ライフサイエンスコーパス: biliary tract

1 ensitivity in vitro (e.g., Salmonella in the biliary tract).

2 transformation of cholangiocytes lining the biliary tract.

3 for patients affected by malignancies of the biliary tract.

4 in human inflammatory diseases involving the biliary tract.

5 ransporters can only extrude Mg(2+) into the biliary tract.

6 epithelial differentiation in the kidney and biliary tract.

7 life-threatening bacterial infection of the biliary tract.

8 us cancers, including those of the liver and biliary tract.

9 ity and specificity in the evaluation of the biliary tract.

10 ntly (P < .05) inhibited colonization of the biliary tract.

11 lems that did not involve the gallbladder or biliary tract.

12 cid transporter (ISBT) in the intestinal and biliary tract.

13 in mouse extraintestinal organs such as the biliary tract.

14 ic disease that involves the kidneys and the biliary tract.

15 of cancer and with 126 controls with normal biliary tracts.

16 onella infection of the gastrointestinal and biliary tracts.

17 Sites most commonly predicted were biliary tract (18%), urothelium (11%), colorectal (10%),

18 lopmental and proliferative disorders of the biliary tract; (2) to foster a better and more comprehen

19 We conclude that biliary tract abnormalities are the primary risk factors

20 to our understanding of gallstones and other biliary tract abnormalities.

21 utaneous liver biopsy in OLT recipients with biliary tract abnormalities.

22 spin-echo (3D FSE) MRC for the evaluation of biliary tract abnormalities.

23 s developed only in patients with underlying biliary tract abnormalities; the frequency of infection

24 tomic defects or evidence of aneuploidy or a biliary tract abnormality.

25 Biliary tract adenocarcinomas (BTAs), although anatomica

26 The effect of HCV infection on the biliary tract after liver transplantation (LT) is not we

27 subjects who underwent right lobe retrieval, biliary tract anatomy determined at CT cholangiography w

28 CT cholangiography accurately depicts biliary tract anatomy in living donor candidates for rig

29 Imaging findings of second-order biliary tract anatomy were compared with intraoperative

30 bs, MBECs 5 to 8, reacted strongly along the biliary tract and by immunoblot analysis, reacted with s

31 ngiography enables accurate depiction of the biliary tract and detection of biliary complications in

32 ted by two radiologists for depiction of the biliary tract and ductal anastomosis and for complicatio

33 inflammatory obstruction of the extrahepatic biliary tract and intrahepatic bile ducts.

34 ver fibrosis is caused by obstruction of the biliary tract and is associated with early activation of

35 represents the most common malignancy of the biliary tract and is highly lethal with less than 5% ove

36 ent was ineffective against infection of the biliary tract and large intestine.

37 arly carcinogenic event in the human hepatic biliary tract and one that is consistent with a frequent

38 hose originating in the oesophagus, stomach, biliary tract and pancreas, but not in the colon, displa

39 lignancies of the esophagus, stomach, colon, biliary tract, and liver.

40 PSC and determine the location of PSC in the biliary tract, and then compared the findings with those

41 ining to the histopathology of the liver and biliary tract are reviewed.

42 y syndrome cholangiopathy, MRCP depicted the biliary tract as clearly as did ERCP (n = 9).

43 d noted the presence of variant second-order biliary tract branching anatomy.

44 l systems of mice revealed tuft cells in the biliary tract but not the normal pancreatic duct.

45 Although infection of the intestinal and biliary tracts by Cryptosporidium parvum is a major prob

46 The study included 411 cases with biliary tract cancer (237 gallbladder, 127 extrahepatic

47 The benefit of adjuvant therapy (AT) for biliary tract cancer (BTC) is unclear, with conflicting

48 ts (pts) with hepatocellular (HCC, n=33) and biliary tract cancer (BTC, n=37) were enrolled into a ph

49 the risk for primary liver and extrahepatic biliary tract cancer among 186,395 patients hospitalized

50 ic differences in these distinct subtypes of biliary tract cancer and demonstrates that approximately

51 ellular biology, diagnosis, and treatment of biliary tract cancer are reviewed.

