ライフサイエンスコーパス: bicuspid

1 lusal contact, and a fistula on a mandibular bicuspid.

2 icuspids and mandibular central incisors and bicuspids.

3 al ligament cells from periodontally healthy bicuspids.

4 dibular second bicuspids and maxillary first bicuspids.

5 bset that were significantly greater in both bicuspid and tricuspid CAS cases with more severe valve

6 e immune response is occurring in cases with bicuspid and tricuspid CAS, involving circulating CD8 T

7 t foods, and particles were removed from all bicuspids and first molars at defined times after swallo

8 n the facial surface of maxillary molars and bicuspids and mandibular central incisors and bicuspids.

9 We have identified an MSX1second bicuspids and mandibular central incisors.

10 udes missing maxillary and mandibular second bicuspids and maxillary first bicuspids.

11 n males, while lippings were seen in molars, bicuspids, and mandibular incisors, with even gender dis

12 ic stenosis were compared with biopsies from bicuspid aortic stenosis.

13 yndrome (21.5% versus 3.1%; P<0.001) but not bicuspid aortic valve (3.6% versus 3.2%; P=0.77).

14 Bicuspid aortic valve (BAV) (39%) and Marfan syndrome (M

15 ay a role contributing to the progression of bicuspid aortic valve (BAV) aortopathy.

16 tic valve (TAV) replacement in patients with bicuspid aortic valve (BAV) disease (TAV-in-BAV).

17 The correlation between bicuspid aortic valve (BAV) disease and aortopathy is no

18 Bicuspid aortic valve (BAV) disease is frequently accomp

19 Ascending aortic dilation is important in bicuspid aortic valve (BAV) disease, with increased risk

20 Familial clustering of HLHS and bicuspid aortic valve (BAV) has been observed, and pedig

21 al tricuspid aortic valve (TAV) in 172 and a bicuspid aortic valve (BAV) in 66 subjects.

22 Bicuspid aortic valve (BAV) is a heritable condition tha

23 Bicuspid aortic valve (BAV) is a heritable congenital he

24 determine whether the morphologic subtype of bicuspid aortic valve (BAV) is associated with valve int

25 Importance: Bicuspid aortic valve (BAV) is considered an autosomal d

26 Bicuspid aortic valve (BAV) is regarded as a relative co

27 Bicuspid aortic valve (BAV) is the most common adult con

28 Bicuspid aortic valve (BAV) is the most common congenita

29 ittle is known about the association between bicuspid aortic valve (BAV) morphologic findings and the

30 ltered ascending aorta (AAo) hemodynamics in bicuspid aortic valve (BAV) patients and its association

31 tic root in first-degree relatives (FDRs) of bicuspid aortic valve (BAV) patients.

32 This large multicenter, international bicuspid aortic valve (BAV) registry aimed to define the

33 y who also exhibited LVOT defects, including bicuspid aortic valve (BAV), coarctation of the aorta (C

34 evelops in most patients with a congenitally bicuspid aortic valve (BAV), in others with this anomaly

35 dies have established familial clustering of bicuspid aortic valve (BAV), presumably indicating genet

36 Bicuspid aortic valve (BAV), the most common congenital

37 RATIONALE: The pathogenesis of bicuspid aortic valve (BAV)-associated aortopathy is poo

38 efined compared to Marfan syndrome (MFS) and bicuspid aortic valve (BAV).

39 te the genetic relationship between HLHS and bicuspid aortic valve (BAV).

40 85), ventricular septal defect (P=0.12), and bicuspid aortic valve (P=0.14) did not carry an increase

41 -dominant aortic valve disease consisting of bicuspid aortic valve and aortic valve calcification was

42 In patients with bicuspid aortic valve and dilated proximal ascending aor

43 One had baseline bicuspid aortic valve and mild aortic regurgitation that

44 In the community, asymptomatic patients with bicuspid aortic valve and no or minimal hemodynamic abno

45 Making the decision regarding the timing of bicuspid aortic valve aneurysm surgery even more difficu

46 Severe calcification of a congenital bicuspid aortic valve continues to be an important cause

47 ng aortic aneurysm surgery in the setting of bicuspid aortic valve disease is complex, with multiple

48 oracic aortopathy after AVR in patients with bicuspid aortic valve disease is substantially different

49 aortic disease such as the Marfan syndrome, bicuspid aortic valve disease, and hereditary aortic ane

50 h aortic regurgitation, and in patients with bicuspid aortic valve disease.

51 Because we have previously found that bicuspid aortic valve experience greater stretch, we inv

52 lities, including Marfan's syndrome in four, bicuspid aortic valve in four, and aortitis in one.

