ライフサイエンスコーパス: beta-FNA

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1 [3H]beta-FNA-labeled receptors were solubilized and purified

2 r ovary cells with high specificity, and [3H]beta-FNA-labeled receptors migrated as one broad band wi

3 monstrated that [3H]beta-funaltrexamine ([3H]beta-FNA) labeled the rat mu opioid receptor expressed i

4 ompletely eliminated covalent binding of [3H]beta-FNA, although these mutants bound beta-FNA with hig

5 idues did not affect covalent binding of [3H]beta-FNA.

6 ptor involved in the covalent binding of [3H]beta-FNA.

7 Since [3H]beta-FNA is a rigid molecule, the information will be ve

8 These results indicate that [3H]beta-FNA binds covalently to Lys233.

9 pioid receptor binding decreased 24 hr after beta-FNA injection and returned to control levels 11 d a

10 In pretrained animals, beta-FNA significantly impaired spatial memory retrieval

11 hermore, pretreatment with the mu antagonist beta-FNA (1.00-2.00 microg) attenuated antinociception i

12 Behaviorally, beta-FNA prevented morphine-induced loss of righting and

13 f [3H]beta-FNA, although these mutants bound beta-FNA with high affinity.

14 lesser degree by NTI, and was unaffected by beta-FNA.

15 id receptor antagonist, beta-funaltrexamine (beta-FNA) also results in opioid analgesia.

16 oid receptor antagonist beta-funaltrexamine (beta-FNA) into area CA3.

17 id receptor antagonist, beta-funaltrexamine (beta-FNA), prior to parturition interfered with the esta

18 id receptor antagonist, beta-funaltrexamine (beta-FNA), prior to systemic morphine injection.

19 mu receptor antagonist, beta-funaltrexamine (beta-FNA), was unilaterally infused into the PAG adjacen

20 e of either naltrexone, beta-funaltrexamine (beta-FNA, mu), nor-binaltorphamine (NBNI, kappa) or nalt

21 ts were pretreated with beta-funaltrexamine (beta-FNA; 15 mg/kg s.c), an irreversible mu-opioid recep

22 NTB, beta-FNA, and nor-BNI were unable to block the cardiopro

23 In the CPu of beta-FNA treated rats, morphine-induced c-Fos and JunB w

24 r selectivity, whereas the agonist effect of beta-FNA is clearly kappa opioid receptor (KOR) mediated

25 Infusion of beta-FNA near specific medial thalamic nuclei attenuated

26 Injections of beta-FNA into the CA3 region, but not into the ventricle

27 The amino acid residue of beta-FNA covalent incorporation was then determined by s

28 5a with the prototypic fumaroylamino opioid beta-FNA (1a) shows that they have similar MOR irreversi

29 Ultra-low-dose NTX, nor-BNI or beta-FNA selectively antagonizes high-efficacy excitator

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