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1 ucose appearance or in glucose disappearance between meals.
2 ly, at 2 h) were not significantly different between meals.
3 ntestinal epithelium and for taurine capture between meals.
4 6Pase-alpha) to maintain glucose homeostasis between meals.
5 concentrations did not differ significantly between meals.
6 RS1-Reg-blocked exocytotic pathway of SGLT1 between meals.
7 se is critical for blood glucose homeostasis between meals.
8 ls during which animals drink; (3) intervals between meals.
9 rts recommend that iron supplements be taken between meals.
10 agents by lowering the set-point for hunger between meals.
11 hunger compared with BS with no differences between meals.
13 - 8.9% after meal 2 (P = 0.01 for difference between meals), and then decreased toward baseline (P <
14 cholecystokinin concentration did not differ between meals, but the 2 low-fat meals elicited a greate
16 e of an aqueous multivitamin (MV) supplement between meals (control group), 2) the same porridge and
17 AAs (n = 16) or placebo (n = 12) twice daily between meals for 1 week before and 2 weeks after TKA.
18 nsulin total AUC and iAUC were not different between meals (iAUC: 159.65 +/- 20.0 mU/L . 3 h for red
19 s no statistical difference (P = 0.34) in SI between meals in type 1 diabetic subjects, the diurnal p
20 derly, administration of dietary supplements between meals instead of with meals may be more effectiv
21 1.46,3.01) and consumption of citrus fruits between meals (OR = 1.69, 95%CI = 1.04, 2.73) were assoc
22 1.18, 3.01) and consumption of citrus fruits between meals (OR = 2.22, 95%CI = 1.30, 3.81) were posit
24 hough meal glucose appearance did not differ between meals, suppression of endogenous glucose product
26 natural satiation and the period of satiety between meals through coupling of ERK activation to both
27 uptake into the three depots did not differ between meals; visceral fat accounted for only approxima
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