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1 tic spine formation and for cocaine-mediated behavioral sensitization.
2 equired for the development or expression of behavioral sensitization.
3 after chronic cocaine exposure that induced behavioral sensitization.
4 rug experience-dependent plasticity, such as behavioral sensitization.
5 function contributing to the development of behavioral sensitization.
6 e following repeated exposure, indicative of behavioral sensitization.
7 -nitroindazole [7-NI]) block cocaine-induced behavioral sensitization.
8 files of PTT and cocaine, both drugs produce behavioral sensitization.
9 changes in specific binding were related to behavioral sensitization.
10 nt manner that paralleled the development of behavioral sensitization.
11 ical areas are related to the development of behavioral sensitization.
12 n may occur only when drug treatments induce behavioral sensitization.
13 en reported to block psychostimulant-induced behavioral sensitization.
14 ronic treatment, indicating the emergence of behavioral sensitization.
15 ne (5 mg/kg) for 5 consecutive days produced behavioral sensitization.
16 nicotine-induced physiological, neural, and behavioral sensitization.
17 excitatory amino acids in the development of behavioral sensitization.
18 hanges in behavior ranging from addiction to behavioral sensitization.
19 aine-induced locomotor activity and occluded behavioral sensitization.
20 xpression of peripheral inflammation-induced behavioral sensitization.
21 d its extent strongly predicts the degree of behavioral sensitization.
22 efrontal cortex (PFC) prevented MPD elicited behavioral sensitization.
23 escending glutamate from PFC in MPD elicited behavioral sensitization.
24 ayed an enhanced response to cocaine-induced behavioral sensitization.
25 played a clear resistance to cocaine-induced behavioral sensitization.
26 that repeated administration of MPD induced behavioral sensitization.
27 RK) as a molecular substrate underlying this behavioral sensitization.
28 five days with daily 2.5 mg/kg MPD to elicit behavioral sensitization.
29 for plasticity underlying the development of behavioral sensitization.
30 Daily injections of cocaine in rats caused behavioral sensitization.
31 the formation of long-term facilitation and behavioral sensitization.
32 to which repeated cocaine injections produce behavioral sensitization.
33 positively correlated with the magnitude of behavioral sensitization.
34 erations depends critically on the extent of behavioral sensitization.
35 re was no correlation between S/I ratios and behavioral sensitization.
36 of experience-dependent plasticity including behavioral sensitization.
37 locomotor activity and also cocaine-induced behavioral sensitization.
38 teins, in a drug-induced phenomenon known as behavioral sensitization.
39 input, (2) chronic treatment of MDMA elicits behavioral sensitization, (3) chronic administration of
40 ibute to the neural adaptations that mediate behavioral sensitization, a model for core aspects of ad
41 roadaptation occurs during the expression of behavioral sensitization, a model of psychostimulant-ind
42 treatment regimen led to the development of behavioral sensitization, a separate set of animals (n=8
43 y developed strikingly different patterns of behavioral sensitization after daily treatment with low
45 ons from animals that did or did not exhibit behavioral sensitization after repeated methamphetamine
47 allenge, normal subjects showed a pattern of behavioral sensitization (an increase in dyskinetic-like
48 or trafficking to plasticity associated with behavioral sensitization, an animal model of drug addict
49 epetitive daily amphetamine injections cause behavioral sensitization and a significant change of cir
52 e of nNOS by 7-NI-attenuated ethanol-induced behavioral sensitization and completely blocked the rewa
53 city during short-term and intermediate-term behavioral sensitization and dishabituation in a semi-in
56 s been shown to effectively induce long-term behavioral sensitization and synaptic facilitation, is n
57 cocaine including escalation of drug intake, behavioral sensitization, and cocaine-primed reinstateme
58 ulation of GIR expression may be involved in behavioral sensitization, and GIR may play a role at the
59 psychostimulant administrations is known as behavioral sensitization, and is an indicator of a drug'
60 ffects of amphetamine, its ability to induce behavioral sensitization, and its ability to induce c-fo
62 ion to cocaine, there are sex differences in behavioral sensitization, and sensitization that develop
63 uisition and expression of context-dependent behavioral sensitization, as this behavior is blocked by
65 ergolide and ondansetron normalized not only behavioral sensitization, but also the increases in thes
66 pamine also failed to exhibit l-DOPA-induced behavioral sensitization, but this may be caused, at lea
67 A) receptor may contribute to l-DOPA-induced behavioral sensitization by facilitating adaptations wit
68 ittent treatment with amphetamine leads to a behavioral sensitization characterized in rats by an inc
69 evaluated for its effects on cocaine-induced behavioral sensitization, conditioned place preference (
70 morphine and cocaine on locomotor activity, behavioral sensitization, conditioned place preference,
71 OX(1), have demonstrated its involvement in behavioral sensitization, conditioned place-preference,
72 t to determine whether the expression of TDF behavioral sensitization could be prevented by the DA D1
73 RO5263397 reduced the expression of cocaine behavioral sensitization, cue- and cocaine prime-induced
74 esioning of PFC eliminated the expression of behavioral sensitization elicited by chronic MPD adminis
75 pamine receptor stimulation, we compared the behavioral sensitization elicited by repeated l-DOPA tre
76 d using Western blots in rats that developed behavioral sensitization following repeated amphetamine
77 hanced hyperlocomotor activity and increased behavioral sensitization following treatment with psycho
81 an endogenous neurotransmitter that mediates behavioral sensitization in Aplysia[1-3], induces long-t
82 Daily amphetamine administration resulted in behavioral sensitization in both Dbh+/- and Dbh-/- mice,
84 shown that the fruitfly Drosophila exhibits behavioral sensitization in response to repeated exposur
86 to the development and robust expression of behavioral sensitization in terms of vertical activity.
