ライフサイエンスコーパス: beta-actin

1 n and one of its major structural components beta-actin.

2 alization against housekeeping genes such as beta-actin.

3 l DNA: non-muscle myosin heavy chain IIA and beta-actin.

4 sed amounts of nuclear, but not cytoplasmic, beta-actin.

5 , consistent with known roles of cytoplasmic beta-actin.

6 mma-actin depolymerizes half as fast as does beta-actin.

7 , and a somewhat slower elongation rate than beta-actin.

8 used a sequence complementary to the mRNA of beta-actin.

9 ingle 5 in AS4.5 results in its release from beta-actin.

10 of the actin-related protein Arp1 and one of beta-actin.

11 tion of endothelial nitric oxide (eNOS) with beta-actin.

12 llular scaffolding proteins beta-catenin and beta-actin.

13 Peripheral axotomy triggers transport of the beta-actin 3'-UTR containing transgene mRNA into axons.

14 These data show for the first time that the beta-actin 3'-UTR is sufficient for axonal localization

15 les and stimulates translation driven by the beta-actin 3'UTR in neurites.

16 Sam68 binds to the mRNA of beta-actin (actb), an integral cytoskeletal component of

17 raldehyde-3-phosphate dehydrogenase (Gapdh), beta-actin (Actb), or beta2-microglobulin (beta2m) showe

18 , myelin-oligodendrocyte glycoprotein [MOG], beta-actin [ACTB], thymosin beta-10 [TB10], and superior

19 ion status of the CpG island of the modified beta-actin allele in differentiated but not in undiffere

20 The modified beta-actin allele was expressed in undifferentiated embr

21 ession in mice heterozygous for the modified beta-actin allele.

22 multaneously followed transcription from the beta-actin alleles in real time and observed transcripti

23 os expressing fluorescent fusion proteins of beta-Actin, alpha-Catenin, and the ERM family member Moe

24 TDC WB can also clearly probe the intrinsic beta-actin, alpha-tubulin, and glyceraldehyde 3-phosphat

25 Gene silencing of beta-actin also affected mitochondrial DNA copy number a

26 Of great interest, ablation of beta-actin altered the ratio of globular actin (G-actin)

27 ACTB encodes beta-actin, an abundant cytoskeletal housekeeping protei

28 Gs glyceraldehyde-3-phosphate dehydrogenase, beta actin and adenosine triphosphate synthase subunit t

29 wed that Stain-Free staining was superior to beta-actin and as good as or better than Ponceau S stain

30 (+/-5000 bps) or at negative control genes (beta-actin and beta-globin).

31 Phosphorylated HtrA2/Omi therefore cleaves beta-actin and decreases the amount of filamentous actin

32 Nevertheless, double-mutant mice lacking beta-actin and expressing fascin-2 p.R109H have a more s

33 This process requires both beta-actin and gamma-actin as knock-out mice lacking eit

34 Fascin-2 binds beta-actin and gamma-actin filaments with similar affini

35 Ubiquitously expressed beta-actin and gamma-actin isoforms play critical roles

36 ich in turn display altered fractionation of beta-actin and gamma-tubulin.

37 beta-actin mRNA depletes axons of endogenous beta-actin and GAP-43 mRNAs and attenuates both in vitro

38 re haploinsufficient for axonal transport of beta-actin and GAP-43 mRNAs and for regeneration of peri

39 ta-actin mRNA 3'UTR competes with endogenous beta-actin and GAP-43 mRNAs for binding to ZBP1 and axon

40 ddress the functions of axonally synthesized beta-actin and GAP-43 proteins.

41 aracterized axonal mRNAs in model organisms, beta-actin and GAP43, within hESC-neuron projections usi

42 Here, beta-actin and GAPDH were evaluated as loading controls

43 sence of serum, and the expression levels of beta-actin and human antigen R (HuR) were analyzed by us

44 and iodixanol gradient analysis suggest some beta-actin and non-muscle myosin heavy chain IIA reside

45 ase, leading to excessive S-nitrosylation of beta-actin and possibly profilin.