52 in, we evaluated the role of beta-catenin in biliary tract cancer by sequencing the third exon of the

53 Exposure of the resistant biliary tract cancer cell line HuCCT1 but not the suscep

54 onfirmed or cytologically confirmed advanced biliary tract cancer from hepatobiliary oncology referra

55 d in Shanghai, China, where the incidence of biliary tract cancer has been increasing in recent decad

56 sion-free survival of patients with advanced biliary tract cancer in combination with cisplatin and g

57 n to chemotherapy for patients with advanced biliary tract cancer remains investigational.

58 y responses may predispose to gallstones and biliary tract cancer, suggesting the need for future stu

59 mmation in the development of gallstones and biliary tract cancer, we examined the risk associated wi

60 ive means for the diagnosis and treatment of biliary tract cancer.

61 ne how H. pylori might influence the risk of biliary tract cancer.

62 SNPs and haplotypes with biliary stones and biliary tract cancer.

63 anisms for the initiation and progression of biliary tract cancer.

64 motherapy regimen for patients with advanced biliary tract cancer; expression of VEGF and its recepto

65 Biliary tract cancers (BTC) comprise a group of uncommon

66 Biliary tract cancers (BTC), which encompass intra- and

67 The management of biliary tract cancers (BTCs) has been challenging partly

68 aining of tissue microarrays (TMAs) from 223 biliary tract cancers (BTCs) was used to analyze candida

69 Biliary tract cancers (BTCs), which encompass intra- and

70 beta-catenin were present in 8 of 107 (7.5%) biliary tract cancers and 4 of 7 (57.1%) gallbladder ade

71 hird exon of the beta-catenin gene among 107 biliary tract cancers and 7 gallbladder adenomas from a

72 ous polyposis coli (APC) gene are present in biliary tract cancers and the APC protein modulates leve

73 Biliary tract cancers are a heterogeneous group of cance

74 The challenges posed by these often lethal biliary tract cancers are daunting, with conventional tr

75 f 5.47 (95% CI: 1.17-25.65) observed for the biliary tract cancers combined.

76 of targeted agents will make the subsets of biliary tract cancers even smaller but is likely necessa

77 ins was associated with an increased risk of biliary tract cancers in ATBC.

78 hepatic bile duct (IBD), and gallbladder and biliary tract cancers outside of the liver (GBTC) in a n

79 a role in the pathogenesis of gallstones and biliary tract cancers.

80 xaliplatin (GEMOX) in patients with advanced biliary-tract cancers (BTCs).

81 Bile acids have been implicated in biliary tract carcinogenesis, in part, by activating the

82 esent a unique new animal model for studying biliary tract carcinogenesis.

83 ne and has become a new standard in advanced Biliary Tract Carcinoma (aBTC).

84 Biliary tract carcinoma carries a poor prognosis, and di

85 h chemotherapeutic options remain limited in biliary tract carcinoma, radiation therapy may provide a

86 Thirty patients were studied who had PSC and biliary tract carcinoma.

87 Most biliary tract carcinomas complicating PSC can be demonst

88 g cholangitis (a premalignant disease of the biliary tract) compared with controls.

89 Biliary tract complications are less well recognized.

90 study were to: (1) evaluate the incidence of biliary tract complications using a new method of side-t

91 Biliary tract complications were categorized as bile lea

92 logic, although a comparable number suffered biliary tract complications, either from obstruction or

93 or not use of the T tube leads to increased biliary tract complications.

94 nt of therapies aimed at halting the ongoing biliary tract destruction found in immune-mediated chola

95 privately insured patients for 4 diagnoses: biliary tract disease (odds ratio, 0.73 [95% CI, 0.55 to

96 with ADPKD had higher rates of admission for biliary tract disease (rate ratio [RR], 2.24; 95% confid

97 antially, but ADPKD remained associated with biliary tract disease (RR, 1.19; 95% CI, 1.08 to 1.31) a

98 ies have evaluated the management options of biliary tract disease after OHT.

