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4 erent input were observed in the homonymous (biceps brachii) and antagonist (triceps brachii) motor n
5 r (Pmajor) and posterior deltoid (Pdeltoid); biceps brachii (Bi) and Tri brachii (Tri), and linking m
6 e recorded in the surface EMG of contracting biceps brachii (Bi), evoked by taps applied to the tendo
7 e acquired high-density EMG signals from the biceps brachii in 5 male transhumeral amputees who under
9 rected to the motor cortex representation of biceps brachii muscle during the adaptation phase of the
10 same recordings were also performed from the biceps brachii muscle of additional 5 able-bodied indivi
12 hibition (ICI) and facilitation (ICF) to the biceps brachii muscle proximal to the level of deafferen
15 magnetic stimulation (TMS) in small hand and biceps brachii muscles before, during and after INB of t
16 array was used to record surface EMG of the biceps brachii muscles from both impaired and non-impair
18 he rotator interval and the long head of the biceps brachii tendon are anatomically closely associate
21 tor pollicis brevis, abductor digiti minimi, biceps brachii, tibialis anterior, extensor dig. brevis,
22 mous, monosynaptic Group Ia projections from biceps brachii to both the antagonist triceps brachii an
24 post-stroke patients with spasticity of the biceps brachii, we found involuntary microscopic contrac
25 scharge characteristics of 53 motor units in biceps brachii were recorded after being recruited durin
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