ライフサイエンスコーパス: bethanechol

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1 specific beta3 antagonist), propranolol, and bethanechol.

2 ates following intravenous administration of bethanechol, a cholinergic agonist, or intralumenal inst

3 These data suggest that bethanechol acts to decrease CTC fluorescence at the sam

4 Bethanechol also reduced gland duct mucin content in the

5 holinergic agents (ACh, nicotine, muscarine, bethanechol and pilocarpine) increased [Ca2+]i.

6 ed gastric relaxation was enhanced following bethanechol and reduced by l-NAME administration, sugges

7 Both bethanechol- and LPS-stimulated intestinal lumenal perfu

8 esponse to direct cholinergic stimulation by bethanechol at 2 days postsympathectomy were increased f

9 e effects of SScIgGs on M(3)-R activation by bethanechol (BeCh), M(3)-R occupancy, and receptor bindi

10 ing doses of the muscarinic receptor agonist bethanechol, but not the nicotinic receptor antagonist m

11 ent with the pro-motility muscarinic agonist bethanechol, but were abolished by systemic administrati

12 EVS applied after pre-contraction with bethanechol caused relaxation of wild type gastric tissu

13 ximal fluorescence loss, however, required a bethanechol concentration three times greater than that

14 Bethanechol depressed this excitation whereas muscarine

15 t with MQC inhibited nicotine-evoked but not bethanechol-evoked secretion.

16 m allografts only generated 23% of the total bethanechol-induced contractile force in vitro compared

17 reatic beta-cells with a muscarinic agonist (bethanechol) led to significantly increased plasma insul

18 CL316243 and isoproterenol inhibited the bethanechol-mediated hypertensive LES.

19 Stimulation with either bethanechol or caerulein resulted in a rapid loss of flu

20 d in UP-LPS-treated TLR4-competent mice, but bethanechol-stimulated contractions were not altered by

21 in neutrophil infiltration and a decrease in bethanechol-stimulated contractions.

22 Spontaneous and bethanechol-stimulated jejunal circular muscle contracti

23 nt 2-deoxy-D-glucose (75 mg kg(-1)), whereas bethanechol-stimulated pancreatic protein output was inh

24 wing the loss of CTC fluorescence induced by bethanechol stimulation.

25 e pronounced when lungs were also exposed to bethanechol to stimulate submucosal gland secretion, whe

26 The fluorescence loss induced by bethanechol was blocked by atropine and was seen at all

27 Bethanechol was used to produce contraction of the smoot

28 l muscle contractions caused by nicotine and bethanechol were similar in segments from P2X2+/+ and P2

29 l muscle contractions caused by nicotine and bethanechol were similar in segments from P2X3+/+ and P2

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