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1 factor) and perhaps MBF (MluI cell cycle box binding factor).
2  nuclear factor 1, Forkhead box A1 and CCCTC-binding factor.
3  targets of E2F corresponds to Adenovirus E2 Binding Factor.
4 actor related to FUS3, and LEC1, a CCAAT box-binding factor.
5 A BINDING WITH ONE FINGER3, and PROLAMIN BOX-BINDING FACTOR.
6 ions identifies hydrophobic effect as a main binding factor.
7  that has high-affinity binding to the CCCTC-binding factor.
8 i allele extensively binds the protein CCCTC-binding factor.
9 king site for TFIID, the major core promoter-binding factor.
10 rred in the putative A protein, an important binding factor.
11 is a gamma-tubulin small complex (gammaTuSC) binding factor.
12  by moderating the activities of E2 promoter-binding factors.
13 interaction with regulatory elements and DNA-binding factors.
14 nucleosome-induced cooperativity between DNA-binding factors.
15 ell-type-specific regulation directed by DNA binding factors.
16 ough interactions with sequence-specific DNA-binding factors.
17 d their interplay with cooperative chromatin binding factors.
18 ing sites where nucleosomes compete with DNA-binding factors.
19 lastic moduli of hydrogels, with and without binding factors.
20                                       RBP AU-binding factor 1 (AUF1) has four isoforms resulting from
21                              The RBP AU-rich-binding factor 1 (AUF1) isoform p37 was found to have hi
22 rget of viral proteinase cleavage is AU-rich binding factor 1 (AUF1), a cellular protein that binds t
23 onal coactivation complex through C promoter-binding factor 1 (CBF1; RBPJ) and strengthens the associ
24 gamma (PPARgamma), leptin, osteonectin, core binding factor 1 (CBFA1), and FBJ murine osteosarcoma vi
25 ctroscopy to study the human telomere repeat binding factor 1 (hTRF1) in complex with Escherichia col
26 n (Pg) in conjunction with lymphoid enhancer-binding factor 1 (Lef-1) differentially regulates the pr
27   The transcription factor lymphoid enhancer-binding factor 1 (LEF-1), which plays a definitive role
28 n via the nuclear protein, lymphoid enhancer-binding factor 1 (LEF-1).
29 odermin (EOMES; mesoderm), lymphoid enhancer-binding factor 1 (LEF1) and mesoderm posterior BHLH tran
30  contain normal amounts of lymphoid enhancer-binding factor 1 (Lef1) and transcription factor 1 (Tcf1
31 ar definition of BL with lymphocyte enhancer-binding factor 1 (LEF1) as a signature gene.
32 ers a binding site for the lymphoid enhancer-binding factor 1 (LEF1) transcription factor, reducing L
33 nuclear proteins, i.e. the lymphoid enhancer-binding factor 1 (Lef1), histone H3, and Brahma-related
34                             Telomeric repeat binding factor 1 (TRF1) is essential to the maintenance
35      Here we establish that telomeric repeat-binding factor 1 (TRF1), a core component of the telomer
36 and the shelterin component telomeric repeat binding factor 1 (TRF1)-interacting nuclear factor 2 (TI
37  the nucleolar transcription factor upstream binding factor 1 (UBF1) on rDNA promoters and recruiting
38      CSNK2A2 phosphorylates telomeric repeat binding factor 1 and plays an important role for regulat
39 the collection of RNAs regulated by AUF1 (AU-binding factor 1), an RBP linked to cancer, inflammation
40 an antigen R) and GRSF1 (G-rich RNA sequence-binding factor 1), that associated with the nuclear DNA-
41   The mammalian RNA-binding protein AUF1 (AU-binding factor 1, also known as heterogeneous nuclear ri
42 roles of RBPs HuR, CUG-binding protein 1, AU-binding factor 1, and several GI epithelial-specific miR
43 llerRed chromophore with the telomere repeat binding factor 1, TRF1, we developed a novel approach to
44 old-regulated transcription factors C-REPEAT BINDING FACTORs 1-3 (CBF1, CBF2, CBF3).
45 f the transcription factor lymphoid enhancer-binding factor-1 (LEF-1).
