ライフサイエンスコーパス: beta-glucan receptor

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1 s dependent upon recognition by the Dectin-1 beta-glucan receptor.

2 olar macrophages is mediated by the Dectin-1 beta-glucan receptor and that the subsequent generation

3 Furthermore, the soluble carbohydrate beta-glucan receptor antagonists laminariheptaose and la

4 ific carbohydrate inhibitors, we show that a beta-glucan receptor, but not the MR, is a predominant r

5 The macrophage Dectin-1 beta-glucan receptor colocalized with the P. carinii cys

6 Dectin-1, a beta-glucan receptor, contributes to host anti-fungal de

7 We recently demonstrated that the beta-glucan receptor Dectin-1 (betaGR) was the major non

8 rmacologically immunosuppressed) lacking the beta-glucan receptor Dectin-1 (Dectin-1(-/-)) are more s

9 n of PC by alveolar macrophages involves the beta-glucan receptor Dectin-1 and macrophage effector fu

10 ype and knock-out mice demonstrates that the beta-glucan receptor Dectin-1 and MyD88 regulate early r

11 The training required the beta-glucan receptor dectin-1 and the noncanonical Raf-1

12 shown that lung responses generated via the beta-glucan receptor Dectin-1 are required for lung defe

13 ollectively, these results indicate that the beta-glucan receptor Dectin-1 contributes to lung inflam

14 eviously reported that mice deficient in the beta-glucan receptor Dectin-1 displayed increased suscep

15 reviously demonstrated the importance of the beta-glucan receptor Dectin-1 in the recognition of path

16 Here, we demonstrate that the beta-glucan receptor Dectin-1, a yeast binding C type le

17 he yeast particle zymosan, by binding to the beta-glucan receptor Dectin-1, activates an ITAM-Syk-dep

18 s not reduce detection of yeasts by the host beta-glucan receptor Dectin-1.

19 RNA interference based depletion of the host beta-glucan receptor dectin-1.

20 hages infected with M smegmatis required the beta-glucan receptor dectin-1.

21 tivity are dependent on CD18, but not on the beta-glucan receptor dectin-1.

22 oduction were enhanced by overexpressing the beta-glucan receptor, dectin-1, but not dectin-1 lacking

23 Toll-like receptors and the beta-glucan receptor, dectin-1, mediate macrophage infla

24 o address the role of the recently described beta-glucan receptor, Dectin-1, we generated a novel ant

25 also investigated the role of the macrophage beta-glucan receptor, Dectin-1.

26 se findings define Dectin-1 as the leukocyte beta-glucan receptor, first described over 50 years ago,

27 Nevertheless, the identity of the beta-glucan receptor has been controversial.

28 Dectin-1, the major beta-glucan receptor in leukocytes, triggers an effectiv

29 t receptor 3, there is evidence that another beta-glucan receptor is present on macrophages.

30 s failed to stimulate TNF-alpha in Dectin-1 (beta-glucan receptor) knockout BMDCs.

31 r the phagocytosis of C. albicans and that a beta-glucan receptor may be required for C. albicans pha

32 inst CR3, demonstrating that CR3 was not the beta-glucan receptor mediating this activity.

33 r Dectin-1 (betaGR) was the major nonopsonic beta-glucan receptor on macrophages (Mphi) for the yeast

34 Dectin-1, a major beta-glucan receptor on macrophages, colocalized to phag

35 ines mainly through interaction with cognate beta-glucan receptors on the phagocyte.

36 as not affected by inhibitors of mannose and beta-glucan receptors or monoclonal antibodies directed

37 ought to determine whether Dectin-1, a major beta-glucan receptor recently identified in leukocytes,

38 that fungi interact with macrophages through beta-glucan receptors, thereby inducing release of tumor

39 In conclusion, CR3 serves as the leukocyte beta-glucan receptor through a cation-independent lectin

40 egrin, CR3 (Mac-1, CD11b/CD18) served as the beta-glucan receptor through one or more lectin sites lo

41 CD18 (Mac-1, CR3) is the most widely studied beta-glucan receptor, we demonstrate that CD11b/CD18 is

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