ライフサイエンスコーパス: beat-to-beat

1 ivation and amplitude of release varied from beat to beat.

2 Ca(2+) release from these sites varied from beat to beat.

3 ts indicating a change in contractility from beat to beat.

4 , increasing refractoriness, and diminishing beat-to-beat action potential variability.

5 cium entry from extracellular spaces for its beat-to-beat activation.

6 rm for addressing heart disease and enabling beat-to-beat adaptation of cardiac pacing in response to

7 ng, and were not merely passive responses to beat-to-beat alterations in AP; 3) the complex Ca(2+) dy

8 t modulation of stroke volume (SV) caused by beat-to-beat alterations in left ventricular filling, wh

9 is study appears to be largely the result of beat-to-beat alternans of [Ca2+]i.

10 own that small depolarizing pulses produce a beat to beat alternation in the amplitude of the systoli

11 Beat-to-beat alternation (alternans) of the cardiac acti

12 tive trigger event for cardiac reentry, is a beat-to-beat alternation in membrane potential and calci

13 A beat-to-beat alternation in the action potential duratio

14 heart, in which rapid stimulation elicits a beat-to-beat alternation in the action potential duratio

15 Ca(2+) alternans, a beat-to-beat alternation in the amplitude of the [Ca(2+)

16 aim of this work was to investigate whether beat-to-beat alternation in the amplitude of the systoli

17 Beat-to-beat alternation in the cardiac intracellular Ca

18 Alternans, a condition in which there is a beat-to-beat alternation in the electromechanical respon

19 henomenon of T-wave alternans (TWA) (i.e., a beat-to-beat alternation in the morphology and amplitude

20 complementary mechanism, CTA is caused by a beat-to-beat alternation in the number of refractory RyR

21 Alternans, the beat-to-beat alternation in the shape of cardiac electri

22 n the surface ECG was explained primarily by beat-to-beat alternation in the time course of cellular

23 CLs of 300 to 250 ms caused more pronounced beat-to-beat alternation of action potential duration (A

24 Although this alternation results from beat-to-beat alternation of intracellular Ca(2+) wave pr

25 uced mild to moderate TWA principally due to beat-to-beat alternation of repolarization of cells in t

26 +) alternans is a potentially arrhythmogenic beat-to-beat alternation of the amplitude of the action

27 ns (TWA), an ECG phenomenon characterized by beat-to-beat alternation of the morphology, amplitude, a

28 a period doubling bifurcation, manifest as a beat-to-beat alternation, or alternans, of cardiac actio

29 ICaCC played a decisive role in shaping the beat-to-beat alternations in AP morphology observed duri

30 Cardiac alternans--periodic beat-to-beat alternations in contraction, action potenti

31 At the cellular level alternans manifests as beat-to-beat alternations in contraction, action potenti

32 Cardiac alternans, described as periodic beat-to-beat alternations in contraction, action potenti

33 ever, it remains an unresolved issue whether beat-to-beat alternations in intracellular Ca(2+) ([Ca(2

34 Beat-to-beat alternations of AP morphology and CaT ampli

35 propagation became completely variable from beat to beat and thus transformed into fibrillatory cond

36 Cardiac myocyte intracellular calcium varies beat-to-beat and calmodulin (CaM) transduces Ca2+ signal

37 as characterized by significant increases in beat-to-beat atrial CL, MAPD, and diastolic interval var

38 Furthermore, the beat-to-beat autonomic reflex control of HR was found to

39 ction to increases in myocardial demand on a beat-to-beat basis and mitochondrial calcium release dep

40 eleration of conduction was compensated on a beat-to-beat basis by an equal degree of slowing in the

41 Atrial flutter is modulated on a beat-to-beat basis by an interplay between the autonomic

42 gesting that NCX is regulated by Ca(2+) on a beat-to-beat basis during excitation-contraction couplin

43 tivity to baroreflex (CSB) were derived on a beat-to-beat basis from these data.

44 d mechanisms for the occurrence of EADs on a beat-to-beat basis have been proposed.

45 low probe placed around the aortic root on a beat-to-beat basis in seven anesthetized open-chested ca

46 egulate cardiac pacemaker cell function on a beat-to-beat basis remains unknown.

47 regulates CL in cardiac pacemaker cells on a beat-to-beat basis, and suggest a more realistic numeric

48 ther mitochondria take up Ca2+ rapidly, on a beat-to-beat basis, or slowly, by temporally integrating

49 f cardiac muscle contraction is altered on a beat-to-beat basis.

50 adjust cardiac performance or perfusion on a beat-to-beat basis.

51 ventricular filling with cardiac output on a beat-to-beat basis.

