ライフサイエンスコーパス: big ET-1

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1 rsion of exogenously added big endothelin-1 (big ET-1) to ET-1 in subcellular fractions obtained by s

2 ne systemic hemodynamics and plasma ET-1 and big ET-1 concentrations were measured using electrical b

3 , systemic hemodynamics, and plasma ET-1 and big ET-1 concentrations.

4 mately 90%) of vasoconstrictions to ET-1 and big ET-1.

5 only slightly reduced responses to ET-1 and big ET-1.

6 e analyzed for immunoreactivity for ET-1 and big ET-1.

7 indicate that there is an increase in ET and big ET-1 associated with fully developed atherosclerotic

8 Levels of endogenous ET and big ET-1 detectable by radioimmunoassay in human aorta c

9 Differences in affinity between big ET-1 and big ET-3 for ECE-1 thus appear to be due so

10 is a bona fide activating protease for both big ET-1 and big ET-3 in vivo, and that the cell-cell co

11 to block the hypertensive effects induced by big ET-1.

12 , and, to a much lesser extent, also cleaved big ET-1 and big ET-2 at Trp(21)-Val(22), yielding ET-1

13 Exogenous big ET-1 was added to permeabilized and nonpermeabilized

14 Immunoreactivity for big ET-1 and ET-1 was ubiquitous in the extracellular sp

15 aled intense immunofluorescence staining for big ET-1 and the 2 isoforms of ECE-1 (ECE-1alpha and ECE

16 except in the C-terminal residues, 34-38 in big ET-1 and 34-41 in big ET-3.

17 efficient hydrolysis of the W21-V22 bond in big ET-1 and which have the sequence QTVP in big ET-3.

18 which has a loop for residues 27-30 (HVVP in big ET-1), which have previously been demonstrated to be

19 eta resulted in an increase in intracellular big ET-1 and a decrease in SMC from the main artery.

20 tracellular site involved in the cleavage of big ET-1 to the biologically active peptide ET-1 by dete

21 nted cGMP, would inhibit ECE-1 conversion of big ET-1 to active ET-1, thus reducing tissue ET-1 conce

22 At pH 6.9, conversion of big ET-1 was inhibited markedly by 30 micromol/L PD15979

23 caused by intravenous infusion of Ang II or big ET-1 to a greater extent and with longer duration th

24 In anesthetized pigs, big ET-1-stimulated increases in systemic blood pressure

25 ET-1 precursor big ET-1 elicited time-dependent vasoconstriction over 2

26 immunoreactivity for ET-1 and its precursor, big ET-1, within the atheromatous plaque.

27 vesicles with a possible role in processing big ET-1 while in transit to the cell surface via the co

28 The findings of this study indicate that big ET-1 is processed to the mature vasoactive peptide b

29 -1 production in vivo as demonstrated by the big ET-1-induced pressor response in rats.

30 owed poor functional activity in vivo in the big ET-1 pressor test.

31 logy modeling were used to determine whether big ET-1 and big ET-3 adopt similar secondary and tertia

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