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1 factor) and perhaps MBF (MluI cell cycle box binding factor).
2 nuclear factor 1, Forkhead box A1 and CCCTC-binding factor.
3 targets of E2F corresponds to Adenovirus E2 Binding Factor.
4 actor related to FUS3, and LEC1, a CCAAT box-binding factor.
5 A BINDING WITH ONE FINGER3, and PROLAMIN BOX-BINDING FACTOR.
6 ions identifies hydrophobic effect as a main binding factor.
7 that has high-affinity binding to the CCCTC-binding factor.
8 i allele extensively binds the protein CCCTC-binding factor.
9 king site for TFIID, the major core promoter-binding factor.
10 rred in the putative A protein, an important binding factor.
11 is a gamma-tubulin small complex (gammaTuSC) binding factor.
12 by moderating the activities of E2 promoter-binding factors.
13 interaction with regulatory elements and DNA-binding factors.
14 nucleosome-induced cooperativity between DNA-binding factors.
15 ell-type-specific regulation directed by DNA binding factors.
16 ough interactions with sequence-specific DNA-binding factors.
17 d their interplay with cooperative chromatin binding factors.
18 ing sites where nucleosomes compete with DNA-binding factors.
19 lastic moduli of hydrogels, with and without binding factors.
22 rget of viral proteinase cleavage is AU-rich binding factor 1 (AUF1), a cellular protein that binds t
23 onal coactivation complex through C promoter-binding factor 1 (CBF1; RBPJ) and strengthens the associ
24 gamma (PPARgamma), leptin, osteonectin, core binding factor 1 (CBFA1), and FBJ murine osteosarcoma vi
25 ctroscopy to study the human telomere repeat binding factor 1 (hTRF1) in complex with Escherichia col
26 n (Pg) in conjunction with lymphoid enhancer-binding factor 1 (Lef-1) differentially regulates the pr
27 The transcription factor lymphoid enhancer-binding factor 1 (LEF-1), which plays a definitive role
29 odermin (EOMES; mesoderm), lymphoid enhancer-binding factor 1 (LEF1) and mesoderm posterior BHLH tran
30 contain normal amounts of lymphoid enhancer-binding factor 1 (Lef1) and transcription factor 1 (Tcf1
32 ers a binding site for the lymphoid enhancer-binding factor 1 (LEF1) transcription factor, reducing L
33 nuclear proteins, i.e. the lymphoid enhancer-binding factor 1 (Lef1), histone H3, and Brahma-related
36 and the shelterin component telomeric repeat binding factor 1 (TRF1)-interacting nuclear factor 2 (TI
37 the nucleolar transcription factor upstream binding factor 1 (UBF1) on rDNA promoters and recruiting
39 the collection of RNAs regulated by AUF1 (AU-binding factor 1), an RBP linked to cancer, inflammation
40 an antigen R) and GRSF1 (G-rich RNA sequence-binding factor 1), that associated with the nuclear DNA-
41 The mammalian RNA-binding protein AUF1 (AU-binding factor 1, also known as heterogeneous nuclear ri
42 roles of RBPs HuR, CUG-binding protein 1, AU-binding factor 1, and several GI epithelial-specific miR
43 llerRed chromophore with the telomere repeat binding factor 1, TRF1, we developed a novel approach to
48 ing putative T-cell factor/lymphoid enhancer binding factor-1 (TCF/LEF) motifs, consistent with canon
49 s with Epstein-Barr virus latency C-promoter binding factor-1/suppressor of hairless/lag-1 (CSL) and
51 t a putative fifth subunit, nuclear receptor binding factor 2 (NRBF2), is a tightly bound component o
52 nthesis, including sterol regulatory element binding factor 2 (Srebf2) and 3-hydroxy-3-methylglutaryl
53 ivator protein 1 (RAP1) and telomeric repeat-binding factor 2 (TRF2) are two subunits in shelterin th
55 e 3' overhang facilitated by telomere repeat-binding factor 2 (TRF2) into telomeric DNA, forming t-lo
56 t a non-telomeric isoform of telomere repeat-binding factor 2 (TRF2-S) is a novel RBP that regulates
57 functionally interacts with telomere repeat-binding factor 2 that in turn regulates its helicase act
58 of the shelterin component telomeric repeat-binding factor 2, cells survived but remained dormant an
59 nother shelterin component, telomeric repeat binding factor 2, interacting protein (p.Ala104Pro and p
60 ed the impact of a sterol regulatory element-binding factor-2 (SREBF-2) polymorphism on the risk of d
61 ion and telomere binding of telomeric repeat-binding factor-2 (TRF2), associated with increased DNA d
62 (Zea mays), several genes, including a G-BOX BINDING FACTOR 3 (GBF3) were identified as candidate dro
63 yeloma tumor protein 2, interleukin enhancer binding factor 3 (IEBP3), guanine nucleotide binding pro
64 NAs, we discovered that interleukin enhancer-binding factor 3 (ILF3) is required for sustained uPA ex
66 homolog of the bacterial RbfA (for ribosome-binding factor A) protein that was identified as a cold-
67 Cocaine exposure reduces binding of CCCTC-binding factor, a chromosomal scaffolding protein, and i
69 se 1 via mTOR (P < 0.02), and total upstream binding factor abundance were significantly greater in E
72 d-Sequential Induction in Patients With Core Binding Factor Acute Myeloid Leukemia and Treating Patie
77 o 60 years of age, excluding those with core binding factor AML and acute promyelocytic leukemia, wer
79 ontain an insulator element that binds CCCTC-binding factor and reduces its insertional genotoxicity.