52 Measured beat-to-beat BF time history derived from real-time phas

53 al Doppler ultrasound along with noninvasive beat-to-beat blood pressure (BP), heart rate, and transc

54 ension, and orthostatic hypotension based on beat-to-beat blood pressure methods in a population-repr

55 urements of heart rate (ECG) and noninvasive beat-to-beat blood pressure recording (Finapres), with 5

56 Beat-to-beat blood pressure responses to the Valsalva ma

57 Heart rate, noninvasive beat-to-beat blood pressure, and muscle sympathetic nerv

58 Beat-to-beat blood pressure, carotid ultrasonography at

59 od leading to vasovagal syncope we monitored beat-to-beat blood pressure, heart rate (HR) and forearm

60 Beat-to-beat BP, R-R interval and respiratory excursions

61 activity is essential in the maintenance of beat-to-beat Ca(2+) homeostasis in cardiac myocytes.

62 ive stress, also have deleterious effects on beat-to-beat [Ca(2+)](c) handling and excitation-contrac

63 We propose that beat-to-beat [Ca(2+)](i) transient alternans during isch

64 Compared with BsCaM-2, BsCaM-45 tracks the beat-to-beat Ca2+-CaM alterations more closely following

65 ulse contour analysis can be used to provide beat-to-beat cardiac output (CO) measurement.

66 c resonance imaging of brain and noninvasive beat-to-beat cardiovascular monitoring, we show that sti

67 Our newly developed assessment of beat-to-beat carotid diameters during baroreflex engagem

68 lays a significant role in the regulation of beat-to-beat CBF in humans.

69 early afterdepolarizations (EADs) result in beat to beat changes in the origin and direction of the

70 phic ventricular arrhythmias may result from beat to beat changes in wave propagation patterns initia

71 CaT alternans leads to complex beat-to-beat changes in Ca(2+)-regulated ion currents th

72 lts in acceleration and amplification of the beat-to-beat changes in cytosolic Ca(2+) in cardiomyocyt

73 Beat-to-beat changes in HR and MAP were recorded through

74 Spectral transfer function gain between beat-to-beat changes in left ventricular end-diastolic p

75 2 (n = 2 out of 5) showed persistent complex beat-to-beat changes in nodal line formation of DA assoc

76 Beat-to-beat changes in renal blood flow velocity (RBV;

77 hroughout the heart at high speeds producing beat-to-beat changes in the activation sequence.

78 total cellular fluorescence failed to track beat-to-beat changes of mitochondrial fluorescence.

79 nsidered an accurate technique for measuring beat-to-beat CO with limited risk to the patient.

80 a highly specialized role in regulating the beat-to-beat contraction of the heart.

81 and exchangers, but are actively involved in beat to beat control of cardiac function by neural and h

82 We investigated the beat-to-beat control of atrial flutter cycle length usin

83 t fluctuations in heart rate responsible for beat-to-beat control of heart activity, both at rest and

84 Analysis of multiscale complexity of beat-to-beat dynamics at high temporal resolution has po

85 or blood pressure, quantification of complex beat-to-beat dynamics using multiscale entropy was able

86 frequency, kurtosis, and higher degree of a beat-to-beat electrogram similarity than areas without o

87 DCM is associated with beat-to-beat fluctuations in QT interval that are larger

88 T-wave alternans (TWA) reflects beat-to-beat fluctuations in the electrocardiographic T-

89 Beat-to-beat haemodynamics (Modelflow), muscle sympathet

90 Beat-to-beat heart rate and blood pressure responses to

91 To investigate the effects of DR on beat-to-beat heart rate and diastolic blood pressure var

92 Supine, resting, 2-min beat-to-beat heart rate data were collected at the basel

93 Baseline, supine, resting beat-to-beat heart rate data were collected.

94 Beat-to-beat hemodynamic functions were determined nonin

95 metoprolol or propranalol) conditions, while beat-to-beat HR and BP were continuously measured.

96 al contractility is constantly changing from beat to beat in atrial fibrillation because of the influ

97 At 13 minutes, hemodynamics was analyzed beat-to-beat in the end-inspiratory and end-expiratory c

98 In these cardiomyocytes, which produce NO beat-to-beat, inhibition of mtNOS increased myocyte shor

99 decreased with decreases in the steady-state beat-to-beat interval (P = .0008).

