コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 mice in two separate murine models: CCl4 and bile duct ligation.
2 hepatic deposition of collagen 2 weeks after bile duct ligation.
3 sed markedly in the livers of mice following bile duct ligation.
4 IP-2 or rendered neutrophil deficient before bile duct ligation.
5 mice compared with wild-type mice following bile duct ligation.
6 y reduces the extent of acute fibrosis after bile duct ligation.
7 liver fibrosis induced by CCl4 treatment or bile duct ligation.
8 growth and mucosal injury in ileum caused by bile duct ligation.
9 lmonary syndrome developed only after common bile duct ligation.
10 ic acid on cholangiocyte proliferation after bile duct ligation.
11 proliferation and ductal mass in vivo after bile duct ligation.
12 cyte marker DPPIV were seen at 30 days after bile duct ligation.
13 and protein significantly decrease following bile duct ligation.
14 as influenced by other factors present after bile duct ligation.
15 ) controls were subjected to sham surgery or bile duct ligation.
16 production have also been found after common bile duct ligation.
17 ent lymphoproliferation (lpr) mice underwent bile duct ligation.
18 th chronic liver failure secondary to common bile duct ligation.
19 latively maintained 3 and even 14 days after bile duct ligation.
20 of bile salt excretion was determined after bile duct ligation.
21 vels, similar to that observed in rat common bile duct ligation.
22 lveolar vascular staining was enhanced after bile duct ligation.
23 butes to intrapulmonary vasodilatation after bile duct ligation.
24 n pulmonary artery rings were assessed after bile duct ligation.
25 rat model of cholestasis secondary to common bile duct ligation.
26 duced cholestasis in mouse livers via common bile duct ligation.
27 ntibody or control IgG and subjected them to bile duct ligation.
28 o duct injury induced by virus infection and bile duct ligation.
29 m mice following administration of CCl(4) or bile duct ligation.
30 ceptor I (TNFRI)-deficient mice subjected to bile duct ligation.
31 d development of sickness behavior following bile-duct ligation.
32 sed mesenteric vascular beds from rats after bile-duct ligation.
33 on of mice increased significantly following bile-duct ligation.
34 lated in three mouse models of liver injury (bile duct ligation, 1% cholic acid [CA] fed, and the Mdr
35 ers with biliary damage (Mdr2(-/-) knockout, bile duct ligation, 3,5-diethoxycarbonyl-1,4-dihydrocoll
37 and liver-specific p38alpha knockout mice by bile duct ligation and animals were sacrificed at 12 and
39 s of hepatic and bile duct injury, including bile duct ligation and CCl(4), D-galactosamine, and meth
41 dramatically increased in SHP(-/-) mice with bile duct ligation and in human cirrhotic livers, which
42 sis, fibrosis, and portal hypertension after bile duct ligation and is regulated by osmotically sensi
44 ormed with mice before and 2 weeks following bile duct ligation and with Fah-/- and Fah/p21-/- mice b
46 with INT-747 or vehicle during 10 days after bile-duct ligation and then were assessed for changes in
47 f PDGFRalpha in murine carbon tetrachloride, bile duct ligation, and 0.1% 3,5-diethoxycarbonyl-1,4-di
48 w, increase with bile acid administration or bile duct ligation, and account for only a small fractio
49 centration increased significantly following bile duct ligation, and both of these were prevented by
51 ary biliary cirrhosis was induced in rats by bile duct ligation, and portal hypertension was induced
52 ation; and 1-, 2-, 3-, 4-, and 5-week common bile duct ligation animals by Northern, Western and immu
53 ibition with tin protoporphyrin IX in common bile duct ligation animals was used to define effects on
54 cular monocytes/macrophages in 3-week common bile duct ligation animals, whereas pulmonary microvascu
56 l lamina propria of wild-type mice following bile duct ligation; bacterial translocation was facilita
58 osis in Alfp-Cre x Rosa26-YFP mice using the bile duct ligation (BDL) (2, 4, and 8 weeks), carbon tet
60 th factor (HGF) on liver fibrosis induced by bile duct ligation (BDL) and investigated potential mech
61 e 1 (ICAM-1) is induced in mouse liver after bile duct ligation (BDL) and plays a key role in neutrop
62 study, we aimed to first validate rats with bile duct ligation (BDL) as a model for hepatic pruritus
63 unctions) and (2) proliferate in response to bile duct ligation (BDL) by activation of cyclic adenosi
