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1 foreign DNA elements increases resistance to bicyclomycin.
2 region as the functional domain inhibited by bicyclomycin.
3 f a gram-positive organism can be blocked by bicyclomycin.
4 ts sensitivity to the Rho-specific inhibitor bicyclomycin.
9 growth and survival of Escherichia coli, and bicyclomycin (1) is its only known selective inhibitor.
15 positive phylogenetic group, is prevented by bicyclomycin, an antibiotic that inhibits the activity o
17 mation with rho to enhance the affinity of a bicyclomycin analogue and determine the binding stoichio
18 icyclomycin has a Rho that is insensitive to bicyclomycin and has the single amino acid residue chang
19 action of two Rho effectors, the antibiotic bicyclomycin and nucleic acids that bind to Rho's primar
21 factors nusA and nusG were hypersensitive to bicyclomycin, and had extensive chromosome fragmentation
23 air (recB and ruvC) increased sensitivity to bicyclomycin, as did loss of the replication fork reload
24 sceptibility or resistance to the antibiotic bicyclomycin (BCM), a potent inhibitor of the transcript
27 weak) imposed on the rho hexamer, the nearby bicyclomycin binding pocket is not affected, and both 1
28 d 3 per rho hexamer indicating the number of bicyclomycin binding sites for the rho hexamer is betwee
33 activities for the C(5a)-extended conjugated bicyclomycin derivatives and the (5E) and (5Z) isomers w
35 bond with unsaturated substituents provides bicyclomycin derivatives with excellent inhibitory activ
38 involved, because the Rho-specific inhibitor bicyclomycin failed to show synergism with either aza-C
39 nthesis, and characterization of a series of bicyclomycin fluorescent probes (BFP) constructed to sen
42 f Rho-dependent transcription termination by bicyclomycin in Escherichia coli induced double-strand b
46 as a different structure and is inhibited by bicyclomycin, indicating that T2 is probably Rho depende
47 assays, limited tryptic digestions, and the bicyclomycin inhibition kinetics of ATPase activity in t
48 ntibiotic concentrations near the I50 value, bicyclomycin inhibition of Rho-dependent transcripts was
54 rac prophage in wild-type E. coli increases bicyclomycin resistance and permits deletion of nusG.
55 r-mediated repression, as well as a putative bicyclomycin resistance gene (so2280) and cation efflux
56 taining the G337S Rho mutation also has high bicyclomycin resistance, and the proximity of L208, S266
59 epared to provide information concerning the bicyclomycin-rho inactivation process and the drug's bin
63 ed-ring (RNA-translocation) states, we found bicyclomycin to be a direct antagonist of ring closure.
64 pondingly, the Km(app) for ribo(C)10 without bicyclomycin was 0.8 microM and with bicyclomycin was 5
66 without bicyclomycin was 0.8 microM and with bicyclomycin was 5 microM at infinite inhibitor concentr
67 The interaction of Rho and the antibiotic bicyclomycin was probed using in vitro transcription ter
68 rmylbicyclomycin, an inhibitor comparable to bicyclomycin, was previously shown to form a stable imin
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