ライフサイエンスコーパス: bieneusi

1 ic gastrointestinal pathogen (Enterocytozoon bieneusi).

2 icularly the enteric parasite Enterocytozoon bieneusi.

3 virus/AIDS disease who are infected with E. bieneusi.

4 all subunit ribosomal RNA of human origin E. bieneusi.

5 phalitozoon cuniculi, E. intestinalis and E. bieneusi.

6 49 (52.1%) were positive for Enterocytozoon bieneusi, 43 (45.8%) were positive for Encephalitozoon i

7 Enterocytozoon bieneusi, a microsporidian, is clinically one of the mos

8 llem, and 9 samples (9.6%) contained both E. bieneusi and E. intestinalis spores.

9 ents with mixed infections of Enterocytozoon bieneusi and E. intestinalis.

10 sis, and the microsporidians, Enterocytozoon bieneusi and Encephalitozoon intestinalis.

11 ents as well as in studying the genome of E. bieneusi and host-parasite interactions.

12 of microsporidiosis caused by Enterocytozoon bieneusi and the pattern of intestinal shedding of spore

13 lli, Cyclospora cayetanensis, Enterocytozoon bieneusi, and Septata intestinalis are intestinal spore-

14 tidium coli, Giardia lamblia, Enterocytozoon bieneusi, and Trichuris trichiura was found in all anima

15 acted by indirect immunofluorescence with E. bieneusi but not with Encephalitozoon spp., Candida albi

16 , we examined captive rhesus macaques for E. bieneusi by PCR, in situ hybridization, and cytochemical

17 od for the simultaneous identification of E. bieneusi, E. intestinalis, and E. hellem, as well as Enc

18 major microsporidian species: Enterocytozoon bieneusi, Encephalitozoon cuniculi, Encephalitozoon hell

19 ty, and zoonotic potential of Enterocytozoon bieneusi, feces from 348 stray and pet dogs and 96 pet c

20 t here successful serial transmissions of E. bieneusi from patients with AIDS and from macaques with

21 any other parasite, including Enterocytozoon bieneusi, fungus, or bacterium tested.

22 An unusual Enterocytozoon bieneusi genotype was found in seven guinea pigs and a 2

23 Most C. hominis subtypes and E. bieneusi genotypes found have been detected in humans in

24 The lack of an animal model for E. bieneusi hinders serious investigations and limits the a

25 ttempts to develop and serially propagate E. bieneusi in rodent models.

26 ed, at least in part because detection of E. bieneusi in stools is more difficult than detection of o

27 ade because of the inability to propagate E. bieneusi in vitro and in vivo, which limits the source o

28 21 (12.8%) patients, than of Enterocytozoon bieneusi, in 2 patients (1.2%), was unexpected.

29 patients with electron microscopic-proven E. bieneusi infection and correlated the results with elect

30 ntly the first documented transmission of E. bieneusi infection between hosts.

31 E. bieneusi infection in the gut was sparse, either because

32 E. bieneusi infection induced no symptoms.

33 Risk factors for E. bieneusi infection segregated by genotype: contact with

34 For over a decade Enterocytozoon bieneusi infections in people with AIDS have been linked

35 Enterocytozoon bieneusi is a common opportunistic pathogen of human pat

36 Enterocytozoon bieneusi is clinically the most significant of the micro

37 Enterocytozoon bieneusi is closely linked with chronic diarrhea and was

38 osporidia that infect humans, Enterocytozoon bieneusi is known to cause a gastrointestinal disease wh

39 Enterocytozoon bieneusi is the most common microsporidian infecting pat

40 Enterocytozoon bieneusi is the most common microsporidian parasite reco

41 Four different E. bieneusi isolates were equally infectious, and one of th

42 alues from 3.5 x 10(3) to 4.4 x 10(5) for E. bieneusi (mean, 8.8 x 10(4)/ml), 2.3 x 10(2) to 7.8 x 10

43 uding Cryptosporidium parvum, Enterocytozoon bieneusi, Mycobacterium avium, Entamoeba histolytica, Ba

44 56 were identified as having Enterocytozoon bieneusi of 10 different genotypes.

45 small subunit ribosomal RNA derived from E. bieneusi of human origin.

46 E. bieneusi prevalence was 35.7% among the HIV(+) TB patien

47 Given the size and shape of the E. bieneusi spores (1.1 to 1.6 by 0.7 to 1.0 microm) and th

48 y purified and contained large numbers of E. bieneusi spores and relatively few bacteria and other de

49 Enterocytozoon bieneusi spores derived from rhesus macaque feces were p

50 Duodenal E. bieneusi spores from an AIDS patient were orally transmi

51 describe a method for the purification of E. bieneusi spores from human stools and the production of

52 ethod for the purification of Enterocytozoon bieneusi spores from stool specimens was developed.

53 or Vittaforma corneae or with Enterocytozoon bieneusi spores in feces, we concluded that an immunoflu

54 Although quantitation of E. bieneusi spores in stool specimens was closely correlate

55 Although the overall recovery of the E. bieneusi spores was low, calcofluor and Gram chromotrope

56 rastructural examination revealed typical E. bieneusi spores.

57 Among these, Enterocytozoon bieneusi, the agent of gastrointestinal (GI) disease, is

58 data suggest the possibility of zoonotic E. bieneusi transmission and an association with poor sanit

59 In six of seven normal rhesus animals, E. bieneusi was detected by PCR in bile obtained through pe

60 E. bieneusi was detected in 15.5% of the dogs, including 20

61 E. bieneusi was detected in the stool at least once in 74%

62 Cryptosporidium hominis, and Enterocytozoon bieneusi were detected in 42 (17.9%), 6 (2.6%), and 29 (

63 The development of an animal model for E. bieneusi will open up new opportunities for investigatin