bpm, while after-div
174 s while resting at the surface were 44 +/- 6 beats min(-1), 179 +/- 31 ml, and 7909 +/- 1814 l min(-1
175  L/min/m) (p = 0.86) for cardiac index; -1.8 beats/min (-3.7 to 0.1 beats/min) (p = 0.06) for heart r
176 1 R37C(+/-) at stimulation frequencies of 55 beats/min (bpm), but these cells showed no restitution w
177 or 3 weeks: sustained AFL; sustained AF (600 beats/min atrial tachypacing); AF superimposed on an AFL
178 beats/min, 24 (interquartile range: 9 to 41) beats/min faster than daytime sinus rates.
179 ositioned at the finger and earlobe) was 2.8 beats/min for over 65% of the time the patient was stabl
180 de ablation and ventricular pacemakers at 80 beats/min to control ventricular rate.
181 tion led to higher mean heart rate (56 +/- 2 beats/min vs. 50.1 +/- 0.4 beats/min; p = 0.05), less ba
182 r cardiac index; -1.8 beats/min (-3.7 to 0.1 beats/min) (p = 0.06) for heart rate; 96.8 mL/min/m (71.
183 te (ventricular tachycardia [VT]; 120 to 179 beats/min) in 27%, and rarely severe (VT >=180 beats/min
184 ats/min) in 27%, and rarely severe (VT >=180 beats/min) in 9%.
185 g AF was 96 (interquartile range: 83 to 114) beats/min, 24 (interquartile range: 9 to 41) beats/min f
186 pected in patients with mean heart rate >100 beats/min, atrial fibrillation, and/or premature ventric
187  (6.8 nmol/l, P < 0.001) and heart rate (7.2 beats/min, P = 0.035) increased significantly after the
188 - 5,497 beats/min/mm Hg vs. 19,519 +/- 4,653 beats/min/mm Hg vs. 17,530 +/- 4,678 beats/min/mm Hg; p
189 est (rate-pressure product: 22,157 +/- 5,497 beats/min/mm Hg vs. 19,519 +/- 4,653 beats/min/mm Hg vs.
190 - 4,653 beats/min/mm Hg vs. 17,530 +/- 4,678 beats/min/mm Hg; p = 0.004), but peak CBF was lowest in
191 rt rate (56 +/- 2 beats/min vs. 50.1 +/- 0.4 beats/min; p = 0.05), less backup pacemaker utilization
192 h ingredient defined by the number of cancer-beating molecules found therein.
193 tform to identify putative food-based cancer-beating molecules.
194 acid-binding experiments, comparing original BEAT, mutated BEAT, and "knobs-into-holes" interfaces, s
195 cessing of a pair of low frequency dual-comb beat notes.
196 slow to be consistent with the activation of beating observed at higher mantATP concentrations; this
197 e virtual self always moved in time with the beat of instrumental music, the virtual other moved eith
198 sible for transport in the cytoplasm and the beating of axonemes in cilia and flagella.
199 endothelium barrier function and spontaneous beating of cardiac muscle cells, which are important fun
200                                 Synchronized beating of cilia on multiciliated cells (MCCs) generates
201 s of energy-consuming behaviors, such as the beating of cilia or the production of surfactant.
202 motility using the polarized and coordinated beating of hundreds of motile cilia.
203 rientation of basal bodies, and synchronized beating of motile cilia.
204 eed as smoothly and reliably as the lifelong beating of our heart or filtration of blood by our kidne
205    Detailed phase mapping during the initial beats of VT/VF identified one or more rotors that were l
206 nformed prescribing and clinical trials were beaten off the mark by social media, rumors, and panic i
207 third ventricles and deceleration of ciliary beating on ependymal cells lining the ventricular walls.
208 l communities are commonly extracted by bead-beating or ultra-sonication, but both showed varying yie
209 tion in CPVT, and whether the arrhythmogenic beats originate from the conduction system or the ventri
210 tion (0.16+/-0.07 versus 0.14+/-0.06 mL O(2)/beat, P<0.0001) despite unchanged stroke work (P=0.24),
211 nsumption (0.15+/-0.06 versus 0.16+/-0.07 mL/beat, P=0.003) and improved myocardial efficiency (43.7+
212 al phenotypic end points included effects on beating parameters and intracellular Ca2+ flux in CMs an
213  = 19.8 Hz versus control = 15.8 Hz) ciliary-beat pattern.
