ライフサイエンスコーパス: binding

1 gNP aggregation responses upon biomolecular binding.

2 es for local structure, dynamics, and ligand binding.

3 litic-specific motifs implicated in receptor binding.

4 wins both prevented anemia and reduced HSP90 binding.

5 ugmented steric-interference of actin-myosin binding.

6 ylation status of Lys-72 may affect ERK1 ATP binding.

7 Striatal dopamine transporter binding, VMAT2 binding, (18)F-FDOPA uptake, and serotonin transporter b

8 been well studied for over a decade, the DNA-binding activities and the biological functions of these

9 general principle that confers the substrate binding adaptability and specificity to OGA in O-GlcNAc

10 mechanism by which Cmpd-15 modulates agonist binding affinity and signalling.

11 Of interest, the binding affinity between Sesn2 and AMPK upstream LKB1 is

12 ination of the sedimentation coefficient and binding affinity of proteins in the micromolar range, th

13 ted membrane fusion and had higher levels of binding affinity with NHR peptide N46.

14 ency directly correlated with target tubulin binding affinity, and the reduction in differential func

15 This intricate modulation of binding affinity, and thus cooperativity, renders our ne

16 metal-induced alterations decrease RcnR-DNA binding affinity, leading to rcnAB expression.

17 proportional to FcRn expression and albumin-binding affinity.

18 differences in striatal dopamine transporter binding (all age ranges in caudate and putamen, p<0.0001

19 mphocytes, which exhibit variable E-selectin binding among CD4(+) and CD8(+) T cells but no binding b

20 Importantly, through in vitro binding and activity assays, we showed that CML36 intera

21 identify the residues of WRN involved in the binding and ATPase-driven unwinding of G4 DNA.

22 such as guanosine 5'-3-O-(thio)triphosphate binding and beta-arrestin2 recruitment.

23 emia significantly enhanced [(3) H]ryanodine binding and Ca(2+) /calmodulin-dependent protein kinase

24 poisomerase for mRNAs, and requires both RNA binding and catalytic activity to promote neurodevelopme

25 245) to interrogate their roles in substrate binding and catalytic activity.

26 vealing dynamic structural changes upon iron binding and core formation, as reflected by a quick alte

27 beta-III-spectrin causes high-affinity actin binding and decreased thermal stability in vitro.

28 time the dynamic connection between FTO RNA binding and demethylation activity that influences sever

29 APP, APLP1 exhibits increased trans-cellular binding and elevated cell-surface levels due to reduced

30 o pg/ml range with insignificant nonspecific binding and false signal in undiluted serum.

31 ccessfully eliminated Fc-associated effector binding and functions.

32 oop in a conformation that sterically blocks binding and neutralization by MAb PL-2.

33 h I mutations had only modest effects on GTP binding and on GTPase activity and did not perturb stabi

34 n STN1 engenders a selective defect in POLA2-binding and PP stimulation, indicating that these activi

35 es (MHC I) help protect jawed vertebrates by binding and presenting immunogenic peptides to cytotoxic

36 ause it has a reduced net charge, and in DNA binding and protein-protein interactions because key res

37 losteric regulation of polypeptide substrate binding and release.

38 homodimer (or "lysibody") with high-affinity binding and specificity for carbohydrate determinants on

39 DNA binding and transcription assays demonstrated that the m

40 tiseizure agents but with slower kinetics of binding and unbinding.

41 that these residues participate in substrate binding and/or catalysis.

42 uences to probe contributions to folding and binding, and identified 2,618 high-affinity binders.

43 ns linked to the function of the Erythrocyte binding antigen family and P. falciparum reticulocyte bi

44 Substrate recognition and binding are critical for the regulation of this reaction

45 Changes in Nab3 binding are largely independent of alterations in transc

46 igned to identify ligands that block protein binding are much more challenging to develop; attenuatin

47 10 bp of DNA was sufficient to support PU.1 binding as a monomer, additional flanking bases were req

48 engineered sequence is used to detect ligand binding as blocking events during DNA unzipping, allowin

49 A URAT1 binding assay using radiolabeled verinurad revealed that

50 ted low-affinity binding to LRP6 in in vitro binding assays, and inhibition of LRP6 or critical signa

51 vity relationship (SAR) effort, and specific binding assessment using a LC-MS/MS "cold tracer" method

52 entify active sites, we first predict the CO binding at a large number of sites and select four exhib

53 additional mechanisms that interfere with GR binding at promoters.

