ライフサイエンスコーパス: biliary stricture

1 ed gastric emptying, pancreatic fistula, and biliary strictures).

2 e incidence of postoperative cholangitis and biliary stricture.

3 ts after bilioenteric anastomoses for benign biliary stricture.

4 onfunction, early allograft dysfunction, and biliary strictures.

5 ining acceptance for the treatment of benign biliary strictures.

6 included 19 (3.3%) bile leaks and 27 (4.6%) biliary strictures.

7 n the diagnostic algorithm for indeterminate biliary strictures.

8 described in the evaluation of indeterminate biliary strictures.

9 anastomoses in the evaluation and therapy of biliary strictures.

10 There have been no reoperations for biliary strictures.

11 s were performed during 112 treatments of 84 biliary strictures.

12 BD) was performed in 85 patients with benign biliary strictures.

13 olangiopancreatography for the evaluation of biliary strictures.

14 se of metallic endobiliary stents for benign biliary strictures.

15 the differentiation of benign and malignant biliary strictures.

16 initial modality for treating posttransplant biliary strictures.

17 ients had 14 (23.7%) bile leaks and 4 (6.8%) biliary strictures.

18 dren had three bile leaks (6%) and six (12%) biliary strictures.

19 Nine patients had developed biliary strictures.

20 12 men, aged 51+/-11 years) with anastomotic biliary stricture after LDLT.

21 was performed to determine the incidence of biliary stricture after PD.

22 le and safe for the treatment of anastomotic biliary strictures after LDLT.

23 f a protocol of initial balloon dilation for biliary strictures after liver transplantation.

24 There was no difference in the incidence of biliary strictures after resection for benign (n = 10, 2

25 e protection against radiological changes of biliary stricture, although possibly to a lesser degree

26 Among patients with benign biliary strictures and a bile duct diameter 6 mm or more

27 Revision of the HA may help prevent biliary strictures and allow for good long-term graft fu

28 tic endotherapy by ERCP for the treatment of biliary strictures and chronic pain in chronic pancreati

29 also assessed the incidence of ischemic type biliary strictures and hepatic artery thrombosis, and ev

30 The incidence of nonanastomotic biliary strictures and hepatic histologic findings sugge

31 Many patients with IAC present with biliary strictures and obstructive jaundice, making chol

32 al or radiographic evidence of postoperative biliary stricture, and all patients underwent successful

33 ary complications: 21 had bile leaks, 22 had biliary strictures, and 4 had both complications.

34 urvival, method of HA revision, incidence of biliary strictures, and long-term HA patency.

35 ences in IC among centers, the importance of biliary strictures as a risk factor for graft failure, a

36 IAC should be suspected in unexplained biliary strictures associated with increased serum IgG4

37 nically significant restenosis after PBBD of biliary strictures at anastomotic and nonanastomotic sit

38 in pancreatic duct, with associated multiple biliary strictures at ERCP.

39 Benign biliary strictures (BBS) respond to placement of multipl

40 al vein thrombosis, primary nonfunction, and biliary stricture between the two groups.

41 Biliary strictures, both anastomotic and nonanastomotic,

42 ed diagnostic features including presence of biliary stricture by using a five-point confidence scale

43 mary outcome was development of intrahepatic biliary strictures consistent with IC.

44 biliary-enteric anastomoses in 79 (20%), and biliary strictures following liver transplantation in 88

45 institution review examined the incidence of biliary stricture formation after pancreaticoduodenectom

46 The pathogenesis of intrahepatic biliary stricture formation in patients with primary scl

47 Biliary stricture formation is an infrequent complicatio

48 e only significant univariate predictors for biliary stricture formation were preoperative and postop

49 reoperative jaundice did not protect against biliary stricture formation.

50 Indeterminate biliary strictures frequently present as a diagnostic co

51 in situ hybridization in brushing samples of biliary strictures had a sensitivity of 50%, 57%, and 86

52 ndardized classification of pCLE findings of biliary strictures has been proposed.