46 of the putative ABSs, specifically bound to beta-actin and prevented eNOS association with beta-acti

47 previously determined structures of profilin-beta-actin and profilin-alpha-actin.

48 ss spectroscopy confirmed high expression of beta-actin and scinla, moderate expression of scinlb, an

49 these elevations were not observed for SPION-beta-actin and SPION-Ran.

50 Here, we establish that beta-actin and the actin crosslinking protein fascin-2 c

51 icient production of cystoskeletal proteins (beta-actin and vimentin) containing bicyclo[6.1.0]nonyne

52 mRNA (SPION-cfos) or beta-actin mRNA (SPION-beta-actin) and to sODN with random sequence (SPION-Ran)

53 ctin, transforming growth factor (TGF) beta1/beta-actin, and FOXP3/beta-actin than did IgG4-negative

54 o transfer include those encoding GFP, mouse beta-actin, and human Cyclin D1, BRCA1, MT2A, and HER2.

55 sminogen and plasmin to bind to cell surface beta-actin, and the truncated kringle 5 in AS4.5 results

56 gamma-actin was translated more slowly than beta-actin, and this slower processing resulted in the e

57 Although ubiquitous beta-actins are essential for cell survival during early

58 We show that myosin II and alpha- and beta-actins are present in the nuclei of colonic smooth

59 ctrometry and immunoprecipitation identified beta-actin as a protein that is specifically deglutathio

60 acellular signal-regulated kinase (ERK 1/2), beta-actin as well as p21/WAF/Cip1 levels were determine

61 o significantly reduced the increase in eNOS-beta-actin association and protein tyrosine nitration.

62 To study whether eNOS-beta-actin association contributes to increased peroxyni

63 d an eNOS mutant that exhibited reduced eNOS-beta-actin association, decreased NO production, and inc

64 ia resulted in significant increases in eNOS-beta-actin association, eNOS activity, and protein tyros

65 revented hyperoxia-induced increases in eNOS-beta-actin association, eNOS activity, NO and peroxynitr

66 ng endothelial monolayer and attenuates eNOS-beta-actin association, peroxynitrite formation, endothe

67 duce a synthesis- and ZBP1 binding-dependent beta-actin asymmetry but with opposite polarities.

68 refore, Shh induces the local translation of beta-actin at the growth cone.

69 actin interaction were inhibited by reducing beta-actin availability or by using a synthetic peptide

70 th real-time RT-PCR for expression levels of beta-actin, Bax, Bcl-xl, Bcl-2 and TNFR1 genes with mean

71 y the cytomegalovirus early enhancer/chicken beta-actin/beta-globin promoter into the Rosa26 locus, r

72 al alanine was unable to interfere with eNOS-beta-actin binding and to prevent beta-actin-induced cha

73 together, this novel finding indicates that beta-actin binding to eNOS through residues 326-333 in t

74 ybrid cytomegalovirus (CMV) enhancer/chicken-beta-actin (CBA) transduced both Muller and ganglion cel

75 orff-perfused mouse heart preparations, Cx43/beta-actin complexing is disrupted during acute ischemia

76 ubunit and beta-tubulin, but very little for beta-actin, consistent with the morphology of their tips

77 beta-Actin contains an Asp(1)-Asp(2)-Asp(3) and Val(10)

78 Colocalization of PDI and beta-actin could be abolished by the membrane-permeable

79 , we provided evidence that PDI binds to the beta-actin Cys(374) thiol.

80 ixed-cell microscopy of wild-type and mutant beta-actin demonstrates that incorporation of actin mono

81 The tagged proteins include alpha tubulin, beta actin, desmoplakin, fibrillarin, nuclear lamin B1,

82 previously reported that antibodies against beta-actin did not reliably identify differences in samp

83 exhibited foci of R2* elevation that matched beta-actin-expressing endothelia in the vascular wall.

84 In wild-type mouse embryos, beta-actin expression was prominent in the kidney, heart

85 nslation system, we found that inhibition of beta-actin folding by PDCL3 can be relieved by exchangin

86 ity in vitro and fascin-2 does not depend on beta-actin for localization in vivo.