46 activation of beta-catenin/lymphoid enhancer-binding factor-1 (LEF-1).
47                            Lymphoid enhancer-binding factor-1 (LEF1) is a key transcription factor of
48 ing putative T-cell factor/lymphoid enhancer binding factor-1 (TCF/LEF) motifs, consistent with canon
49 s with Epstein-Barr virus latency C-promoter binding factor-1/suppressor of hairless/lag-1 (CSL) and
50               We identified nuclear receptor binding factor 2 (Nrbf2) as a Beclin 1-interacting prote
51 t a putative fifth subunit, nuclear receptor binding factor 2 (NRBF2), is a tightly bound component o
52 nthesis, including sterol regulatory element binding factor 2 (Srebf2) and 3-hydroxy-3-methylglutaryl
53 ivator protein 1 (RAP1) and telomeric repeat-binding factor 2 (TRF2) are two subunits in shelterin th
54      To examine the role of telomeric repeat-binding factor 2 (TRF2) in epithelial tumorigenesis, we
55 e 3' overhang facilitated by telomere repeat-binding factor 2 (TRF2) into telomeric DNA, forming t-lo
56 t a non-telomeric isoform of telomere repeat-binding factor 2 (TRF2-S) is a novel RBP that regulates
57  functionally interacts with telomere repeat-binding factor 2 that in turn regulates its helicase act
58  of the shelterin component telomeric repeat-binding factor 2, cells survived but remained dormant an
59 nother shelterin component, telomeric repeat binding factor 2, interacting protein (p.Ala104Pro and p
60 ed the impact of a sterol regulatory element-binding factor-2 (SREBF-2) polymorphism on the risk of d
61 ion and telomere binding of telomeric repeat-binding factor-2 (TRF2), associated with increased DNA d
62 (Zea mays), several genes, including a G-BOX BINDING FACTOR 3 (GBF3) were identified as candidate dro
63 yeloma tumor protein 2, interleukin enhancer binding factor 3 (IEBP3), guanine nucleotide binding pro
64 NAs, we discovered that interleukin enhancer-binding factor 3 (ILF3) is required for sustained uPA ex
65                                         GATA-binding factor 6 (GATA6) is a zinc-finger transcription
66  homolog of the bacterial RbfA (for ribosome-binding factor A) protein that was identified as a cold-
67    Cocaine exposure reduces binding of CCCTC-binding factor, a chromosomal scaffolding protein, and i
68                                      The ABA Binding Factor/ABA-Responsive Element Binding Proteins (
69 se 1 via mTOR (P < 0.02), and total upstream binding factor abundance were significantly greater in E
70 function is highly reliant on continued core binding factor activity.
71                   Not all patients with core binding factor acute myeloid leukemia (CBF-AML) display
72 d-Sequential Induction in Patients With Core Binding Factor Acute Myeloid Leukemia and Treating Patie
73      To identify cooperating lesions in core-binding factor acute myeloid leukemia, we performed sing
74  should provide important insights into core-binding factor acute myeloid leukemia.
75                                Although core-binding factor-acute myeloid leukemia (CBF-AML) (t[8;21]
76 ML and are usually reported together as core binding factor AML (CBF-AML).
77 o 60 years of age, excluding those with core binding factor AML and acute promyelocytic leukemia, wer
78                                  In non-core binding factor AML, subclone formation was associated wi
79 ontain an insulator element that binds CCCTC-binding factor and reduces its insertional genotoxicity.
80 appa gene V-J intervening region binds CCCTC-binding factor and specifies locus contraction and long-
81 rs on stimulation; it is stabilized by CCCTC-binding factor and TFIIB and poises the gene for a promp
82 n DNA sequence (cis-elements) as well as DNA binding factors and ATP-dependent chromatin modifiers (t
83                    Here, we identify two OEE-binding factors and determine their contribution to GC i
84 ions of CDKs and cyclins, and of E2 promoter-binding factors and dimerization partners confirmed inte
85 tation experiments for tens of different DNA-binding factors and from ribosome profiling experiments
86 e coordinated integration and release of ES6 binding factors and likely plays a pivotal role in remod
87 probably enhances Ana2 avidity for centriole-binding factors and may bridge multiple factors as requi
88 tors such as corepressors, coactivators, DNA-binding factors and PTM modifying enzymes.