54 ions (T124N/T174I) directly weaken inhibitor binding at the dimer interface of the catalytic core dom

55 transfer resistance decreased after antibody binding, because there was an additional amount of prote

56 Characterizing the binding behaviors of RNA-binding proteins (RBPs) is impo

57 degradation, the potentially different ADAR1 binding behaviors related to its editing activity, as we

58 The ligand-binding betaI and alphaI domains of integrin are the bes

59 Specific binding between biomolecules, i.e., molecular recognitio

60 Substrate binding breaks the six-fold symmetry of the complex, all

61 on of Arg60 (R60Q) significantly reduced DNA binding, but retained a preference for the 5caC modified

62 nding among CD4(+) and CD8(+) T cells but no binding by B cells.

63 face form pathways that facilitate glutamate binding by effectively reducing a three-dimensional diff

64 ar affinity and is saturable in vitro Sterol binding by MpPR-1 requires the presence of a flexible lo

65 Lipid binding by purified MpPR-1 occurs with micromolar affini

66 nt in the dissolved form as a result of iron binding by the organic exudate.

67 Fragments of Ef-Tu retain binding capabilities to host proteins.

68 -alphaPEG, hybrid-type cells) to improve the binding capacity and detection limit for free PEG and PE

69 d (palmitic acid analog) to Uox with the HSA binding capacity and retained enzyme activity.

70 B4 (MDR3) is an adenosine triphosphate (ATP)-binding cassette (ABC) transporter expressed at the cana

71 The subfamily C ATP-binding cassette (ABCC) transporters mediate multidrug r

72 a consequence, it is possible that collagen-binding cells may change their phenotypic traits.

73 ific amino acid polymorphisms in the antigen-binding clefts.

74 In all conjugates, the ligand binding competence toward alphaVbeta3 (using both isolat

75 ple, short measurement times and equilibrium binding conditions.

76 Cl(-) anions can be attributed to the higher binding constant of Cl(-) with the anion receptor and th

77 affinity of SA to BSA was demonstrated by a binding constant value (1.09x10(3) at 310 degrees K).

78 d effects of full-length PTEN but a membrane-binding defective mutant of the C2 domain abrogated thes

79 ress high levels of the canonical E-selectin binding determinant sialyl Lewis X (sLe(X)) and display

80 ia type 5 (SCA5) L253P mutation in the actin-binding domain (ABD) of beta-III-spectrin causes high-af

81 pe-specific residues in the PPARdelta ligand-binding domain (LBD).

82 able docking and undocking of its nucleotide-binding domain (NBD) and substrate-binding domain (SBD).

83 ucleotide-binding domain (NBD) and substrate-binding domain (SBD).

84 We apply this method to study the cAMP binding domain A (CBD-A) of Protein kinase A.

85 We show here that while the central DNA binding domain is essential for anchoring at parS, this

86 t has similarities to the cysteine-rich zinc-binding domain of DnaJ chaperones.

87 d knockin mice with a mutation in the TR DNA-binding domain that abrogates binding to DNA and leads t

88 that TRIP8b binds the HCN cyclic nucleotide-binding domain through a 37-residue domain and the HCN C

89 bits the NLS and the neighboring microtubule-binding domain, and RhoA-GTP binding may relieve this in

90 Anillin contains a RhoA-GTP binding domain, which autoinhibits the NLS and the neigh

91 through a small peptide motif within its MT-binding domain.

92 o its coiled-coil 2-leucine zipper ubiquitin binding domain.

93 of an rcSso7d binding module and a cellulose-binding domain.

94 In this study, we demonstrate that binding domains from autolysins and lysins can be fused

95 regulatory functions of RBPs, including the binding effects of the RNA helicase MOV10 on mRNA degrad

96 tes provides a remarkable enhancement of its binding efficiency.

97 the proton is 938 MeV) also revealed a large binding energy of about 130 MeV between the two charm qu

98 g strong photoluminescence and large exciton binding energy.

99 lision point contribute significantly to the binding energy.

100 ALK2 mutants defective in binding FKBP12 increase hepcidin expression in a ligand-

101 at the enzyme represents a new class of zinc-binding flavin-dependent halogenases and provides new in

102 ctivators and blockers with strong, specific binding for engineering and therapeutic applications.

