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1 e of mu-opioid receptors with the antagonist beta-funaltrexamine.
2 e opioid receptor antagonists, naltrexone or beta-funaltrexamine.
3 eversed by the opioid mu receptor antagonist beta-funaltrexamine.
4 ltrexone (0.1-20 microgram) or mu-selective (beta-funaltrexamine, 0.1-20 microgram), mu(1)-selective
5 d general (naltrexone: 1-50 micrograms), mu (beta-funaltrexamine: 1-20 micrograms), mu 1 (naloxonazin
7 orin against SNL-induced pain was blocked by beta-funaltrexamine, a selective mu-opioid receptor anta
8 l gray (PAG) since general (naltrexone), mu (beta-funaltrexamine) and delta2 (naltrindole isothiocyan
9 palatable (10% sucrose) conditions with mu (beta-funaltrexamine) and kappa (nor-binaltorphamine), bu
10 r-BNI) or the mu-opioid receptor antagonist, beta-funaltrexamine (beta-FNA) also results in opioid an
12 ng-lasting micro opioid receptor antagonist, beta-funaltrexamine (beta-FNA), prior to parturition int
13 infusing the mu opioid receptor antagonist, beta-funaltrexamine (beta-FNA), prior to systemic morphi
14 e-putamen (CPu), the mu receptor antagonist, beta-funaltrexamine (beta-FNA), was unilaterally infused
15 ment with a dose range of either naltrexone, beta-funaltrexamine (beta-FNA, mu), nor-binaltorphamine
16 -opioid receptors, rats were pretreated with beta-funaltrexamine (beta-FNA; 15 mg/kg s.c), an irrever
17 oreover, the selective mu opioid antagonist, beta-funaltrexamine (betaFNA: 2-20 mug, i.c.v.), signifi
18 st, naltrexone, the mu-selective antagonist, beta-funaltrexamine (BFNA) or the delta 2 antagonist, na
19 reatment with general (naltrexone: NTX), mu (beta-funaltrexamine: BFNA), kappa (nor-binaltorphamine:
20 tment with the mu-opioid receptor antagonist beta-funaltrexamine blocked morphine potentiation of CHS
21 pretreated with increasing concentrations of beta-funaltrexamine followed by functional testing after
22 mbens pretreatment with either selective mu (beta-funaltrexamine), mu(1) (naloxonazine), delta(1) ([D
23 nor-binaltorphimine (nor-BNI)] and mu- (e.g. beta-funaltrexamine) opioid receptors have previously be
26 ve opioid antagonists norbinaltorphimine and beta-funaltrexamine, respectively, but not the delta-sel
27 oid receptors by the irreversible antagonist beta-funaltrexamine would decrease spontaneous receptor
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