87 Arp7A, which has 43% sequence identity with beta-actin, forms a complex with the cytoskeletal protei

88 PAmKate-transferrin receptor, and PAmCherry1-beta-actin fusion constructs, revealing correlations bet

89 mmunogold labeling of tropomyosin, spectrin, beta-actin, gamma-actin, espin, and prestin.

90 that regenerating spinal cord axons contain beta-actin, GAP-43, Neuritin, Reg3a, Hamp, and Importin

91 evious studies have shown that disruption of beta-actin gene critically impacts cell migration and mo

92 The beta-actin gene expression was analyzed at the transcrip

93 cassette into the 3'-untranslated region of beta-actin gene led to widespread but not ubiquitous lac

94 mouse lacking beta-actin protein by editing beta-actin gene to encode gamma-actin protein, and vice

95 e amount of genomic DNA quantitated with the beta-actin gene was almost 20 times lower in dry samples

96 The expression of the beta-actin gene was downregulated in the stroma of the s

97 the 3' untranslated region of the essential beta-actin gene.

98 For endogenous beta-actin genes in mouse embryonic fibroblasts, we obse

99 FP) was primarily in neurons, whereas scAAV9/beta-actin-GFP drove transgene expression in astrocytes.

100 detected in NeuN(+) neurons, whereas scAAV9/beta-actin-GFP drove transgene expression in GFAP(+) ast

101 ng neonatal rat hair cells in culture with a beta-actin-GFP fusion, and evidence was found that actin

102 itoring incorporation of inducibly expressed beta-actin-GFP in adult mouse hair cells in vivo and by

103 hair cells in vivo and by directly measuring beta-actin-GFP turnover in explants.

104 ods confirmed this: in hair cells expressing beta-actin-GFP we bleached fiducial lines across hair bu

105 t 1, DEAD (Asp-Glu-Ala-Asp) box polypeptide, beta-actin, glyceraldehyde 3-phosphate dehydrogenase (G3

106 ld in Cln3(Deltaex7/8) mice receiving scAAV9/beta-actin-hCLN3 versus scAAV9/MeCP2-hCLN3 after a singl

107 ld in Cln3(Deltaex7/8) mice receiving scAAV9/beta-actin-hCLN3 versus scAAV9/MeCP2-hCLN3.

108 s) injection of scAAV9/MeCP2-hCLN3 or scAAV9/beta-actin-hCLN3, with green fluorescent protein (GFP)-e

109 ation reporter analysis reveals that de novo beta-actin hotspots colocalize with docked RNA granules

110 lammatory response in DSS-induced colitis in beta-actin-hPepT1 and villin-hPepT1 transgenic mice, ind

111 nduced more severe levels of inflammation in beta-actin-hPepT1 transgenic mice than wild-type litterm

112 Nod2-/-, beta-actin-hPepT1 transgenic/Nod2-/-, and villin-hPepT1

113 gher expression ratios of interleukin (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transfor

114 tios of interleukin (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transforming growth factor

115 presented the same samples and therefore the beta-actin immunoblot was reused.

116 At 4 h after estradiol treatment, both beta-actin immunofluorescence and filopodial spines were

117 was used to visualize spine shape, and then beta-actin immunoreactivity was used as a semiquantitati

118 In the present study, we used both beta-actin immunostaining and Golgi staining to visualiz

119 expression pattern of LIMK-1, cofilin-1, and beta-actin in all the experimental groups by semiquantit

120 gration defects upon conditional ablation of beta-actin in highly motile T cells.

121 cate that increased association of eNOS with beta-actin in PAEC contributes to hyperoxia-induced incr

122 These data support an essential role for beta-actin in regulating cell migration and gene express

123 that NKCC1 interacted with beta-tubulin and beta-actin in spinal cords.

124 We propose that loss of beta-actin in the corneal stroma might be a triggering f

125 mine the expression levels and regulation of beta-actin in the stroma of keratoconus (KC) and normal

126 of LIMK-1, cofilin-1, phospho-cofilin-1, and beta-actin in the whole brain lysates as well as formati

127 ta-actin and prevented eNOS association with beta-actin in vitro.

