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1 ynthesis was blocked by aliskiren but not by benazepril.
2 ce response system in a 1:1 ratio to receive benazepril (20 mg) plus amlodipine (5 mg; n=5744) or ben
3 il (20 mg) plus amlodipine (5 mg; n=5744) or benazepril (20 mg) plus hydrochlorothiazide (12.5 mg; n=
4 ere treated with insulin, candesartan (ARB), benazepril (ACE inhibitor), or aliskiren (renin inhibito
6 There were 552 primary-outcome events in the benazepril-amlodipine group (9.6%) and 679 in the benaze
7 dose adjustment were 131.6/73.3 mm Hg in the benazepril-amlodipine group and 132.5/74.4 mm Hg in the
8 representing an absolute risk reduction with benazepril-amlodipine therapy of 2.2% and a relative ris
9 ine, but rates were significantly lower with benazepril and amlodipine in overweight patients (hazard
10 both benazepril and hydrochlorothiazide and benazepril and amlodipine, but rates were significantly
13 , primary event rates were similar with both benazepril and hydrochlorothiazide and benazepril and am
14 ment with single-pill combinations of either benazepril and hydrochlorothiazide or benazepril and aml
17 fects of a renin-angiotensin system blocker, benazepril, combined with amlodipine (B+A) or hydrochlor
18 ociated with the response of diastolic BP to benazepril (diastolic BP response: -7.4 mm Hg for subjec
19 l-amlodipine combination was superior to the benazepril-hydrochlorothiazide combination in reducing c
20 epril-amlodipine group (9.6%) and 679 in the benazepril-hydrochlorothiazide group (11.8%), representi
22 rs [SD 0.4]) because of superior efficacy of benazepril plus amlodipine compared with benazepril plus
23 of chronic kidney disease progression in the benazepril plus amlodipine group compared with 215 (3.7%
24 sease, angio-oedema was more frequent in the benazepril plus amlodipine group than in the benazepril
26 ular events to receive treatment with either benazepril plus amlodipine or benazepril plus hydrochlor
28 d that initial antihypertensive therapy with benazepril plus amlodipine was superior to benazepril pl
29 hronic kidney disease was peripheral oedema (benazepril plus amlodipine, 189 of 561, 33.7%; benazepri
30 43 (1%) patients who were lost to follow-up (benazepril plus amlodipine, n=70; benazepril plus hydroc
31 dipine group compared with 215 (3.7%) in the benazepril plus hydrochlorothiazide group (HR 0.52, 0.41
32 a, and hypotension were more frequent in the benazepril plus hydrochlorothiazide group than in the be
34 h benazepril plus amlodipine was superior to benazepril plus hydrochlorothiazide in reducing cardiova
35 dipine should be considered in preference to benazepril plus hydrochlorothiazide since it slows progr
36 nazepril plus amlodipine, 189 of 561, 33.7%; benazepril plus hydrochlorothiazide, 85 of 532, 16.0%).
40 ted 1447 hypertensive patients from a 3-year benazepril postmarket surveillance trial and genotyped t
41 were partially inhibited by candesartan and benazepril, whereas aliskiren produced complete inhibiti
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