89 E2 Y102F protein interacted with cellular E2-binding factors and the viral helicase E1; however, in c
90 o chromatin structural proteins, Ctcf (CCCTC-binding factor) and Hmgb2 (high mobility group protein B
91 S-phase activators, SBF (Swi4 cell cycle box binding factor) and perhaps MBF (MluI cell cycle box bin
92  the transcriptional insulator protein CCCTC-binding factor, and interacted with each other, creating
93 cluding histone modifications and associated binding factors, and their functional contribution to tr
94                         Other identified HVD-binding factors are also involved in EEEV replication, b
95                                          DNA binding factors are essential for regulating gene expres
96 nding protein homolog-1) and FBF (fem-3 mRNA binding factor) are evolutionary conserved regulators of
97 ybrid assays with the ABA responsive element binding factor AREB2/ABF4, which binds to a G-box absolu
98 members, and the ethylene-responsive element binding factor-associated amphiphilic repression (EAR-li
99  presence of the ethylene-responsive element-binding factor-associated amphiphilic repression motif.
100          PF74's potency depended on these CA-binding factors at low doses.
101 ine finger-like motif in the ParG centromere-binding factor augments ParF ATPase activity but is inef
102 plex is composed of the unique cofactor core binding factor beta (CBF-beta) and canonical ligase comp
103  Vif critically depends on the cellular core binding factor beta (CBFbeta).
104 nscription factor fusion CBFbeta-SMMHC (core binding factor beta and the smooth-muscle myosin heavy c
105 on factor 1 (RUNX1) and its associating core binding factor-beta (CBFB) play pivotal roles in leukemo
106   Here, we show that deletion of CTCF (CCCTC-binding factor)-binding sites at TAD and sub-TAD topolog
107 ding of HvGAMYB and the Barley Prolamine-Box binding Factor (BPBF) to their target sequences.
108 se, and TAF12, an RNA polymerase II TATA-box binding factor, cause CIN when overexpressed in human ce
109                                However, core-binding factor (CBF) AML treated with FLAG/DNX resulted
110 rd genetic screen for regulators of C-REPEAT BINDING FACTOR (CBF) gene expression (RCFs), we identifi
111 ns of gene expression in the poplar C-repeat binding factor (CBF) gene family.
112 MTA3) in the rapid cold induction of CRT/DRE BINDING FACTOR (CBF) genes CBF1 and CBF2, and the repres
113 h leukemogenic fusion proteins, such as core-binding factor (CBF) leukemia and MLL fusion leukemia, r
114 ort of recurring FLT3 N676 mutations in core-binding factor (CBF) leukemias and suggests a defined su
115                                         Core binding factor (CBF) leukemias, those with translocation
116 e map to a region that includes the C-repeat binding factor (CBF) locus.
117                                 The C-REPEAT-BINDING FACTOR (CBF) pathway has important roles in plan
118 lerance that involves action of the C-repeat binding factor (CBF) regulatory pathway.
119                   Expression of the C-repeat-binding factor (CBF) transcription factors is induced by
120  thaliana, acting downstream of the C-repeat binding factor (CBF) transcription factors to recruit th
121  including rearrangements involving the core-binding factor (CBF) transcriptional complex.
122  the C-REPEAT/DEHYDRATION-RESPONSIVE ELEMENT BINDING FACTOR (CBF) transcriptional regulators that con
123 ene expression under cold stress by C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathw
124 ble in 165/1026 (16.1%) of aberrant non-core-binding-factor (CBF) karyotype patients.
125                          RUNX1/CBFbeta (core binding factor [CBF]) is a heterodimeric transcription f
126 rowth defect in cells lacking the centromere binding factor Cbf1 and synthetic lethality when combine
127 epeat (CRT)/drought-responsive element (DRE) binding factor CBF1/DREB1b is a transcriptional regulato
128                     The beta-subunit of core binding factor (CBFbeta), that heterodimerizes with RUNX
129                                     C-Repeat Binding Factors (CBFs) are DNA-binding transcriptional a
130 cleus-encoded transcription factors C-REPEAT BINDING FACTORS (CBFs).