128 gtin, and refold the physiological substrate beta-actin in vitro.

129 ase domain and that association of eNOS with beta-actin increases eNOS activity and nitric oxide (NO)

130 ously reported that association of eNOS with beta-actin increases eNOS activity and NO production in

131 with eNOS-beta-actin binding and to prevent beta-actin-induced changes in NO and superoxide formatio

132 Peptide 326 also prevented beta-actin-induced decrease in superoxide formation and

133 In the present study, we found that beta-actin-induced increase in NO production was accompa

134 Disruption of eNOS-beta-actin interaction in endothelial cells using ABS pe

135 We found that disruption of eNOS-beta-actin interaction prevented hyperoxia-induced incre

136 to increased peroxynitrite production, eNOS-beta-actin interaction were inhibited by reducing beta-a

137 rporation of (15)N-labelled protein and EGFP-beta-actin into nascent stereocilia.

138 gamma-actin are highly homologous, but only beta-actin is amino-terminally arginylated in vivo, whic

139 We have previously reported that beta-actin is associated with eNOS oxygenase domain and

140 We demonstrate that beta-actin is associated with YY1 specifically in the nu

141 inked to the 3' untranslated region (UTR) of beta-actin is delivered into cells fated to become the r

142 Our results show that beta-actin is downregulated in the corneal stroma of pat

143 ggest that the essential in vivo function of beta-actin is provided by the gene sequence independent

144 ckout (Actb(-/-)) mice and demonstrated that beta-actin is required for early embryonic development.

145 alysis and in vitro validation revealed that beta-actin is the receptor of C7H2 in the tumor cells.

146 organization and induction of the nonmuscle, beta actin isoform.

147 We generated whole-body beta-actin-knockout (Actb(-/-)) mice and demonstrated th

148 e growth impairment and migration defects in beta-actin-knockout primary mouse embryonic fibroblasts

149 at significant levels (expression >1% of the beta-actin level).

150 mice at P4, 5 months, and 12 months, whereas beta-actin levels were significantly decreased at P4.

151 in fluorescence signal intensity between the beta-actin LNA probe and a biotinylated, nonspecific con

152 and protein synthesis-dependent increase in beta-actin localization in growth cones.

153 vivo to regulate actin filament properties, beta-actin localization, and lamella formation in motile

154 As beta-actin-MBS was ubiquitously expressed, we could uniq

155 rons were produced by tamoxifen treatment of beta-actin-MerCreMer/LpL(flox/flox) mice.

156 hybridization to demonstrate that dendritic beta-actin messenger RNA (mRNA) and ribosomes are in a m

157 ipt in vitro and in vivo SHAPE-MaP for human beta-actin messenger RNA that revealed similar global fo

158 for the thiol-disulfide rearrangement in the beta-actin molecule of adhering cells.

159 complementary to c-fos mRNA (SPION-cfos) or beta-actin mRNA (SPION-beta-actin) and to sODN with rand

160 Ns) with sequence complementary to c-fos and beta-actin mRNA (SPION-cfos and SPION-beta-actin, respec

161 a reporter mRNA with the axonally localizing beta-actin mRNA 3'UTR competes with endogenous beta-acti

162 This GFP-3'-beta-actin mRNA accumulates in injured PNS axons before

163 for assessing the functional consequences of beta-actin mRNA and protein localization by tethering th

164 P1), an mRNA-binding protein that transports beta-actin mRNA and releases it for local translation up

165 Further, our work demonstrates that beta-actin mRNA and ribosomes are in a masked state that

166 that the two proteins cooperate to localize beta-actin mRNA and sought to address where and how this

167 not Cal PA-XFS, induced degradation of host beta-actin mRNA and suppressed host protein synthesis, s

168 cin D (actD) on the interactions of HuR with beta-actin mRNA and with poly(A)+ mRNA at both native an

169 e of these transcripts exceeded the level of beta-actin mRNA by more than 150-fold.

170 duction of a reporter mRNA with the 3'UTR of beta-actin mRNA competes with endogenous mRNAs for bindi

171 mRNA, and introducing GFP with the 3'UTR of beta-actin mRNA depletes axons of endogenous beta-actin

172 length of axons, while overall depletion of beta-actin mRNA from DRGs decreased the number of axon b

173 for netrin-1 stimulated local translation of beta-actin mRNA in axonal growth cones.