66 s, male Sprague-Dawley rats underwent common bile duct ligation (BDL) for 14 days and were treated wi
69 with secondary biliary cirrhosis induced by bile duct ligation (BDL) for 4 weeks (n = 5) and in pair
72 changes in the GSH synthetic enzymes during bile duct ligation (BDL) in mice and how treatment with
76 Mrp3(Abcc3) is markedly induced following bile duct ligation (BDL) in the rat and in some human ch
78 the cholestatic phenotype induced by common bile duct ligation (BDL) is reduced in mice genetically
82 isposition toward infections, the effects of bile duct ligation (BDL) on bulk intrahepatic T cells ha
83 estasis and the PiZZ phenotype, we performed bile duct ligation (BDL) on C57BL/6 mice possessing a tr
85 icroscopy in fibrotic livers from mice after bile duct ligation (BDL) or CCl(4) administration and in
88 late cells from rat livers injured by either bile duct ligation (BDL) or repeated carbon tetrachlorid
92 e obtained from serum and bile of rats after bile duct ligation (BDL) or sham surgery and applied to
93 Male Sprague-Dawley rats were subjected to bile duct ligation (BDL) or sham surgery, and liver and
97 Biliary hyperplasia was induced in rats via bile duct ligation (BDL) surgery, and galanin was increa
99 te cotransporting polypeptide (Ntcp), common bile duct ligation (BDL) was performed in pregnant rats
100 e mapped in vivo during the first week after bile duct ligation (BDL) when peak BEC DNA synthesis is
102 ocytes isolated from rats that had undergone bile duct ligation (BDL), an experimental model of bilia
103 ely functional hepatectomy (HepX), and 2-day bile duct ligation (BDL), as well as cultured human fibr
104 s were performed in wild-type (WT) mice with bile duct ligation (BDL), BDL SR(-/-) mice, or Mdr2(-/-)
106 (HA) and MCs infiltrate the liver following bile duct ligation (BDL), increasing intrahepatic bile d
110 massive hepatic necrosis and mortality after bile duct ligation (BDL), whereas treatment of these mic
111 estasis, hepatocytes cultured from livers of bile duct ligation (BDL)- or ethinyl estradiol (EE)-inje
113 nduced liver injury and fibrosis, a model of bile duct ligation (BDL)-induced hepatic fibrosis in viv
138 Adult Sprague-Dawley rats 4 weeks after bile duct ligation (BDL)/Sham-operation, or naive rats f
140 the cerebral cortex using rat models of HE (bile duct ligation [BDL] and induced hyperammonemia) and
142 ld-type controls to compensate for long-term bile duct ligation because of significantly greater hepa
144 ein levels increased severalfold with common bile duct ligation but were unchanged with either endoto
145 ent of hepatopulmonary syndrome after common bile duct ligation, but not in thioacetamide-induced cir
146 nd S-adenosylmethionine are anti-fibrotic in bile duct ligation, but this effect was nearly lost if G
147 n response to secretin and proliferate after bile duct ligation by activation of cyclic adenosine 3',
148 mental hepatopulmonary syndrome after common bile duct ligation by stimulating pulmonary endothelial
151 ion were assessed in rat livers after common bile duct ligation (CBDL) from 1-7 days, and taurocholat
154 s of endotoxin, ethinylestradiol, and common bile duct ligation (CBDL) on Mrp2 protein, messenger RNA
156 Cirrhosis was induced in rats by common bile duct ligation (CBDL), and they were compared with s
157 In a rat model of HPS induced by common bile duct ligation (CBDL), but not thioacetamide (TAA)-i
161 s study examined rats 1 to 3 wk after common bile-duct ligation (CBDL), at which time they had hyperb
163 totoxin (carbon tetrachloride) injection and bile duct ligation, constitutive FGFR1 signalling in liv
165 O mice treated with carbon tetrachloride and bile duct ligation developed reduced fibrosis versus wil
168 ce that were injected with iNKT cells before bile duct ligation exhibited significant decreases in ne
171 ce showed increased liver fibrosis following bile duct ligation for 21 days or chronic carbon tetrach
175 d hyperplastic cholangiocytes isolated after bile duct ligation from either syngeneic Wistar or allog
176 -alpha (TGF-alpha) was observed 7 days after bile duct ligation in adult rats, the expression of all
177 e established by thioacetamide injection and bile duct ligation in Balb/C mice and treated with soraf
178 h SCF and c-kit were clearly increased after bile duct ligation in both control and mutant mice.