131 he viral envelope contains the mammalian LPS-binding factors CD14, TLR4, and MD-2, which, in conjunct
132 pproach uncovered the chromatin helicase DNA-binding factor CHD1 as a putative synthetic-essential ge
133 cycle box-binding factor/MluI cell-cycle box binding factor complexes.
134  activator Sub1, recently identified as a G4-binding factor, contributes to genome maintenance at G4-
135 in an inter-chromosomal Sox-2 enhancer/CCCTC-binding factor CTCF/cohesin loop) to an active (within a
136 ropic "tension globule." In the other, CCCTC-binding factor (CTCF) and cohesin act together to extrud
137 erved chromosome loop anchors bound by CCCTC-binding factor (CTCF) and cohesin are vulnerable to DNA
138 ence specificity in vitro and in vivo, CCCTC-binding factor (CTCF) and its paralog brother of the reg
139 set of DNA-binding proteins, including CCCTC-binding factor (CTCF) and pluripotency factors.
140 germline transcripts, and proximity to CCCTC-binding factor (CTCF) and Rad21 sites were compared with
141 fic chromatin interactions mediated by CCCTC-binding factor (CTCF) and RNA polymerase II (RNAPII) wit
142 mains are enriched for binding sites of CTCC-binding factor (CTCF) and the cohesin complex, implicati
143  in part by the architectural proteins CCCTC binding factor (CTCF) and the cohesin complex, which co-
144              Two of these targets, the CCCTC-binding factor (CTCF) and the epidermal growth factor re
145                   Sequence variants in CCCTC-binding factor (CTCF) and transcription factor binding s
146 inding sites for the insulator protein CCCTC-binding factor (CTCF) are constitutively occupied, a sub
147 ed the DNA-binding zinc-finger protein CCCTC-binding factor (CTCF) as a crucial regulator of long-ran
148 tin fiber and the proteins cohesin and CCCTC-binding factor (CTCF) as key players anchoring such chro
149  elements, many of which coincide with CCCTC-binding factor (Ctcf) binding sites, for example, the cl
150 methylated regions to be enriched with CCCTC-binding factor (CTCF) binding sites.
151                          The host cell CCCTC-binding factor (CTCF) functions in genome-wide chromatin
152           Here we demonstrate that the CCCTC-binding factor (CTCF) functions to insulate these adjace
153 th the architectural/insulator protein CCCTC-binding factor (CTCF) genome-wide.
154            MIR insulators appear to be CCCTC-binding factor (CTCF) independent and show a distinct lo
155 ial dysfunction.SIGNIFICANCE STATEMENT CCCTC-binding factor (CTCF) is a DNA-binding protein that orga
156                                        CCCTC-binding factor (CTCF) is a key regulator of nuclear chro
157                                        CCCTC-binding factor (CTCF) is a multi-functional chromatin re
158                                    The CCCTC-binding factor (CTCF) is a zinc finger DNA binding prote
159                                        CCCTC-binding factor (CTCF) is an 11 zinc finger DNA-binding d
160 er determined that chromatin insulator CCCTC-binding factor (CTCF) is TGF-beta inducible and facilita
161 ss of architectural proteins, of which CCCTC-binding factor (CTCF) is the best characterized.
162 romatin bound by the insulator protein CCCTC-binding factor (CTCF) on the inactive X chromosome (Xi).
163                                        CCCTC-binding factor (CTCF) plays an essential role in regulat
164 F-induced activation of the epigenetic CCCTC binding factor (CTCF) plays an important role in corneal
165                      Epigenetic factor CCCTC binding factor (CTCF) plays important roles in the genet
166      Chromatin looping mediated by the CCCTC binding factor (CTCF) regulates V(D)J recombination at A
167 s of which are enriched in cohesin and CCCTC-binding factor (CTCF) required for long-range interactio
168 f2 ICR [Delta2,3] removing two of four CCCTC-binding factor (CTCF) sites and the intervening sequence
169                                        CCCTC-binding factor (CTCF) sites were predicted in silico and
170 ed TAD borders reveals the presence of CCCTC-binding factor (CTCF) sites with diverging orientations
171  polymerase III (Pol III) elements and CCCTC binding factor (CTCF) sites.
