ライフサイエンスコーパス: beta-adrenergic blocker

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1 s, angiotensin-converting enzyme inhibitors, beta-adrenergic blockers, aldosterone antagonists, and d

2 beta-adrenergic blockers, amiodarone, and sotalol are th

3 and medications for CVD treatment, including beta-adrenergic blockers and ACE inhibitors, potentially

4 glutamate could be reversibly suppressed by beta-adrenergic blockers and the order of inhibitory pot

5 be achieved using calcium-channel blockers, beta-adrenergic blockers, and class I or III antiarrhyth

6 beta-adrenergic blockers are still the most common medic

7 hibitors, angiotensin-receptor blockers, and beta-adrenergic blockers, are routinely used and are qui

8 Initial treatment included a beta-adrenergic blocker (beta-blocker) alone in 24%, a c

9 hough initially thought to be harmful in HF, beta-adrenergic blockers (beta-blockers) have consistent

10 alone and in combination with propranolol (a beta-adrenergic blocker), both given systemically, to re

11 Nonselective beta-adrenergic blockers decrease portal pressure and pr

12 perimental conditions for SERS, we chose the beta-adrenergic blocker drug propranolol as the target a

13 laxis, angiotensin-receptor blockers (ARBs), beta-adrenergic blockers, epinephrine, and Kounis syndro

14 angiotensin-converting enzyme inhibitors and beta-adrenergic blockers, has been shown to improve the

15 In addition, recent studies suggest that beta-adrenergic blockers have a beneficial effect on bot

16 Thiazide diuretics, niacin, and beta-adrenergic blockers impair glucose homeostasis.

17 ellular basis for the therapeutic actions of beta-adrenergic blockers in LQT1 and suggest that sodium

18 as observed with propranolol, a nonselective beta-adrenergic blocker, in pretreated animals such that

19 Additionally, infusion of beta-adrenergic blockers into the amygdala blocks the me

20 ty in patients with CHF who are treated with beta-adrenergic blockers is currently unknown.

21 th previous reports that betaxolol and other beta-adrenergic blockers may exert its neuroprotective a

22 fight-or-flight stress response such as the beta-adrenergic blocker metoprolol and the beta-adrenerg

23 Use of antianginal medications (beta-adrenergic blockers, nitrates, and calcium-channel

24 ned the effectiveness of betaxolol and other beta-adrenergic blockers on glutamate-induced calcium si

25 at 6 months, 39% of patients were receiving beta-adrenergic blockers (preprocedure proportion, 43%;

26 aline, the centrally and peripherally acting beta-adrenergic blocker propranolol (4 or 10 mg/kg ip),

27 notypes were reversed by the addition of the beta-adrenergic blocker propranolol.

28 ects can be inhibited by the addition of the beta-adrenergic blocker propranolol.

29 d be greater after the administration of the beta-adrenergic blocker propranolol.

30 ick from a mammalian host, we administered a beta-adrenergic blocker, propranolol, to infected mice.

31 In patients with myocardial infarction, beta-adrenergic blockers reduce recurrent myocardial inf

32 Prophylactic use of beta-adrenergic blockers reduces the incidence of postop

33 Prophylactic treatment with beta-adrenergic blockers should be continued during the

34 or 10 mg/kg ip), or the peripherally acting beta-adrenergic blocker sotalol (4 or 10 mg/kg ip) immed

35 udies of left ventricular assist devices and beta-adrenergic blockers suggest that attenuation of pro

36 Even at high doses, beta-adrenergic blockers, the first-line therapy, often

37 Long-term administration of beta-adrenergic blockers to patients after myocardial in

38 vagotonic agents, alpha-adrenergic agonists, beta-adrenergic blockers, tricyclic or tetracyclic antid

39 The beta-adrenergic blocker was only effective when the drug

40 Treatment with beta-adrenergic blockers was independently associated wi

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