174 s been implicated in the axonal transport of beta-actin mRNA in both primary and transformed neuronal

175 expression level and spatial distribution of beta-actin mRNA in HeLa cells and to observe the real-ti

176 ls and to observe the real-time transport of beta-actin mRNA in mouse embryonic fibroblasts.

177 We found that beta-actin mRNA in primary fibroblasts localizes predomi

178 or the transport and/or local translation of beta-actin mRNA in the growth cones of motor neurons.

179 ZBP1 colocalizes with nascent beta-actin mRNA in the nucleus but is predominantly a cy

180 mRNA increased length and axonally targeted beta-actin mRNA increased branching of sensory axons gro

181 ve developed a transgenic mouse in which all beta-actin mRNA is fluorescently labeled.

182 have provided the first direct evidence that beta-actin mRNA is present in SMN cytoplasmic complexes

183 hydrogenase, wild-type HSV infection reduced beta-actin mRNA levels to between 20 and 30% of those in

184 transfected with any of three eIF4H siRNAs, beta-actin mRNA levels were indistinguishable in infecte

185 Furthermore, BDNF-stimulated beta-actin mRNA localization was attenuated in Zbp1-defi

186 equence element necessary and sufficient for beta-actin mRNA localization.

187 a-actin positively correlated with c-fos and beta-actin mRNA maps obtained from in situ hybridization

188 llowing detection and localization of single beta-actin mRNA molecules in various mouse tissues.

189 Ribosomes and single beta-actin mRNA motility increase after stimulation, ind

190 Importantly, beta-actin mRNA near focal adhesions exhibited sub-diffu

191 duct of a chimeric green fluorescent protein-beta-actin mRNA showed similar accumulation or depletion

192 studies indicate that axonal translation of beta-actin mRNA supports axon branching and axonal trans

193 " in the 3' untranslated region (UTR) of the beta-actin mRNA that is important for its localization a

194 hat the zipcode-binding protein 1 transports beta-actin mRNA to the focal adhesion compartment, where

195 Inhibition of axonal beta-actin mRNA translation disrupts arbor dynamics prim

196 n 1 (ZBP1) is required for the regulation of beta-actin mRNA transport and local translation underlyi

197 ZBP1 interaction with beta-actin mRNA was enhanced perinuclearly in neurons co

198 beta-Actin mRNA was targeted in whole A549 epithelial ce

199 de binding protein 1 (ZBP1) and ribosomes to beta-actin mRNA within subcellular compartments of prima

200 als the Src-dependent phosphorylation of the beta-actin mRNA zipcode binding protein 1 (ZBP1), which

201 ZBP1 and HuD bind the zipcode element in rat beta-actin mRNA's 3' UTR.

202 ZBP1 is needed for axonal localization of beta-actin mRNA, and introducing GFP with the 3'UTR of b

203 required ZBP1-mediated local translation of beta-actin mRNA, and therefore ZBP1 regulates protein sy

204 used these improvements to study endogenous beta-actin mRNA, which has 24xMS2 binding sites inserted

205 GAP-43 mRNA can deplete axons of endogenous beta-actin mRNA.

206 ced levels of a stabilizing factor (HuR) for beta-actin mRNA.

207 ucleoprotein-associated assembly factors and beta-actin mRNA.

208 ease or decrease axonal levels of endogenous beta-actin mRNA.

209 live cell method for imaging translation of beta-actin mRNA.

210 own for its role in the local translation of beta-actin mRNA.

211 d acute brain slices, we found that multiple beta-actin mRNAs can assemble together, travel by active

212 We show that beta-actin mRNAs freely access the entire nucleus and fe

213 Therefore, we imaged single beta-actin mRNAs tagged with MS2 stem loops colocalizing

214 limited spot, and the localization of single beta-actin mRNAs was measured in space and time.

215 me occurs naturally in the 3'-UTR of Vg1 and beta-actin mRNAs.