99 d tomography may improve staging accuracy in biliary tract disease and plays a definite role in diagn

100 Absolute excess risk of biliary tract disease associated with ADPKD was larger t

101 Two patients died of reasons unrelated to biliary tract disease before the completion of treatment

102 Endoscopic management of biliary tract disease continues to be influenced by new

103 Biliary tract disease developed in 17 patients (5%) who

104 cholecystectomy has transformed the care of biliary tract disease in Mongolia.

105 Overall, biliary tract disease seems to be a distinct and importa

106 Major updates in the management of biliary tract disease using biliary endoscopy are discus

107 The ADPKD versus non-ADPKD RRs for biliary tract disease were larger for men than women (he

108 gh commonly occurring in patients with known biliary tract disease, is often cryptogenic in origin (i

109 Hospitalization rates for biliary tract disease, serious liver complications, and

110 spontaneous and fatal autoimmune polycystic biliary tract disease, with lymphocytic peribiliary infi

111 ture related to the diagnosis and therapy of biliary tract disease.

112 d a hypothesis that ADPKD is associated with biliary tract disease.

113 warrant its use in the routine diagnosis of biliary tract disease.

114 associated with increased ICC risk included biliary tract diseases (adjusted odds ratio [AOR]: 81.8;

115 he mechanisms regulating the pathogenesis of biliary tract diseases and in devising new treatment app

116 mechanisms that underlie the pathogenesis of biliary tract diseases.

117 s (VEGFs) participate in the pathogenesis of biliary tract diseases.

118 s are more susceptible than males to several biliary tract diseases.

119 nesis play an important role in a variety of biliary tract diseases.

120 er the past year in endoscopic approaches of biliary tract diseases.

121 ndoscopy are discussed over a broad range of biliary tract diseases.

122 rstanding and treatment of several liver and biliary tract disorders of childhood.

123 er, hemochromatosis, Wilson disease, several biliary tract disorders, and pathology of liver tumors,

124 (ICD-9) codes: laparoscopic cholecystectomy, biliary tract disorders, pneumonia, and cellulitis.

125 thways, as well as a possible involvement in biliary tract disorders, require further studies.

126 formation is required, such as management of biliary tract dysplasia and cancer chemoprevention in PS

127 ngiocarcinomas are malignancies arising from biliary tract epithelia that are associated with a poor

128 erexpression of ErbB-2 in the basal layer of biliary tract epithelium led to the development of gallb

129 langiocarcinoma is a malignancy arising from biliary tract epithelium that is increasing in incidence

130 pulate the entire length of the extrahepatic biliary tract, except the gallbladder.

131 e and effective technique to reconstruct the biliary tract following hepatic transplantation.

132 carcinoma is an aggressive malignancy of the biliary tract for which effective treatment is lacking.

133 Although the effects of progesterone on the biliary tract have been implicated in the increased inci

134 (GBC), the most aggressive malignancy of the biliary tract, have a poor prognosis.

135 is a specific pattern of involvement of the biliary tract in patients with PSC and to evaluate featu

136 ls with stones and from controls with normal biliary tracts in their serum and bile biochemistries.

137 4 % (2/58), pancreatitis in 8.6 % (5/58) and biliary tract infection (BTI) in 1.7 % (1/58) of patient

138 of sialic acid in reovirus encephalitis and biliary tract infection in mice.

139 eloped early postoperative IAI (peritonitis, biliary tract infection, abdominal abscess, or enteritis

140 rse events (one acute coronary syndrome, one biliary tract infection, one other neoplasms, and two co

141 isfortune to have, during cholecystectomy, a biliary tract injury which required four subsequent bili

142 erol in hepatocytes and secreted through the biliary tract into the small intestine, where they aid i

143 suppressed host is frequently complicated by biliary tract involvement.

144 us strain-specific replication in the murine biliary tract is determined by both viral entry mediated

145 tient admission, with particular emphasis on biliary tract issues.

146 holangiocarcinoma (CCA), a malignancy of the biliary tract, knowledge of these receptors in biliary e

147 increased risk of chromosomal aneuploidy or biliary tract malformation.

148 ns suggest a recent increase in intrahepatic biliary tract malignancies.