172  mediated by the architectural protein CCCTC-binding factor (CTCF) that binds to the boundaries of to
173                             Binding of CCCTC-binding factor (CTCF) to the first exon of the hTERT gen
174 tment of the transcriptional repressor CCCTC-binding factor (CTCF) to the MSMP enhancer region was de
175          The transcriptional insulator CCCTC binding factor (CTCF) was shown previously to be critica
176 nt) database identified association of CCCTC-binding factor (CTCF) with Ahr promoter in mouse livers.
177 c changes regulated the association of CCCTC-binding factor (CTCF) with select genomic sites.
178                                        CCCTC-binding factor (CTCF), a sequence-specific transcription
179 data of H3K4 di-methylation (H3K4me2), CCCTC binding factor (CTCF), AR and FoxA1 cistrome data as wel
180                        The multidomain CCCTC-binding factor (CTCF), containing a tandem array of 11 z
181               Distinct combinations of CCCTC-binding factor (CTCF), Mediator, and cohesin show widesp
182 DNF chromatin by DNA insulator protein CCCTC-binding factor (CTCF), which is associated with suppress
183 alized by simultaneous mutation of two CCCTC-binding factor (CTCF)-binding elements (CBE1 and CBE2) w
184                          We found weak CCCTC-binding factor (CTCF)-binding sites and reduced CTCF bin
185  gene desert form a 1 Mbp loop between CCCTC-binding factor (CTCF)-binding sites that is separated fr
186 xhibit hypermethylation at cohesin and CCCTC-binding factor (CTCF)-binding sites, compromising bindin
187 cterized by clusters of hypomethylated CCCTC-binding factor (CTCF)-bound elements, which were predomi
188 n start sites (TSS), and the number of CCCTC-binding factor (CTCF)-cohesin complexes between the inte
189 activates HPIP gene transcription in a CCCTC-binding factor (CTCF)-dependent manner.
190 ated by long noncoding RNAs (lncRNAs), CCCTC binding factor (CTCF)-mediated boundaries, and DNA methy
191  domain (TAD) boundaries, enriched for CCCTC-binding factor (CTCF).
192 tifs for the enhancer blocking protein CCCTC-binding factor (CTCF).
193 ovement by transcription and DNA-bound CCCTC-binding factor (CTCF).
194                                        CCCTC-binding factor, CTCF, has received much attention in thi
195  by the binding of hundreds of different DNA binding factors (DBFs) to the genome.
196 eavage domain with a series of Pumilio/fem-3-binding factor domains that specifically recognize diffe
197 sis that apical HS in the airway serves as a binding factor during infection, and HS modulating compo
198 rotein that functions dually as a centromere binding factor during segrosome assembly and as a transc
199 ion by competing with eIF5 for the Met-tRNAi-binding factor eIF2.
200 itiate translation in the absence of the cap-binding factor eIF4E.
201  recruited to the miR-31 promoter by the DNA binding factor ETS-1, and it represses miR-31 transcript
202  gene pABRE:NCED enhanced NCED and ABF (ABRE-binding factor) expression in Arabidopsis Columbia-0 see
203 r of C-repeat/dehydration responsive element-binding factor expression1/scream2 that are required for
204  and ABA-INSENSITIVE5/ABA-responsive element binding factor family identified specific members that c
205  interaction between PUF family member fem-3 binding factor (FBF) and germline development defective-
206                                       Adding binding factors from collagen and RGD peptides increases
207  Zic family of proteins as enhancer-specific binding factors functioning in development.
208 dies on a beta-globin enhancer- and promoter-binding factor, GATA-1, the founding member of the GATA
209 ii evades complement-mediated destruction by binding factor H (FH), a host-derived negative regulator
210 hbB protein contributes to immune evasion by binding factor H (FH).
211 t the identification of the minor groove DNA-binding factor high mobility group AT-hook 2 (HMGA2) as
212 quences in a synergistic manner with the RNA-binding factor hnRNP A1.