53 Bicuspid aortic valve is frequent and is reported to cau

54 Bicuspid aortic valve is frequently an antecedent to aor

55 1,000 live births if the potentially serious bicuspid aortic valve is included), and of all forms inc

56 Bicuspid aortic valve is the most common type of cardiac

57 Bicuspid aortic valve occurs in 1% of the population, ma

58 In addition, bicuspid aortic valve occurs in more than one-half of th

59 METHODS AND We studied 969 consecutive bicuspid aortic valve patients (50+/-13 years; 87% men)

60 In bicuspid aortic valve patients with dilated proximal asc

61 with age, and it is often associated with a bicuspid aortic valve present in 1-2% of the population.

62 rates of Marfan syndrome and lower rates of bicuspid aortic valve than those undergoing bio-CVG or m

63 Bicuspid aortic valve was associated with more intense h

64 County, Minn (age, 32+/-20 years; 65% male), bicuspid aortic valve was diagnosed between 1980 and 199

65 Bicuspid aortic valve was diagnosed in 227 patients (73.

66 During cardiac surgery, a congenital bicuspid aortic valve was found to be the predisposing f

67 Bicuspid aortic valve was identified in 74 individuals (

68 ortic aneurysms variably associated with the bicuspid aortic valve was used for identification of add

69 r malformation (i.e., bicuspid aortic valve, bicuspid aortic valve with coarctation of the aorta, or

70 tic valve, partially fused aortic valve, and bicuspid aortic valve+unicuspid aortic valve, respective

71 ); partially fused aortic valve, 12% (n=25); bicuspid aortic valve, 23% (n=47); and unicuspid aortic

72 ar septal defects, patent ductus arteriosus, bicuspid aortic valve, and coarctation of the aorta as w

73 ts were more likely to have Marfan syndrome, bicuspid aortic valve, and prior aortic surgery (all, p

74 ch1, or RBPJ displayed enlarged valve cusps, bicuspid aortic valve, and septal defects, indicating th

75 Partially fused aortic valve, bicuspid aortic valve, and unicuspid aortic valve were s

76 idney and cardiovascular malformation (i.e., bicuspid aortic valve, bicuspid aortic valve with coarct

77 similar to the pattern seen in nonsyndromic bicuspid aortic valve, is equally prevalent (20-30%) in

78 ricle, aberrant semilunar valve development, bicuspid aortic valve, ventricular septal defects, and e

79 rm and ectoderm caused glandular defects and bicuspid aortic valve, which indicates that the FGF8 end

80 women, 161 (46%) had either a unicuspid or a bicuspid aortic valve.

81 ying aortic valve abnormalities as seen with bicuspid aortic valve.

82 ociated cardiac malformation that included a bicuspid aortic valve.

83 ne had truncus arteriosus, and another had a bicuspid aortic valve.

84 hymal transition, and NOTCH1 mutations cause bicuspid aortic valve; however, the temporal requirement

85 ression) and, as a clinical correlate, human bicuspid aortic valves (63% reduction).

86 tion in the ascending aorta in patients with bicuspid aortic valves (BAV) have influenced strategies

87 ic aortic aneurysms (ATAAs) in patients with bicuspid aortic valves (BAV) versus patients with tricus

88 Bicuspid aortic valves (BAVs) are associated with premat

89 Explanted, stenotic bicuspid aortic valves (BAVs) from pediatric patients we

90 tricuspid aortic valves (n = 27), calcified bicuspid aortic valves (n = 23), and control tissue from

91 Patients with bicuspid aortic valves also showed significantly increas

92 s imply higher energy losses associated with bicuspid aortic valves and dilated ascending aortic geom

93 e risk of aortic dissection in patients with bicuspid aortic valves and severe aortic enlargement: th

94 Bicuspid aortic valves are associated with valve dysfunc

95 Bicuspid aortic valves calcify at a significantly higher

96 ith Marfan syndrome compared with those with bicuspid aortic valves confirm that operative management

97 m that operative management of patients with bicuspid aortic valves should not be extrapolated from M

98 neurysmal progression in Npr2(+/-) mice with bicuspid aortic valves than those with tricuspid valves.

99 Furthermore, pediatric stenotic bicuspid aortic valves that have lost normal extracellul

100 Incompetent bicuspid aortic valves with dilated aortic annuli are al

101 Bicuspid aortic valves with raphe had a significantly hi

102 rison, outcomes of 13,205 adults (2,079 with bicuspid aortic valves, 73 with Marfan syndrome, and 11,

103 isk factors for dissection: Marfan syndrome, bicuspid aortic valves, and larger aortic dimensions.

