ライフサイエンスコーパス: beta3 adrenergic receptor

1 cells to decrease CXCL12 levels through the beta3-adrenergic receptor.

2 ment-binding protein 1, glycerol kinase, and beta3-adrenergic receptor.

3 n WAT and could be reversed by antagonism of beta3 adrenergic receptors.

4 nd by genetic deletion of stromal beta2- and beta3-adrenergic receptors.

5 olerant, expended less energy in response to beta3-adrenergic receptor activation, and were more insu

6 e, the liver fatty acid-binding protein, the beta3-adrenergic receptor, adipsin and the peroxisome pr

7 Sustained beta3 adrenergic receptor (ADRB3) activation simultaneou

8 y to a twofold increase in the expression of beta3 adrenergic receptor (Adrb3) at both the mRNA and p

9 er a beiging response: cold temperatures and beta3-adrenergic receptor (Adrb3) agonists.

10 ptin (OB), the leptin receptor (OBR/DB), the beta3-adrenergic receptor (ADRB3), lipoprotein lipase (L

11 Acute activation of beta3-adrenergic receptors (ADRB3) triggers lipolysis an

12 The beta3-adrenergic receptor (ADRbeta3) is a seven-membrane

13 moneutrality were treated with the selective beta3 adrenergic receptor agonist CL 316, 243 and underw

14 We used CL-316,243, a beta3 adrenergic receptor agonist, to acutely elevate bl

15 m-adapted rats by infusions of the selective beta3-adrenergic receptor agonist CL 316,243.

16 ital, and given intraperitoneally either the beta3-adrenergic receptor agonist CL-316,243, 1 mg/kg (n

17 Treatment with the beta3-adrenergic receptor agonist CL316,243 increased Cx

18 ghly enantioselective total synthesis of the beta3-adrenergic receptor agonist Solabegron.

19 of these receptors with BRL37344, a specific beta3-adrenergic receptor agonist, promoted migration th

20 mors were treated with CL-316243, a specific beta3-adrenergic receptor agonist, which sensitizes insu

21 -regulation of UCP1 after stimulation with a beta3-adrenergic receptor agonist.

22 e consume less oxygen after treatment with a beta3-adrenergic-receptor agonist and that they are sens

23 etanilides were synthesized and evaluated as beta3-adrenergic receptor agonists (beta3-AR) for the tr

24 c signaling molecules, i.e., norepinephrine, beta3-adrenergic receptor, and cAMP; the transcriptional

25 adipogenic genes (LpL, adipsin, GLUT-4, aP2, beta3-adrenergic receptor, and peroxisome proliferator-a

26 ed the effects of treatment with a selective beta3 adrenergic receptor (AR) agonist (CL 316,243 [1 mg

27 Epinephrine or the beta3-adrenergic receptor (AR) agonist CL 316,243 (CL) s

28 nergic receptor agonist isoproterenol or the beta3-adrenergic receptor (AR)-specific agonist CL 31624

29 beta1- and beta3-adrenergic receptors (AR) are the predominant beta

30 ulated by the sympathetic nervous system via beta3-adrenergic receptors (beta3-AR).

31 beta3-Adrenergic receptors (beta3-ARs) are expressed pre

32 uences of dual Gs/Gi protein coupling of the beta3-adrenergic receptor (beta3AR) in 3T3-F442A adipocy

33 ogenesis is transduced by the beta2, but not beta3, adrenergic receptor expressed on stromal cells of

34 women with the Trp64Arg polymorphism of the beta3-adrenergic receptor gene have lower daily energy e

35 In mice with genetic deficiency of the beta3 adrenergic receptor, hematopoietic stem cells did

36 Stimulation of beta3-adrenergic receptors increases metabolic rate via

37 hese data indicate that neural activation of beta3-adrenergic receptors is an important determinant o

38 The beta3 adrenergic receptor, located on chromosome 8, is a

39 These data show that alpha1 and beta3-adrenergic receptors may contribute to the mediati

40 Activation of beta3 adrenergic receptors on the surface of adipocytes

41 We show, for the first time, expression of beta3-adrenergic receptors on cultured retinal endotheli

42 nt study, we hypothesized that activation of beta3-adrenergic receptors on retinal endothelial cells

43 (2+) cycling by activation of alpha1- and/or beta3-adrenergic receptors or the SERCA2b-RyR2 pathway s

44 to be regulated by insulin (e.g. Glut-1 and beta3-adrenergic receptor), other novel insulin-sensitiv

45 These results support the hypothesis that beta3-adrenergic receptors play a role in proliferation

46 ived brown adipocytes, displayed an impaired beta3-adrenergic receptor response that was characterize

47 Moreover, cold exposure and beta3-adrenergic receptor signaling, conditions that ind

48 Here we show that BAT activation by beta3-adrenergic receptor stimulation protects from athe

49 ion about which signals activate BA, besides beta3-adrenergic receptor stimulation, is limited.

50 NE enhances thermogenesis through beta3-adrenergic receptors to activate brown adipose tis

51 r comparison, polymorphisms in the beta2 and beta3 adrenergic receptors were also evaluated.

52 receptor nor polymorphisms in the beta2 and beta3 adrenergic receptors were associated with resting

53 Beta3-adrenergic receptors were not detected.