216 tropomyosin complexes containing filamentous beta-actin, nonmuscle myosin-2B (NM-2B) constructs, and

217 Ca-form, phosphate release from polymerizing beta-actin occurs much more rapidly and extensively than

218 AFM image two specific components, BRG1 and beta-actin, of the human Swi-Snf ATP-dependent nucleosom

219 Live-imaging EGFP-beta-actin or dendra2-beta-actin reveal stable F-actin c

220 ing with siRNA-resistant constructs encoding beta-actin or GAP-43 proteins, but only if the mRNAs wer

221 eptides, for example, from the C terminus of beta-actin or NLSIII of the p53 protein, slide along DNA

222 of Stain-Free staining as an alternative to beta-actin or the protein stain Ponceau S showed that St

223 which hPepT1 expression was regulated by the beta-actin or villin promoters; colitis was induced usin

224 , moesin, plastin 1, lamin B1, vimentin, and beta-actin) or in protein biosynthesis (glutamyl-prolyl-

225 ding RhoA, dynamin-1, kinesin, beta-tubulin, beta-actin, oxysterol-binding protein (OSBP)-related pro

226 Formation of the beta-actin-PDI complex was mediated by integrin-dependen

227 of NO and superoxide formation from eNOS by beta-actin plays an important role in endothelial functi

228 ns, aberrant NDRG4 and BMP3 methylation, and beta-actin, plus a hemoglobin immunoassay.

229 or knockdown leads to a marked reduction in beta-actin polymerization and cytoskeleton formation.

230 C7H2 induces beta-actin polymerization and F-actin stabilization, lin

231 Disruption of estradiol-induced beta-actin polymerization with cytochalasin D attenuated

232 striatum; retention of SPION-cfos and SPION-beta-actin positively correlated with c-fos and beta-act

233 identified small molecule ligands that bound beta-actin present in cytoplasmic cell extracts of Ramos

234 and soluble forms and tested for binding of beta-actin present in Ramos B-cell extracts and for acti

235 Animals that received the SPION-beta-actin probe exhibited the highest R2* values, follo

236 ing the human BBS4 gene under control of the beta actin promoter.

237 ybrid cytomegalovirus (CMV) enhancer chicken beta-actin promoter (CAGGS) and full-length beta-globin

238 sitional independence is linked to the human beta-actin promoter and is most likely a result of its t

239 ssion of the fragments driven by the chicken beta-actin promoter at nearly identical levels.

240 , T2/Onc3, in which the CMV enhancer/chicken beta-actin promoter drives oncogene expression.

241 ere isolated from transgenic FVB mice with a beta-actin promoter driving firefly luciferase and green

242 TG mice that overexpressed GLUT12 under a beta-actin promoter were generated.

243 ss murine Ngb under the control of a chicken beta-actin promoter, resulting in enhanced Ngb expressio

244 orescent protein (GFP) using a small chicken beta-actin promoter.

245 biquitously express human PEDF driven by the beta-actin promoter.

246 vector with a modified CMV enhancer/chicken beta-actin promoter.

247 ansgenic mice were produced with the chicken beta-actin promoter.

248 omposite cytomegalovirus-IE enhancer/chicken beta-actin promoter.

249 erexpressed under the control of the chicken beta-actin promoter.

250 Tomato-ttk) driven by a constitutive chicken beta-actin promoter.

251 Overexpressing Sox2 from a constitutive (beta-actin) promoter induces the expression of the prone

252 the methyl-CpG-binding protein 2 (MeCP2) and beta-actin promoters to drive low versus high transgene

253 the methyl-CpG-binding protein 2 (MeCP2) and beta-actin promoters.

254 In particular, Shh stimulation increases beta-actin protein at the growth cone even when the cell

255 rate here that generation of a mouse lacking beta-actin protein by editing beta-actin gene to encode

256 icient neurons, which impaired enrichment of beta-actin protein in the growth cone.