103 trophysiology to measure resting affinities (binding free energies) of these and other agonists in ad

104 e molecular dynamics method to calculate the binding free energy of a series of alpha-ketoamide analo

105 opattern and the maackia amurensis lectin-II binding glycoproteins (MBGs) in 65 children with ASD and

106 hich block substrate access to the substrate-binding groove.

107 her investigation into the basis for ASO-FUS binding illustrated the importance of ASO backbone and h

108 18)F-FDOPA uptake, and serotonin transporter binding in multiple brain regions were compared by ANCOV

109 ast six (35%) alter transcription factor-DNA binding in neuroblastoma cells.

110 found an overall reduction in D1 and 5-HT2A binding in the hippocampus of zQ175 compared with WT ani

111 mGluR5 availability (that is, [(11)C]ABP688 binding) in both MDD and control subjects (average of 14

112 In the absence of vivax invasion assays, binding-inhibitory activity of antibodies has been repor

113 her affinity compared with other Arg-Gly-Asp binding integrins.

114 domain that are located distal to the Galpha-binding interface.

115 nd an X-ray cocrystal structure reveals that binding is biased toward occupation of the adenine subpo

116 While DNA binding is not essential for H3-H4 tetrasome deposition

117 when working with dynamic proteins or weakly binding ligands.

118 including salicylates and diclofenac, as MR1-binding ligands.

119 ntigen family and P. falciparum reticulocyte binding-like families.

120 the beta-sheet structure between the Ca(2+) binding loops particularly at C-domain of CaM, enabling

121 ing microtubule-binding domain, and RhoA-GTP binding may relieve this inhibition during mitosis.

122 ssue) with the agonist whereas 12 had a high binding (mean density, 4,447 +/- 1,128 dpm/mg of tissue)

123 In 13 breast cancers, 8 had a low binding (mean density, 844 +/- 168 dpm/mg of tissue) wit

124 fold of SH3 domains, they display different binding mechanisms and affinities for their interaction

125 re recruited to the cellulosome via a single-binding mode mechanism with an adaptor scaffoldin.

126 ed on the apex of the molecule; the receptor-binding mode might be different from that of retroviruse

127 s, which binds choline in a unique dual-site-binding mode.

128 a fusion construct consisting of an rcSso7d binding module and a cellulose-binding domain.

129 ted in multiple key residues in the receptor-binding motif (RBM) of RBD and demonstrated their strong

130 induces transcriptional coactivator with PDZ-binding motif (TAZ) expression, which is required for os

131 RNA binding motif protein 25 (RBM25) is a putative splicing

132 ontaining aromatic amino acids, the caveolin-binding motif.

133 ) or Arg35 (R35H/L) completely abolished DNA binding, mutation of Arg60 (R60Q) significantly reduced

134 tedly, we identified PABPN1-dependent ALYREF binding near the 3' end of the mRNA.

135 The binding occured within the PLA2R NC3 fragment and was in

136 We investigated whether variation in binding of a transcription factor, the vitamin D recepto

137 We observe that binding of agonists to VFD2 of TAS1R2 leads to major con

138 on of the signal transduction cascade, after binding of all investigated products to the receptor pre

139 novel mechanism of MAPK activation requiring binding of an activator and also shows that MAPKs can be

140 onclude that phosphorylation can disrupt the binding of an E3 ubiquitin ligase to an E2-conjugating e

141 (ii) Selective and cooperative binding of both an acetato ligand and an amino ligand to

142 species in human-inflamed tissue and allows binding of complement factor 1q (C1q) and activation of

143 in its coiled-coil 1 domain that impedes the binding of linear (M1-linked) di-ubiquitin to its coiled

144 e adhesins has been shown to be required for binding of multiple glycan receptors.

145 jacent residues that facilitate the specific binding of proteins and modulate the global folding and

146 Binding of STIM1 to SUR1 was enhanced by poly-lysine.

147 Thus, we demonstrated peptide-dependent binding of the activating NK cell receptor KIR2DS1, prov

148 Glycan array analysis confirmed selective binding of the CRD to glycans that contain Manalpha1-2Ma

149 Competitive binding of the human monoclonal antibody (mAb) LE2E9 rev

150 riments revealed that Spt6 could compete for binding of the PRC2 methyltransferase Ezh2 to Suz12 and

151 noprecipitation experiments reveal increased binding of the repressor factor RE1-silencing transcript

152 al at the RI, hypoxia-induced expression and binding of the splicing factor SRSF3, and increased bind

153 of the splicing factor SRSF3, and increased binding of total and phospho-Ser2 RNA polymerase II spec

154 ise that stereospecific but necessarily weak binding of tropomyosin to F-actin is required for effect

155 's disease susceptibility protein nucleotide-binding oligomerization domain-containing 2 (NOD2); howe

156 y, as well as the antagonizing effect of RBP binding on miRNA repression.

