ライフサイエンスコーパス: bifidobacteria

1 fied essential genes that appear specific to bifidobacteria.

2 sential functions not previously examined in bifidobacteria.

3 lose relationship between HMO and infant-gut bifidobacteria.

4 microorganisms for prebiotic approaches are bifidobacteria.

5 ey in order to ensure effective detection of bifidobacteria.

6 carbohydrates by comparison with adult-borne bifidobacteria.

7 by a microbial consortium often dominated by bifidobacteria.

8 s (P = 0.069) and proportions (P = 0.029) of bifidobacteria.

9 h a specific "healthy" microbiota containing bifidobacteria, a genus commonly observed in the feces o

10 lted in a significant increase in numbers of bifidobacteria after a 24h fermentation compared to a ne

11 BlG16BP homologues occur predominantly in bifidobacteria and a few Firmicutes but lack in other HG

12 e are warranted because the low abundance of bifidobacteria and butyrate-producing species could adve

13 hypothesis, we determined salivary levels of Bifidobacteria and caries-associated organisms for 156 o

14 l molecular tool for scientific discovery of bifidobacteria and identifies targets for further studie

15 d with reduced numbers of beneficial colonic bifidobacteria and impaired immunity.

16 testinal microbiota, including reductions in bifidobacteria and key butyrate producers.

17 Bifidobacteria and lactobacilli are purportedly benefici

18 vely, are preferentially fermented by mainly bifidobacteria and lactobacilli in the human gut.

19 d glucagon like peptide-1 content as well as Bifidobacteria and Lactobacilli populations in the caecu

20 ficial barrier commensal gut bacteria (e.g., bifidobacteria and lactobacilli) and increase the abunda

21 s, in addition to their selective effects on bifidobacteria and lactobacilli, influence many aspects

22 iet rich in yacon FOS promoted the growth of bifidobacteria and lactobacilli, resulting in high level

23 ministered with prebiotics, or by endogenous bifidobacteria and lactobacilli, whose metabolic activit

24 sugar to enumerate total anaerobes, aerobes, bifidobacteria, and enterobacteria, and to assay for bet

25 ally pathogenic bacteria and the increase of bifidobacteria, and possible beneficial commensals, conf

26 ely correlated with Sutterella, Akkermansia, Bifidobacteria, and Roseburia abundance, and positively

27 Combinations of Lactobacilli, Bifidobacteria, and Streptococcus salivarius prevent rel

28 e storage period, while the viability of the bifidobacteria ( approximately 10(7)cfu/g) also remained

29 In the gastrointestinal tract, bifidobacteria are a key genus, but are often under-repr

30 Bifidobacteria are aciduric bacteria that might play a r

31 her levels of beneficial gut bacteria called Bifidobacteria are associated with the human lactase non

32 The glycan-degrading abilities of bifidobacteria are believed to reflect available carbon

33 Bifidobacteria are common and frequently dominant member

34 Bifidobacteria are common gut commensals with purported

35 that when commercially available strains of bifidobacteria are cultured in milk, spiked with perchlo

36 Bifidobacteria are dominant members of the microbial com

37 Bifidobacteria are frequently proposed to be associated

38 Bifidobacteria are important members of human gut microb

39 Bifidobacteria are important members of the human gut fl

40 Bifidobacteria are the dominant intestinal bacteria in b

41 Because bifidobacteria are thought to reduce the risk for intest

42 e numbers of beneficial bacteria, especially bifidobacteria, at the expense of less beneficial groups

43 and subjected to quantitative PCRs to detect bifidobacteria, bacteroides, lactobacilli, Escherichia c

44 To test the hypothesis that Bifidobacteria behave as caries-associated organisms, as

45 Bifidobacteria comprise a significant proportion of the

46 ined.We sought to determine the effects of a bifidobacteria-containing formula on the healthy human i

47 Bifidobacteria counts were lower in cases at all time po

48 genetic attenuation that may be occurring in bifidobacteria cultures, we obtained the complete genome

49 Bifidobacteria demonstrate an antagonistic correlation w

50 isation (FISH) shows that the proportions of bifidobacteria detected in faecal samples were in agreem

51 roidetes), Clostridium leptum, C. coccoides, bifidobacteria, Escherichia coli and Archaea in stool.