213 g competent substrate, human telomere repeat binding factor (hTRF1), which is bound to DnaK in a glob
214  differential expression of inhibitor of DNA binding factors Id1 and Id3, as well as Notch-regulated
215                     The interleukin enhancer binding factors ILF2 (NF45) and ILF3 (NF90/NF110) have b
216               Besides other mechanisms, GAGA-binding factors in animals can guide PRC members in a se
217 g similarities between animal and plant GAGA-binding factors in the recruitment of PRC1 and PRC2 comp
218 ETX) is one of the best characterised R-loop-binding factors in vivo.
219 ctions of RIOK3 with actin and several actin-binding factors including tropomyosins (TPM3 and TPM4) a
220 Ethylene Response Factor 1 (ERF1), a GCC-box binding factor is in synchrony with expression of AtNudt
221                                  CTCF (CCCTC-binding factor) is a highly conserved multifunctional DN
222                           The host chromatin-binding factor LEDGF/p75 interacts with HIV-1 integrase
223  T cell factor (TCF)-1 and lymphoid enhancer-binding factor (LEF)-1 transcription factors have redund
224 report on the tethering of MLL1 to chromatin binding factor lens epithelium-derived growth factor (LE
225                           Patients with core binding factor leukemia achieving complete remission (CR
226 cally interacts with the MluI cell cycle box-binding factor (MBF) transcription complex that regulate
227 ted by Cds1, resulting in activation of MluI-binding factor (MBF)-dependent transcription.
228                Loss of STAG2 inhibited CCCTC-binding-factor-mediated expression of dual specificity p
229 n clinical specimens showed that the heparin-binding factor midkine is a systemic inducer of neo-lymp
230  not recruit Swi4/6-dependent cell-cycle box-binding factor/MluI cell-cycle box binding factor comple
231   In this study, we investigate the distinct binding factors needed on apposed membranes for Rab effe
232 hich hair cells produce an otolith precursor-binding factor, normally localised to tether cell kinoci
233 ions for the transcriptional regulator CCCTC-binding factor of ESR1 and ESR2, consistent with sustain
234 on with pituitary tumour-transforming 1, the binding factor of PTTG1lP, and with vimentin and E-cadhe
235  and Ska complexes are the major microtubule-binding factors of the kinetochore responsible for maint
236 ot affect hydrophobic interactions as a main binding factor or the location of binding sites in Abeta
237 s VAL1/2 possibly in combination with GT-box binding factors, other AtBMI1 regulatory networks requir
238 KT) and phosphorylated cAMP response element binding-factor (p-CREB) than similar cancers that lacked
239                            The CBF (C-repeat binding factor) pathway has a major role in plant cold a
240 tion of abscisic acid-dependent and C-REPEAT-BINDING FACTOR pathways.
241  the vertebrate genome organizer CTCF (CCCTC-binding factor) played an important role for the evoluti
242 lastoma tumor suppressor protein/E2 promoter binding factor (pRb/E2F1) pathway, which we have previou
243 rabidopsis (Arabidopsis thaliana) GAGA-motif binding factor protein basic pentacysteine6 (BPC6) inter
244                           Pumilio/fem-3 mRNA binding factor (PUF) proteins bind RNA with sequence spe
245 minization of XX and XO animals (fem)-3 mRNA-binding factor (PUF) proteins bind sequence specifically
246                           Pumilio-fem-3 mRNA binding factor (PUF) RNA-binding proteins achieve that s
247 i of insect and vertebrate Pumilio and Fem-3 binding factors (PUFs) are related, and we show that cor
248  a context-dependent manner, and cis-element-binding factors recruit chromatin regulators that mediat
249        Tau downregulation decreases upstream binding factor recruitment, ribosomal RNA synthesis, rib
250 ments, but their functional significance and binding factors remain largely unknown.
251   Suppressor of Hairy-wing [Su(Hw)] is a DNA-binding factor required for gypsy insulator function and
252 nding protein hnRNPK as the principal XR-PID binding factor required to recruit PCGF3/5-PRC1.