89 contingent cocaine injections, which lead to behavioral sensitization, increase AMPA receptor (AMPAR)
91 e network-level adaptations suggest that the behavioral sensitization induced by repeated psychomotor
92 hibitors and suggest that the development of behavioral sensitization is a uniform characteristic of
95 udies have demonstrated that cocaine-induced behavioral sensitization is associated with persistent f
96 he finding that cocaine- and ethanol-induced behavioral sensitization is associated with upregulation
99 ocaine administration to wild types produced behavioral sensitization, knock-outs exhibited no additi
100 duce forms of cellular plasticity related to behavioral sensitization may also contribute to addictio
102 tations compared with controls and exhibited behavioral sensitization of contralateral rotations indu
104 eated ethanol (EtOH) injections that induced behavioral sensitization on subsequent acquisition of Et
105 on cellular function that may be relevant in behavioral sensitization or cocaine self-administration.
106 propensity of a given rat to exhibit either behavioral sensitization or conditioned place preference
107 lter cocaine-induced psychomotor activation, behavioral sensitization, or conditioned place preferenc
108 l test environment, a protocol that produces behavioral sensitization, or in the home cage, a protoco
109 dbrain DA neurons, although not required for behavioral sensitization per se, may contribute to the a
112 ed the development but not the expression of behavioral sensitization produced by repeated administra
113 The data add support to the inference that behavioral sensitization represents not only a quantitat
114 are involved in induction and expression of behavioral sensitization respectively and glutamate from
115 opa-induced dyskinesia (LID) is a persistent behavioral sensitization that develops after repeated le
116 is the enhanced locomotor response known as behavioral sensitization that occurs with prolonged expo
117 ections of 2.5 mg/kg methamphetamine induced behavioral sensitization that was demonstrated (expresse
118 bens (NAc) neurons have been correlated with behavioral sensitization, the molecular pathways governi
124 cross-reactivity with chronic FRAs, and that behavioral sensitization to amphetamine is not accompani
125 n-like immunoreactivity (FRALI) accompanying behavioral sensitization to amphetamine was assessed in
126 nvestigate neural mechanisms associated with behavioral sensitization to amphetamine, we studied the
127 a variety of neuroeffectors are involved in behavioral sensitization to amphetamine-induced stereoty
128 cal studies suggested that the initiation of behavioral sensitization to cocaine and amphetamine invo
130 t that NT-3 contributes to the initiation of behavioral sensitization to cocaine by activating the Ra
132 signal transduction pathway in long-lasting behavioral sensitization to cocaine in rats, an animal m
148 en free access to high-fat diets demonstrate behavioral sensitization to D2/3R agonists but not to an
151 rtant role in the neuroplasticity underlying behavioral sensitization to L-DOPA, supporting considera
152 t only when mice developed context-dependent behavioral sensitization to morphine in which morphine t
161 (NMDARs) is essential for the development of behavioral sensitization to psychostimulants such as amp
162 stigated the role of PFC DA in mediating the behavioral sensitization to repeated administration of M
163 nd serotonin neurons of living flies blocked behavioral sensitization to repeated cocaine exposures.
164 elopment and particularly the persistence of behavioral sensitization to repeated l-DOPA treatment.
166 lphenidate (MPD) has been reported to elicit behavioral sensitization to their locomotor and stereoty
169 ocedures, limbic brain regions implicated in behavioral sensitization were assayed for extracellular
171 nucleus accumbens abolish the development of behavioral sensitization when they are administrated dur
172 triatopallidal neuronal activity facilitated behavioral sensitization, whereas decreasing excitabilit
173 ed behaviors known as 'reverse tolerance' or behavioral sensitization, which may model the behavioral
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