ライフサイエンスコーパス: biliary cancer

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1 tor have each been shown to have activity in biliary cancer.

2 GFR blockade with erlotinib in patients with biliary cancer.

3 in liver to later-developed mucin-producing biliary cancer.

4 ollow-up, 13 patients (3.3%), presented with biliary cancer.

5 peutic alternative in patients with advanced biliary cancer.

6 4 adverse effects in patients with advanced biliary cancers.

7 ts of genetic inactivation in pancreatic and biliary cancers.

8 We included 64 biliary cancers, 122 liver cancers, and 224 age-matched

9 ven the possibility of future development of biliary cancer after CC resection.

10 NPs examined, 14 were related to the risk of biliary cancer and stones.

11 (21%) were performed for primary hepatic or biliary cancers and 161 (9%) for benign disease.

12 differentially up-regulated genes in primary biliary cancers and biliary cancer cell lines and their

13 served in sporadic pulmonary, pancreatic and biliary cancers and melanomas.

14 oup for gallstone disease, susceptibility to biliary cancer, and show variants that alter sterolin fu

15 nt study has not been previously reported in biliary cancers, and represent novel potential screening

16 Cholangiocarcinoma (CCA) is a biliary cancer arising from damaged bile ducts.

17 Biliary cancers (BCs) carry a poor prognosis, but target

18 eceptor 1 and ligand expression is common in biliary cancers (BILI) and may be associated with worse

19 genes was confirmed in tissue microarrays of biliary cancers by immunohistochemical analysis (n = 4)

20 ly resected biliary carcinomas (n = 11), and biliary cancer cell lines (n = 9).

21 gulated genes in primary biliary cancers and biliary cancer cell lines and their expression profiles

22 4) or in situ hybridization (n = 1), and in biliary cancer cell lines by reverse transcriptase PCR (

23 type: metastatic neuroendocrine (81 months), biliary cancer (cholangiocarcinoma) (63 months), gallbla

24 plant hepatocellular (HR 2.92, P = 0.001) or biliary cancer (HR 12.7, P < 0.001), glomerular filtrati

25 11/LKB1 in the development of pancreatic and biliary cancer in patients with and without the PJS.

26 al to tumorigenesis were up-regulated in the biliary cancers, including proliferation and cell cycle

27 eukemia or lymphoma, and those with liver or biliary cancer; it was longest for those with chronic lu

28 0), miscellaneous liver metastases (N = 42), biliary cancer (N = 20), and benign tumors (N = 176).

29 Biliary cancers overexpress epidermal growth factor rece

30 subgroup with native-liver hepatocellular or biliary cancer (P = 0.02).

31 GABA decreases biliary cancer proliferation and reduces the metastatic

32 e allelotype of 82 xenografted pancreatic or biliary cancers using 386 microsatellite markers and spa

33 noma (CCA) includes a heterogeneous group of biliary cancers with poor prognosis.

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