80 appa gene V-J intervening region binds CCCTC-binding factor and specifies locus contraction and long-
81 rs on stimulation; it is stabilized by CCCTC-binding factor and TFIIB and poises the gene for a promp
82 n DNA sequence (cis-elements) as well as DNA binding factors and ATP-dependent chromatin modifiers (t
84 ions of CDKs and cyclins, and of E2 promoter-binding factors and dimerization partners confirmed inte
85 tation experiments for tens of different DNA-binding factors and from ribosome profiling experiments
86 e coordinated integration and release of ES6 binding factors and likely plays a pivotal role in remod
87 probably enhances Ana2 avidity for centriole-binding factors and may bridge multiple factors as requi
89 E2 Y102F protein interacted with cellular E2-binding factors and the viral helicase E1; however, in c
90 o chromatin structural proteins, Ctcf (CCCTC-binding factor) and Hmgb2 (high mobility group protein B
91 S-phase activators, SBF (Swi4 cell cycle box binding factor) and perhaps MBF (MluI cell cycle box bin
92 the transcriptional insulator protein CCCTC-binding factor, and interacted with each other, creating
93 cluding histone modifications and associated binding factors, and their functional contribution to tr
96 nding protein homolog-1) and FBF (fem-3 mRNA binding factor) are evolutionary conserved regulators of
97 ybrid assays with the ABA responsive element binding factor AREB2/ABF4, which binds to a G-box absolu
98 members, and the ethylene-responsive element binding factor-associated amphiphilic repression (EAR-li
99 presence of the ethylene-responsive element-binding factor-associated amphiphilic repression motif.
101 ine finger-like motif in the ParG centromere-binding factor augments ParF ATPase activity but is inef
102 plex is composed of the unique cofactor core binding factor beta (CBF-beta) and canonical ligase comp
104 nscription factor fusion CBFbeta-SMMHC (core binding factor beta and the smooth-muscle myosin heavy c
105 on factor 1 (RUNX1) and its associating core binding factor-beta (CBFB) play pivotal roles in leukemo
106 Here, we show that deletion of CTCF (CCCTC-binding factor)-binding sites at TAD and sub-TAD topolog
108 se, and TAF12, an RNA polymerase II TATA-box binding factor, cause CIN when overexpressed in human ce
110 rd genetic screen for regulators of C-REPEAT BINDING FACTOR (CBF) gene expression (RCFs), we identifi
112 MTA3) in the rapid cold induction of CRT/DRE BINDING FACTOR (CBF) genes CBF1 and CBF2, and the repres
113 h leukemogenic fusion proteins, such as core-binding factor (CBF) leukemia and MLL fusion leukemia, r
114 ort of recurring FLT3 N676 mutations in core-binding factor (CBF) leukemias and suggests a defined su
120 thaliana, acting downstream of the C-repeat binding factor (CBF) transcription factors to recruit th
122 the C-REPEAT/DEHYDRATION-RESPONSIVE ELEMENT BINDING FACTOR (CBF) transcriptional regulators that con
123 ene expression under cold stress by C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathw
126 rowth defect in cells lacking the centromere binding factor Cbf1 and synthetic lethality when combine
127 epeat (CRT)/drought-responsive element (DRE) binding factor CBF1/DREB1b is a transcriptional regulato
131 he viral envelope contains the mammalian LPS-binding factors CD14, TLR4, and MD-2, which, in conjunct
132 pproach uncovered the chromatin helicase DNA-binding factor CHD1 as a putative synthetic-essential ge
134 activator Sub1, recently identified as a G4-binding factor, contributes to genome maintenance at G4-
135 in an inter-chromosomal Sox-2 enhancer/CCCTC-binding factor CTCF/cohesin loop) to an active (within a
136 ropic "tension globule." In the other, CCCTC-binding factor (CTCF) and cohesin act together to extrud
137 erved chromosome loop anchors bound by CCCTC-binding factor (CTCF) and cohesin are vulnerable to DNA
138 ence specificity in vitro and in vivo, CCCTC-binding factor (CTCF) and its paralog brother of the reg
140 germline transcripts, and proximity to CCCTC-binding factor (CTCF) and Rad21 sites were compared with
141 fic chromatin interactions mediated by CCCTC-binding factor (CTCF) and RNA polymerase II (RNAPII) wit
142 mains are enriched for binding sites of CTCC-binding factor (CTCF) and the cohesin complex, implicati
143 in part by the architectural proteins CCCTC binding factor (CTCF) and the cohesin complex, which co-
146 inding sites for the insulator protein CCCTC-binding factor (CTCF) are constitutively occupied, a sub
147 ed the DNA-binding zinc-finger protein CCCTC-binding factor (CTCF) as a crucial regulator of long-ran
148 tin fiber and the proteins cohesin and CCCTC-binding factor (CTCF) as key players anchoring such chro
149 elements, many of which coincide with CCCTC-binding factor (Ctcf) binding sites, for example, the cl
155 ial dysfunction.SIGNIFICANCE STATEMENT CCCTC-binding factor (CTCF) is a DNA-binding protein that orga
160 er determined that chromatin insulator CCCTC-binding factor (CTCF) is TGF-beta inducible and facilita
162 romatin bound by the insulator protein CCCTC-binding factor (CTCF) on the inactive X chromosome (Xi).