52 n of orthologous genes differed between both bifidobacteria even when grown on identical substrates.

53 samples, thereby supporting the notion that bifidobacteria expand the human glycobiome.

54 many substances that stimulate the growth of bifidobacteria in vitro and also in the small intestine

55 gosaccharides (GOSs) stimulate the growth of bifidobacteria in younger adults, but little is known ab

56 Consumer interest in probiotic bifidobacteria is increasing, but industry efforts to se

57 f fatty acid metabolism to administration of bifidobacteria is strain-dependent, and strain-strain di

58 growth of health-promoting lactobacilli and bifidobacteria is supported by FOS, giving it the classi

59 ization of glycolipids from the cell wall of bifidobacteria is the first step in correlating glycolip

60 Salivary Bifidobacteria levels were positively associated with th

61 iarrhea had differences in the proportion of bifidobacteria (median: 0.4% and 3.7%; interquartile ran

62 n early gut microbiota including E. coli and bifidobacteria might promote this maturation.

63 Salivary levels of Bifidobacteria, mutans streptococci, lactobacilli, and y

64 concentrations were linked to changes in the bifidobacteria number.

65 indeed show selective growth of infant-borne bifidobacteria on milk oligosaccharides or core componen

66 We have designed modified, 'bifidobacteria-optimised' universal primers, which we ha

67 05), Streptococcus sobrinus (p < 0.005), and Bifidobacteria (p < 0.0001) were associated with S-ECC,

68 sed fecal pH (P < 0.001) and increased fecal bifidobacteria (P < 0.001) and fecal lactate (P < 0.001)

69 significantly associated with only salivary Bifidobacteria (p < 0.001) and yeast (p < 0.001) levels

70 gsiae (p = 0.003), Streptococcus mutans with bifidobacteria (p < 0.001), and S. mutans with S. wiggsi

71 tervention), or to a control formula without bifidobacteria (placebo).

72 Bifidobacteria represent one of the dominant groups of m

73 Bifidobacteria should be regarded as caries-associated o

74 ver, clinical feeding studies with exogenous bifidobacteria show they don't remain in the intestine,

75 Finally, use of bifidobacteria slightly increased CLA relative content i

76 In contrast, adult-associated bifidobacteria such as the closely related B. longum sub

77 Bifidobacteria-supplemented animals had a significant re

78 Infants exposed to bifidobacteria-supplemented formula showed decreased occ

79 gastrointestinal tract is often dominated by bifidobacteria that flourish on milk glycans.

80 et caused an increase in total anaerobes and bifidobacteria, the highest densities occurred during su

81 This has encouraged scientific research into bifidobacteria, though recalcitrance to genetic manipula

82 thesis of saccharidic resource sharing among bifidobacteria through species-specific metabolic specia

83 often facilitated by mobile elements, allows bifidobacteria to adapt to fermentation environments in

84 variables (P > 0.05).The supplementation of bifidobacteria to infant diet can modulate the occurrenc

85 the influence of inulin on iron absorption, bifidobacteria, total bacteria, short-chain fatty acids

86 phospholipase A(2) expression were lower in bifidobacteria-treated pups than in controls, supporting

87 Infant-type bifidobacteria utilize these soluble carbohydrate oligom

88 multistrain preparation of lactobacilli and bifidobacteria was effective in prevention of AAD or CDD

89 t increase on the growth of lactobacilli and bifidobacteria was observed after exposition to the bark

90 nd facilitate functional genomic analyses in bifidobacteria, we created a large Tn5 transposon mutant

91 early colonization with Escherichia coli and bifidobacteria were associated with higher numbers of CD

92 While bifidobacteria were the dominant genus in the infant gut

93 fants from these countries were dominated by bifidobacteria, were different from each other, and were

94 multistrain preparation of lactobacilli and bifidobacteria, with a total of 6 x 10(10) organisms, on