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1 nificantly lower than that induced by either biguanide.
2 Serine synthesis was not inhibited by biguanides.
3 K) is known to be a major cellular target of biguanides.
4 ga officinalis L.) led to the development of biguanides.
5 osphorylation are regulated independently by biguanides.
6 eated to determine whether polyhexamethylene biguanide, 0.02%, chlorhexidine digluconate, 0.02%, hexa
9 ed drugs (insulins, sulfonylureas, glinides, biguanides, alpha-glucosidase inhibitors, thiazolidinedi
11 s, C-fiber responses to injections of phenyl biguanide and lactic acid and to constant-pressure lung
12 ent, local irrigation with polyhexamethylene biguanide, and the systemic administration of voriconazo
17 ord and colleagues show, paradoxically, that biguanides are more effective in the treatment of mouse
18 anipulation that can enhance the efficacy of biguanides as antineoplastic agents that target cancer c
20 have investigated the effects of the diamino biguanide compound metformin and of hyperglycemia on MG
21 Bacillus subtilis , to develop guanidine and biguanide compounds with up to 20-fold increased potency
22 ese results suggest that cellular effects of biguanides depend on their metal-binding properties.
23 ed us to identify an anti-cancer drug of bis-biguanide dihydrochloride (BBD) as potent anti-mycobacte
28 r cell lines, the global metabolic impact of biguanides during the process of neoplastic transformati
31 he delocalization of cationic charges in the biguanide groups of PolyMet reduces the toxicity of PEI
34 g) or the 5-HT(3) agonist l-(m-chlorophenyl)-biguanide hydrochloride (mCPBG; 5.0-15.0 mg/kg), alone o
39 ular depletion of aPKC (>90%) led to loss in biguanide-induced aPKC phosphorylation, it had no effect
40 for serine to allow cells to compensate for biguanide-induced decrease in oxidative phosphorylation
44 c profiles are consistent with the idea that biguanides inhibit mitochondrial complex 1, but they ind
46 tochondrial oxidative phosphorylation (using biguanides) led to a complex response that could improve
47 he 5-HT3 receptor agonist 1-m-(chlorophenyl) biguanide (m-CPGB, 1 microM), markedly increased (300%)
52 % [14 of 15 isolates]) and polyhexamethylene biguanide (median growth grade, 0.0; kill incidence rate
54 CT1) affects the response to the widely used biguanide metformin (see the related article beginning o
55 onse to the sulphonylurea gliclazide and the biguanide metformin differed in HNF-1alpha diabetes and
57 In recent decades, the antihyperglycemic biguanide metformin has been used extensively in the tre
58 ion between TEA and Arg was found, while the biguanide metformin was able to strongly inhibit uptake
60 AMPK activators, including the antidiabetic biguanide metformin, inhibited FXR agonist induction of
62 tabolic effects of metformin and the related biguanide phenformin have been investigated in establish
66 widely used antimicrobial polyhexamethylene biguanide (PHMB) kills bacteria selectively over host ce
67 lyethyleneimine (PEI) and poly(hexamethylene biguanide) (PHMBG) and are prepared by a two-step proced
68 f chemosensitive vagal afferents with phenyl biguanide produced an increase (n=3), decrease (n=2), or
70 The objective of this study was to quantify biguanide related compounds (BRCs) in experimentally or
75 ication of the metal-liganding groups of the biguanide structure, supporting recent data that AMPK an
80 d pressure, statins adjusted for lipids, and biguanides, sulfonylureas, alpha-glycosidase inhibitors
81 kers, diuretics, nitrates, statins, insulin, biguanides, sulfonylureas, aspirin, and other nonsteroid
82 eported a series of (bis)guanidines and (bis)biguanides that are potent inhibitors of LSD1 and induce
83 and upregulation of ACAD10 are required for biguanides to reduce viability in melanoma and pancreati
84 vidence that this intrinsic property enables biguanides to regulate AMPK, glucose production, glucone
85 ring the process of cellular transformation, biguanide treatment prevents the boost in glycolytic int
88 in reducing glucose production has been the biguanides, which include phenformin and metformin, the
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