164 F-induced activation of the epigenetic CCCTC binding factor (CTCF) plays an important role in corneal
166 Chromatin looping mediated by the CCCTC binding factor (CTCF) regulates V(D)J recombination at A
167 s of which are enriched in cohesin and CCCTC-binding factor (CTCF) required for long-range interactio
168 f2 ICR [Delta2,3] removing two of four CCCTC-binding factor (CTCF) sites and the intervening sequence
170 ed TAD borders reveals the presence of CCCTC-binding factor (CTCF) sites with diverging orientations
172 mediated by the architectural protein CCCTC-binding factor (CTCF) that binds to the boundaries of to
174 tment of the transcriptional repressor CCCTC-binding factor (CTCF) to the MSMP enhancer region was de
176 nt) database identified association of CCCTC-binding factor (CTCF) with Ahr promoter in mouse livers.
179 data of H3K4 di-methylation (H3K4me2), CCCTC binding factor (CTCF), AR and FoxA1 cistrome data as wel
182 DNF chromatin by DNA insulator protein CCCTC-binding factor (CTCF), which is associated with suppress
183 alized by simultaneous mutation of two CCCTC-binding factor (CTCF)-binding elements (CBE1 and CBE2) w
185 gene desert form a 1 Mbp loop between CCCTC-binding factor (CTCF)-binding sites that is separated fr
186 xhibit hypermethylation at cohesin and CCCTC-binding factor (CTCF)-binding sites, compromising bindin
187 cterized by clusters of hypomethylated CCCTC-binding factor (CTCF)-bound elements, which were predomi
188 n start sites (TSS), and the number of CCCTC-binding factor (CTCF)-cohesin complexes between the inte
190 ated by long noncoding RNAs (lncRNAs), CCCTC binding factor (CTCF)-mediated boundaries, and DNA methy
196 eavage domain with a series of Pumilio/fem-3-binding factor domains that specifically recognize diffe
197 sis that apical HS in the airway serves as a binding factor during infection, and HS modulating compo
198 rotein that functions dually as a centromere binding factor during segrosome assembly and as a transc
201 recruited to the miR-31 promoter by the DNA binding factor ETS-1, and it represses miR-31 transcript
202 gene pABRE:NCED enhanced NCED and ABF (ABRE-binding factor) expression in Arabidopsis Columbia-0 see
203 r of C-repeat/dehydration responsive element-binding factor expression1/scream2 that are required for
204 and ABA-INSENSITIVE5/ABA-responsive element binding factor family identified specific members that c
205 interaction between PUF family member fem-3 binding factor (FBF) and germline development defective-
208 dies on a beta-globin enhancer- and promoter-binding factor, GATA-1, the founding member of the GATA
209 ii evades complement-mediated destruction by binding factor H (FH), a host-derived negative regulator
211 t the identification of the minor groove DNA-binding factor high mobility group AT-hook 2 (HMGA2) as
213 g competent substrate, human telomere repeat binding factor (hTRF1), which is bound to DnaK in a glob
214 differential expression of inhibitor of DNA binding factors Id1 and Id3, as well as Notch-regulated
217 g similarities between animal and plant GAGA-binding factors in the recruitment of PRC1 and PRC2 comp
219 ctions of RIOK3 with actin and several actin-binding factors including tropomyosins (TPM3 and TPM4) a
220 Ethylene Response Factor 1 (ERF1), a GCC-box binding factor is in synchrony with expression of AtNudt
223 T cell factor (TCF)-1 and lymphoid enhancer-binding factor (LEF)-1 transcription factors have redund
224 report on the tethering of MLL1 to chromatin binding factor lens epithelium-derived growth factor (LE
226 cally interacts with the MluI cell cycle box-binding factor (MBF) transcription complex that regulate
229 n clinical specimens showed that the heparin-binding factor midkine is a systemic inducer of neo-lymp
230 not recruit Swi4/6-dependent cell-cycle box-binding factor/MluI cell-cycle box binding factor comple
231 In this study, we investigate the distinct binding factors needed on apposed membranes for Rab effe
232 hich hair cells produce an otolith precursor-binding factor, normally localised to tether cell kinoci