104 paroxysmal, persistent, or chronic AF, with bicuspid aortic valves, and patients who died within 48

105 addition, another 20/1,000 live births have bicuspid aortic valves, isolated anomalous lobar pulmona

106 of mice heterozygous for Npr2 had congenital bicuspid aortic valves, with worse aortic valve function

107 stify surgical intervention in patients with bicuspid aortic valves.

108 ng aorta with ventricular septal defect, and bicuspid aortic valves.

109 the development of atrial septal defects and bicuspid aortic valves.

110 athogenetically and prognostically linked to bicuspid aortopathy.

111 eived in both maxillary and mandibular molar-bicuspid areas LDS or BGP on one side and non-anesthetic

112 ectiveness of LDS directly with BGP in molar-bicuspid areas of both arches.

113 were mainly seen around maxillary molars and bicuspids, especially in males, while lippings were seen

114 e extraction socket) of the maxillary teeth (bicuspids forward) immediately following tooth extractio

115 Within the bicuspid group, multivariate analysis demonstrated that

116 unicommissural in 42; acommissural in 4] and bicuspid in 458); 417 (45%) had tricuspid valves (either

117 The aortic valve was bicuspid in 74 of 250 (30%) adequately imaged subjects.

118 lve was congenitally malformed (unicuspid or bicuspid) in 58 (98%) of the 59 AS patients, and in 38 (

119 The morphology of the bicuspid or bicommissural aortic valve (BAV) may predict

120 did not affect survival, but patients with a bicuspid or unicuspid valve required operative intervent

121 A subgroup analysis of younger patients with bicuspid sAS showed a similar pattern of significantly l

122 l/lingual sites, higher proportions of lower bicuspid teeth demonstrated attachment loss compared wit

123 ntributed subgingival samples from molar and bicuspid teeth presenting interproximal periodontitis le

124 interproximal attachment loss, whereas lower bicuspid teeth were at risk for gingival recession on bu

125 adavers were edentulous distal to the second bicuspid teeth, and 14 out of 20 were edentulous distal

126 PVR with a man-made polytetrafluoroethylene bicuspid valve and percutaneous PVR.

127 ion of clonal expansions in the much younger bicuspid valve CAS cases.

128 estimation of aortic dissection incidence in bicuspid valve patients irrespective of diagnosis status

129 alva, congenital aortic valve stenosis (with bicuspid valve) and myocarditis.

130 been described for a subset of cases with a bicuspid valve, data are limited on the overall familial

131 R was defined as TAVR in patients with known bicuspid valve, moderate aortic stenosis, severe mitral

132 63 +/- 11 years, 73% were male, and 38% had bicuspid valve.

133 4 men, 343 (59%) had either a unicuspid or a bicuspid valve; of the 348 women, 161 (46%) had either a

134 rome (5.5 +/- 2.7%) compared with those with bicuspid valves (0.55 +/- 0.21%) and control group patie

135 s significantly lower for flat versus doming bicuspid valves (0.73 +/- 0.14 vs. 0.94 +/- 0.14, p < 0.

136 -up (10.4 +/- 4.3%) compared with those with bicuspid valves (2.5 +/- 0.6%) and control group patient

137 ome (10.8 +/- 4.4%) compared with those with bicuspid valves (4.8 +/- 0.8%) and control group patient

138 ficantly >0 in 21 patients and was lower for bicuspid valves (slope 0.21 cm2/100 ml per s) than for t

139 eas less flow dependence was associated with bicuspid valves and the features of rheumatic disease.

140 d for aortic complications of patients whose bicuspid valves had gone undiagnosed.

141 The results of this study demonstrate that bicuspid valves induced significantly altered ascending

142 search for aortic dissections in undiagnosed bicuspid valves revealed 2 additional patients, allowing

143 Time-resolved (cine) 2D images of the bicuspid valves were coregistered with 4D flow data, dir

144 tive endocarditis, 46 patients (38%; 15 with bicuspid valves); probable rheumatic heart disease, 8 pa

145 ents, in 20 (53%) of the 38 AR patients with bicuspid valves, and in all 12 AR patients with tricuspi

146 but interest is increasing, particularly for bicuspid valves.

147 the mandibular cuspids and first and second bicuspids was 275.88 mm2, 251.45 mm2, and 271.81 mm2, re

148 -sided measurements for the mandibular first bicuspid were 252.55 mm2 and 247.02 mm2, respectively (P

149 of severe aortic stenosis (6 tricuspid and 2 bicuspid) were created using dual-material fused 3D prin

150 he frequent (75%) absence of maxillary first bicuspids, while the most distinguishing feature of PAX9