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1 in biliary epithelial cell infection causing bile duct obstruction.
2 ts are placed for palliation of extrahepatic bile duct obstruction.
3 -/-) mice did not have hyperbilirubinemia or bile duct obstruction.
4 tic intrahepatic cholangiocarcinomas without bile duct obstruction.
5 ent typical clinical and radiologic signs of bile duct obstruction and cholangitis, her blood analysi
6 wth within liver, paralleled by increases in bile duct obstruction and gross peritoneal metastases.
8 n of cholestasis, decreased the incidence of bile duct obstruction, and improved survival above wild-
9 Lower survival is also determined by distal bile duct obstruction, Bismuth- Corlette type IV strictu
11 f recombinant IFN-gamma led to recurrence of bile duct obstruction following rotavirus infection of I
18 ally covered) for palliation of extrahepatic bile duct obstruction initially is more expensive than p
20 otal bilirubin, indicating that extrahepatic bile duct obstruction leads to down-regulation of NTCP m
21 ecular mechanisms of injury and repair after bile duct obstruction, microarray analysis was performed
23 he onset of BEC proliferation in response to bile duct obstruction was associated with diminished HNF
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