157 Fe-2S] clusters in Miner2, with each cluster binding one nitric oxide.

158 aging effect was observed in [(18) F]Nifene binding over 5 decades.

159 The endothelial binding parameter Kb varied highly across brain regions.

160 ntly identified as an aberrant extracellular binding partner of mutant GlyRS.

161 ent induced c-FOS to replace JDP2 as a c-JUN binding partner, forming transcriptionally active AP-1.

162 ing cell nuclear antigen (PCNA) as a nIGF-1R-binding partner.

163 olved information and probe multiple protein binding partners simultaneously, using small amounts of

164 nitial prediction of human leukocyte antigen-binding peptides by in silico algorithms, but the predic

165 (18)F-florbetapir (Amyvid) is an amyloid-binding PET ligand with a half-life suitable for clinica

166 and a subset of nucleotides in the cobalamin-binding pocket.

167 Large-scale RNA-binding pockets on protein surfaces are grouped by measu

168 s revealed that these ligands adopt distinct binding poses and engage different subsets of residues,

169 methods to estimate the distribution volume, binding potential (BPND), and SUVR.

170 mpetition between actin and microtubules for binding profilin.

171 ional complexation with human growth hormone binding protein (hGHBp) to the different NOTA-modified s

172 by its naturally occurring antagonist, IL-18 Binding Protein (IL-18BP).

173 er-inducing interferon-beta (TRIF) and Z-DNA-binding protein 1 (ZBP1)/DNA-dependent activator of IFN-

174 tion initiation factor 4E, phosphorylated 4E-binding protein 1, and p-S6 ribosomal protein.

175 encoding the transcription factor Methyl CpG Binding Protein 2 (MECP2).

176 hermore, we report that calcium and integrin-binding protein 2 binds to the components of the hair ce

177 2 coactivated the transcription factors GATA-binding protein 4 (GATA-4) and hypoxia-inducible factor

178 3p must be in complex with the small calcium-binding protein Cdc31p to be active.

179 ring a cavity resembling that of the choline-binding protein ChoX, as revealed by crystal and density

180 nitrite reductase gene (aniA), the factor H-binding protein gene (fHbp), and the capsule biosyntheti

181 ce, we show that TERF1 evolved as a telomere-binding protein in the common stem lineage of marsupial

182 The Human antigen R protein (HuR) is an RNA-binding protein that recognizes U/AU-rich elements in di

183 tubule network and the microtubule minus end-binding protein, Patronin.

184 ncy, B cell-specific CD79a-Cre x XBP1 (X-box binding protein-1) floxed mice (XBP1-conditional knockou

185 us sequence in the sterol regulatory-element binding protein-1c (Srebp-1c) promoter.

186 ing the need to separate the marker from its binding protein.

187 ), a negative regulator of the Ras small GTP-binding protein.

188 ssess significant chemical diversity; glycan binding proteins (GBPs) recognize specific glycans to tr

189 Characterizing the binding behaviors of RNA-binding proteins (RBPs) is important for understanding t

190 ble the rapid characterization of engineered binding proteins and interaction networks.

191 PNA-binding proteins may also participate in the patterning

192 RNA-binding proteins of the Musashi (Msi) have been implicat

193 This removal is controlled in part by RNA-binding proteins that regulate alternative splicing deci

194 ple neurite-targeted non-coding RNAs and RNA-binding proteins with potential regulatory roles.

195 ) is a member of the fragile X family of RNA-binding proteins, which includes FMRP and FXR2P.

196 eukaryotes, and recognized as small-molecule binding proteins.

197 with standardized parameters before striatal binding ratios were quantified against a normal database

198 um sensing via antagonism of the autoinducer-binding receptors, LasR and RhlR.