100 The short-term variations of beat-to-beat interval exhibited strongly and consistentl

101 multiscale complexity dynamics) measures of beat-to-beat interval variability were analyzed in two m

102 including standard deviation of the average beat to beat intervals over a 5-minute period, percentag

103 he NOX4 embryos displayed much more variable beat-to-beat intervals (mean S.D. of beat-to-beat interv

104 , the distributions of the variations in the beat-to-beat intervals for all healthy subjects are desc

105 However, sequences of beat-to-beat intervals obtained from these recordings ar

106 ariable beat-to-beat intervals (mean S.D. of beat-to-beat intervals was 0.027 s/beat in control embry

107 fraction </= 45%) and sleep apnea underwent beat-to-beat measurement of SV by digital photoplethysmo

108 based techniques are less invasive and offer beat-to-beat measurements and excellent trending ability

109 ed neck chamber that was developed to enable beat-to-beat measurements of stroke volume using pulse-d

110 namics of the Starling mechanism, namely the beat-to-beat modulation of stroke volume (SV) caused by

111 a short, regular cycle length with identical beat-to-beat morphology, and the rest of the atria were

112 Beat-to-beat NO production and altered shortening by NOS

113 A by microneurography, and blood pressure by beat-to-beat noninvasive technique.

114 val and nonlinear analyses (newly developed, beat-to-beat nonlinear measurement of the repetitiveness

115 Dynamic, beat-to-beat or electrocardiogram-to-electrocardiogram,

116 We hypothesized that beat-to-beat oscillations in APD may explain AF substrat

117 arization instability, manifested by TWA and beat-to-beat oscillations of T-wave amplitudes at other

118 sing a semiautomated algorithm that measured beat-to-beat QT duration in 817 MADIT II patients.

119 We sought to test the hypothesis that beat-to-beat QT interval variability is increased in DCM

120 Beat-to-beat QT interval variability was measured by aut

121 ibit labile ventricular repolarization using beat-to-beat QT variability analysis.

122 udy was to determine the predictive value of beat-to-beat QT variability in heart failure patients ac

123 Beat-to-beat QT variability index (QTVI), log-transforme

124 noatrial node and atria play a major role in beat-to-beat regulation of the heart rate.

125 As expected, cardiac pacing eliminated beat-to-beat RR interval variability.

126 the fibrillatory cycle lengths with varying beat-to-beat sequences suggestive of unstable trajectori

127 Alternans is the periodic beat-to-beat short-long alternation in action potential

128 rnative mechanisms, we measured simultaneous beat-to-beat stroke volume (flow) using Doppler echocard

129 However, beat-to-beat stroke volume (SV) has not been assessed in

130 Accurate beat-to-beat stroke volume and BF were estimated using V

131 Beat-to-beat systolic BP, diastolic BP, and heart rate r

132 similar baseline characteristics but greater beat-to-beat TCL variability.

133 2+) uniporter catalyzes Ca(2+) uptake during beat-to-beat transients of mitochondrial free Ca(2+), wh

134 Thus, beat-to-beat V(m) fluctuations during late DD phase refl

135 phase to the DD later part, which exhibited beat-to-beat V(m) fluctuations with an amplitude of appr

136 Beat-to-beat validation of VMHDVCG-derived BF was perfor

137 Beat-to-beat variability in atrial cycle length (baselin

138 horter effective refractory periods, greater beat-to-beat variability of action potential durations,

139 horter effective refractory periods, greater beat-to-beat variability of action potential durations,

140 max boundaries was associated with increased beat-to-beat variability of conduction velocity and dire

141 Beat-to-beat variability of left-ventricular monophasic-

142 ease the action potential duration (APD) and beat-to-beat variability of repolarization (BVR) of APD

143 Excessive beat-to-beat variability of repolarization duration (BVR

144 assessed by spectral analysis of spontaneous beat-to-beat variability of RR and QT intervals from sta

145 fidence interval, 1.7 to 9.3), and decreased beat-to-beat variability of the heart rate (odds ratio,

146 heir effect on the beating frequency and the beat-to-beat variability seemed largely independent of t

147 ery near the pulmonary vein ostia, and their beat-to-beat variability was greater than control (1.93+

148 28S abbreviated refractoriness and increased beat-to-beat variability, leading to early afterdepolari

149 ice insertion, transmitral flow showed rapid beat to beat variation in each patient, from abnormal re

150 nchronous LV assistance produced significant beat to beat variation in filling indexes, but overall a

151 The cardiac beat-to-beat variation evoked at the moment of lung infl

152 Moreover, the beat-to-beat variation in arterial and throbbing events

153 Beat-to-beat variation in heart rate (f H ) has been use

154 A beat-to-beat variation in the cardiac action potential d

155 is crucial for stable heart rate and regular beat-to-beat variation.

156 The mechanism of the small beat-to-beat variations in cycle length of atrial flutte

157 whereas ERP restitution underlies temporal, beat-to-beat variations in refractoriness during rapid p

158 In these examples, we observed large beat-to-beat variations in the cell activation times, de

159 The maps showed incessantly changing beat-to-beat wave fronts and varying spatiotemporal beha

160 Ca(2+) transients from beat to beat were comparable in amplitude with identical

161 Forearm blood flow (FBF) was measured beat-to-beat with Doppler ultrasound, while saline or dr

162 hat varied its location and orientation from beat to beat, with the majority of ventricular myocardiu