53 Biliary strictures have a negative effect on HCV fibrosi

54 years was associated with increased rate of biliary strictures (hazard ratio [HR], 1.67; 95% confide

55 Biliary strictures (HR = 2.25, P = 0.0006), creatinine a

56 ] = 1.38 for doubling of AST, P = 0.005) and biliary strictures (HR = 2.68, P = 0.0001) were associat

57 low less than 400 mL/min was associated with biliary strictures (HR, 1.53; 95% CI, 1.04-2.24; P = 0.0

58 Intrahepatic biliary strictures (IHBS) without hepatic artery thrombo

59 All cases with biliary stricture in DCD group finally led to graft loss

60 The development of a biliary stricture in patients who have undergone PD for

61 most frequent indications were postoperative biliary strictures in 123 (31%), stenosed biliary-enteri

62 n with advances, such as stent placement for biliary strictures in a patient after liver transplantat

63 hould exert a therapeutic role in preventing biliary strictures in liver allografts.

64 incidence and later onset of nonanastomotic biliary strictures in patients with PSC compared with a

65 crease the intraoperative HA flow to prevent biliary strictures in such patients.

66 L/min were associated with increased rate of biliary strictures in younger donors (<50 years old), an

67 00 mL/min was associated with higher rate of biliary strictures in younger donors with duct-to-duct r

68 is the first-line treatment for most benign biliary strictures; it is possible that fully covered, s

69 rtal vein stenosis/thrombosis, ischemic-type biliary stricture (ITBS), and primary nonfunction were s

70 luding chronic pancreatitis (n = 10), benign biliary stricture (n = 1), pancreatic complex cyst (n =

71 AC (n = 1) and a benign chronic inflammatory biliary stricture (n = 1), respectively.

72 detection of bile duct dilatation (n = 27), biliary strictures (n = 10), and intraductal abnormaliti

73 olving patients with treatment-naive, benign biliary strictures (N = 112) due to orthotopic liver tra

74 transplant, 162 (21.8%) patients developed a biliary stricture, of which 88 (11.8%) exhibited intrahe

75 ng was favored with pretest probabilities of biliary stricture or malignancy 0%-73% for patients aged

76 t seen in patients with either postoperative biliary strictures or recurrence of PSC.

77 of hepatic artery thrombosis, nonanastomotic biliary strictures, or 4-month allograft survival.

78 prolonged transplantation time (P=0.02), and biliary stricturing (P=0.016) were identified as signifi

79 tion of varying etiologies (including benign biliary stricture, papillary stenosis, choledocholithias

80 c pancreatitis and pancreatic cancer-induced biliary strictures, postoperative bile leaks, the use of

81 percentile (P=0.035), reoperation (P=0.005), biliary stricturing postoperatively (P=0.046), and gastr

82 rs in 11 countries, 187 patients with benign biliary strictures received FCSEMS.

83 No patient developed a biliary stricture, required re-exploration for intra-abd

84 Steroid-sensitive biliary strictures resembling primary sclerosing cholang

85 mptomatic and are still alive, 1 developed a biliary stricture that was stented and is alive 105 mont

86 clinical dilemma because they harbor chronic biliary strictures that are difficult to distinguish fro

87 ch in the endoscopic management of malignant biliary strictures, the broadening therapeutic indicatio

88 cted against the formation of nonanastomotic biliary strictures versus CC/CT patients (12.6%; P = 0.0

89 The overall rate of biliary strictures was greater in the DCD group at 1 yea

90 By univariate analysis, biliary strictures were associated with preoperative per

91 At presentation, biliary strictures were confined to the intrapancreatic

92 nown causes of posttransplant nonanastomotic biliary strictures were excluded leaving 120 patients fo

93 Complete records from 96 patients with biliary strictures were retrospectively reviewed.

94 hybridization (FISH) can be used to evaluate biliary strictures with greater accuracy than convention

95 Distinguishing benign from malignant biliary strictures with routine biliary cytology in this

96 ic cholestasis, but many develop progressive biliary strictures with time, leading to recurrent chola