199 Binding refers to the operation that groups different fe

200 By comparison, the zinc binding region of the P73G mutant, even under native con

201 display near-identical extracellular ligand-binding regions but have intracellular sequences with op

202 ected by sequence mismatches in primer/probe binding regions, RT-dPCR may be the optimal molecular me

203 Collectively, our results indicate that ZO-1 binding regulates channel accrual, while disengagement f

204 Stoichiometric binding requires conditions consistent with coalescence

205 Ca(2+) binding rigidifies elements along this pathway, thereby

206 ged in a radial manner from a minimal flavin-binding scaffold.

207 e, it has proven challenging to achieve high binding selectivity for different isoforms of this prote

208 e and were correlated with the thermodynamic binding selectivity.

209 ormation capable of transmitting the hormone binding signal to the catalytic domain.

210 alytic glutamate of the canonical nucleotide binding site 2 was mutated to glutamine.

211 Transcriptome and genome-wide GABP-binding site analyses identify GABP direct targets encod

212 eceptor (NMDAR) is controlled by a glutamate-binding site and a distinct, independently regulated, co

213 est the potential existence of an additional binding site and provide new insights into GB1:IgG compl

214 Nonetheless, the evolution of the receptor-binding site and the stem region on HA is severely const

215 pation of the adenine subpocket of the AcCoA binding site by an aromatic ring.

216 otein by a conserved kinase, DDK, provides a binding site for the Scc2/4 cohesin loading complex, the

217 2149092 was predicted to disrupt a consensus binding site for the transcription factor ETS within an

218 Furthermore, Nedd8 binding site in Smurf is shown to be necessary for its u

219 Results demonstrated that a miR398 binding site is eliminated in AhCSD1-2.2 as a consequenc

220 sPs and PtdInsPs interact with the polyanion-binding site located on an inner chamber wall of the enz

221 Finally, analysis of transcription factor-binding site motifs of differentially dysregulated genes

222 he N terminus of chECL1, suggesting that the binding site of ALV-J gp85 on chNHE1 is probably located

223 facilitate the formation of an intracellular binding site that enhances G-protein coupling.

224 Although it shares a binding site with other imide-based natural product tran

225 We identified a centrin-binding site within H. sapiens Prp40 homolog A (HsPrp40A

226 und -260 and -230 mV, respectively, in the Q-binding site, respectively, suggesting that release of t

227 istinct, independently regulated, co-agonist-binding site.

228 fficiently to beta-tubulin at the colchicine binding site.

229 which was reduced after mutation of the Sp-1-binding site.

230 Published data reveal TET2 binding sites and hydroxymethylcytosine proximal to KLK3

231 9-amino-acid protein containing five F-actin-binding sites and two G-actin-binding sites, and interac

232 expression, we identified transcriptome-wide binding sites for RNA polymerase II and the exosome cofa

233 ence motifs enriched in the PREs are cognate binding sites for the identified transcription factors a

234 o identify canonical and non-canonical miRNA-binding sites from peaks identified by Ago2 Cross-Linked

235 al, 7622 proteins from the scPDB database of binding sites have been evaluated using both a distance

236 nalysis of enriched transcription factor DNA-binding sites in the promoters of differentially express

237 nteraction energy is weak because only a few binding sites near the collision point contribute signif

238 cers active in liver cells, enriched for the binding sites of the FOXA1, FOXA2 and HNF4A transcriptio

239 Deletion of YY1 binding sites or depletion of YY1 protein disrupts enhan

240 tation-sequencing analysis reveals that LSD1 binding sites overlap significantly with those bound by

241 ethod for prediction of transcription factor binding sites using an integrative energy function that

242 g five F-actin-binding sites and two G-actin-binding sites, and interacts with wheat (Triticum aestiv

243 by ClusterTAD have a high enrichment of CTCF binding sites, promoter-related marks, and enhancer-rela

244 (1)h interactions and identify their genomic binding sites.

245 ChRs having a mutation(s) at the transmitter-binding sites.

246 ner, preventing its recruitment to canonical binding-sites in the promoters of Nanog, Oct4 and Sox2.

247 ilization requires the conserved microtubule-binding Ska complex, which enriches at attachment sites

248 Characterizing the binding specificities of transcription factors (TFs) is

249 de constitutes a key signature providing PIC binding specificity in the human genome.

250 , particularly of sites corresponding to the binding specificity of the overexpressed SH2 domain.

