コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 with somatostatin analogues in order to stop bile secretion.
2 ns", including AQPs, involved in canalicular bile secretion.
3 volved in the molecular mechanisms of ductal bile secretion.
4 through unknown mechanisms, in disorders of bile secretion.
5 rred in the absence of any defect in hepatic bile secretion.
6 xists regarding insulin regulation of ductal bile secretion.
7 e involved in agonist-stimulated canalicular bile secretion.
8 of liver metabolism, hepatic blood flow, and bile secretion.
9 dysfunction, suggesting a slower recovery of bile secretion.
10 ne and for generation of bile acid-dependent bile secretion.
11 oposed that this was a key process in ductal bile secretion.
12 response to a temporal demand for increased bile secretion.
13 l functions including stimulatory effects on bile secretion.
14 mportant component of hormonal regulation of bile secretion.
15 ological functions and is known to stimulate bile secretion.
16 ng to an increase in secretin-induced ductal bile secretion.
24 ts, resulting in modification of canalicular bile secretion and modulation of ductal bile secretion a
25 nts, Atp8b1(G308V/G308V) mice had unimpaired bile secretion and no liver damage, but showed mild abno
26 brane conductance regulator (CFTR) regulates bile secretion and other functions at the apical membran
27 to be essential for secretin-induced ductal bile secretion and suggests that AQP1 can be regulated b
28 de insights into the molecular mechanisms of bile secretion and the development of new experimental m
29 de insights into the molecular mechanisms of bile secretion and the development of new experimental m
30 s were associated with functional changes in bile secretion and with decreases of intracellular cycli
31 the major determinant of bile salt-dependent bile secretion, and its deficiency leads to cholestatic
33 though secretin is known to stimulate ductal bile secretion by directly interacting with cholangiocyt
34 the molecular mechanisms of hormone-induced bile secretion, development of new experimental models,
35 re likely involved in canalicular and ductal bile secretion, gluconeogenesis and microbial infection
39 iocyte proliferation and increases in ductal bile secretion in large but not small cholangiocytes.
40 epithelia appears to be important for normal bile secretion in the liver, and loss of InsP3Rs may be
42 omeostasis without impairment of canalicular bile secretion; in humans this process is likely to be o
45 iocytes responsible for regulation of ductal bile secretion is an initial and required step in genera
46 tes responsible for the regulation of ductal bile secretion is an initial and required step in genera
48 cholangiocyte transport systems involved in bile secretion may provide a molecular correlate for the
49 cholangiocyte transport systems involved in bile secretion may provide a molecular correlate for the
50 es provide evidence that certain products of bile secretion may undergo a "biliohepatic" circulation.
51 omy, the increase in secretin-induced ductal bile secretion observed during bile duct renewal results
52 iocytes is responsible for the impairment in bile secretion that occurs in a number of liver diseases
53 es insights into the molecular mechanisms of bile secretion, the development of new experimental mode
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。