253 s an imprint-specific, sequence-specific DNA binding factor responsible for maintaining methylation a
254 proliferating cell nuclear antigen, upstream binding factor, RNA polymerase II, and 8-oxo-deoxyguanos
255  nuclear factor, erythroid 2 (Nfe2) and core-binding factor, runt domain, alpha subunit 2, translocat
256 )/t(16;16) rearrangements affecting the core-binding factors RUNX1 and CBFB, respectively, are found
257 ndence in EMPs is not mediated by other core binding factors (Runx2 and/or Runx3); however, deleting
258 ng development by the nuclear matrix and DNA binding factor Satb2 (ISR(Satb2)).
259                        They assess how a DNA-binding factor Satb2 controls cell position, molecular i
260 or the SCB (Swi4/6-dependent cell cycle box) binding factor (SBF) complex (composed of Swi4 and Swi6)
261 cription factors CREB (cAMP response element binding factor), SRF (serum response factor), and MEF2 (
262 ith the recruitment of sequence-specific DNA-binding factors such as CTCF, EVI1 and YYI, and second,
263  thought to be generally inaccessible to DNA-binding factors, such as micrococcal nuclease (MNase).
264                                     Many DNA-binding factors, such as transcription factors, form oli
265 f 53BP1 to methyl K810 occurs on E2 promoter binding factor target genes and allows pRb activity to b
266 ed to the ribosomal protein genes by the DNA binding factor Tbf1.
267        Surprisingly, plants lacking this TL1-binding factor, TBF1, respond normally to heat stress bu
268 egulation is mainly achieved through two DNA-binding factors, Tbf1 and Mcm1.
269 We now discover that the telomere duplex DNA-binding factor, TbTRF, also plays a critical role in VSG
270 sent interactions with the site-specific DNA-binding factors that establish and maintain epigenomic m
271                 We discovered classes of DNA-binding factors that strictly avoided, could tolerate or
272  strong preferences toward proximal promoter binding, factors that target intergenic and intronic DNA
273           Transcription is regulated through binding factors to gene promoters to activate or repress
274 dging molecule that cooperates with other PS-binding factors to promote the phagocytosis of apoptotic
275  by coregulators, which are recruited by DNA-binding factors to propagate signaling to basal machiner
276 pathways converge upon sequence-specific DNA binding factors to reprogram gene expression.
277 ize nucleosomes to mediate the access of DNA-binding factors to their sites in vivo.
278        Tau is recruited, along with upstream-binding factor, to ribosomal DNA loci.
279 mpling of TRFs, both DNA-binding and non-DNA-binding factors, to provide a framework for the subseque
280 complex, competing with the telomeric-repeat binding factors TRF1 and TRF2.
281 wo proteins in this complex, telomere repeat binding factors (TRF1 and TRF2), specifically recognise
282 tra-telomeric binding of the telomere repeat binding factor TRF2 within the promoter of the cyclin-de
283 s and insufficient retention of the telomere-binding factor TRF2.
284      PPAN depletion induces NPM and upstream-binding factor (UBF) degradation, which is independent o
285               Herein, we found that upstream binding factor (UBF) interacts with ESET, a histone H3K9
286        PHF6 directly interacts with upstream binding factor (UBF) through its PHD1 domain and suppres
287 A polymerase I transcription factor upstream binding factor (UBF).
288 forms for the rational design of diverse DNA binding factors useful for synthetic biology and other p
289             Identification of differentially binding factors was performed using mass spectrometry (L
290                                  To identify binding factors, we conducted affinity chromatography us
291                                          OEE-binding factors were isolated from human retinal endothe
292 ins defined by constitutive binding of CCCTC binding factor, which appears to restrict the regulatory
293 tein-associated splicing factor (PSF) as OEE-binding factors, which was confirmed by ChIP assay from
294 ents, however, have diverse requirements for binding factors, which, combined with previous data on o
295 shift analysis identified Pdx1 as the Site 4 binding factor, while an 80-88 kilodalton (kDa) beta-cel
296 at this molecule is a general immunoglobulin-binding factor with highest affinity for IgE.
297 s, unifying classical gene regulation by DNA-binding factors with ATP-dependent nucleosome remodeling
298                     CTCF and cohesin are DNA binding factors with central roles in chromatin organiza
299  distinct platelet subpopulations capable of binding factor Xa.
300 ispensable for subsequent DVT propagation by binding factor XII (FXII) and by supporting its activati

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