214                 We observed normal intrinsic beating patterns under control conditions in TNNI1 R37C(
215 5% confidence interval: 1.03 to 1.45] per 10 beat per min [bpm] increase, p = 0.01) were independentl
216 20+/-2 mL ( P<0.001) and heart rate of 7+/-2 beats per minute (bpm) ( P<0.001).
217  rate-based therapy delivery for rates >=250 beats per minute and morphology discrimination for rates
218  mean increase in heart rate was 23.9+/-11.4 beats per minute and the mean decreases in systolic and
219 illator treated ventricular tachycardia >250 beats per minute) in follow-up was assessed by Cox regre
220 heart rates (98.6+/-19.4 versus 112.0+/-20.3 beats per minute).
221  atrial rate during tachycardia was 170+/-21 beats per minute, and the mean left ventricular ejection
222 rmed under constant and pulsatile flow at 60 beats per minute, thus yielding eight distinct testing c
223  with a CIED with heart rates as high as 102 beats per minute.
224 logy discrimination for rates >=200 and <250 beats per minute.
225 eased incrementally until HR increased by 30 beats per minute.
226  with a CIED with heart rates as high as 102 beats per minute.
227 controls (0.156+/-0.133 versus 0.254+/-0.166 beats per minute/[isoproterenol ng/mL]; P<0.001).
228  Peak VO(2) and HR (117+/-15 versus 156+/-15 beats per minute; P<0.001) were lower in HFpEF than seni
229 had lower peak HRs (122+/-14 versus 156+/-15 beats per minute; P<0.001).
230 fied brain regions that may be involved with beat perception and representation.
231 s the timing of beats, yet current models of beat perception do not specify how this is neurally impl
232                                              Beat perception offers cognitive scientists an exciting
233        Rise time, action potential duration, beat period, and triangulation were used to quantify com
234 onalized MAP scores as the proportion of MAP beats predicting each end point.
235             Custom software modelled beat-by-beat pressure (photoplethysmography) and R-R interval in
236                             The cantilevers' beating prevents the initial stage of bacterial adhesion
237 own anticipation influences ongoing auditory beat processing in beta-band networks.
238 jects underwent a 6 min recording of beat-by-beat pulmonary artery diastolic pressure (PAD), stroke v
239 respiratory complex I, reducing cardiac-cell beat rate, with prolonged exposure resulting in cell dea
240 , contraction and relaxation parameters, and beat rate.
241 m/mM), calcium changes markedly affected the beating rate (46 bpm/mM ionized calcium without autonomi
242 ccurring in these patients affect sinus node beating rate and could be responsible for severe bradyca
243 on of the heart involves a sharp increase in beating rate and significant enhancement of contractilit
244 ection-assisted diffusion at high mechanical beating rates.
245                                        While BeAT regulation is well understood in adults, the critic
246  sequences were compared in terms of beat-to-beat restitution (btb-ER) and of the collections of sER
247 s is reduced, and the time series of beat-to-beat RR intervals (RRIs) become highly non-stationary.
248 e gain medium and control over the intermode beat synchronization enable transform-limited picosecond
249 1) simple tapping, 2) two-person alternating beat-tapping, and 3) two-person alternating rhythm-clapp
250 egarding the tissue origin of the arrhythmic beats that initiate ventricular tachycardia, and regardi
251 y information and for some proteins even did beat the best.
252 rprisingly, in cells that exhibited periodic beating, the frequency of beating was similar for all le
253 well-suited for mechanoregulation of ciliary beating through physical collisions with the CP.
254 ere length-dependent manner and on a beat-by-beat time-scale.