233 ions for the transcriptional regulator CCCTC-binding factor of ESR1 and ESR2, consistent with sustain
234 on with pituitary tumour-transforming 1, the binding factor of PTTG1lP, and with vimentin and E-cadhe
235 and Ska complexes are the major microtubule-binding factors of the kinetochore responsible for maint
236 ot affect hydrophobic interactions as a main binding factor or the location of binding sites in Abeta
237 s VAL1/2 possibly in combination with GT-box binding factors, other AtBMI1 regulatory networks requir
238 KT) and phosphorylated cAMP response element binding-factor (p-CREB) than similar cancers that lacked
241 the vertebrate genome organizer CTCF (CCCTC-binding factor) played an important role for the evoluti
242 lastoma tumor suppressor protein/E2 promoter binding factor (pRb/E2F1) pathway, which we have previou
243 rabidopsis (Arabidopsis thaliana) GAGA-motif binding factor protein basic pentacysteine6 (BPC6) inter
245 minization of XX and XO animals (fem)-3 mRNA-binding factor (PUF) proteins bind sequence specifically
247 i of insect and vertebrate Pumilio and Fem-3 binding factors (PUFs) are related, and we show that cor
248 a context-dependent manner, and cis-element-binding factors recruit chromatin regulators that mediat
251 Suppressor of Hairy-wing [Su(Hw)] is a DNA-binding factor required for gypsy insulator function and
253 s an imprint-specific, sequence-specific DNA binding factor responsible for maintaining methylation a
254 proliferating cell nuclear antigen, upstream binding factor, RNA polymerase II, and 8-oxo-deoxyguanos
255 nuclear factor, erythroid 2 (Nfe2) and core-binding factor, runt domain, alpha subunit 2, translocat
256 )/t(16;16) rearrangements affecting the core-binding factors RUNX1 and CBFB, respectively, are found
257 ndence in EMPs is not mediated by other core binding factors (Runx2 and/or Runx3); however, deleting
260 or the SCB (Swi4/6-dependent cell cycle box) binding factor (SBF) complex (composed of Swi4 and Swi6)
261 cription factors CREB (cAMP response element binding factor), SRF (serum response factor), and MEF2 (
262 ith the recruitment of sequence-specific DNA-binding factors such as CTCF, EVI1 and YYI, and second,
263 thought to be generally inaccessible to DNA-binding factors, such as micrococcal nuclease (MNase).
265 f 53BP1 to methyl K810 occurs on E2 promoter binding factor target genes and allows pRb activity to b
269 We now discover that the telomere duplex DNA-binding factor, TbTRF, also plays a critical role in VSG
270 sent interactions with the site-specific DNA-binding factors that establish and maintain epigenomic m
272 strong preferences toward proximal promoter binding, factors that target intergenic and intronic DNA
274 dging molecule that cooperates with other PS-binding factors to promote the phagocytosis of apoptotic
275 by coregulators, which are recruited by DNA-binding factors to propagate signaling to basal machiner
279 mpling of TRFs, both DNA-binding and non-DNA-binding factors, to provide a framework for the subseque
281 wo proteins in this complex, telomere repeat binding factors (TRF1 and TRF2), specifically recognise
282 tra-telomeric binding of the telomere repeat binding factor TRF2 within the promoter of the cyclin-de
284 PPAN depletion induces NPM and upstream-binding factor (UBF) degradation, which is independent o
288 forms for the rational design of diverse DNA binding factors useful for synthetic biology and other p
292 ins defined by constitutive binding of CCCTC binding factor, which appears to restrict the regulatory
293 tein-associated splicing factor (PSF) as OEE-binding factors, which was confirmed by ChIP assay from
294 ents, however, have diverse requirements for binding factors, which, combined with previous data on o
295 shift analysis identified Pdx1 as the Site 4 binding factor, while an 80-88 kilodalton (kDa) beta-cel
297 s, unifying classical gene regulation by DNA-binding factors with ATP-dependent nucleosome remodeling
300 ispensable for subsequent DVT propagation by binding factor XII (FXII) and by supporting its activati
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