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1 mmation in the lung of patients with chronic beryllium disease.
2 ould serve as a possible therapy for chronic beryllium disease.
3 ify both beryllium sensitization and chronic beryllium disease.
4 at position 69 (HLA-DPB1(Glu69)) in chronic beryllium disease.
5 response to beryllium and in progression to beryllium disease.
6 orted to be strongly associated with chronic beryllium disease.
8 osure in the workplace can result in chronic beryllium disease, a granulomatous lung disorder charact
9 for sarcoidosis and, in contrast to chronic beryllium disease, a non-E(69)-containing allele, HLA-DP
10 1) idiopathic pulmonary fibrosis, 2) chronic beryllium disease and sarcoidosis, 3) control subjects w
11 time between those who progressed to chronic beryllium disease and those who remained sensitized with
12 biopsies to determine progression to chronic beryllium disease as evidenced by granulomatous inflamma
13 LA DPB1 locus, a SNP associated with chronic beryllium disease, as well as HLA DPA1 alleles using the
14 ls with beryllium sensitization have chronic beryllium disease at the time of their initial clinical
15 me (ACE) genotype is associated with chronic beryllium disease (CBD) and disease severity, we studied
16 B1 gene (Glu(69)) is associated with chronic beryllium disease (CBD) and possibly beryllium sensitiza
17 ic and clinical similarities between chronic beryllium disease (CBD) and sarcoidosis suggest that sim
18 suggests a genetic predisposition to chronic beryllium disease (CBD) and sarcoidosis, which are clini
36 CII allele, HLA-DP2, are at risk for chronic beryllium disease (CBD), a debilitating inflammatory lun
37 ecific granulomatous inflammation of chronic beryllium disease (CBD), and compared it with that in he
38 ies demonstrate associations between chronic beryllium disease (CBD), beryllium sensitization (BeS),
39 beryllium-specific CD4+ T cells and chronic beryllium disease (CBD), which is characterized by the p
47 in idiopathic pulmonary fibrosis and chronic beryllium disease lung tissues and interacted with hepar
49 ryllium-specific CD4(+) T cells from chronic beryllium disease patients remain CD28-dependent, while
50 lar lavage (BAL) CD4(+) T cells from chronic beryllium disease patients to identify possible therapeu
51 blood and bronchoalveolar lavage of chronic beryllium disease patients up-regulate 4-1BB expression,
52 cell lines derived from the lungs of chronic beryllium disease patients, beryllium presentation to th
53 (+) T cells recruited to the lung in chronic beryllium disease recognize beryllium in an Ag-specific
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