255  single sER (type 2), and high rates (~20 ms/beat) to hysteretic btb-ER under periodic pacing and to
256                      Despite the key role of beat-to-beat action potential (AP) variability in the on
257                Cardiac alternans refers to a beat-to-beat alternation in contraction, action potentia
258  the cellular level alternans is observed as beat-to-beat alternations in contraction, action potenti
259                                              Beat-to-beat analysis of up to 4 full days of electrocar
260  I(NaL) inhibition significantly reduced the beat-to-beat and short-term variabilities of APD.
261  of Ca(2+) -activated Cl(-) channels reduced beat-to-beat AP alternations, but prolonged APD and fail
262  daily living, especially when measured on a beat-to-beat basis.
263 reflex sensitivity (BRS) with ambulatory and beat-to-beat blood pressure (BP).
264 microneurography, 12 paired recordings), and beat-to-beat blood pressure (BP; photoplethysmography) d
265  temporal sequence over 5 years by recording beat-to-beat blood pressure and R-R intervals over 10 mi
266 s were instrumented for electrocardiography, beat-to-beat blood pressure, respiratory rate, CO-Modelf
267 tic nerve activity (MSNA; microneurography), beat-to-beat BP (photoplethysmography) and heart rate (e
268                                   Ambulatory beat-to-beat BP was recorded using a portable device for
269 nd frequency domain HRV indices, BRS, office beat-to-beat BP, and heart rate (HR) were measured for 1
270 ons among HRV, HR, BRS and ambulatory/office beat-to-beat BP.
271 orresponding to progressively higher rate of beat-to-beat CL changes.
272                                              Beat-to-beat heart period, systolic blood pressure, and
273 tion of the instantaneous variability of the beat-to-beat heart rate): spontaneous swallowing 12.02 +
274 4% for daytime, sleeping, 24-hour and office beat-to-beat measurements.
275       AP sequences were compared in terms of beat-to-beat restitution (btb-ER) and of the collections
276 ctuations is reduced, and the time series of beat-to-beat RR intervals (RRIs) become highly non-stati
277 ticity and intranodal conduction, leading to beat-to-beat variability and reentry.
278 mal culture conditions, MFS CMs show a lower beat-to-beat variability compared to corrected CMs using
279                        HIV+ men have greater beat-to-beat variability in QT interval (QTVI) than HIV-
280 d afterdepolarizations (DADs) and has a high beat-to-beat variability of repolarization (BVR) during
281 e of delayed afterdepolarizations (DADs) and beat-to-beat variability of repolarization (BVR) was hig
282 nd intranodal conduction, leading to beat-to-beat variability and reentry.
283 ure conditions, MFS CMs show a lower beat-to-beat variability compared to corrected CMs using multi e
284                HIV+ men have greater beat-to-beat variability in QT interval (QTVI) than HIV- men, es
285 epolarizations (DADs) and has a high beat-to-beat variability of repolarization (BVR) during adrenerg
286 ayed afterdepolarizations (DADs) and beat-to-beat variability of repolarization (BVR) was higher in t
287  dynein ATPases direct ciliary and flagellar beating via adenosine triphosphate (ATP) hydrolysis.
288 P45 supports mammalian ciliary and flagellar beating via an adenine nucleotide homeostasis module.
289                       The frequency of cilia beating was estimated from the motion of the cell body a
290 exhibited periodic beating, the frequency of beating was similar for all lengths with only a slight d
291                                    Premature beats were introduced at 10-ms steps during intrinsic rh
292 extracellular calcium fluxes during a single beat which is away from homeostatic balance.
293 r processes such as cell division or ciliary beating while performing their function.
294 ment with experiments, including the quantum beats, while revealing the interplay between excitons an
295 iated patch in their hind wing clicks as the beating wing rotates and bends.
296 tor cortices were apparent after isochronous beats with anticipation in all conditions, generally rep
297 trate video-rate 4D microscopic imaging of a beating Xenopus embryo heart at a rate of 30 volumes/s.
298 ieve the motor system predicts the timing of beats, yet current models of beat perception do not spec
299 d 3D + time video time-lapse datasets of the beating zebrafish heart.
300 -time blood flow and calcium dynamics in the beating zebrafish heart.

 
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