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1 injected intravenously with trastuzumab 24 h before administration of ((111)In-DTPA)(n)-trastuzumab-(
2 ents, unlabeled free folic acid was injected before administration of (111)In-DTPA-folate to also ass
4 mg/kg intravenously) over the 45-min period before administration of 18F-FCWAY almost obliterated de
5 njection of 100 ng CX3CL1/fractalkine 30 min before administration of 400 ng DAMGO, 100 ng DPDPE or 2
7 bronchoprotective effect, DIs were performed before administration of a single dose of methacholine,
10 ells/mm3; placebo recipients, 657 cells/mm3) before administration of antiretroviral therapy (ART) an
16 CTH were diagnostic for Cushing disease (> 2 before administration of CRH and > 3 after administratio
21 f COX-2 before administration of OA alone or before administration of Etx and OA did not have any sig
26 otherapy strategy was used to debulk disease before administration of granulocyte colony-stimulating
27 mbers of circulating B and activated T cells before administration of HgCl2, and less autoreactivity
28 ppearance (active control group) about 1-2 h before administration of highly emetogenic chemotherapy.
32 istered neutralizing antibodies to TNF-alpha before administration of low, but hepatotoxic, doses of
33 ine (6 mg/kg, intraperitoneally) immediately before administration of LPS (0.83 mg/kg, intraperitonea
36 l in appearance (active control group) 1-2 h before administration of moderately emetogenic chemother
37 luteal phase of the menstrual cycle, 40 min before administration of NMDA (10 mg/kg b.wt., i.v.).
42 transit, and colonic transit were determined before administration of study drug and after 1 week on
45 vealed that binding of bacteria to E was <1% before administration of the bispecific reagent, but wit
48 nd 3 were tested on serum specimens obtained before administration of the first dose of IPV and 28-45
49 r viral load, which was assessed immediately before administration of the first dose through the 12th
51 agonist salvinorin A (0.01-1.8 mg/kg, i.v.) before administration of the KOR selective radiotracer [
53 de were performed in 11 normal male subjects before administration of the serotonin-releasing agent a
54 w that, if resistant populations are present before administration of therapy, treatments designed to
56 norphine taper followed by a week-long delay before administration of XR-naltrexone, consistent with
57 pooledhIgG, or mixing pooledhIgG with SScIgG before administration to tissues, significantly reduced
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