257 Most notably, this treatment also restores beta-actin protein levels in axonal growth cones of SMN-

258 changes in newly synthesized HIF-1alpha and beta-actin protein levels mirror alterations in correspo

259 rat neurons, increasing axonal synthesis of beta-actin protein while decreasing axonal synthesis of

260 Decreasing axonal synthesis of beta-actin protein while increasing axonal synthesis of

261 tor; reported separately), nuclear myosin I, beta-actin (reported separately), calponin 3, and SIKE.

262 os and beta-actin mRNA (SPION-cfos and SPION-beta-actin, respectively) (14-22 nm).

263 Live-imaging EGFP-beta-actin or dendra2-beta-actin reveal stable F-actin cores with turnover and

264 ased on the use of a recently developed pN1p beta actin-rtTA2S-M2-IRES-EGFP vector (where IRES is an

265 We show that beta-actin's 3'-UTR can drive axonal localization of GFP

266 ng mutant fascin-2 (p.R109H) or mice lacking beta-actin share a common phenotype including progressiv

267 Immunofluorescence detection of beta-actin showed that it was absent in the KC fibroblas

268 ing protein 1 (ZBP1), which is necessary for beta-actin synthesis and growth cone turning.

269 We report that Netrin-1 triggers a burst of beta-actin synthesis at multiple non-repetitive sites, p

270 Local beta-actin synthesis in growth cones of developing axons

271 f plasticity and may be facilitated by local beta-actin synthesis, but dynamic information is lacking

272 wth factor (TGF) beta1/beta-actin, and FOXP3/beta-actin than did IgG4-negative MZL (p < 0.05).

273 cause a selective energy barrier relative to beta-actin that retards the equilibration between G- and

274 After beta-actin, the cytosolic brain isoform of creatine kina

275 he presence of a dominant-negative mutant of beta-actin, the repositioning of activated U2 genes is m

276 ulsion is accompanied by a local decrease in beta-actin; thus, both produce a synthesis- and ZBP1 bin

277 isolated mRNA was used for amplification of beta-actin to confirm the compatibility.

278 slices revealed immediate early induction of beta-actin transcription after depolarization.

279 cing factor KSRP, binds initially to nascent beta-actin transcripts and facilitates the subsequent bi

280 n (IL)-4/beta-actin, IL-10/beta-actin, IL-13/beta-actin, transforming growth factor (TGF) beta1/beta-

281 SDF-1, VEGF, and beta-actin up-regulation was detected at early time poin

282 on of stromal cell-derived factor-1 (SDF-1), beta-actin, vascular endothelial growth factor (VEGF), g

283 anization by the thiol-disulfide exchange in beta-actin via a redox-dependent mechanism.

284 We found that beta-actin was arginylated in vivo to regulate actin fil

285 reporter with the 3' untranslated region of beta-actin was attenuated with the Src family kinase-spe

286 The LNA bound to beta-actin was then stained using phycoerythrin-conjugat

287 The expression levels of HuD, ATG5, LC3, and beta-actin were determined by Western blot and quantitat

288 and integrin subunits alphav and beta5) and beta-actin were quantified by Western blotting at 0.5 an

289 ctions among FAK, VE-cadherin, vinculin, and beta-actin were simultaneously decreased, resulting in a

290 e cell, where it forms a disulfide bond with beta-actin when MEG-01 cells adhere via the alphaIIbbeta

291 e mRNA localization and local translation of beta-actin where the new actin participates in stabilizi

292 ion, attraction involves a local increase in beta-actin, whereas repulsion is accompanied by a local

293 active species leading to S-nitrosylation of beta-actin, which causes temporary inhibition of beta(2)

294 posed to hyperoxia causes S-nitrosylation of beta-actin, which increases formation of short actin fil

295 One gene with a 5'-CpG island is cytoplasmic beta-actin, which is an abundantly expressed protein and

296 at PDI forms a disulfide-bonded complex with beta-actin with a molecular mass of 110 kDa.

297 tent to which the association of filamentous beta-actin with these different tropomyosin cofilaments

298 BP1 and HuD bind to overlapping sites in the beta-actin zipcode, but they recognize different feature

299 binding protein 1 (ZBP1/IMP1/IGF2BP1) to the beta-actin zipcode.

300 terized the interaction between ZBP1 and the beta-actin zipcode.