179 procedure to reconstruct biliary flow after bile duct ligation in C57BL/6 mice to generate a model o
180 n-transposase complex was coupled with lobar bile duct ligation in C57BL/6 mice, followed by administ
182 y expressed in small bile ducts 7 days after bile duct ligation in immature rats up to 5 weeks of age
184 d from liver and kidney 14 days after common bile duct ligation in rats and assessed by RNA protectio
187 hepatic endothelin 1 production after common bile duct ligation in relation to thioacetamide cirrhosi
189 vo by carbon tetrachloride i.p. injection or bile duct ligation in wild-type and SEMA7A knockout (KO)
190 from the TGFbeta receptor is increased after bile duct ligation in wild-type mice but not in beta6(-/
192 opolysaccharide-resistant C3H/HeJ mice after bile duct ligation, indicating that Toll-like receptor 4
193 nse to both chronic carbon tetrachloride and bile duct ligation induced injury was also impaired and
196 motile hepatic stellate cells, but not from bile duct ligation-induced fibrosis, in which portal fib
197 alizing PDGF-C antibodies are protected from bile duct ligation-induced liver fibrosis, and we compar
201 creased serum levels of Ang II and augmented bile duct ligation-induced liver injury, as assessed by
202 in livers of wild-type and Fmod(-/-) mice by bile duct ligation, injection of CCl(4), or administrati
204 ter, some mice also received left and median bile duct ligation (LMBDL) and, then 1 week later, were
205 expression increased in activated HSCs from bile duct ligation mice and during HSC activation in vit
206 spite the same insult of long-term (12-week) bile duct ligation, mice prone to decompensation showed
209 er models of chronic liver injury, including bile duct ligation, nonalcoholic steatohepatitis, and ob
210 m obstructive extrahepatic cholestasis after bile duct ligation or administration of alpha-naphthylis
216 levels varied after liver injury induced by bile duct ligation or repeated CCl4 administration, incl
217 othelin 1 levels were evaluated after common bile duct ligation or thioacetamide administration.
220 ed in the livers of mice that have undergone bile duct ligation or were fed a 3,5-diethoxycarbonyl-1,
223 l of acute cholestatic liver injury, partial bile duct ligation (pBDL), with a novel longitudinal bio
224 iary fibrosis and early cirrhosis induced by bile duct ligation, preproET-1 mRNA and immunoreactive E
226 progressively from 3 to 5 weeks after common bile duct ligation relative to controls (5-week protein
227 nd portal hypertension due to chronic common bile duct ligation reproduce the features of human hepat
228 genes in hepatocytes and in the liver after bile duct ligation required early growth response factor
229 Administering Ad-SMAdnPI3K to mice following bile duct ligation resulted in reduced HSC activation an
233 oreover, both Sl and W mice responded to the bile duct ligation, similar to the control mice, by deve
235 ng common bile duct ligation or left hepatic bile duct ligation, the expression of p53, c-Myc, and cy
240 due to either carbon tetrachloride injury or bile duct ligation, we demonstrate de novo expression of
242 ls isolated from adult rats with and without bile duct ligation were incubated with Hh ligand-enriche
246 stablished chimeric livers were subjected to bile duct ligation, with or without pretreatment with th
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。