149 igher LDLT admission rates were observed for biliary tract morbidity throughout the second posttransp

150 nts had associated extraintestinal location (biliary tract [n = 3] and lung [n = 1]).

151 ciated with acute fulminant hepatic failure, biliary tract necrosis and leaks, or relapsing bacteremi

152 sults suggest that the molecular pathways of biliary tract neoplasms vary by anatomical subsite and h

153 cause of neonatal liver disease, results in biliary tract obstruction and hepatic fibrosis.

154 Nonsurgical biliary drainage in malignant biliary tract obstruction can be performed endoscopicall

155 n might be protective in liver diseases with biliary tract obstruction even without increased HGF pro

156 e enterohepatic bile acid circulation during biliary tract obstruction leads to profound perturbation

157 ase, whilst imaging confirmed the absence of biliary tract obstruction.

158 gen activator (uPA) might be important after biliary tract obstruction.

159 urally and functionally heterogeneous in the biliary tract of normal rats.

160 nic fibroinflammatory syndrome involving the biliary tract, often accompanied by inflammatory bowel d

161 tract injury which required four subsequent biliary tract operations.

162 without symptoms or signs referrable to the biliary tract or pancreatic duct.

163 3 in a ciliary disease affecting the kidney, biliary tract, pancreas, and retina.

164 r the other 9 cancer sites studied (stomach, biliary tract, pancreas, cervix, endometrium, prostate,

165 relationship between vascular growth and the biliary tract, particularly the molecular mechanisms tha

166 enty three Roux-en-Y patients with suspected biliary tract pathology underwent balloon-assisted enter

167 of hemolysis, recurrent viral hepatitis, and biliary tract pathology were identified; 10 other post-o

168 creased TGF-beta signaling in the kidney and biliary tract, respectively.

169 nflammation and fibrosis of the extrahepatic biliary tract, resulting in cirrhosis and end-stage live

170 They occur throughout the biliary tract, share some histologic and clinical featur

171 tients have a high prevalence of symptomatic biliary tract stone disease.

172 The authors report their experience with biliary tract stones in adult and pediatric heart transp

173 nd September 1994 to determine prevalence of biliary tract stones, management strategies used, and ou

174 emains a challenge for even the most skilled biliary tract surgeon.

175 ficant 38% increase in the relative risk for biliary tract surgery (P = 0.05).

176 otal of 147 women (7%) were hospitalized for biliary tract surgery in HERS.

177 gen plus progestin, one additional woman had biliary tract surgery per year.

178 The overall RH for biliary tract surgery was 1.48 (95% CI, 1.12-1.95); for

179 e were associated with an increased risk for biliary tract surgery, whereas statin use was associated

180 ased the rates of venous thromboembolism and biliary tract surgery.

181 rapy in postmenopausal women on the risk for biliary tract surgery.

182 ginally significant increase in the risk for biliary tract surgery.

183 fected with PSC independent of pretransplant biliary tract surgery.

184 Seventy-nine (62%) had undergone previous biliary tract surgery.

185 e infection (SSI) after gastrointestinal and biliary tract surgery.

186 iliary FGF19 has a signaling function in the biliary tract that differs from its established signalin

187 ocarcinoma is an enigmatic malignancy of the biliary tract that has recently been shown to be increas

188 Commonly found in the biliary tract, tuft cells are absent from normal murine

189 cholangiography enables significantly better biliary tract visualization than conventional or excreto

190 Interobserver agreement for overall biliary tract visualization was good for CT, conventiona

191 Ectopic oncogene expression in the biliary tract was accomplished by the Sleeping Beauty tr

192 Like in humans, abnormalities of the biliary tract were an invariant finding.

193 , and morphological studies of the liver and biliary tract were assessed.

194 gression of malignancy is exemplified in the biliary tract where persistent inflammation strongly pre

195 Recent papers on disorders of the liver and biliary tract which clarify their pathogenesis and atten

196 polycystic disease affecting the kidneys and biliary tract with an estimated incidence of 1 in 20,000

197 are susceptible to chronic infections of the biliary tract with Cryptosporidium parvum (CP) that may

198 leading to fibrosis and obliteration of the biliary tract with the development of biliary cirrhosis.