251 We report here a few Zika NS1-binding ssDNA aptamers selected using the conventional S

252 d scaffold, in both noncovalent and covalent binding states, and for two highly homologous proteases,

253 the absence of PPi, suggests that nucleotide binding stimulates PPi dissociation and occurs before po

254 Most striking was the decline of binding stoichiometry with linking number.

255 umber of sites and select four exhibiting CO binding stronger than the (211) step surface.

256 Radioligand binding studies and functional assays that use human rec

257 Binding studies disclosed that the competitive inhibitor

258 In vitro binding studies showed that whereas mutation of Arg36 (R

259 enting a simple constraint of continuity for binding successive sounds in a probabilistic manner.

260 rsatile scaffolds to display diverse antigen-binding surfaces.

261 result of significant levels of cooperative binding that is present in both solvents.

262 s found to affect synaptic integrity through binding the ColQ tail of acetylcholine esterase.

263 mechanisms and calculate the free energy of binding the hypothesized ligands to YKL-40, addressing t

264 romoting serum response transcription factor binding to a cryptic cis-element.

265 BPA exposure increased Nrf2 binding to a putative antioxidant response element conse

266 ls of the interplay between membrane and CaM binding to Akt may help in the development of potential

267 Id1 suppresses BAT thermogenesis by binding to and suppressing PGC1alpha transcriptional act

268 elements of the pathways that couple agonist binding to channel gating.

269 in the TR DNA-binding domain that abrogates binding to DNA and leads to complete loss of canonical T

270 Thus, binding to GAGs in the extracellular matrix can direct a

271 ering of a protein scaffold for preferential binding to K-Ras G12D.

272 usly used to investigate peptide and protein binding to lipid membranes, as it allows for very low am

273 Domain 4 exhibited low-affinity binding to LRP6 in in vitro binding assays, and inhibiti

274 Its binding to mRNA is regulated by tyrosine 396 phosphoryla

275 el method of kinetic analysis of radioligand binding to neuroreceptors in brain in vivo, here applied

276 uenza induced cytotoxicity and showed strong binding to platelets within thrombi in infected mouse lu

277 , or polymersomes, which are internalized by binding to scavenger receptors and subsequently escape t

278 ese structures illustrate the flexibility of binding to sequences outside of-but adjacent to-the kina

279 Specific ion binding to such sites induces changes in the filter conf

280 and TH receptors (TRs) alpha and beta act by binding to TH response elements (TREs) in regulatory reg

281 w that blocking of the Galpha13 protein from binding to the cytoplasmic domain of the beta3 integrin

282 A1 by >/=75% and were evaluated for in vitro binding to the enzyme active site and for inhibition con

283 sembly process shepherds primary transposase binding to the inner 12DRs (where cleavage does not occu

284 eration of angiotensin-(1-7) by ACE2 and its binding to the Mas receptor (MasR) improves glucose home

285 reatment of breast cancer cells increased ER binding to the NEMO promoter, thereby increasing NEMO ex

286 Moreover, poly(ADP-ribose) binding to the Parp9 macrodomains increases E3 activity.

287 onserved amino acids that couple RNA and ATP binding to the protein (Motif III).

288 Finally, DDX21 was shown to enhance Rev binding to the RRE in a manner similar to that previousl

289 rted similar effects as progesterone, likely binding to the same site.

290 as the bridging molecule that mediates IL-37 binding to the TGF-beta receptor complex.

291 d conditions, is inhibited by intramolecular binding to the Vms1 leucine-rich sequence (LRS).

292 ues in the C-terminal helix to engage in DNA binding, triggering a major reprogramming of gene expres

293 yrazone and probenecid all inhibit verinurad binding via a competitive mechanism.

294 Striatal dopamine transporter binding, VMAT2 binding, (18)F-FDOPA uptake, and serotoni

295 to investigate general properties of ligand binding: we observe both a direct stabilizing effect of

296 a 50% reduction of (11)C-BMT-136088 specific binding were 73 +/- 30 nmol/kg and 28 +/- 12 nM, respect

297 rocedure to purify trypsin based on affinity binding with ferromagnetic particles of azocasein compos

298 ino acid substitutions abolished alphaLbeta2 binding with soluble and immobilized intercellular cell

299 In 14 prostate cancers, none had sst2 binding with the agonist and only 4 had a weak binding w

300 nding with the agonist and only 4 had a weak binding with the antagonist.