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1 in biliary epithelial cell infection causing bile duct obstruction.
2 ts are placed for palliation of extrahepatic bile duct obstruction.
3 -/-) mice did not have hyperbilirubinemia or bile duct obstruction.
4 tic intrahepatic cholangiocarcinomas without bile duct obstruction.
5 ent typical clinical and radiologic signs of bile duct obstruction and cholangitis, her blood analysi
6 wth within liver, paralleled by increases in bile duct obstruction and gross peritoneal metastases.
7 nd SA11-FM caused clinical manifestations of bile duct obstruction and high mortality.
8 n of cholestasis, decreased the incidence of bile duct obstruction, and improved survival above wild-
9  Lower survival is also determined by distal bile duct obstruction, Bismuth- Corlette type IV strictu
10                                              Bile duct obstruction causes neutrophilic inflammation o
11 f recombinant IFN-gamma led to recurrence of bile duct obstruction following rotavirus infection of I
12             The etiology and pathogenesis of bile duct obstruction in children with biliary atresia a
13       In this study, we investigated whether bile duct obstruction in rats induces portal expression
14 tes undergo adaptive regulation after common bile duct obstruction in the rat.
15                                           In bile duct obstruction, increased biliary bile acid conce
16         We tested the hypothesis that during bile duct obstruction, increased biliary bile acids trig
17                                 We show that bile duct obstruction induces a striking increase in cho
18 ally covered) for palliation of extrahepatic bile duct obstruction initially is more expensive than p
19                                    Malignant bile duct obstruction is a common problem among cancer p
20 otal bilirubin, indicating that extrahepatic bile duct obstruction leads to down-regulation of NTCP m
21 ecular mechanisms of injury and repair after bile duct obstruction, microarray analysis was performed
22        In bile duct-incannulated (BDI) rats, bile duct obstruction present for 7 days was relieved fo
23 he onset of BEC proliferation in response to bile duct obstruction was associated with diminished HNF
24              Cholangiocytes respond to acute bile duct obstruction with markedly increased expression

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